ライフサイエンスコーパス: polysomnography

1 racheal aspirate cultures, bronchoscopy, and polysomnography).

2 as the risk factor of OSA (diagnosed through polysomnography).

3 apnea-hypopnea index >/=15 events per hour (polysomnography).

4 f rapid eye movement sleep without atonia on polysomnography.

5 th functional magnetic resonance imaging and polysomnography.

6 All of the subjects underwent polysomnography.

7 h as electroencephalographic recordings from polysomnography.

8 inical-biochemical assessment and a standard polysomnography.

9 nd obstructive sleep apnoea, as confirmed by polysomnography.

10 sleep obtained from overnight in-laboratory polysomnography.

11 y selected sleep-lab subjects underwent full polysomnography.

12 me on actigraphy and apnea-hypopnea index on polysomnography.

13 of SV by digital photoplethysmography during polysomnography.

14 atories and validated the measurements using polysomnography.

15 ilated subjects were monitored by continuous polysomnography.

16 SDB was assessed at baseline with full polysomnography.

17 ltaNC) and leg fluid volume before and after polysomnography.

18 s assessed on separate nights using standard polysomnography.

19 ging from normal to obese underwent attended polysomnography.

20 raphy that followed 1 night of accommodation polysomnography.

21 iary care medical center underwent overnight polysomnography.

22 red questions instead of the 'gold standard' polysomnography.

23 n a sleep laboratory using sleep diaries and polysomnography.

24 presence of OSA was determined by overnight polysomnography.

25 concurrent UES and esophageal manometry and polysomnography.

26 lance Test, diaries of patients, and daytime polysomnography.

27 ts (14 women) undergoing combined stereo-EEG/polysomnography.

28 per hour of sleep, measured by in-laboratory polysomnography.

29 nd without underlying lung disease underwent polysomnography.

30 nnaire, and underwent oximetry and two-night polysomnography.

31 BP) in 4,409 subjects referred for overnight polysomnography.

32 5 to February 1998 and characterized by home polysomnography.

33 Thirty-five patients underwent polysomnography.

34 r hour of sleep, was measured during in-home polysomnography.

35 on) [corrected], obtained by unattended home polysomnography.

36 of spontaneous awakenings was determined by polysomnography.

37 osed by reported dream-enactment symptoms or polysomnography.

38 ness characterized by insomnia, confirmed by polysomnography.

39 somnia with demonstration of the disorder by polysomnography.

40 ian time (phase), and sleep was monitored by polysomnography.

41 s per hour of sleep, measured during in-home polysomnography.

42 breathing was excluded by complete overnight polysomnography.

43 ning by specialists in patients referred for polysomnography.

44 ilateral cortical windows and electrodes for polysomnography.

45 ssure swings were measured via in-laboratory polysomnography.

46 had undiagnosed sleep apnea, as detected by polysomnography.

47 lts utilizing objective assessments, such as polysomnography.

48 tials, intracranial electroencephalogram and polysomnography.

49 cle signals show comparable performance with polysomnography.

50 isk were recruited at 3 public hospitals for polysomnography.

51 ld OSA were difficult to distinguish without polysomnography.

52 prodromal criteria and a standard overnight polysomnography.

53 ipants completed two overnight in-laboratory polysomnographies (1-week washout), with an epiglottic c

54 n tests, transthoracic echocardiography, and polysomnography 3 months after ICU discharge.

55 In 89 patients with NAION who underwent polysomnography, 67 (75%) had OSAS.

56 Measurements included polysomnography, a multiple sleep latency test, an oral

57 smography (RIP) with simultaneously recorded polysomnography-acquired nasal end-tidal CO(2) (PET(CO(2

58 thods to measure sleep in the ICU, including polysomnography, actigraphy, and questionnaires.

59 All patients received follow-up polysomnography after 6 months.

60 roblem severity and depression severity, and polysomnography after at least 2 weeks of abstinence.

61 rice-weekly hemodialysis underwent overnight polysomnography along with measurement of total body ext

62 Children underwent polysomnography and 24-hour ambulatory BP monitoring (AB

63 We investigated 59 persons simultaneously by polysomnography and 3-D-camera and visual perceptive com

64 osed with RBD (19.8%), including 30 (35%) by polysomnography and 56 (65%) as probable.

