ライフサイエンスコーパス: post-transcriptional gene silencing

1 t hypotheses to account for the mechanism of post transcriptional gene silencing.

2 A interference (RNAi) is a powerful tool for post-transcriptional gene silencing.

3 ) proteins that mediate RNA interference and post-transcriptional gene silencing.

4 oRNAs are small non-coding RNAs that mediate post-transcriptional gene silencing.

5 son silencing, heterochromatin formation and post-transcriptional gene silencing.

6 ory processes, including transcriptional and post-transcriptional gene silencing.

7 e cleavage of complementary mRNA, leading to post-transcriptional gene silencing.

8 nificant contribution to transcriptional and post-transcriptional gene silencing.

9 NA (siRNA) molecules are potent effectors of post-transcriptional gene silencing.

10 scriptional activator and/or a suppressor of post-transcriptional gene silencing.

11 of the transcribed region is associated with post-transcriptional gene silencing.

12 rocessing, transcriptional co-activation and post-transcriptional gene silencing.

13 zoa, animals, plants and fungi, resulting in post-transcriptional gene silencing.

14 synthase, a process called cosuppression or post-transcriptional gene silencing.

15 that the DNA itself is a potent activator of post-transcriptional gene silencing.

16 nce to further infection, factors typical of post-transcriptional gene silencing.

17 ayed several phenotypes that we attribute to post-transcriptional gene silencing.

18 ) of the N gene displayed resistance through post-transcriptional gene silencing.

19 utant plants affected in DNA methylation and post-transcriptional gene silencing.

20 ulted in small(s) RNA production, leading to post-transcriptional gene silencing.

21 NA interference (RNAi) reagents for directed post- transcriptional gene silencing.

22 ns in eukaryotes are known as key players in post-transcriptional gene silencing(1), while recent stu

23 NAs (siRNA) are potent reagents for directed post-transcriptional gene silencing and a major new gene

24 MicroRNAs (miRNAs) act in post-transcriptional gene silencing and are proposed to

25 genes implicate cellular energy homeostasis, post-transcriptional gene silencing and fatty-acid amide

26 ) associate with ARGONAUTE1 (AGO1) to direct post-transcriptional gene silencing and regulate numerou

27 Recently, oligonucleotide post-transcriptional gene silencing, antisense oligonucl

28 MicroRNAs are responsible for post-transcriptional gene silencing as part of critical

29 xisRNA induction possibly mediates post-transcriptional gene silencing but they do not broa

30 of endogenous, non-coding RNAs that mediate post-transcriptional gene silencing by inhibiting mRNA t

31 l and evolutionarily conserved phenomenon of post-transcriptional gene silencing by means of sequence

32 ssues (with the probable involvement of some post-transcriptional gene silencing by pathways related

33 Post-transcriptional gene silencing by small interfering

34 of transformants spontaneously triggers Nia post-transcriptional gene silencing (class II) whereas a

35 load miRNAs on target mRNAs without causing post-transcriptional gene silencing, due to its inabilit

36 desaturase (PDS) gene capable of catalyzing post-transcriptional gene silencing if RNA escapes into

37 ile microRNAs (miRNAs) generally function in post-transcriptional gene silencing in both somatic and

38 A mutation that disrupts post-transcriptional gene silencing in Neurospora crassa

39 , in addition to well-characterized roles in post-transcriptional gene silencing in the cytoplasm.

40 Post-transcriptional gene silencing in transgenic plants

41 Unlike post-transcriptional gene silencing in worms, elt-2::gfp

42 RNA interference (RNAi) is a process of post-transcriptional gene silencing initiated by double-

43 Post-transcriptional gene silencing is a promising thera

44 d with the potential of transgenes to confer post-transcriptional gene silencing, is a cause or a con

45 ), the double-stranded RNA (dsRNA) triggered post-transcriptional gene silencing, is becoming a power

46 of "sensing" activates meiotic silencing, a post-transcriptional gene silencing-like mechanism that

47 These responses, called RNA interference or post-transcriptional gene silencing, may provide anti-vi

48 and consequent gene silencing, and (ii) the post-transcriptional gene silencing mechanism can trans-

49 n evolutionarily conserved sequence-specific post-transcriptional gene silencing mechanism that is we

50 tes protection against viral infection via a post-transcriptional gene silencing mechanism.

51 ween DNA viral activity, transcriptional and post-transcriptional gene silencing mechanisms.

