ライフサイエンスコーパス: polymorphonuclear

1 The infiltration of polymorphonuclear and macrophage cells was associated wi

2 m of ICOS (ICOS-Fc) inhibits the adhesion of polymorphonuclear and tumor cell lines to HUVECs; thus,

3 ere immunostained for IL-6R, gp130, CD15(+) (polymorphonuclear), and CD3(+) (T cell) inflammatory cel

4 CD21(-)CD11b(+) cells can be subdivided into polymorphonuclear (CADO48A(+)CD14(-)) and monocytic (CAD

5 D11b+CD33+) phenotype and a subpopulation of polymorphonuclear CD11b+CD33+CD15+ cells.

6 human leukocytes to purified Pic resulted in polymorphonuclear cell activation, but impaired chemotax

7 coli, an abnormal ultrasonographic finding, polymorphonuclear cell count of greater than 60%, C-reac

8 Polymorphonuclear cell Ecto expressed MHC class I and in

9 that IL-36gamma treatment promoted transient polymorphonuclear cell infiltration to the vaginal cavit

10 els of proinflammatory mediators (decreasing polymorphonuclear cell infiltration), increasing anti-in

11 istopathological analysis, a predominance of polymorphonuclear cell inflammatory infiltrate and a red

12 Enriched NPs dramatically reduced polymorphonuclear cell influx in murine peritonitis, sho

13 es in vivo and platelet/P-selectin-dependent polymorphonuclear cell migration in vitro were exclusive

14 reased IL-1beta and MIP-2 proteins, reducing polymorphonuclear cell number in the infected cornea.

15 yanidin also decreased KC concentrations and polymorphonuclear cell recruitment in bronchoalveolar la

16 acrine manner in the suppressive activity of polymorphonuclear cell-derived Ecto.

17 n comparison with ADCC by mononuclear cells, polymorphonuclear cell-mediated ADCC and complement-depe

18 Polymorphonuclear cells (neutrophils) are the first cell

19 eptible to phagocytosis and killing by human polymorphonuclear cells (P = 0.01 and P = 0.006, respect

20 In 22/29 cases of active cryptitis, polymorphonuclear cells (PMN) clearly expressed HSP70i,

21 The engulfment of apoptotic polymorphonuclear cells (PMN) during the resolution of i

22 or cells such as mononuclear cells (MNC) and polymorphonuclear cells (PMN) to exert Ab-dependent cell

23 of select genes in PBMC and peripheral blood polymorphonuclear cells (PMN).

24 CD97 was upregulated on polymorphonuclear cells (PMNC) of patients with psoriasi

25 loroquine (CQ) on the antifungal capacity of polymorphonuclear cells (PMNs) and on the inflammatory r

26 formed to determine the total levels of oral polymorphonuclear cells (PMNs) before and 3 months after

27 BLP activated human polymorphonuclear cells (PMNs) ex vivo to adhere to dena

28 ase (MMP)-8 and -9 released by degranulating polymorphonuclear cells (PMNs) promote pericellular prot

29 In inflammation, they activate and recruit polymorphonuclear cells (PMNs) through binding of the ch

30 ers in the lung, a robust alveolar influx of polymorphonuclear cells (PMNs), and a risk of systemic s

31 addition, in the presence of complement and polymorphonuclear cells (PMNs), antibodies to Ata were h

32 We have previously reported that neutrophil (polymorphonuclear cells [PMNs]) accumulation in culprit

33 robust alveolar infiltration of neutrophils (polymorphonuclear cells [PMNs]) that can promote systemi

34 te flow, clearance of apoptotic neutrophils (polymorphonuclear cells [PMNs]), production of specializ

35 nfiltration, consisting of mainly neutrophil polymorphonuclear cells and monocytes/macrophages, cente

36 ages and dendritic cells, and recruitment of polymorphonuclear cells are likely to contribute to this

37 totoxicity (ADCC) by NK cells, monocytes, or polymorphonuclear cells as well as complement-dependent

38 2, 3, and 8, and E-NPP1, 2, and 3, in their polymorphonuclear cells by immunofluorescence and qPCR.

