ライフサイエンスコーパス: post infection

1 (DEGs) was highest during late mycosis (72 h post-infection).

2 de follicles throughout the follow-up (240 d post-infection).

3 ctional shortening were observed eight weeks post infection.

4 id not affect viral contents at 4 and 8 days post infection.

5 pha, and cyclooxygenase-2 expression at 24 h post infection.

6 animals is tracked as a function of the time post infection.

7 gained a numerical advantage as early as 8 h post infection.

8 ressed both PRRSV and PCV2 infection at 12 h post infection.

9 d only from donor hosts between 3 and 9 days post infection.

10 ] vs 413.5 [70.5], p=0.0476) in BAL at day 7 post infection.

11 arly between the groups of 4, 7, and 60 days post infection.

12 H1N1 and were followed up for up to 21 days post infection.

13 ncentration significantly decreased at day 3 post infection.

14 er survival and less weight loss at 24 hours post infection.

15 related with decreased lung function 21 days post infection.

16 pression and cell surface markers 8-16 hours post infection.

17 ely, CFU was significantly lower by 48 hours post infection.

18 etioles of wild-type and coi1 plants at 10 d post infection.

19 from animals euthanized on or before 15 days post infection.

20 acked vasculitis and fibrosis 15 to 120 days post infection.

21 ia-infected mice lived until at least day 30 post infection.

22 ant compared with control strains at 21 days post infection.

23 extravasating pial vessels as early as 1 day post infection.

24 ymal vessels were not observed until 5 weeks post infection.

25 vival) when administered between day 0 and 3 post infection.

26 as ten parasites and as early as five weeks post infection.

27 ed from VV-infected cells at 1 hr and 3.5 hr post infection.

28 3 was characterized at different time points post infection.

29 en when treatment is started as late as 48 h post infection.

30 mission, viral subtype, or time of isolation post infection.

31 serum IL-6 are 7.6 times more likely to die post infection.

32 and SUDV infection when delivered four days post infection.

33 sessed at a systemic and tissue level day 45 post infection.

34 for ROCK-mediated cell contraction from 2 hr post infection.

35 neutrophil trafficking to the brain at day 8 post infection.

36 n C. jeuni numbers in the ceca by nine weeks post infection.

37 ge of all IBVs tested when administered 72 h post infection.

38 TmCactin expression were detected at 9 hours post infection.

39 a slight reduction in activity over a decade post infection.

40 pecimens from inoculated bats from 5-19 days post infection.

41 k-out mutant for analyses at different times post-infection.

42 NAs) of ileal tissues collected at one-month post-infection.

43 Pg impaired pHAEC viability 24 hours post-infection.

44 tly reduced by 6.5-11.4% beginning in week 3 post-infection.

45 Fusobacteria was only present 12 h post-infection.

46 after therapeutic administration up to 24 h post-infection.

47 y and late genes when added at various times post-infection.

48 tralization titers at day one, three and six post-infection.

49 (TLR) expression were measured at 6-20 weeks post-infection.

50 expression was completely absent by 6 months post-infection.

51 ly reached the titre of wild-type virus late post-infection.

52 7 was silenced by approximately 85% on day 3 post-infection.

53 ining the bacterial loads at different times post-infection.

54 rnea when applied topically starting 8 hours post-infection.

55 Both models are monitored for up to 5 d post-infection.

56 ashes, saliva, urine, and feces up to 8 days post-infection.

57 sacs infected with R. prowazekii at ten days post-infection.

58 iminated from peripheral blood within 3 days post-infection.

59 l activation and decreased humoral responses post-infection.

60 orders of magnitude above base-line 24-48 h post-infection.

61 observed only when BIP was added within 3 h post-infection.

62 y, we observed an increase in apoptosis 48 h post-infection.

63 P. berghei-infected mice on days 3, 4, and 5 post-infection.

64 became predominantly hypophosphorylated 48 h post-infection.

65 igens and the antibodies lasted out to 52 wk post-infection.

66 clining to 50% of control levels by 16 weeks post-infection.

67 ame rounded within 1-2 h and detached by 5 h post-infection.

68 but the expression was downregulated by 12 h post-infection.

