ライフサイエンスコーパス: post-traumatic

1 f proinflammatory mediators and inhibits the post-traumatic activation of JNK in a rodent model of TB

2 d further investigations were in favour of a post-traumatic aetiology.

3 Retrospective assessment of post-traumatic amnesia (PTA) must take into account fact

4 predominantly mild-to-moderate TBI (52% with post-traumatic amnesia (PTA)</=24 hours), but including

5 e novel insights into the pathophysiology of post-traumatic amnesia and evidence that memory impairme

6 traumatic brain injury were classified into post-traumatic amnesia and traumatic brain injury contro

7 the hypothesis that the mnemonic symptoms of post-traumatic amnesia are caused by functional disconne

8 pairments in associative memory, patients in post-traumatic amnesia demonstrated impairments in infor

9 Clinically, post-traumatic amnesia is an important predictor of func

10 unctional importance, the pathophysiology of post-traumatic amnesia is not understood.

11 FOR A SCIENTIFIC COMMENTARY ON THIS ARTICLE: Post-traumatic amnesia is very common immediately after

12 Patients in post-traumatic amnesia showed abnormal functional connec

13 Patients with post-traumatic amnesia showed evidence of widespread tra

14 normality was also observed in patients with post-traumatic amnesia.

15 ere statistically significant differences in post-traumatic and spontaneous CCF regarding age (p = 0.

16 Delayed presentation of post-traumatic aortic pseudoaneurysm and its fistulous c

17 n which we performed RPICIOL implantation in post-traumatic aphakia (141 eyes, group 1), post-catarac

18 evidence of IL-1 signaling as a mediator of post-traumatic astrogliosis and seizure susceptibility.S

19 for limiting the devastating consequences of post-traumatic autonomic hyperreflexia and post-injury i

20 njury, including diffusion MRI assessment of post-traumatic axonal injury.

21 lls by MHC-II on Schwann cells thus promotes post-traumatic axonal loss and neuropathic pain.

22 ts after repetitive surgery or who developed post-traumatic blepharoptosis.

23 as been proposed as a treatment strategy for post-traumatic brain injured patients in minimally consc

24 s work, we present a finite element model of post-traumatic brain injury and decompressive craniectom

25 icacy of catecholaminergic drugs in treating post-traumatic brain injury cognitive impairments.

26 n achieved despite repetitive surgery and in post-traumatic cases where levator muscle and aponeurosi

27 lly conscious state with language (n = 4) or post-traumatic confusional state (n = 4).

28 edullary damage such as haemorrhage, oedema, post-traumatic cystic cavities, and tissue bridges.

29 pression of the spinal cord and formation of post-traumatic cysts.

30 s mature inhibitory interneurons and restore post-traumatic decreases in synaptic inhibition.

31 However, clinical diagnosis of post-traumatic dementia is problematic.

32 The neuropathology of post-traumatic dementias is increasingly well characteri

33 t primed ASCs injected into our rat model of post-traumatic elbow contracture decreased histological

34 spp. are common causes of post-operative and post-traumatic endophthalmitis.

35 This study aimed to characterize post-traumatic epilepsy in a mouse model of pediatric br

36 t always model heritable absence epilepsy or post-traumatic epilepsy in humans, and may instead refle

37 res (CPSs) following traumatic brain injury (post-traumatic epilepsy).

38 s early seizures and is the leading cause of post-traumatic epilepsy.

39 the organotypic hippocampal culture model of post-traumatic epileptogenesis.

40 Post-traumatic headache (PTH) is a highly disabling seco

41 Post-traumatic hypopituitarism (PTHP) can have major con

42 halanx I-1 have a morphology consistent with post-traumatic infection in the form of osteomyelitis.

43 romised fracture healing because of systemic post-traumatic inflammation.

44 in mice and investigated how this influenced post-traumatic intraneural inflammation and neuropathic

45 t study, we hypothesized that PAI-1 mediates post-traumatic malfunction of coagulation, with inhibiti

46 powerful therapeutic strategy for correcting post-traumatic memory and seizure disorders.