39 NA) in THP-1 (human monocytic cell line) and polymorphonuclear cells from patients with sepsis enhanc

40 of cortical bone, and the diagnostic role of polymorphonuclear cells in implant-associated osteomyeli

41 nflammatory genes, decreased infiltration of polymorphonuclear cells into the T-cell zone of lymphoid

42 gocytosis of platelets using FcgammaRIIIb(+) polymorphonuclear cells or FcgammaRIIIa(+) monocytes as

43 e >40 years, African American race, and >/=5 polymorphonuclear cells per high-power field on urethral

44 ondition where macrophages, eosinophils, and polymorphonuclear cells play an important role in its pa

45 Properdin released from human polymorphonuclear cells stimulated with PMA did not bind

46 the migration of IL-17(+)CD8(+) T cells and polymorphonuclear cells to infected tissues.

47 tion of c-Jun, leading to the recruitment of polymorphonuclear cells to the cochlea.

48 Furthermore, secondary tethering of polymorphonuclear cells was blocked by DREG-200 but sign

49 city (ADCC) by isolated monocytes, activated polymorphonuclear cells, and human whole blood.

50 In the presence of complement and polymorphonuclear cells, antisera to 9Glc-NH(2)-TT media

51 lates in phagocytes, monocytes, fibroblasts, polymorphonuclear cells, macrophages, and lymphocytes.

52 Also, the islets of Langerhans attracted polymorphonuclear cells, possibly via release of IL-6, I

53 At day 3, the grafts were surrounded by polymorphonuclear cells, which were replaced by a notabl

54 influenzae-induced cochlear infiltration of polymorphonuclear cells.

55 as, sclerosis of the surrounding tissue, and polymorphonuclear cells.

56 such as natural killer cells, monocytes, and polymorphonuclear cells.

57 uction of TRAIL, and reduced infiltration of polymorphonuclear cells.

58 parent differences between splenic and tumor polymorphonuclear cells/granulocytic myeloid-derived sup

59 ion would enable gonococci to survive within polymorphonuclear cells; however, an active LgtD in a fe

60 to monocytic (mononuclear) and granulocytic (polymorphonuclear) cells using the Ly6C and Ly6G markers

61 Using resting human polymorphonuclear granulocytes (PMN) from peripheral blo

62 alpha, Il-1beta, and Il-6 and attenuation of polymorphonuclear inflammatory cells into the placental

63 Recently, we demonstrated that blood polymorphonuclear leucocytes (PMNs) in ARDS are basally

64 accompanied by impaired defense functions of polymorphonuclear leucocytes (PMNs), increased patient s

65 pheral insulin resistance and alterations in polymorphonuclear leukocyte (PML) function.

66 ids participate in the suppression of murine polymorphonuclear leukocyte (PMN) bactericidal activitie

67 Polymorphonuclear leukocyte (PMN) is the predominant inn

68 Polymorphonuclear leukocyte (PMN) migration across the i

69 eltaybcL) failed to suppress transepithelial polymorphonuclear leukocyte (PMN) migration in vitro, a

70 ation but had no effect on bacterial load or polymorphonuclear leukocyte (PMN) numbers in the lung.

71 es (CORMs) suppress inflammation by reducing polymorphonuclear leukocyte (PMN) recruitment to the aff

72 lular adhesion molecule 1 (ICAM-1)-dependent polymorphonuclear leukocyte (PMN) recruitment, functiona

73 n species (ROS) are critical for neutrophil (polymorphonuclear leukocyte (PMN)) microbicidal function

74 rate that epinephrine alters the neutrophil (polymorphonuclear leukocyte (PMN))-dependent inflammator

75 ant throughout the observation period, while polymorphonuclear leukocyte (PMN), S100A8, and interleuk

76 entify novel virulence factors in evasion of polymorphonuclear leukocyte (PMN)-mediated innate immuni

77 Neutrophil [polymorphonuclear leukocyte (PMN)] transepithelial migra

78 y with IL-1beta to promote early neutrophil (polymorphonuclear leukocyte [PMN]) recruitment.

79 In contrast, polymorphonuclear leukocyte adhesion and chemotaxis were

80 but reduced cytokines/chemokines as well as polymorphonuclear leukocyte and macrophage numbers.