69 y few brain cysts identified in mice 5 weeks post-infection.

70 issue, skeletal muscle, and liver by 9 weeks post-infection.

71 h survival recorded from three to seven days post-infection.

72 rozoites and infected cell monolayers 2-48 h post-infection.

73 heir colonies and sampled 5, 10, and 21 days post-infection.

74 samples 4 days prior to infection and day 6 post-infection.

75 and were detected from 20 days to four years post-infection.

76 uginosa bacterial counts 1000 times on day 4 post-infection.

77 ange was seen when IBRV-A4 was added 2 hours post-infection.

78 ered by virus and dsRNA at three time-points post-infection.

79 acquire influenza antigen in the lungs early post-infection.

80 of measuring virus neutralization at 6 hours post-infection, a duration likely confined to a single v

81 ar in both study groups, peaking by 10 weeks post-infection, a higher plateau (set-point) being achie

82 ion when treatments were initiated nine days post-infection, a time when animals were demonstrating a

83 genes, such as Irf7 and Cxcl9; 7 and 60 days post infection, acquired immunity-related genes, such as

84 Finally, topical and post-infection administration of rolipram into the middl

85 recovery of infectious virus 28 days or more post infection and has been a useful construct for exper

86 related host response and elevated SA levels post infection and in the down-regulation of jasmonic ac

87 vel of PML protein was expressed within 24 h post-infection and a detectable level remained at day 16

88 become committed to a B-blast fate <12 days post-infection and are unable to de-repress p18INK4c or

89 rhans cells were first detected at about 2 y post-infection and comprised about one-third of the Lang

90 eatment was started by the oral route at 6 h post-infection and continued for 7 days, bilosome levofl

91 pacts proIL-1beta expression as early as 2 h post-infection and delays activation of AIM2 and NLRP3-i

92 infection activates PKR at very early times post-infection and despite the presence of E3 and K3.

93 cally treated with MAb F429 starting 8 hours post-infection and evaluated for bacterial levels and co

94 s of gene expression were detected at 3 days post-infection and increased over time, suggesting this

95 atrilysin mRNA levels were detectable at 3 h post-infection and peaked at a 25-fold induction between

96 sults identify a mechanism of neuronal death post-infection and point to a role for tissue-resident M

97 Protein synthesis ceases at late times post-infection and the translation initiation factor eIF

98 conditions (mouse and bacterial strain, time post infection) and was validated in outbred mice (AUC>0

99 activation during progression (days 6 and 12 post-infection) and regression (days 20-34 post-infectio

100 s were seen at the earliest time points (4 h post-infection) and the number of differentially express

101 esus monkeys euthanized between 3 and 8 days post-infection, and 3 uninfected controls were enrolled

102 days prior to infection and 3, 6 and 14 days post-infection, and blood samples 4 days prior to infect

103 in approximately half the animals five weeks post-infection, and morphine/saline administration conti

104 rus and persists at least through day eleven post infection; and (4) the transient nature of the infe

105 fection/disease via immunization and less on post-infection antibiotics.

106 The post-infection antiviral activity of h5B3.1 was evaluate

107 Percent neutrophils in lung lavage fluid post-infection are significantly higher in CF mice, but

108 tor T cell phenotypes at various time points post infection as promising indicators of infection outc

109 increase in fungal burden beginning 2 weeks post-infection (as compared with mice infected with urea

110 % probability of presentation within 3 hours post-infection, as observed experimentally.

111 PRV-IR neurons were first observed on day 3 post-infection at L6 in the SPN.

112 Compared to day 1 post infection, bacterial colony counts, BALF total cell

113 Our results demonstrated that at 48 hours post-infection Brucella adjusts its metabolism in order

114 splayed minimal bacterial clearance for 1 mo post-infection but eventually resolved genital tract inf

115 irus decayed significantly from 1 to 2 weeks post infection, but decreased minimally thereafter.

116 t 5 weeks post infection followed at 9 weeks post infection by infusions of a primatized monoclonal a

117 y expressed genes at one or more time points post infection compared to the day 0 baseline.