47 been described to enlarge in the presence of post-traumatic meningeal hemorrhages or deformities of t

48 of alternative pharmacodynamic approaches in post-traumatic minimally conscious state patients should

49 we evaluated 13 normal volunteers and seven post-traumatic minimally conscious state patients using

50 ange of potential predictors of longitudinal post-traumatic neurodegeneration and compared the varian

51 ental models, diffuse axonal injury triggers post-traumatic neurodegeneration, with axonal damage lea

52 phenotypes associated with distinct types of post-traumatic neurodegeneration.

53 al injury predicts the degree of progressive post-traumatic neurodegeneration.

54 volumetric T1 scans as a measure of measure post-traumatic neurodegeneration.

55 fuse axonal injury are a strong predictor of post-traumatic neurodegeneration.

56 Inhibition of IFN-beta reduces post-traumatic neuroinflammation and neurodegeneration,

57 ns only of IgG control mice accompanied with post-traumatic neuroinflammation and was not seen in mid

58 arly and late infection, indicating enhanced post-traumatic neuroinflammation.

59 -deficient (IFN-beta(-/-)) mice and assessed post-traumatic neuroinflammatory responses, neuropatholo

60 the immune receptor TLR4 directly regulates post-traumatic neuronal excitability.

61 RORA multisite longitudinal study of adverse post-traumatic neuropsychiatric sequelae (APNS) among pa

62 ion (CL) were tested for its ability to slow post-traumatic OA (PTOA) in obese mice models.

63 affected knees in a rat model of established post-traumatic OA (PTOA).

64 rgically induced knee OA in mice, a model of post-traumatic OA in humans, increased expression of VEG

65 ole for IKKalpha in cartilage degradation in post-traumatic OA, and also suggest intrinsic, cell-auto

66 ilage mechanotransduction in age-related and post-traumatic OA, and point to a novel disease-modifyin

67 of these cells attenuated the development of post-traumatic OA, reduced pain and increased cartilage

68 bilization of medial meniscus (DMM) model of post-traumatic OA.

69 to cartilage degradation and progression of post traumatic osteoarthritis (PTOA).

70 ed the spectrum of lesions characteristic of post-traumatic osteoarthritis (PTOA) across the knee joi

71 ntrations have been reported in experimental post-traumatic osteoarthritis (PTOA) animal models and i

72 Post-traumatic osteoarthritis (PTOA) is associated with

73 revious in vivo studies in a murine model of post-traumatic osteoarthritis and suggest that the abili

74 mproved treatment option planning to prevent post-traumatic osteoarthritis.

75 ticles were tested in vivo in a rat model of post-traumatic osteoarthritis; intra-articular injection

76 anxious about the most likely cause of their post-traumatic problems.

77 paired fibrinolysis as a critical process in post-traumatic secondary brain damage and suggests that

78 tive deficits, and reduced the occurrence of post-traumatic seizure activity.

79 Early post-traumatic seizures are one potential mechanism for

80 sociations between immunogenetic factors and post-traumatic sepsis and septic shock.

81 region is associated with the development of post-traumatic sepsis and septic shock.

82 novel as they implicate adaptive immunity in post-traumatic sepsis.

83 ed by progressive neurodegeneration or other post-traumatic sequelae such as psychiatric impairment.

84 processing after TBI might contribute to TBI-post traumatic stress disorder (PTSD) comorbidity.

85 r Disorder (BD), Schizophrenia, anxiety, and Post Traumatic Stress Disorder (PTSD).

86 last exposure had an antemortem diagnosis of post traumatic stress disorder.

87 injury showed more severe neurobehavioural, post-traumatic stress and depression symptoms along with

88 t/hyperactivity disorder (ADHD), aggression, post-traumatic stress and substance abuse.

89 validation of an algorithm for prediction of post-traumatic stress course over 12 months using two in

90 Depressive symptoms (17%) and post-traumatic stress disorder (18%) persisted at 1 year

91 4, p=0.011), higher risk of comorbidity with post-traumatic stress disorder (eight studies, 2494 part

92 , 95% CI 1.14-1.23, p < 0.001), particularly post-traumatic stress disorder (HR 9.33, 95% CI 7.96-10.

