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1 (anovulation with either hyperandrogenism or polycystic ovaries).
2 However, little is known about the cause of polycystic ovaries.
3 PR-4 females studied did not ovulate and had polycystic ovaries.
4 49 (21%) of the women had polycystic ovaries.
5 oligo-/anovulation, hyper-androgenism and/or polycystic ovaries.
6 y androgen excess, ovulatory dysfunction and polycystic ovaries(1), and is often accompanied by insul
7 es and regular cycles and from 32 women with polycystic ovaries, 16 of whom had regular, ovulatory cy
8 idence of vascular dysfunction in women with polycystic ovaries and are compatible with the hypothesi
10 eproductive function in women with epilepsy (polycystic ovaries and hyperandrogenemia), this was not
15 women with high ZAG had fewer MetS, IGT and polycystic ovaries as compared with the low ZAG PCOS wom
16 rinology clinics, 20 asymptomatic women with polycystic ovaries attending the family planning clinic,
17 owing 60 subjects: 20 symptomatic women with polycystic ovaries attending the reproductive endocrinol
18 ased density of small preantral follicles in polycystic ovaries could result from increased populatio
19 isease, ovarian primordial follicle loss and polycystic ovary disease were increased in F1 generation
20 emales, ovarian primordial follicle loss and polycystic ovary disease were increased in F3 generation
23 of female infertility and is associated with polycystic ovaries, hirsutism, obesity, and insulin resi
24 tages, was six-fold greater in biopsies from polycystic ovaries in anovulatory women than in normal o
26 nosed after irregular menses, hirsutism, and polycystic ovaries, is concerned about weight gain despi
28 elopment of dysfunctional ovulation, classic polycystic ovaries, reduced large antral follicle health
30 onfidence interval (95% CI): 1.22-1.53)) and polycystic ovary syndrome (OR = 1.51 (95% CI: 1.33-1.72)
31 eptibility loci that are associated with the polycystic ovary syndrome (PCOS) affection status by scr
34 tatic model assessment [HOMA]) in women with polycystic ovary syndrome (PCOS) and chronic periodontit
35 y and densitometry features in patients with polycystic ovary syndrome (PCOS) and compare them with h
36 terleukin-6 (IL-6) in non-obese females with polycystic ovary syndrome (PCOS) and either clinically h
37 he understanding of the relationship between polycystic ovary syndrome (PCOS) and endometrial cancer
38 red in common reproductive disorders such as polycystic ovary syndrome (PCOS) and hypothalamic amenor
39 tor of MMP-1 (TIMP)-1 ratio in patients with polycystic ovary syndrome (PCOS) and systemically health
42 teristics differed in women with and without polycystic ovary syndrome (PCOS) between a Caucasian and
43 As a heterogeneous reproductive disorder, polycystic ovary syndrome (PCOS) can be caused by geneti
46 EPS) are altered in myotubes from women with polycystic ovary syndrome (PCOS) compared to healthy con
51 ars, it has been clearly documented that the polycystic ovary syndrome (PCOS) has major metabolic seq
53 female first-degree relatives of women with polycystic ovary syndrome (PCOS) have hyperandrogenemia
56 or metformin for comprehensive management of polycystic ovary syndrome (PCOS) in women with obesity i
75 Despite affecting ~11-13% of women globally, polycystic ovary syndrome (PCOS) is a substantially unde
93 ly, some studies have revealed the effect of polycystic ovary syndrome (PCOS) on gingival inflammatio
96 women in their reproductive age suffer from polycystic ovary syndrome (PCOS) that, alongside subfert
97 ations in B cell numbers are associated with polycystic ovary syndrome (PCOS) through unknown mechani
98 arous women with diagnosed, probable, and no polycystic ovary syndrome (PCOS) throughout 15-year foll
100 hyperandrogenic insulin-resistant women with polycystic ovary syndrome (PCOS) who are at increased ri
106 ent study, we evaluate a group of women with polycystic ovary syndrome (PCOS), a condition characteri
107 n to this model because it is usually due to polycystic ovary syndrome (PCOS), a condition in which i
108 nd stroma are also prominent features of the polycystic ovary syndrome (PCOS), a leading cause of inf
110 orioretinopathy (CSCR) among transmasculine, polycystic ovary syndrome (PCOS), and androgen-exposed p
112 alleles are associated with type 2 diabetes, polycystic ovary syndrome (PCOS), and size at birth.