118 ificant attenuation in BALB/c mice at 1 week post infection compared with the virulent parental strai

119 were not statistically different at 18 days post-infection compared to uninfected animals indicating

120 strate increased PKR expression through 96 h post-infection, concomitant with an increase in eIF-2alp

121 Gross findings post-infection confirmed extensive hemorrhage in the lun

122 lectron microscopy structures of the pre and post-infection conformations of the virus.

123 nd DbpB were only seen at the 1 h time point post infection, consistent with its low infectivity phen

124 rpoD transcripts were detected at all times post-infection, consistent with their expected function

125 03 expression in the crypts at days 6 and 12 post infection correlated with reduced levels of its tar

126 riend retrovirus (FV) replication at 2 weeks post-infection correlated with stronger natural killer,

127 Between 16-20 h post-infection, CteG-2HA mostly associated with the Golg

128 lation, ZIKV RNA is detected in plasma 1 day post infection (d.p.i.) in all animals (N=8, including 2

129 e and analyzed at multiple time points up to post-infection day 20.

130 ype (WT) and Tetherin KO mice at 3 to 7 days post-infection despite removal of a potent restriction f

131 IgG2b levels relative to controls by week 9 post-infection, despite similar viral loads.

132 dissected syncytial cells at 3, 6 and 9 days post infection (dpi) during the course of the resistant

133 uctive tissues were harvested 14 and 35 days post infection (DPI) for inoculation studies and 14, 35

134 m samples were collected at 7, 14 and 21 day post infection (DPI) from infected and control mice and

135 F2 family was evaluated at two- and six-days post infection (dpi) in the spleen from both lines, eith

136 n H129, and at daily intervals up to 16 days post infection (dpi) rose bengal or lissamine green B wa

137 levels of host immune-related genes at 1 day post infection (dpi) were higher in H5N8-infected than H

138 At 3, 4, or 5 days post infection (dpi), both optic tracts (OT) were dissec

139 clear accumulation of TP53 at 6- and 14-days post infection (dpi), indicating a prolonged arrest in t

140 enza A virus at 0, 6, 10, 14, 21 and 28 days post infection (dpi), representing the major stages of I

141 r to YHV infection had 50% survival at 8 day-post infection (dpi), whereas 84.1% mortality was observ

142 isolated from draining lymph nodes at 2 days post infection (dpi).

143 om spleen tissues taken at 10-day and 21-day post infection (dpi).

144 tion, the tissue FA depots depleted at 3days post-infection (DPI) and were then restored to their pre

145 d from infected rats were elevated by 4 days post-infection (dpi), but were not elevated in serum and

146 isera per animal, at 30 minutes, 1 or 2 days post-infection (dpi), in which all animals survived.

147 ng FcgammaRs were expressed maximally at 5 d post-infection (dpi), while the inhibitory receptor (Fcg

148 t-virus interactions which result in various post-infection effects in humans and animals.

149 entical adsorption kinetics in the first 2 h post infection, EhV-207 gained a numerical advantage as

150 l of 62 miRNAs were differentially expressed post infection (false discovery rate <0.1).

151 At 24 h post infection, fluticasone treatment suppressed rhinovi

152 p study and who had a median of 24 months of post-infection follow-up.

153 a 90-day course of ART initiated at 5 weeks post infection followed at 9 weeks post infection by inf

154 ted the highest titers at three and six days post-infection for the type A viruses and lower titers f

155 es for prophylaxis, and 24, 48, and 72 hours post-infection for treatment.

156 ta), Wnt and Notch pathways on 12 or 34 days post infection formed monolayers in vitro, and underwent

157 hallenge (day 0) and at 4, 7, 11 and 14 days post infection from 44 pigs revealed 6,430 differentiall

158 re sampled and cultured at 6, 13 and 20 days post-infection from the 4 groups.

159 inning at 2 weeks and continuing for 6 weeks post infection, however, GR106 and 2308 displayed equiva

160 By 3-4 wk post infection, however, iNOS knockout mice did succumb

161 ed at all time points except on days 4 and 7 post-infection; however, duIL-17A and IL-17F expression

162 llowed in intact leaf tissue from 12 to 36 h post infection (hpi) by using confocal microscopy.

163 ed over four timepoints (4, 24, 48, and 72 h post infection (hpi)).