65 d patients and 13 healthy subjects underwent polysomnography and [(18)F]fluorodeoxyglucose positron e

66 Polysomnography and activity monitoring revealed a profo

67 , 10th Revision, code for OSA confirmed with polysomnography and an additional code for continuous po

68 History of OSA was determined by polysomnography and associated conditions, including apn

69 mean age = 60.3 +/- 9.9) that underwent full polysomnography and brain magnetic resonance imaging to

70 orrelation was found between PLM measured by polysomnography and by the 3-D-camera (RLS: r = 0.654; p

71 ndex (mean score=49; range=15-111)-underwent polysomnography and completed the Center for Epidemiolog

72 were randomized to WWSC or AT and underwent polysomnography and completed validated QOL and symptom

73 who were diagnosed with OSA after overnight polysomnography and control children matched on the basi

74 Full montage home-polysomnography and fasting glucose were available on al

75 dren (ages 4.8-12 years) underwent overnight polysomnography and fasting homeostatic model (HOMA) of

76 a fasting morning blood sample at diagnostic polysomnography and follow-up 1.3 +/- 0.6 yr later.

77 o completed baseline and follow-up overnight polysomnography and had complete questionnaire-based dat

78 of 112 Minnesota residents who had undergone polysomnography and had died suddenly from cardiac cause

79 n=10) subjects were assessed with concurrent polysomnography and LFP recordings from the DBS electrod

80 Polysomnography and lumbar puncture were performed in OS

81 with a wide range of SDB severity underwent polysomnography and measurement of high-sensitivity CRP.

82 eep Heart Health Studies underwent overnight polysomnography and measurement of high-sensitivity trop

83 nts (mean age, 62.5 +/- 5.5 years) underwent polysomnography and measurement of hs-TnT.

84 e cohort study, patients underwent overnight polysomnography and MRI using a Dixon sequence.

85 ence analysis on 127 patients with nocturnal polysomnography and MSLT, including 25 with narcolepsy w

86 Polysomnography and multiple sleep latency testing are u

87 f 2 weeks of actigraphy at home, 2 nights of polysomnography and multiple sleep latency tests in the

88 recruited from public schools and underwent polysomnography and neurocognitive assessments of intell

89 the literature provides the available normal polysomnography and oximetry data for reference and docu

90 to adolescence, where participants underwent polysomnography and performed a declarative word-pair le

91 criterion validity in companion dogs against polysomnography and physical activity monitors (PAMs).

92 n the apnea hypopnea index (AHI) measured by polysomnography and respiratory events measured with the

93 ects (9 women, age 22 to 45 years) underwent polysomnography and simultaneous recording of ECG, blood

94 Polysomnography and sleep physiology were assessed in we

95 Polysomnography and spirometry results from 5,954 partic

96 The patients underwent polysomnography and studies of their ventilatory respons

97 5.71) healthy subjects underwent a nocturnal polysomnography and T1 MRI.

98 rior history of hypothyroidism who underwent polysomnography and thyroid function testing, four new c

99 ) underwent 3 consecutive nights of standard polysomnography and weight and height assessments as par

100 Participants underwent baseline polysomnography and were followed for a mean 7.3 years f

101 ltisite community-based study that conducted polysomnography and wrist actigraphy.

102 a Index (AHI) greater than 15 as assessed by polysomnography, and in 14 older adults (age +/- SD: 62.

103 diffusion tensor imaging and structural MRI, polysomnography, and neuropsychological assessments.

104 affeine versus placebo underwent actigraphy, polysomnography, and parental sleep questionnaires.