93 Anxiety Disorder [GAD]-2 anxiety scale), and post-traumatic stress disorder (measured by the Post-Tra

94 thy control subjects (n = 20), patients with post-traumatic stress disorder (n = 25) demonstrated int

95 e randomised controlled trial; n=30), 12 for post-traumatic stress disorder (one randomised controlle

96 Resource loss is a core driver of post-traumatic stress disorder (PTSD) after large-scale

97 Outcomes included depression, anxiety, and post-traumatic stress disorder (PTSD) among family 3 and

98 s characterized by social withdrawal such as post-traumatic stress disorder (PTSD) and depression.

99 ation of two clinically relevant subtypes of post-traumatic stress disorder (PTSD) and major depressi

100 isk of some psychiatric disorders, including post-traumatic stress disorder (PTSD) and psychosis, com

101 mation contributes to the pathophysiology of post-traumatic stress disorder (PTSD) and that anti-infl

102 tive protein (CRP) have been associated with post-traumatic stress disorder (PTSD) and traumatic expe

103 candidate that has enhanced extinction in a post-traumatic stress disorder (PTSD) animal model and w

104 Part of the symptomatology of post-traumatic stress disorder (PTSD) are alterations in

105 KEY POINTS: Patients with post-traumatic stress disorder (PTSD) are at a significa

106 ermining if traumatic brain injury (TBI) and post-traumatic stress disorder (PTSD) are risk factors f

107 e begin to elucidate the mechanisms by which post-traumatic stress disorder (PTSD) at a young age con

108 nts and are at increased risk for developing post-traumatic stress disorder (PTSD) compared with the

109 Stress modifies nociception, and humans with post-traumatic stress disorder (PTSD) exhibit co-morbid

110 Women are at increased risk of developing post-traumatic stress disorder (PTSD) following a trauma

111 Post-traumatic stress disorder (PTSD) has been an exempl

112 Post-traumatic stress disorder (PTSD) impacts many veter

113 he present study investigates the effects of post-traumatic stress disorder (PTSD) in combat-exposed

114 on due to acute stress disorder (ASD) and/or post-traumatic stress disorder (PTSD) in firearm-injury

115 sex and gender biases in disorders including post-traumatic stress disorder (PTSD) in humans.

116 factors associated with trauma exposure and post-traumatic stress disorder (PTSD) in young people.

117 Screen to assess for probable depression and post-traumatic stress disorder (PTSD) indicators.

118 Post-traumatic stress disorder (PTSD) is a common, debil

119 Post-traumatic stress disorder (PTSD) is a debilitating

120 Post-traumatic stress disorder (PTSD) is a heterogeneous

121 Post-traumatic stress disorder (PTSD) is a major problem

122 Post-Traumatic Stress Disorder (PTSD) is a psychiatric d

123 Post-traumatic stress disorder (PTSD) is an anxiety diso

124 ABSTRACT: Post-traumatic stress disorder (PTSD) is associated with

125 Post-traumatic stress disorder (PTSD) is characterized b

126 Post-traumatic stress disorder (PTSD) is characterized b

127 Post-traumatic stress disorder (PTSD) is characterized b

128 Post-traumatic stress disorder (PTSD) is thought to deve

129 isorder (MDD), Parkinson's Disease (PD), and Post-Traumatic Stress Disorder (PTSD) patients with inso

130 sorders.SIGNIFICANCE STATEMENT Patients with post-traumatic stress disorder (PTSD) show heightened am

131 Structural brain studies of adult post-traumatic stress disorder (PTSD) show reduced gray

132 ter a child's premature birth can present as post-traumatic stress disorder (PTSD) symptoms and can i

133 predictors, and correlates of cancer-related post-traumatic stress disorder (PTSD) symptoms and diagn

134 kers of associative threat learning to overt post-traumatic stress disorder (PTSD) symptoms in combat

135 Primary outcome was post-traumatic stress disorder (PTSD) symptoms or casene

136 We assessed patients' mood, post-traumatic stress disorder (PTSD) symptoms, and QOL

137 examines associations between war exposures, post-traumatic stress disorder (PTSD) symptoms, depressi

138 The increased prevalence of post-traumatic stress disorder (PTSD) that is observed i

139 Post-traumatic stress disorder (PTSD) was measured with

140 disorders, including anxiety, depression or post-traumatic stress disorder (PTSD)(2-4).