164 ed with 25 mg/kg of either L or V at 2 hours post infection (hpi).

165 ins (ICPs) synthesized between 3 and 6 hours post infection (hpi).

166 nduced > or =2-fold in HFF between 2 and 6 h post-infection (hpi) in repeated experiments while immed

167 Litopenaeus vannamei at 1.5, 18 and 56 hours-post-infection (hpi), between WSSV-challenged and contro

168 gnal response detected as early as ~30 hours post-infection (hpi).

169 mpared to those of non-infected bAM 24 hours post-infection (hpi).

170 Four weeks post infection, Hu-NSG mice of both sexes developed left

171 ft towards stromal staining at peak (12 days post infection) hyperplasia, whereas staining for Lgr5 a

172 After 8 days post infection, Il10(-/-) mice infected with Cj-P1 demon

173 loids between the orange layers after 4 days post infection in concentrations > LOQ.

174 ly infected with HSV-1 and surveyed at times post infection in the nervous system and lymph node for

175 al plasma samples collected over three years post-infection in a cohort of MSM HIV-1 seroconvertors,

176 ore, expression of Stmn2 was reduced 30-fold post-infection in a mouse cellular model of prion diseas

177 y [pre-infection, post-infection in latency, post-infection in latency or recovered, or pre-infection

178 status required for efficacy [pre-infection, post-infection in latency, post-infection in latency or

179 d therapeutic efficacy when dosed up to 18 h post-infection in the pneumonia model.

180 in learning and memory and that by 20 months post infection, in the surviving mice, we found no evide

181 ral morning core clock genes as early as 1 h post-infection, including all members of the NIGHT LIGHT

182 breadth of an infant at approximately 1 year post-infection, including one with cross-clade breadth.

183 ted in Arabidopsis root tips as early as 6 h post infection, indicating that ET signaling was activat

184 the separation were as follows: 4 and 7 days post infection, innate immunity-related genes, such as I

185 Analysis of shorter post-infection intervals showed that levels of viral RNA

186 Three weeks post infection, intestinal tissue samples were collected

187 mes are completely converted, within 2 weeks post-infection, into non-rearranged circular concatamers

188 Five days post-infection, labeling was seen in the IML and lamina

189 ceived cART beginning between 4- and 27 days post-infection, leading to the establishment of differen

190 35 days post infection, mice were treated with low dose hydrocor

191 Three days post-infection, mice were given saline or ethanol (5 g/k

192 animals sacrificed between 16 and 44 months post infection (mpi) were examined for the presence of t

193 ognized for its association with the serious post-infection neurological complications of the Miller-

194 In the lumbosacral cord, 3 days post-infection, neurons labeled by virus from the extern

195 We show that at later times post-infection NF-kappaB activation is blocked in reovir

196 s constructed from Arabidopsis at 6 and 14 h post infection of non-pathogenic, virulent and avirulent

197 er liter of culture medium collected at 72 h post-infection of BTI-Tn-5B1-4 cells with the basic prot

198 cellular bacteria during the first few hours post-infection of macrophages but the expression was dow

199 Ninety-six h post-infection of Sf9 insect cells with recombinant bacu

200 By 7 days post-infection of sponge implants in Flk-1(LacZ) knock-i

201 m Legionella pneumophila were induced at 4 h post-infection of the U937 macrophage-like cells.

202 Multiple observations indicate poorer post-infection outcomes for patients with cancer than fo

203 h the largest difference of 100-fold at 72 h post infection (p.i.) and peak titers decreased by appro

204 Ranavirus was first detected at 3 days post infection (p.i.) in the oral cavity and upper respi

205 our group II animals died by 8 and 10 months post infection (p.i.), all four group III animals remain

206 The mice were euthanized 10 days post infection (p.i.), and the urethra, bladder, epididi

207 ice exhibited reduced weight loss at 15 days post-infection (p < 0.01) and demonstrated reduced histo

208 ghly discriminatory for sera taken from pigs post-infection (P < 0.05) indicating that these peptides

209 imary HIV infection (PHI) and up to one year post-infection (p.i).