105 topographer, apnea-hypopnea index (AHI) with polysomnography, and serum HIF-1alpha, MMP2, and desmosi

106 of SDB was based on the results of overnight polysomnography, and severe SDB was defined as an apnea-

107 ions by magnetoencephalography together with polysomnography, and source-localized the origins of osc

108 cohort study, consecutive patients underwent polysomnography, and subsequent events (strokes and deat

109 The subjects underwent clinical assessment, polysomnography, and wrist actigraphy.

110 ter sleep onset, derived from sleep diaries; polysomnography; and symptoms of fatigue, sleepiness, an

111 In a clinical research facility, overnight polysomnography, anthropometry, and 9 blood pressure mea

112 Findings on nocturnal polysomnography are described.

113 Using polysomnography as the criterion standard, we prospectiv

114 seline to 6 months, measured by a full-night polysomnography assessed by masked investigators in a co

115 n age = 72.3 +/- 5.8) completed an overnight polysomnography assessment with emotional memory tested

116 mmunities (ARIC) study who completed in-home polysomnography assessments.

117 ransplant recipients who underwent one-night polysomnography at baseline and were followed for a medi

118 ividuals who underwent overnight, unattended polysomnography at home.

119 ta, personal and treatment history, and full polysomnography at home.

120 ires, 24-hour blood pressure monitoring, and polysomnography at the end of follow-up.

121 ) was determined by unattended, single-night polysomnography at the participant's home.

122 era are less time-consuming in comparison to polysomnography because they do not require sophisticate

123 a group of patients with OSA diagnosed with polysomnography between 1992 and 2004 (apnea-hypopnea in

124 eep center in Zaragoza, Spain, for nocturnal polysomnography between January 1, 1994, and December 31

125 ocardiography, range-of-motion measurements, polysomnography, clinical laboratory evaluations, measur

126 Polysomnography, cognitive performance, and sleepiness w

127 PVI between 2007 and 2010, 62 patients had a polysomnography-confirmed diagnosis of OSA.

128 by lumbar puncture from patients with video polysomnography-confirmed IRBD recruited at a specialise

129 Eighty-seven patients with polysomnography-confirmed IRBD underwent (123) I-FP-CIT

130 signature was identified in 48 patients with polysomnography-confirmed iRBD using partial least squar

131 an age 70.5 +/- 6.3 years, 70.5% males) with polysomnography-confirmed RBD who eventually phenoconver

132 patients 72.8% of leg movements confirmed by polysomnography could be detected by 3-D-video and a sig

133 Overnight polysomnography data containing EEG across sleep and wak

134 Polysomnography data from 2121 people were included in t

135 We explored associations between overnight polysomnography-derived measures of OSA and the optic di

136 Children aged 3.0 to 12.9 years with polysomnography-diagnosed (AHI <3) mild obstructive slee

137 Blood pressure and overnight polysomnography did not change after fat gain or loss.

138 ng sound recordings during in-lab full-night polysomnography, drug-induced sleep endoscopy (DISE), an

139 r an unfamiliar voice (UFV), while recording polysomnography during a full night of sleep.

140 rwent combined stereo-electroencephalography/polysomnography during presurgical evaluation.

141 vity in the early auditory cortex along with polysomnography during thousands of episodes when male r

142 Eleven OSA subjects underwent a night of polysomnography during which the physiological traits we

143 as superior to placebo on all subjective and polysomnography end points at night 1/week 1, month 1, a

144 patients at night 1, month 1, and month 3 by polysomnography end points of wakefulness after persiste

145 nstrate that Somnotate sets new standards in polysomnography, exhibiting annotation accuracies that e

146 ts were tested with 1 night of in-laboratory polysomnography followed by a cognitive evaluation the n

147 , the apnea-hypopnea index was determined by polysomnography followed by determination of anatomic (p

148 to investigate BP homeostasis, we conducted polysomnography followed by tilt-table testing on 15 sub

149 istory alone, but some may require nocturnal polysomnography for accurate diagnosis and determining a

150 RECOMMENDATION 2: ACP recommends polysomnography for diagnostic testing in patients suspe

151 rty patients undergoing nocturnal diagnostic polysomnography for sleep apnea underwent transcranial D

152 Clinical practices utilise polysomnography for sleep assessment, which is intrusive

153 ipants were assessed overnight by 18-channel polysomnography for sleep-disordered breathing, as defin

154 es in each of 38 adult patients evaluated by polysomnography for sleep-disordered breathing.