81 r early source of chemokines associated with polymorphonuclear leukocyte and monocyte/macrophage infi

82 lves (1) enhancement of SCD erythrocytes and polymorphonuclear leukocyte efferocytosis, (2) blunting

83 (active matrix metalloproteinase-8 [aMMP-8], polymorphonuclear leukocyte elastase [PMN elastase], and

84 The most polymorphonuclear leukocyte infiltration in periodontal

85 In zymosan-initiated peritonitis, neutrophil polymorphonuclear leukocyte infiltration in response to

86 ion, increased cell apoptosis, and decreased polymorphonuclear leukocyte infiltration in the healing

87 phagocytosis of Escherichia coli, decreased polymorphonuclear leukocyte infiltration, and counter-re

88 attenuated pulmonary membrane thickening and polymorphonuclear leukocyte infiltration, reduced NF-kap

89 ls of erythrocyte lysis, resistance to human polymorphonuclear leukocyte killing, and pathogenesis in

90 We show that the specific immunodepletion of polymorphonuclear leukocyte neutrophils using anti-Ly6G

91 ng inflammation, characterized by increasing polymorphonuclear leukocyte recruitment, is a major caus

92 ntercellular adhesion molecule-1 expression, polymorphonuclear leukocyte transendothelial migration,

93 MRSA exotoxins also caused neutrophil (polymorphonuclear leukocyte) activation, as measured by

94 novel pathogenic entity, the activated PMN (polymorphonuclear leukocyte, i.e., neutrophil)-derived e

95 volving either platelet-monocyte or platelet-polymorphonuclear leukocyte.

96 Neutrophil (polymorphonuclear leukocyte; PMN) inflammatory functions

97 cytosis of G. bethesdensis by normal and CGD polymorphonuclear leukocytes (CGD PMN) required heat-lab

98 ce expressing human alpha-defensins in their polymorphonuclear leukocytes (Def(+/+)).

99 Polymorphonuclear leukocytes (neutrophils) are the prima

100 trates the epidermis and reaches the dermis, polymorphonuclear leukocytes (PMLs) accumulate and an ab

101 gue associated with extensive recruitment of polymorphonuclear leukocytes (PMN or neutrophils) to the

102 Polymorphonuclear leukocytes (PMN) achieve an intermedia

103 Polymorphonuclear leukocytes (PMN) are the first respond

104 Polymorphonuclear leukocytes (PMN) from patients with ch

105 tussis is able to avoid bacterial killing by polymorphonuclear leukocytes (PMN) if specific opsonic a

106 expression in human uroepithelial cells and polymorphonuclear leukocytes (PMN) in vitro and in bladd

107 flammation is traditionally characterized by polymorphonuclear leukocytes (PMN) influx followed by ph

108 Transendothelial migration (TEM) of polymorphonuclear leukocytes (PMN) involves a carefully

109 xperiments, conditioned media collected from polymorphonuclear leukocytes (PMN) selectively increased

110 . bethesdensis resists killing by serum, CGD polymorphonuclear leukocytes (PMN), and antimicrobial pe

111 dal polypeptides secreted by macrophages and polymorphonuclear leukocytes (PMN).

112 , sustained recruitment to the lymph node of polymorphonuclear leukocytes (PMN, or neutrophils), the

113 st immune responses, including the influx of polymorphonuclear leukocytes (PMN; neutrophils).

114 by the provider, who recorded the number of polymorphonuclear leukocytes (PMNLs) per epithelial cell

115 HEK293) cell line expressing 5-LO, and human polymorphonuclear leukocytes (PMNLs) we investigated the

116 We hypothesized that polymorphonuclear leukocytes (PMNs) and less mature myel

117 nstrate its ability in separating/recovering polymorphonuclear leukocytes (PMNs) and mononuclear leuk

118 s a host foreign body response, during which polymorphonuclear leukocytes (PMNs) and then monocytes (

119 Polymorphonuclear leukocytes (PMNs) are innate immune ce

120 e, we demonstrate that RvD1 actions on human polymorphonuclear leukocytes (PMNs) are pertussis toxin

121 oeae recruits and interacts extensively with polymorphonuclear leukocytes (PMNs) during infection.

122 We tested cytotoxicity of LukGH toward polymorphonuclear leukocytes (PMNs) from mice, rabbits,

123 progenitor cells (HSPCs) differentiate into polymorphonuclear leukocytes (PMNs) in the bone marrow.

