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1 the absence of other risk factors, including polydrug abuse, hypertension, or cerebrovascular disease
2 cts of cocaine, ruling out confounds such as polydrug abuse, preexisting differences in DAT levels, o
5 fects of nicotine alone and nicotine+cocaine polydrug combinations in a nonhuman primate model of dru
6 eatment of coexisting heroin and amphetamine polydrug dependence in 100 heroin- and amphetamine-depen
9 creases in overdose (OD) mortality caused by polydrug exposure to synthetic opioids such as fentanyl
11 n 2024 versus 2023 were similar to those for polydrug OD (-42% and -39% APC in males and females, res
12 oint regressions detected increases in these polydrug OD after 2018, then decreases from 2023 in male
13 pioids in this time period, the decreases in polydrug OD mortality are more likely to be caused by ch
15 icate that after increases from 2018 onward, polydrug OD mortality caused by synthetic opioids and st
18 to understand the recent trajectory in this polydrug OD mortality, especially as associated with dec
19 Stratification into 10-year age groups for polydrug OD revealed that mortality peaked at age 35-44
22 100 000 person-years), including opioid and polydrug overdoses, and homicide deaths (-23 per 100 000
24 in cocaine abuse patients is complicated by polydrug use and other comorbid factors, hindering the d
26 We observed similar patterns for alcohol and polydrug use disorders, separately, although differences
27 the prevalence of recreational drug use and polydrug use in the previous 3 months and associations w
36 smokers and ketamine users, individuals with polydrug users scored significantly higher on impulsivit
37 ceptor, might be upregulated in brains of MA polydrug users, although lower dopamine levels in MA use
38 ding is above normal in methamphetamine (MA) polydrug users, using PET and the D3-preferring ligand [