155 336 consecutive adult patients who underwent polysomnography for suspected OSA.

156 ay be a simpler alternative to in-laboratory polysomnography for the management of more symptomatic p

157 e we develop non-invasive fluorescence-based polysomnography for zebrafish, and show-using unbiased,

158 and respiratory parameters were recorded by polysomnography from 4 PM to 9 AM on the second, third,

159 ants were individuals referred for overnight polysomnography from March 1 to August 30, 2015.

160 l factors, was conducted using in-laboratory polysomnography from March 20, 2017, to June 3, 2019.

161 nd slow sleep spindle duration in full-night polysomnography has only been reported in females but no

162 Polysomnography has specific preoperative indications.

163 dogs, including development of non-invasive polysomnography; however, basic understanding of dog sle

164 l corticosteroids improve SDB as measured by polysomnography; however, the effect on symptoms and qua

165 We monitored Pes during polysomnography in 155 patients and compared their sleep

166 challenge (nasal occlusion) during overnight polysomnography in 31 women (45 to 55 yr).

167 herefore measured blood pressure (BP) during polysomnography in 41 children with OSAS, compared to 26

168 We also assessed its accuracy compared with polysomnography in a sample of the study population.

169 e (OSAS) is usually diagnosed with overnight polysomnography in a sleep laboratory.

170 er adults was assessed using objective sleep polysomnography in combination with longitudinal trackin

171 mated with oesophageal pressure, Pes) during polysomnography in four adult volunteers and applied the

172 sual Analogue Scale measures) and sleep (via polysomnography), including increased REM and NREM sleep

173 The cost-utility of polysomnography instead of home study or no testing in t

174 The introduction of polysomnography into psychiatric research confirmed a di

175 Polysomnography is invaluable for the evaluation of slee

176 increased neck circumference), but overnight polysomnography is needed to confirm presence of the dis

177 es as an alternative to polysomnography when polysomnography is not available for diagnostic testing.

178 Therefore, polysomnography is not necessary for most patients with

179 populations is essential; however, access to polysomnography is often limited due to its high cost, t

180 Polysomnography is the diagnostic standard, but is often

181 Although polysomnography is the gold standard for arousal detecti

182 Stereo-electroencephalography paired with polysomnography is the ideal tool to study this relation

183 ss (6 mo) of home respiratory polygraphy and polysomnography management protocols in patients with in

184 These results suggest that a night of polysomnography may aid in evaluating tau and Abeta burd

185 SD] age: 82.3 [3.2] years) who had overnight polysomnography measured between January 2002 and April

186 Confirmatory tests such as polysomnography, multiple sleep latency test, and actigr

187 = 4,307-10,332), actigraphy (n = 1,513), or polysomnography (n = 3,021).

188 of 114 subjects with 2 consecutive nocturnal polysomnographies (New York, NY) without esophageal mano

189 and dermatitis, sleep variables measured by polysomnography, nocturnal urinary levels of 6-sulfatoxy

190 t were adequately specific (77%) to rule out polysomnography noninsomnia.

191 MSLTs were conducted following nocturnal polysomnography (NPSG) and daily sleep diaries in 289 ma

192 ur asymptomatic subjects underwent nocturnal polysomnography (NPSG) with monitoring of flow (nasal ca

193 leep Latency Test (MSLT) following nocturnal polysomnography (NPSG).

194 Methods: DeltaHR was measured from baseline polysomnography of the RICCADSA (Randomized Intervention

195 mode of mechanical ventilation, we performed polysomnography on 11 critically ill patients.

196 d matched control participants using in-home polysomnography on 4 nights.

197 sleep apnea, have improvements documented by polysomnography on the night of surgery following adenot

198 patients and 17 healthy volunteers underwent polysomnography on two consecutive nights.

199 n a population of 10,701 adults referred for polysomnography, OSA predicted incident SCD, and the mag

200 The incremental charges for polysomnography over home study or no testing were about

201 the mean oxygen desaturation index among the polysomnography parameters (P = 0.023).