124 ntratracheal administration of LPS increased polymorphonuclear leukocytes (PMNs) in the bronchoalveol

125 nococcus), is characterized by the influx of polymorphonuclear leukocytes (PMNs) to the site of infec

126 A few polymorphonuclear leukocytes (PMNs) were already present

127 On necropsy, a large number of necrotic polymorphonuclear leukocytes (PMNs) were observed in the

128 ite the fundamental function of neutrophils (polymorphonuclear leukocytes (PMNs)) in innate immunity,

129 ing in septic plasma required C5a receptors, polymorphonuclear leukocytes (PMNs), and the Nacht-, LRR

130 Neutrophil granulocytes, also called polymorphonuclear leukocytes (PMNs), extrude molecular l

131 Our method reproducibly recovers 94.0% of polymorphonuclear leukocytes (PMNs), with up to 10(5) PM

132 , as they prevented an influx of Siglec-F(+) polymorphonuclear leukocytes (PMNs).

133 infections depends on bacterial clearance by polymorphonuclear leukocytes (PMNs); however, excessive

134 Neutrophil (polymorphonuclear leukocytes [PMN]) infiltration plays a

135 ed to increased infiltration of neutrophils (polymorphonuclear leukocytes [PMN]), macrophages (MPhi),

136 Human neutrophils (polymorphonuclear leukocytes [PMNs]) generate inflammato

137 n to avoid destruction by human neutrophils (polymorphonuclear leukocytes [PMNs]), which are crucial

138 n by human epithelial cells and neutrophils (polymorphonuclear leukocytes [PMNs]).

139 ous system (CNS) include infiltrating cells (polymorphonuclear leukocytes [PMNs], macrophages, and na

140 associated with decreases in infiltration of polymorphonuclear leukocytes and activation of microglia

141 ly 70% by 2C7-Ximab-E430G in the presence of polymorphonuclear leukocytes and complement.

142 re susceptible to oxidative killing by human polymorphonuclear leukocytes and displays decreased tiss

143 Munro's microabscesses contain polymorphonuclear leukocytes and form specifically in th

144 Primary polymorphonuclear leukocytes and keratinocytes were used

145 ment and reduced intrahepatic recruitment of polymorphonuclear leukocytes and NKT cells after islet i

146 Measurement of sputum polymorphonuclear leukocytes and other analytes, cortiso

147 it both apoptosis and the oxidative burst in polymorphonuclear leukocytes but were unable to identify

148 labeled bacteria was tested in primary mouse polymorphonuclear leukocytes by flow cytometry.

149 recruitment and the functional activities of polymorphonuclear leukocytes during infections caused by

150 blood and during phagocytic interaction with polymorphonuclear leukocytes ex vivo We conclude that Fa

151 , and decreases interstitial infiltration of polymorphonuclear leukocytes in a mouse model of TNF-alp

152 Large numbers of polymorphonuclear leukocytes in the amnion and chorion d

153 uired to counter toxic effectors secreted by polymorphonuclear leukocytes in the tissues.

154 ice had increased efferocytosis of apoptotic polymorphonuclear leukocytes instilled into the peritone

155 act of fruit reduced the edema, migration of polymorphonuclear leukocytes into the peritoneal cavity,

156 laque rupture and consequent thrombosis, but polymorphonuclear leukocytes likely promote endothelial

157 Neutrophils are polymorphonuclear leukocytes of the phagocytic system th

158 Cases had >=5 polymorphonuclear leukocytes on Gram stain plus symptoms

159 with NGU (visible urethral discharge or >/=5 polymorphonuclear leukocytes per high-power field [PMNs/

160 NGU was defined as >=5 polymorphonuclear leukocytes per high-power field on ure

161 nt score, and cervicitis was defined as >=30 polymorphonuclear leukocytes per high-power microscopic

162 The total number of bronchoalveolar lavage polymorphonuclear leukocytes recovered from the mice exp

163 Bacterial invasion of host tissues triggers polymorphonuclear leukocytes to release DNA [neutrophil

164 iae-infected Mmp2/9(-/-) mice recruited more polymorphonuclear leukocytes to the lung but had higher

165 ly, specific immunodepletion of neutrophils (polymorphonuclear leukocytes) blocked hCNS-SCns astrogli

166 ular phagocytosis with mouse NK cells, mouse polymorphonuclear leukocytes, and mouse macrophages.

