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1 e to settings where the exposure variable is polytomous and where the assumption of independence betw
2 atment is dichotomous, treatment is actually polytomous as there are at least 3 levels: no treatment,
3 ferent morphological habitus, including long polytomous body branches and a maximum body length of mo
5 lassic (Levin's) epidemiological formula for polytomous exposures, with relative risks (RRs) reported
7 ted the biomarker panel to LV geometry using polytomous logistic regression adjusting for clinical co
8 h case-only logistic regression analyses and polytomous logistic regression analyses (with one contro
9 f a mutation in any of the LS genes by using polytomous logistic regression analysis of clinical and
13 In this paper, the authors describe how a polytomous logistic regression method previously develop
21 ral log-1 unit increase) were assessed using polytomous logistic regression models, joint effects usi
27 authors propose an extension to quantitative polytomous logistic regression that allows testing for m
31 p but not enrollment (n = 688; 40%) and used polytomous logistic regression to estimate odds ratios (
32 to investigate etiologic heterogeneity or do polytomous logistic regression to estimate odds ratios s
33 egorical outcome typically entails fitting a polytomous logistic regression via maximum likelihood es
42 Factors associated with surgery use (from polytomous logistic regression); overall and breast canc
43 analyses to support differences in risk: (1) polytomous logistic regression, (2) homogeneity tests, o
50 entional) were estimated using multivariable polytomous logistic regressions and multilevel models.
53 e control group examined by colonoscopy in a polytomous model with several case groups (newly diagnos
55 ype, for example, binary, count, continuous, polytomous, ordinal, time-to-onset, multivariate and oth
56 ment before and after rehabilitation, by the polytomous rating scale measurement model of Wright and
63 A three-parameter logistic model (3PL) for polytomous response was calculated to evaluate a model o
64 bias away from the null; and, if exposure is polytomous, the bias produced by independent nondifferen
65 ngness, and trajectories were modelled using polytomous variable latent class analysis (poLCA) in bot