202 urther optic disc changes in relationship to polysomnography parameters to be documented and associat

203 ng a night of sleep, which we monitored with polysomnography, participants encoded a second set of fa

204 Polysomnography plays an important role in addressing pi

205 Using combined stereo-EEG/polysomnography possible only in the human brain during

206 y polygraphy protocol was noninferior to the polysomnography protocol based on the Epworth scale.

207 were randomized to respiratory polygraphy or polysomnography protocols.

208 orientation was assessed in 21 patients with polysomnography-proven idiopathic REM sleep behaviour di

209 Under almost all modeled conditions, polysomnography provided maximal quality-adjusted life-y

210 ive care unit (ICU) patients with continuous polysomnography (PSG) and environmental noise measuremen

211 ical sleep diagnosis traditionally relies on polysomnography (PSG) and expert manual classification o

212 Among 14,152 patients who had undergone polysomnography (PSG) and whose data were registered on

213 n patients with advanced cancer, but 24-hour polysomnography (PSG) examinations of these patterns hav

214 asingly conducted at home) versus laboratory polysomnography (PSG) for diagnosing obstructive sleep a

215 d with two measures of arousal; conventional polysomnography (PSG) for electroencephalogram (EEG) ass

216 l patients and controls underwent over-night polysomnography (PSG) for the diagnosis of OSAS and calc

217 However, current gold standard polysomnography (PSG) has limited spatial resolution to

218 Efforts to simplify standard polysomnography (PSG) in laboratories, especially for ob

219 To date, polysomnography (PSG) is the most commonly used sleep-mo

220 p monitoring and analysis using multivariate polysomnography (PSG) records has achieved significant e

221 apnea (OSA) depends on clinical examination, polysomnography (PSG) results, and imaging analysis.

222 ingham Heart Study who completed 2 overnight polysomnography (PSG) studies in the time periods 1995 t

223 hiatric evaluations, simultaneous ambulatory polysomnography (PSG), and near-infrared spectroscopy (N

224 esults of the complementary tests, including polysomnography (PSG), brain imaging and genetic analysi

225 current gold standard for measuring sleep is polysomnography (PSG), but it can be obtrusive and costl

226 and tolerances in volunteers after overnight polysomnography (PSG).

227 ho underwent overnight unattended 12-channel polysomnography (PSG).

228 Measurements: Polysomnography, pulmonary function tests, arterial bloo

229 Among individuals referred for overnight polysomnography, quantitative markers of eyelid laxity w

230 ekly episodes (72 to 96 hours) of continuous polysomnography (r=0.94) and work logs that were validat

231 nasal CPAP, recalibration of nasal CPAP with polysomnography, regular downloading of home data, and a

232 leep slow waves, objectively quantified with polysomnography, relate to longitudinal changes in Unifi

233 The proper interpretation of infant polysomnography requires an understanding of normative d

234 tain information on thyroid function status, polysomnography results, levothyroxine use, and clinical

235 nce and severity of OSA were determined from polysomnography results.

236 Using polysomnography, retrospective questionnaires, and tau-

237 Despite careful screening, polysomnography revealed that 1 of 25 normal-weight subj

238 seline to 2-year change in AHI on diagnostic polysomnography scored by staff blinded to randomization

239 AS is common in patients with NAION and that polysomnography should be considered in these patients.

240 In five of five patients, video polysomnography showed features of obstructive sleep apn

241 Finally, overnight polysomnography showed that sleep-related memory enhance

242 health-related quality-of-life assessments, polysomnography, spirometry, 6-minute-walk distance, dro

243 identifying the disease severity stage, but polysomnography still remains the definitive gold standa

244 isconsin state employees to attend overnight polysomnography studies at 4-year intervals.

245 Adult patients undergoing diagnostic polysomnography studies at a private Australian universi

246 d covariate information were assessed during polysomnography studies through March 2013.

247 nts who were 30-70 years of age had baseline polysomnography studies to assess the presence of sleep-

248 ep diagnoses and sleep architecture based on polysomnography studies, actigraphy assessment, and 24-h

249 f <5 events/h and not treated) by 2 baseline polysomnography studies.

