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1 s not express p185 are strong suppressors of position effect variegation.
2 act in these domains, as shown by monitoring position effect variegation.
3 d by suppression of heterochromatin-mediated position effect variegation.
4 allelic to Lighten-up, a known suppressor of position effect variegation.
5 ression, a heterochromatin phenomenon called position effect variegation.
6 ncer (TE) and a region that protects against position effect variegation.
7 netic gene silencing classically observed as position effect variegation.
8  reminiscent of, but clearly different from, position effect variegation.
9 l involvement of the P0 protein in modifying position effect variegation.
10           In addition, Low weakly suppresses position effect variegation.
11 n, eye development, chromosomal proteins and position effect variegation.
12 9me3, and is a classic genetic suppressor of position-effect variegation.
13 yed by nuclear organization in cis and trans position-effect variegation.
14 acts as both an enhancer and a suppressor of position-effect variegation.
15 to further methylation, suggests a model for position-effect variegation.
16 cus, initially identified as a Suppressor of Position-Effect Variegation.
17 lls, an effect that is probably analogous to position-effect variegation.
18  nearby gene in cis, a hallmark of classical position-effect variegation.
19 a HP1, a heterochromatin protein involved in position-effect variegation.
20   By using a combination of two modifiers of position effect variegation, adding an extra Y chromosom
21 to demonstrate that dLDH and L-2HG influence position effect variegation and DNA methylation, suggest
22                 LCRs protect transgenes from position effect variegation and heterochromatinization a
23  natural or synthetic PATCs are resistant to position effect variegation and stochastic silencing in
24 ls of retinal degeneration and mechanisms of position-effect variegation and demonstrate the utility
25      The lawc gene behaves as an enhancer of position-effect variegation and interacts genetically wi
26 gmentation patterns similar to those seen in position-effect variegation and yet most inserts were in
27 ivity to DNA-damaging agents, suppression of position-effect variegation, and female sterility in whi
28 L-1 histone H3S10 kinase act as enhancers of position-effect variegation at pericentric sites whereas
29 tion in flies acts as a dominant enhancer of position effect variegation but in a more context-specif
30 overed in HP2 act as dominant suppressors of position effect variegation, confirming a role in hetero
31 of silencers in trans or by the spreading of position effect variegation from rearrangements having h
32  in Drosophila melanogaster for modifiers of position-effect variegation have revealed the basis of m
33  a variety of biological processes including position-effect variegation, heterochromatin formation a
34 erature, such as vernalization in plants and position effect variegation in animals.
35                         Similarities between position effect variegation in Drosophila and gene silen
36 hat the loss of Otu1 and Usp5 induces strong position effect variegation in Drosophila eye following
37 transcriptional silencing, as exemplified by position effect variegation in Drosophila melanogaster a
38 s enhanced by lower temperatures, similar to position effect variegation in Drosophila.
39 to the postulated role of this DNA repeat in position effect variegation in facio- scapulohumeral mus
40 as been described in diverse systems such as position effect variegation in insects, silencing near y
41 letes H3K9 methylation levels and suppresses position-effect variegation in various Drosophila tissue
42  presence of Su(z)12, a strong suppressor of position effect variegation, in PRC2 suggests that PRC2
43 y nor qualitatively affected by modifiers of position effect variegation including the Y chromosome,
44                Mutations in HP2 can suppress position effect variegation, indicating a role in gene s
45 tation in Drosophila PR-Set7 that suppresses position effect variegation, indicating that PR-Set7 ind
46 r full function of the AE1 promoter and that position effect variegation is associated with RNA trans
47                                              Position effect variegation may be considered an abnorma
48 tic DNA instability described here underlies position effect variegation, molds the structure of poly
49                                              Position effect variegation of most Drosophila melanogas
50                                              Position-effect variegation of the w(m4h) allele and dif
51                  In Drosophila melanogaster, position-effect variegation of the white gene has been a
52 riptional repression of nearby marker genes (position-effect variegation or silencing).