210 cellular replication of bacilli during day 7 post-infection (p.i.) within RAW264.7 and THP-1 macropha

211 r C. trachomatis and harvested at t = 0-48 h post-infection (p.i; active).

212 Variation in post-infection parasite growth and killing time depended

213 2 post-infection) and regression (days 20-34 post-infection) phases of TMCH.

214 a persistent cytopathic infection at 21-day post infection (PI) based upon the quantitative detectio

215 oducible distinct succession of peaks at 12h post infection (PI), 23h PI and 31h PI and local minima

216 By day 28 post infection (PI), 5-HT turnover had normalized, but 5

217 ected in a patchy fashion beginning on day 7 post infection (pi), and the infection progressed to inv

218 e were elevated significantly at 1 to 3 days post infection (PI), peaked at 3 and decreased at 5 days

219 diographically and grossly apparent by day 7 post infection (PI).

220 ory pathways was examined following 2-5 days post-infection (PI) and the virus was located immunocyto

221 experiments, vancomycin was injected 4 hours post-infection (PI) for each passively immunized group.

222 infection were observed as early as 12 hours post-infection (PI) in osteoblasts, skeletal muscle myoc

223 on of neoplastic lesions in vitro at 27 days post-infection (PI), providing new evidence of the role

224 at HC-23 mRNA levels decreased from 6 to 12h post-infection (pi), were maximally expressed at 24h pi,

225 tarted on day 15 and was completed by day 35 post-infection (pi), whereas the KO mice remained partia

226 male and female mice at days 90, 150 and 180 post-infection (PI).

227 ation, there is a transient period (day 1-20 post-infection) prior to the establishment of persistenc

228 activation of mTORC1 occurs as early as 8 h post-infection, prior to AC formation.

229 eloped severe colitis and hepatitis by 10 wk post-infection, progressing into colon carcinoma by 20 w

230 , initiated at the time of infection or 24 h post-infection, promoted long term survival in candidemi

231 single dose of WNV-86 administered two days post-infection protected mice from lethal WNV challenge.

232 that the qRT-PCR signal detected at 6 hours post-infection reflected an amplification of at least 10

233 296, 400, to 1086 at 24, 48, 72 and 96 hours post infection, respectively).

234 241 and 141 RISC-bound genes at 7 h and 10 h post-infection, respectively); it affected several cell

235 ype B viruses in MDCK cells before day three post-infection, resulting in the systematic underestimat

236 hological analyses of hearts seventeen weeks post infection revealed coronary microvascular leakage,

237 sessment of the LCMV-specific memory at 65 d post infection revealed many more LCMV-specific WT memor

238 O and WT CD4 and CD8 T cell responses at 8 d post infection revealed modest but significant differenc

239 d nucleotide reductase NrdEF was observed in post-infection samples compared to the pre-infection sta

240 217 cohort with paired longitudinal pre- and post-infection samples.

241 necrotizing fasciitis and myositis, and the post-infection sequelae glomerulonephritis and rheumatic

242 sults observed for neutralization at one day post-infection showed MDCK cells were similar (<1 log(2)

243 etion was noted for periods of up to 6 weeks post-infection, significantly longer than that which occ

244 By six weeks post infection, SIV-infected cells were no longer detect

245 At 0, 24, 72, and 144 hours post-infection, spleen and lung samples were removed for

246 epresents the pre-infection state, while the post-infection state is thick.

247 and CMV replication, when GA is administered post-infection, suggest a possible secondary mechanism t

248 asma bnAbs frequently and as early as 1-year post-infection suggesting factors governing bnAb inducti

249 III secretion (T3S) in the RB stage at 18 h post infection, suggesting a reduced T3S capacity or a l

250 res unable to transmit malaria within 5 days post-infection, surpassing the World Health Organization

251 ls to neonatal CNS infection with PVC-211 at post-infection survival times 7, 14, 21, and 28 days.

252 s is essential for antimicrobial defence and post-infection survival(1).

253 ated from rotavirus-infected cells six hours post-infection (termed in vivo 6 hr large RNAs), also ac

254 are induced but rapidly wane and by 3 months post-infection the bats are seronegative.