141 f anxiety and fatigue (but not depression or post-traumatic stress disorder (PTSD)).

142 depressive disorder (MDD), 50 patients with post-traumatic stress disorder (PTSD), and 122 healthy c

143 tes were calculated for depression, anxiety, post-traumatic stress disorder (PTSD), and suicidal beha

144 ibing recovery from ICU included the risk of post-traumatic stress disorder (PTSD), anxiety, depressi

145 assess whether post-deployment screening for post-traumatic stress disorder (PTSD), depression, anxie

146 e study of the neurobiological correlates of post-traumatic stress disorder (PTSD), little is known a

147 dering the effect of traumatic experience on post-traumatic stress disorder (PTSD), this study aims t

148 e epigenetic response to traumatic stress on post-traumatic stress disorder (PTSD), this study examin

149 eceptor type 2 (CRFR2) to be associated with post-traumatic stress disorder (PTSD)-like symptoms.

150 of psychiatric disorders such as phobias and post-traumatic stress disorder (PTSD).

151 ted with major depressive disorder (MDD) and post-traumatic stress disorder (PTSD).

152 s of traumatic stress and the development of post-traumatic stress disorder (PTSD).

153 als experiencing trauma subsequently develop post-traumatic stress disorder (PTSD).

154 , such as autism spectrum disorder (ASD) and post-traumatic stress disorder (PTSD).

155 stress-related psychopathologies, including post-traumatic stress disorder (PTSD).

156 ds threat is one of the defining features of post-traumatic stress disorder (PTSD).

157 phenotype is consistent with some aspects of post-traumatic stress disorder (PTSD).

158 nt is a hallmark of emotional disorders like post-traumatic stress disorder (PTSD).

159 r functional neurological disorder (FND) and post-traumatic stress disorder (PTSD).

160 tivity and is involved in the development of post-traumatic stress disorder (PTSD).

161 y adapt to the trauma is a characteristic of post-traumatic stress disorder (PTSD).

162 nd prepulse inhibition) that are observed in post-traumatic stress disorder (PTSD).

163 ation (TBS) is a potential new treatment for post-traumatic stress disorder (PTSD).

164 impactful and commonly reported symptoms in post-traumatic stress disorder (PTSD).

165 a common feature of panic disorder (PD) and post-traumatic stress disorder (PTSD).

166 asure: presence of daily pain; screening for post-traumatic stress disorder (PTSD); new functional li

167 ions highlight a unique sensory pathology of post-traumatic stress disorder (ruling out effects merel

168 eported symptoms of depression, anxiety, and post-traumatic stress disorder 1 year after flooding.

169 2.93), for anxiety 1.66 (1.12-2.46), and for post-traumatic stress disorder 1.70 (1.17-2.48) than peo

170 stress disorder (PTSD) was measured with the Post-traumatic Stress Disorder 8 items (PTSD-8) and seve

171 sorders (major depression, bipolar disorder, post-traumatic stress disorder [PTSD], substance and alc

172 A vast expansion of research into post-traumatic stress disorder accompanied revisions to

173 interventions that might offset the risk of post-traumatic stress disorder after cardiovascular dise

174 Interestingly, patients with post-traumatic stress disorder also showed heightened fr

175 ) for mild forms of depression, anxiety, and post-traumatic stress disorder and 4.0% (95% UI 2.9-5.5)

176 Post-traumatic stress disorder and anxiety are more prev

177 ny candidate mechanisms for the link between post-traumatic stress disorder and cardiovascular diseas

178 Post-traumatic stress disorder and cocaine use disorder

179 ses conditionability, a phenotype related to post-traumatic stress disorder and propose that this phe

180 abis' effects on two psychiatric conditions: post-traumatic stress disorder and psychotic disorders.