113 g bacterial vaginosis, gestational diabetes, polycystic ovary syndrome (PCOS), anxiety, depression, a
115 Girls with T2D are at risk of developing polycystic ovary syndrome (PCOS), but the prevalence of
118 ment parameters across various phenotypes of polycystic ovary syndrome (PCOS), considering body mass
119 ty, and hirsutism, common characteristics of polycystic ovary syndrome (PCOS), improve with even mode
120 iome and chronic metabolic disease including polycystic ovary syndrome (PCOS), is well documented, ho
121 similar to those displayed in patients with polycystic ovary syndrome (PCOS), such as follicular gro
122 oxidative stress, in the pathophysiology of polycystic ovary syndrome (PCOS), the most common endocr
123 upport faster and more accurate diagnosis of polycystic ovary syndrome (PCOS), with a focus on both p
124 resents a significant concern for women with polycystic ovary syndrome (PCOS), with deleterious psych
140 of healthy women (n = 9), and in women with polycystic ovary syndrome (PCOS; n = 6) or hypothalamic
143 Naylor's valuable critique of our study "Polycystic Ovary Syndrome and Endometrial Cancer Risk: R
144 2-1.16; P = 0.007); and genetic liability to polycystic ovary syndrome and endometrioid carcinoma (OR
146 disrupted in pathological conditions such as polycystic ovary syndrome and hypothalamic amenorrhea.
148 pecially primary ovarian insufficiency), and polycystic ovary syndrome are also relevant, with good e
150 ed, in part, by PCOS Challenge: The National Polycystic Ovary Syndrome Association and by the Foundat
151 ene can be increased in obese women with the polycystic ovary syndrome by decreasing insulin secretio
153 bout limitations of the test, which included polycystic ovary syndrome falsely inflating AMH levels (
155 MI 33 kg/m(2)), insulin-resistant women with polycystic ovary syndrome had aberrant skeletal muscle m
162 nce thought to affect primarily adult women, polycystic ovary syndrome is frequently diagnosed during
165 the University of California, San Francisco, Polycystic Ovary Syndrome Multidisciplinary Clinic over
166 ndomly assigned 626 infertile women with the polycystic ovary syndrome to receive clomiphene citrate
168 evels, that they hypothesized could point to polycystic ovary syndrome underpinning these association
169 s described in the literature and found that polycystic ovary syndrome was associated with increased
171 ed controlled trial, 60 infertile women with polycystic ovary syndrome were randomly selected and ass
172 proate, is associated with the occurrence of polycystic ovary syndrome when used in young adulthood o
173 ons, linking dementia with bone development, polycystic ovary syndrome with cardiovascular developmen
177 eving live birth in infertile women with the polycystic ovary syndrome, although multiple birth is a
179 g obesity, type 2 diabetes, hepatitis C, and polycystic ovary syndrome, and is a primary feature of m
180 a) improves ovulatory function in women with polycystic ovary syndrome, and specific dietary fatty ac
181 ecological conditions-such as endometriosis, polycystic ovary syndrome, and uterine fibroids-have rem
182 drenal hyperplasia, premature adrenarche and polycystic ovary syndrome, as well as in androgen-depend
183 line infertility treatment in women with the polycystic ovary syndrome, but aromatase inhibitors, inc
185 of start of follow-up, parity, diagnosis of polycystic ovary syndrome, diabetes, chronic hypertensio
186 ental disorders, endometriosis, menorrhagia, polycystic ovary syndrome, dysmenorrhea, leiomyoma, and
187 diagnosis, disease duration, treatment, and polycystic ovary syndrome, endometriosis, bilateral ooph
188 56), after adjustment for education, parity, polycystic ovary syndrome, energy intake, and physical a
189 ease, hypertension, stroke, type 2 diabetes, polycystic ovary syndrome, heart failure, atrial fibrill
191 an overview of our current understanding of polycystic ovary syndrome, its epidemiology and natural
192 genes associated with spontaneous abortion, polycystic ovary syndrome, myocardial infarction and mel
194 sed overwhelmingly on affective disorders in polycystic ovary syndrome, overlooking a substantial por
195 eted educational qualification, nulliparity, polycystic ovary syndrome, physical activity, and body m
196 ideline for the Assessment and Management of Polycystic Ovary Syndrome, requiring two out of three cr
197 rched PubMed using a string of variations of polycystic ovary syndrome, therapy/treatment, and adoles
198 s the action of insulin in patients with the polycystic ovary syndrome, thereby improving ovulatory f
199 netic liability to 3 factors (endometriosis, polycystic ovary syndrome, type 2 diabetes) scaled to re
200 sex hormone-dependent cancers and diseases (polycystic ovary syndrome, uterine fibroids, endometrios
201 enetically correlated with endometriosis and polycystic ovary syndrome, we find limited genetic overl
202 ulin resistance is observed in patients with polycystic ovary syndrome, we hypothesized that TAC or S
225 y in symptomatic and asymptomatic women with polycystic ovaries than in the controls (10.7, 14.1, and
226 In both ovulatory and anovulatory women with polycystic ovaries, we noted a significant increase in t
227 n disease (primary ovarian insufficiency and polycystic ovaries) were increased in the F3 generation