167 reus abscesses, including the involvement of polymorphonuclear leukocytes, and provide a brief overvi

168 gion increase resistance to killing by human polymorphonuclear leukocytes, increase bacterial prolife

169 Protection was mediated by Gr-1(+) polymorphonuclear leukocytes, indicating a novel form of

170 ructure displayed cellular activity in human polymorphonuclear leukocytes, oral bioavailability, and

171 In polymorphonuclear leukocytes, SipA or other Salmonella p

172 n, epithelial exfoliation, and the influx of polymorphonuclear leukocytes.

173 the assembly of the LT synthetic complex in polymorphonuclear leukocytes.

174 ed potent ADCC with mouse NK cells and mouse polymorphonuclear leukocytes.

175 the alpha-subunit after G-CSF expression on polymorphonuclear leukocytes.

176 P12-associating lectin-1 (MDL-1) on immature polymorphonuclear leukocytes.

177 strong activity for stimulating migration of polymorphonuclear leukocytes.

178 enuated for survival upon challenge by human polymorphonuclear leukocytes.

179 nuated in ability to resist killing by human polymorphonuclear leukocytes.

180 ive inflammation with granulation tissue and polymorphonuclear leukocytes.

181 ls (PMVECs), alveolar macrophages (AMs), and polymorphonuclear leukocytes.

182 or-bearing mice with reduced accumulation of polymorphonuclear MDSC but not monocytic MDSC (M-MDSC),

183 t not monocytic MDSC (M-MDSC), and decreased polymorphonuclear MDSC suppression in vitro through the

184 cells (MDSCs), monocytic MDSCs (M-MDSCs) and polymorphonuclear MDSCs (PMN-MDSCs) regulate immune resp

185 t also an accumulation of tumor-infiltrating polymorphonuclear mononuclear cells caused by Cxcl-1 rel

186 Polymorphonuclear myeloid-derived suppressor cells (PMN-

187 T cells, B cells, natural killer (NK) cells, polymorphonuclear myeloid-derived suppressor cells (PMN-

188 y identified cells were eosinophils (n = 4), polymorphonuclears (n = 1), and giant cells (n = 1).

189 r knowledge, of how Leishmania-induced human polymorphonuclear neutrophil (hPMN) autophagy regulates

190 0.023), serum CRP level (P < 0.001) and oral polymorphonuclear neutrophil (oPMN) count (P < 0.001) wa

191 adenosine triphosphate (ATP) contributes to polymorphonuclear neutrophil (PMN) activation leading to

192 iphosphate (ATP) and adenosine in regulating polymorphonuclear neutrophil (PMN) activation, fibrin fo

193 Myeloperoxidase (MPO) is released during polymorphonuclear neutrophil (PMN) degranulation, and me

194 Polymorphonuclear neutrophil (PMN) extravasation require

195 To determine whether platelet and polymorphonuclear neutrophil (PMN) functions require spe

196 metabolite, adenosine, are key regulators of polymorphonuclear neutrophil (PMN) functions.

197 cin treatment also increased clinical score, polymorphonuclear neutrophil (PMN) infiltration (determi

198 heral organs in the two groups, WNV-infected polymorphonuclear neutrophil (PMN) infiltration and vira

199 onfirmed with synthetic MaR1, i.e., limiting polymorphonuclear neutrophil (PMN) infiltration in murin

200 observations revealed that a robust vaginal polymorphonuclear neutrophil (PMN) migration occurs in s

201 in response to C. albicans, which stimulate polymorphonuclear neutrophil (PMN) migration to the vagi

202 -surface protein, plays an important role in polymorphonuclear neutrophil (PMN) transmigration across

203 tly expressed on pulmonary epithelium and on polymorphonuclear neutrophil (PMNs) after transepithelia

204 tic ketoacidosis is associated with systemic polymorphonuclear neutrophil activation and degranulatio

205 2-triggered neutrophilia and monocytosis and polymorphonuclear neutrophil and monocyte renal infiltra

206 Of all the polymorphonuclear neutrophil azurophilic enzymes examine

207 , possibly due to the release of destructive polymorphonuclear neutrophil azurophilic enzymes.