53 r the spread of inactivation associated with position-effect variegation or X chromosome inactivation
54 tic screens that relied on mosaic silencing (position-effect variegation, or PEV) of the yellow gene
55                                              Position effect variegation (PEV) in Drosophila results
56 d HSS3), which is required for prevention of position effect variegation (PEV) in transgenic mice.
57 l are lethal, heterozygotes display enhanced position effect variegation (PEV) indicative of the broa
58                                              Position effect variegation (PEV) is the clonal inactiva
59                                              Position effect variegation (PEV) occurs when a gene is
60 some (Dp(1;f)1187) dramatically increase the position effect variegation (PEV) of a yellow(+) body-co
61 stone H3S10 kinase are strong suppressors of position effect variegation (PEV) of the wm4 allele and
62 d that loss of 8 out of 13 JmjC genes modify position effect variegation (PEV) phenotypes, consistent
63 able gene repression, such as is observed in position effect variegation (PEV) when the Drosophila me
64  Drosophila melanogaster chromosomes exhibit position effect variegation (PEV), a mosaic silencing ch
65 terochromatin-localized protein required for position effect variegation (PEV), colocalized with DmOR
66         Reduction of roX function suppresses position effect variegation (PEV), revealing functional
67  a PcG gene and mutations in His2Av suppress position effect variegation (PEV), suggesting that this
68 uchromatin and heterochromatin can result in position effect variegation (PEV), the variable expressi
69                This contrasts with classical position effect variegation (PEV), where a given gene is
70        It is a strong dominant suppressor of position effect variegation (PEV).
71 as exemplified in Drosophila melanogaster by position effect variegation (PEV).
72 transgene are silenced-a phenomenon known as position effect variegation (PEV).
73 sed as the explanation for such phenomena as position-effect variegation (PEV) and control of segment
74                              Heterochromatic position-effect variegation (PEV) describes the mosaic p
75                                              Position-effect variegation (PEV) describes the stochast
76           This would be similar to a loss of position-effect variegation (PEV) in Drosophila.
77                  The classical phenomenon of position-effect variegation (PEV) is the mosaic expressi
78 gaster, heterochromatin-induced silencing or position-effect variegation (PEV) of a reporter gene has
79                 BID expression also enhances position-effect variegation (PEV) of the w(m4h) allele a
80                                              Position-effect variegation (PEV) results from the juxta
81 in into a euchromatic gene, which results in position-effect variegation (PEV), also causes the aberr
82 osome in regulating rRNA gene transcription, position-effect variegation (PEV), and the link among rD
83 iable, fertile, and recessive suppressors of position-effect variegation (PEV), indicating that, as i
84 eterochromatin and regulates heterochromatin position-effect variegation (PEV), organization of repet
85  in an understanding of aneuploid syndromes, position-effect variegation (PEV), quantitative traits,
86 , we found that dhtt acts as a suppressor of position-effect variegation (PEV), suggesting that it in
87  antagonistic and counterbalancing effect on position-effect variegation (PEV).
88 n/euchromatin at three distinct loci showing position-effect variegation (PEV).
89 t required for survival and does not control position-effect variegation (PEV).
90 ases is highly site dependent, and resembles position-effect variegation (PEV).
91  effect of both Su(var)3-9 and Su(var)2-5 on position-effect variegation, providing evidence that a f
92                                         With position-effect variegation, similar responses were foun
93                                   Studies of position effect variegation suggest that promoters of he
94             Rga also acts as a suppressor of position effect variegation, suggesting that a possible
95     Mutations in Nap-1 are shown to suppress position effect variegation, suggesting that Nap-1 funct
96 IR2 mutations were recently shown to perturb position effect variegation, suggesting that the role of
97                          Telomere-associated position-effect variegation (TPEV) in budding yeast has
98 ear telomeres is attenuated through telomere position-effect variegation (TPEV).
99 bed tyrosyl tRNA gene, SUP4-o, is subject to position effect variegation when located near a telomere
100 y RNA polymerase II (RNAP II) are subject to position effect variegation when located near yeast telo
101 is, and it decreased (but did not alleviate) position effect variegation within the expressing cell t
102 emplified by piRNAs (piwi-interacting RNAs), position effect variegation, X-chromosome inactivation,

 
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