255 he A protein is absent from the virus shell (post-infection), the process of crystallization exerts s

256 After either 4- or 5-day survival period post-infection, the rats were euthanized by transcardial

257 ced its presence in the sera, over ten years post infection; the prevalence of other GP-specific IgG

258 -infected cells were detectable at two weeks post infection, their abundance increased with time, and

259 were infected with Mp and, twenty-four hours post infection, their host defense responses were compar

260 By day 5 post infection, there was a significant loss in T, NK, a

261 the lymph node of both species at two weeks post infection, they were confined to the lymph node par

262 force to convert the protein shell from the post-infection (thick) state to the pre-infection (thin)

263 host cell plasma membrane, and at 30 or 40 h post-infection this was its predominant localization.

264 ared whole-blood RNA-seq profiles at pre-and post-infection time points from Malian adults who were e

265 ase, but generally remaining elevated at all post-infection time points.

266 Post-infection titres started higher against H3N2Pe09 bu

267 MCMV activates the IRE1-XBP1 pathway early post infection to relieve repression by XBP1u, the produ

268 ency virus (SIV)mac239 genomes at four weeks post-infection to determine the extent of acute CTL esca

269 At 6 days post infection, transcription of viral genes was detecte

270 herapy, such as pre-exposure prophylaxis and post infection treatments.

271 t viral load was assessed at 4- and 10-weeks post-infection using quantitative PCR.

272 e vs. early chronic infection (7 vs. 28 days post-infection), using neurological and behavioral pheno

273 Instead, an efficacious post-infection vaccine delivered to older adults will be

274 Furthermore, with older adult vaccination, post-infection vaccines provided substantially greater m

275 tinct immunological correlates compared with post-infection virological control and required the incl

276 V-1) vaccine candidates have typically shown post-infection virological control, but protection again

277 ly distributed within the cytoplasm even 6 h post-infection, VLPs comprising both L1 and L2 exhibited

278 FTL_0325 mutant-infected macrophages at 24 h post-infection was independent of both AIM2 and NLRP3.

279 encephalomyelitis virus on 4, 7, and 60 days post infection, we conducted bioinformatics analyses, in

280 e expression changes over the first 12 hours post-infection, we developed a statistically rigorous en

281 n latency or recovered, or pre-infection and post-infection]) were assessed.

282 peak infection density in gut at 10-12 days post-infection when blood viral loads were low.

283 icles per Coxiella call was observed at 12 h post-infection when compared with the starting inoculum.

284 ASC response was obviously detected on day 7 post-infection when the virus was completely cleared fro

285 were not readily detectable by ET at 5-days post-infection, whereas HIV-1-infected cells surrounded

286 oHV-1 infected MDBK cells at 0 and 0.5 hours post-infection, whereas no change was seen when IBRV-A4

287 M1 classical macrophage activation two days post infection, which has the collateral effect of suppr

288 at the wild type persists in mouse lung 24 h post infection while the mutant strain is cleared by hos

289 monary CD11c(+) cells revealed that at day 7 post infection, wild-type cells up-regulated type-I IFN-

290 replication is readily detectable by one day post infection with HBV baculovirus and persists at leas

291 specimens from rhesus macaques prior to and post infection with pathogenic SIVmac239.

292 mutated ancestor emerged between weeks 30-38 post-infection with a 35-residue CDR H3, and neutralized

293 ssue were assessed over a period of 16 weeks post-infection with ASRV.

294 and WNT4 in colonic crypts at days 6 and 12 post-infection with Citrobacter rodentium (CR) and tende

295 hological changes in LX-2 cells within 24 hr post-infection with HCV, HIV or HCV/HIV in MLH co-cultur

296 he culture medium of BTI-Tn-5B1-4 cells 72 h post-infection with the basic protein promoter-uPA-IgG v

297 ntigen levels increase up to 2-fold at day 3 post-infection with toxigenic Sterne 34F(2) strain, wher

298 d the in vivo antiviral host response 7 days post infection, with no induction of interferon-stimulat

299 n, progressing into colon carcinoma by 20 wk post-infection, with pronounced pathology in the cecum a

300 1 in early infection and 0 around two years post infection, yielding MDRI of 420 [361, 467] days.