208 Polymorphonuclear neutrophil granulocytes (neutrophils)

209 nds on phagocytosis of invading pathogens by polymorphonuclear neutrophil granulocytes (PMNs), follow

210 translates into increased transmigration of polymorphonuclear neutrophil granulocytes across endothe

211 demonstrated an attenuated extravasation of polymorphonuclear neutrophil granulocytes in Sema7a-defi

212 of Blood, Sapey et al. report that the human polymorphonuclear neutrophil leukocyte (or neutrophil) u

213 utrophils, tumor-associated neutrophils, and polymorphonuclear neutrophil myeloid-derived suppressor

214 endothelial cells that appear essential for polymorphonuclear neutrophil recruitment in IC disease.

215 To test individual mosaic subpopulations of polymorphonuclear neutrophil responses, we also develope

216 Interestingly, C5a-stimulated endothelial polymorphonuclear neutrophil transmigration, but not che

217 the activity of neutrophil serine proteases polymorphonuclear (neutrophil) elastase, cathepsin G, an

218 Polymorphonuclear neutrophilic granulocytes (PMN) as cel

219 h reactive oxygen species production by oral polymorphonuclear neutrophils (oPMNs) in chronic periodo

220 /80(-)I-A(-) intraglomerular macrophages and polymorphonuclear neutrophils (PMN) are important in ind

221 Polymorphonuclear neutrophils (PMN) are potent inflammat

222 Polymorphonuclear neutrophils (PMN) have distinct phenot

223 Because polymorphonuclear neutrophils (PMN) interaction with ECs

224 Polymorphonuclear neutrophils (PMN) play a central role

225 e of alphaMbeta2 affected the recruitment of polymorphonuclear neutrophils (PMN) to bacterial and fun

226 t-mediated phagocytosis and firm adhesion of polymorphonuclear neutrophils (PMN) under physiological

227 er PGE(2) could inhibit bacterial killing by polymorphonuclear neutrophils (PMN) using a mouse model

228 is expressed on human leukocytes, including polymorphonuclear neutrophils (PMN), monocytes, and macr

229 iated with significant tumor infiltration by polymorphonuclear neutrophils (PMN).

230 how that stimulation of alpha7nAChR on human polymorphonuclear neutrophils (PMNs) and blood mononucle

231 vitro model of transepithelial migration of polymorphonuclear neutrophils (PMNs) and in human intest

232 Polymorphonuclear neutrophils (PMNs) and macrophages are

233 Polymorphonuclear neutrophils (PMNs) and NK cells are bo

234 Polymorphonuclear neutrophils (PMNs) are critical for th

235 Polymorphonuclear neutrophils (PMNs) are increasingly re

236 Polymorphonuclear neutrophils (PMNs) are innate effector

237 Polymorphonuclear neutrophils (PMNs) are innate immune s

238 Polymorphonuclear neutrophils (PMNs) are largely conside

239 this infection METHODS: To determine whether polymorphonuclear neutrophils (PMNs) are required for di

240 ET formation in vivo and the infiltration of polymorphonuclear neutrophils (PMNs) but also reduces fi

241 Human polymorphonuclear neutrophils (PMNs) counteracted the pr

242 its functional relevance for recruitment of polymorphonuclear neutrophils (PMNs) during acute inflam

243 Trafficking of polymorphonuclear neutrophils (PMNs) during inflammation

244 Polymorphonuclear neutrophils (PMNs) express a number of

245 Polymorphonuclear neutrophils (PMNs) form the first line

246 We observed that polymorphonuclear neutrophils (PMNs) from FcgammaRIIIB w

247 nfection in skin involves the recruitment of polymorphonuclear neutrophils (PMNs) from the bone marro

248 Polymorphonuclear neutrophils (PMNs) from these KO mice

249 Polymorphonuclear neutrophils (PMNs) have previously bee

250 uces pro-inflammatory cytokine production by polymorphonuclear neutrophils (PMNs) of diabetic individ

251 DGKalpha function is altered in polymorphonuclear neutrophils (PMNs) of patients with lo

252 Polymorphonuclear neutrophils (PMNs) play a critical rol

253 Bone marrow (BM) holds a large reserve of polymorphonuclear neutrophils (PMNs) that are rapidly mo

254 Recruitment of polymorphonuclear neutrophils (PMNs) to sites of acute i

255 ammatory cytokines followed by attraction of polymorphonuclear neutrophils (PMNs) to the site of infl

256 During inflammation polymorphonuclear neutrophils (PMNs) traverse venular wa

257 count for this absence, we hypothesized that polymorphonuclear neutrophils (PMNs), recruited massivel

258 ADAM9 is also a product of human and murine polymorphonuclear neutrophils (PMNs).

259 ogous to CD16A and a major FcgammaR on human polymorphonuclear neutrophils (PMNs).