52 In order to achieve efficient therapeutic post-transcriptional gene-silencing mediated by the RNA

53 Among these regulatory events are post-transcriptional gene silencing, mediated by small n

54 nown variously as RNA interference (RNAi) or post-transcriptional gene silencing, mediates resistance

55 Post-transcriptional gene silencing, most recently via R

56 viously reported initial clinical results of post-transcriptional gene silencing of BCL11A expression

57 ion of miRNA, molecular silencers that enact post-transcriptional gene silencing of coding genes.

58 MA) tumor model was used to demonstrate that post-transcriptional gene silencing of dicer, the enzyme

59 stability and translation, and siRNAs cause post-transcriptional gene silencing of transposons, viru

60 Our results define the core components of a post-transcriptional gene silencing pathway in Arabidops

61 h a 5' overhang, and is required for several post-transcriptional gene silencing pathways.

62 hly evolutionary conserved family engaged in post-transcriptional gene silencing pathways.

63 DNA methylation and post-transcriptional gene silencing play critical roles

64 A interference (RNAi) is a sequence-specific post-transcriptional gene silencing process.

65 with a transgene and the ability to promote post-transcriptional gene silencing (PTGS) and transgene

66 tion can be caused by many factors including post-transcriptional gene silencing (PTGS) and/or rapid

67 In plants, both transcriptional (TGS) and post-transcriptional gene silencing (PTGS) can be self-r

68 on') reproducibly and consistently activates post-transcriptional gene silencing (PTGS) in every plan

69 achineries that regulate transcriptional and post-transcriptional gene silencing (PTGS) in plants, ai

70 ediated defence mechanism that is related to post-transcriptional gene silencing (PTGS) in transgenic

71 Post-transcriptional gene silencing (PTGS) is a fundamen

72 Here, we show that post-transcriptional gene silencing (PTGS) is a major ca

73 Post-transcriptional gene silencing (PTGS) is a sequence

74 Post-transcriptional gene silencing (PTGS) is a sequence

75 Post-transcriptional gene silencing (PTGS) is an importa

76 In plants, post-transcriptional gene silencing (PTGS) is mediated b

77 the basis of genetic studies appears to be a post-transcriptional gene silencing (PTGS) mechanism.

78 Post-transcriptional gene silencing (PTGS) of a green fl

79 s of petunia (Petunia hybrida) are caused by post-transcriptional gene silencing (PTGS) of the key en

80 Post-transcriptional gene silencing (PTGS) provides prot

81 ic tobacco plants expressing a suppressor of post-transcriptional gene silencing (PTGS) with a PVX am

82 st suppressors identified that could inhibit post-transcriptional gene silencing (PTGS), but with lit

83 ith inverted repeats can efficiently trigger post-transcriptional gene silencing (PTGS), presumably v

84 To induce post-transcriptional gene silencing (PTGS), the TRV2 gen

85 d small-interfering RNA (siRNA) pathways for post-transcriptional gene silencing (PTGS), whereas AGO4

86 RNAi can be categorized into post-transcriptional gene silencing (PTGS), which involv

87 indicating that it also exerts an effect on post-transcriptional gene silencing (PTGS).

88 is large family of low expressed genes using post-transcriptional gene silencing (PTGS).

89 n L3 (RPL3) genes in Nicotiana tabacum using post-transcriptional gene silencing (PTGS).

90 cco and Arabidopsis thaliana, was subject to post-transcriptional gene silencing (PTGS).

91 ora crassa QDE-1, two proteins implicated in post-transcriptional gene silencing (PTGS).

92 for flavonoid biosynthesis, is inhibited by post-transcriptional gene silencing (PTGS).

93 ripts and inhibiting their translation (i.e. post-transcriptional gene silencing, PTGS).

94 a sequence-specific RNA degradation process (post-transcriptional gene silencing, PTGS).

95 results strongly suggest that miRNA-mediated post-transcriptional gene silencing relies primarily on

96 In plants, post-transcriptional gene silencing results in RNA degra

97 relate the processes of transcriptional and post-transcriptional gene silencing (TGS, PTGS) in persp

98 RNA interference (RNAi) is a form of post-transcriptional gene silencing that has been descri

99 nce (RNAi) is an intracellular mechanism for post-transcriptional gene silencing that is frequently u

100 MicroRNAs regulate post-transcriptional gene silencing through base-pair bi

101 ) proteins are well known for their roles in post-transcriptional gene silencing through microRNA-med

102 volutionary conserved microRNA that mediates post-transcriptional gene silencing to regulate a wide r

103 Here we investigate the potential of post-transcriptional gene silencing using RNA interferen

104 Argonaute (Ago) proteins, where they direct post-transcriptional gene silencing via base-pairing wit

105 c modifications referred to as virus-induced post-transcriptional gene silencing (ViPTGS) and virus-i

106 Small RNAs associated with post-transcriptional gene silencing were first discovere