250 Participants then underwent an overnight polysomnography study that obtained measurements of apne

251 ctive- and placebo-controlled, dose-finding, polysomnography study was conducted from November 2017 t

252 Of 1,001 polysomnography subjects, 90 with SDB defined as a respi

253 ctive sleep measures (e.g. wrist actigraphy, polysomnography) support links between disturbed sleep a

254 nts underwent a single night of experimental polysomnography that followed 1 night of accommodation p

255 video-electroencephalographic telemetry and polysomnography, the differential diagnostic challenges

256 Using polysomnography, the gold standard for sleep assessment,

257 Prior to overnight polysomnography, the participants were given a battery o

258 en ages 30 to 60 yr was studied by overnight polysomnography to assess the frequency of apneas and hy

259 Patients underwent polysomnography to detect OSAS and were prospectively fo

260 ldren from the community underwent overnight polysomnography to determine SDB severity (obstructive a

261 Nocturnal polysomnography to evaluate apnea-hypopnea index.

262 All participants underwent polysomnography to exclude obstructive sleep apnea or ot

263 All control subjects underwent overnight polysomnography to exclude the existence of occult OSA.

264 ervational study, we used clinical and video polysomnography to identify a novel sleep disorder in th

265 plus nocturnal pulse oximetry against using polysomnography to identify patients without apnea (Obje

266 (working memory measure) tests and overnight polysomnography to investigate the specific sleep-depend

267 easurement unit, is used in conjunction with polysomnography to understand the relationship between l

268 HI) of at least 20 events per h, tested by a polysomnography, underwent device implantation and were

269 Polysomnography was assumed as the gold standard.

270 prevalence of objective insomnia assessed by polysomnography was higher than the prevalence of subjec

271 Home unattended polysomnography was performed and scored using similar p

272 Overnight polysomnography was performed during the 2 sessions.

273 Full night polysomnography was performed during two consecutive nig

274 Polysomnography was performed throughout the study.

275 teral local injection of orexin-saporin, and polysomnography was performed to measure baseline sleep

276 months after the LT; in each phase, standard polysomnography was performed, and anthropometric, patho

277 Full polysomnography was used to characterize SDB.

278 Polysomnography was used to characterize sleep architect

279 ed neuroimaging techniques [7, 8] as well as polysomnography, we found that the temporary sleep distu

280 Using simultaneous fiber photometry and polysomnography, we observed time-delineated dorsal raph

281 Here, in parallel with whole-night video polysomnography, we recorded local field potentials from

282 2omSOREMPs on an MSLT that follows nocturnal polysomnography, we reviewed data from 1,145 consecutive

283 ide ventriculography, Holter monitoring, and polysomnography were done.

284 se and heart failure at the time of baseline polysomnography were followed up for a median of 8.7 yea

285 ation averaged over 5 minutes, and overnight polysomnography were obtained in participants, each with

286 onnaires from patients and bed partners, and polysomnography were obtained on all subjects in compari

287 Quality of life, blood pressure, and polysomnography were similar between protocols.

288 of sleep apnea, derived from 12-channel home polysomnography, were the apnea-hypopnea index (average

289 t serious comorbidities as an alternative to polysomnography when polysomnography is not available fo

290 These five patients had been assessed with polysomnography, which was done in our sleep unit in one

291 ure (Pes) monitoring can be performed during polysomnography with a thin, water-filled catheter conne

292 in 146 participants who underwent overnight polysomnography with an epiglottic catheter to measure t

293 Some patients referred for polysomnography with complaints of excessive daytime sle

294 removed by UF, followed by repeat overnight polysomnography with fluid measurements.

295 d recurrence of exacerbations.Methods: Video polysomnography with neck-muscle EMG was performed in pa

296 (MrOS) Sleep Study cohort underwent in-home polysomnography with PLMS measurement and were followed

297 olved two consecutive nights of high density polysomnography with training on the Motor Sequence Task

298 ygraphy management is similarly effective to polysomnography, with a substantially lower cost.

299 and related symptoms (all P < .01), but not polysomnography, with similar improvements in both group

300 clinical evaluation, subjective scales, four polysomnographies without nasal CPAP, recalibration of n