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1 ion was done in 52 (37%) of those undergoing portoenterostomy.
2 r patients (86%) had undergone prior hepatic portoenterostomy.
3 f recipient age, weight, or previous hepatic portoenterostomy.
4 autoantibodies in the first year after Kasai portoenterostomy.
5 actors that drive liver fibrosis after Kasai portoenterostomy.
6 ents with BA, collected at the time of Kasai portoenterostomy, along with liver biopsies from infants
7 d with improved outcomes following the Kasai portoenterostomy and longer survival with the native liv
8 reatment of this disease have been the Kasai portoenterostomy and orthotopic liver transplantation.
9 en will achieve biliary drainage after Kasai portoenterostomy and will have serum bilirubin within th
10 ort of subjects (6-12 months following Kasai portoenterostomy) and correlations of autoantibodies wit
11 ts depend on early referral and timely Kasai portoenterostomy, and thus a high index of suspicion is
12 study of 43 infants who underwent the Kasai portoenterostomy as treatment for biliary atresia at the
14 of minimally invasive approaches to hepatic portoenterostomy but there has been little comparative s
17 de advantages for newborn procedures such as portoenterostomy for biliary atresia and repair of esoph
19 investigated risk factors for failure after portoenterostomy for biliary atresia using univariate an
25 natomic features of infants undergoing Kasai portoenterostomy (KPE) for biliary atresia (BA) and to e
27 es of surgical management, typically a Kasai portoenterostomy (KPE), vary considerably across the wor
32 ding transplantation as primary therapy, but portoenterostomy remains the standard of care as first-l
33 espite multiple modifications of the hepatic portoenterostomy, two thirds of treated patients still d