260 is a risk factor for TRALI and neutrophils (polymorphonuclear neutrophils [PMNs]) are considered to

261 Neutrophils (polymorphonuclear neutrophils [PMNs]) play an elaborate

262 cluded albumin leak into lung and buildup of polymorphonuclear neutrophils and cytokines/chemokines i

263 With human polymorphonuclear neutrophils and macrophages, RvD1, RvD

264 icates the inflammatory response mediated by polymorphonuclear neutrophils and monocytes as the key e

265 hlamydia-specific immunoglobulin G (IgG) and polymorphonuclear neutrophils and show the importance of

266 Polymorphonuclear neutrophils constitute the first line

267 ed as visible urethral discharge and/or >/=5 polymorphonuclear neutrophils per high-powered field; n

268 Blood polymorphonuclear neutrophils provide immune protection

269 whereas adoptive transfer of MD-2-competent polymorphonuclear neutrophils restored it.

270 te inflammation by blocking the migration of polymorphonuclear neutrophils to initiate resolution.

271 rane response increased the ability of human polymorphonuclear neutrophils to readily clear both E. f

272 Elevated counts of polymorphonuclear neutrophils were detectable at 15 minu

273 Myeloperoxidase, a heme enzyme released by polymorphonuclear neutrophils, accumulates within ischem

274 ing the migration of monocytes, neutrophils, polymorphonuclear neutrophils, and dendritic cells into

275 d Y. pestis' ability to counter lysozyme and polymorphonuclear neutrophils, but it did not affect the

276 and possibly inflammatory monocytes, but not polymorphonuclear neutrophils, in clearing HSV-1 from th

277 the adhesion of a type of white blood cell--polymorphonuclear neutrophils--to the lining of the circ

278 profile and impaired antifungal activity of polymorphonuclear neutrophils.

279 rs (degree of fibrosis) and concentration of polymorphonuclear neutrophils.

280 ollagenase thought to be expressed mainly by polymorphonuclear neutrophils.

281 is and reactive oxygen species generation in polymorphonuclear neutrophils.

282 ctively inhibited CXCL8-induced migration of polymorphonuclear neutrophils.

283 sis in children was associated with elevated polymorphonuclear neutrophils.

284 that this oxysterol increases the number of polymorphonuclear-neutrophils and gammadelta-T cells at

285 tions of 27-hydroxycholesterol requires both polymorphonuclear-neutrophils and gammadelta-T cells, an

286 hrough its actions on gammadelta-T cells and polymorphonuclear-neutrophils, 27-hydroxycholesterol fun

287 CD11b(+) Ly6C(int) Ly6G(+) cells exhibited a polymorphonuclear or band-shaped nuclear morphology.

288 decreased accumulation and/or activation of polymorphonuclear (PMN) and CD4(+) and CD8(+) T cells, a

289 epithelial cells (SECs) and high numbers of polymorphonuclear (PMN) cells are regarded as indicative

290 ungs during infection but also accumulate in polymorphonuclear (PMN) cells.

291 neal epithelial wound closure and attenuated polymorphonuclear (PMN) leukocyte responses, a phenotype

292 Neutrophils, polymorphonuclear (PMN) leukocytes, play an important ro

293 antitative and qualitative reconstitution of polymorphonuclear (PMN), CD4, CD8, and natural killer (N

294 hat have been identified in humans and mice: polymorphonuclear (PMN)-MDSCs and monocytic (M)-MDSCs.

295 we demonstrate that monocytic (M)-MDSCs and polymorphonuclear (PMN)-MDSCs can be detected using seve

296 consist of two major subsets, monocytic and polymorphonuclear (PMN).

297 receptor I (FcgammaRI) CD64 on neutrophils (polymorphonuclear [PMN] CD64 index) in flow cytometry wa

298 ver disease (ALD) with intense neutrophilic (polymorphonuclear; PMN) inflammation and high mortality.

299 eratinocyte-derived cytokine measurement and polymorphonuclear recruitment in bronchoalveolar lavage

300 indicate that properdin in serum as well as polymorphonuclear-released properdin is unable to bind a