181 ematic reviews for two psychiatric outcomes (post-traumatic stress disorder and schizophrenia) and on

182 c reviews on only two psychiatric disorders (post-traumatic stress disorder and schizophrenia) and on

183 Fear-related psychopathologies such as post-traumatic stress disorder are characterized by impa

184 ence of symptoms of depression, anxiety, and post-traumatic stress disorder between participants disp

185 t-traumatic stress disorder (measured by the Post-Traumatic Stress Disorder Checklist [PCL]-6 scale).

186 Post-traumatic stress disorder develops in response to a

187 whom carried mild traumatic brain injury and post-traumatic stress disorder diagnoses.

188 and thus it appears that the development of post-traumatic stress disorder drives cocaine use vulner

189 is association, as well as the prevalence of post-traumatic stress disorder due to cardiovascular dis

190 ng to passive picture viewing, patients with post-traumatic stress disorder failed to demonstrate alp

191 The onset of post-traumatic stress disorder generally occurs prior to

192 Primary outcome was symptoms of post-traumatic stress disorder in family members 90 days

193 s to predict the emergence of depression and Post-Traumatic Stress Disorder in Twitter users.

194 We conclude that post-traumatic stress disorder is a risk factor for inci

195 Post-traumatic stress disorder is characterized by exagg

196 lity are associated with pathologies such as post-traumatic stress disorder or autism; however, the u

197 Post-traumatic stress disorder patients experience chron

198 in 8 days after the ICU discharge to predict post-traumatic stress disorder symptoms at 3 months, the

199 executive control, fuelling and perpetuating post-traumatic stress disorder symptoms.

200 to 35 at 3 months were considered as having post-traumatic stress disorder symptoms.

201 detection of patients at risk of developing post-traumatic stress disorder symptoms.

202 < 0.001) were identified as risk factors for post-traumatic stress disorder symptoms.

203 at ICU discharge are more at risk to develop post-traumatic stress disorder symptoms.

204 s in four studies, more women presented with post-traumatic stress disorder than did men in two studi

205 causes a greater than expected frequency of post-traumatic stress disorder than other traumatic even

206 berrations help construct a vicious cycle in post-traumatic stress disorder that is in action even at

207 Scores for depression and post-traumatic stress disorder were higher in people who

208 Symptoms of post-traumatic stress disorder were significantly higher

209 Rates of depression, anxiety, and post-traumatic stress disorder were similar to other cri

210 e look at the evidence for an association of post-traumatic stress disorder with incident cardiovascu

211 We recently characterized a rat model of post-traumatic stress disorder with segregation of rats

212 ping treatments for memory disorders such as post-traumatic stress disorder(5).

213 ., early symptoms of anxiety, depression, or post-traumatic stress disorder) critical illness that ca

214 evere depression, severe anxiety, and severe post-traumatic stress disorder) was 5.1% (95% UI 4.0-6.5

215 flooding (p=0.04 for depression, p=0.01 for post-traumatic stress disorder), although the difference

216 ession Scale; Impact of Event Scale-Revised (post-traumatic stress disorder); 6-minute walk; and/or t

217 that examined the apolipoprotein E4 allele, post-traumatic stress disorder, and genetic risk for sch

218 ons characterized schizophrenia, depression, post-traumatic stress disorder, and obsessive-compulsive

219 UXO victims reported a greater prevalence of post-traumatic stress disorder, anxiety, or depression t

220 ssion, poor sleep quality, somatic symptoms, post-traumatic stress disorder, being overweight and neg

221 Other psychiatric disorders, such as post-traumatic stress disorder, bipolar disorder and dep

222 prevalence of depression, anxiety disorder, post-traumatic stress disorder, bipolar disorder, and sc

223 ce of mental disorders (depression, anxiety, post-traumatic stress disorder, bipolar disorder, and sc

224 Intrusive memories are prominent features of post-traumatic stress disorder, but the mechanisms suppo

225 e exposure to a salient stressor, such as in post-traumatic stress disorder, can have lasting impacts

226 hysiologic and psychologic diagnoses such as post-traumatic stress disorder, cardiovascular disease,

227 alcohol and used a diagnostic tool to assess post-traumatic stress disorder, depression, and anxiety.

228 Six of the studies screened for post-traumatic stress disorder, depression, and anxiety;

229 iological disease states such as depression, post-traumatic stress disorder, hypertension, diabetes,

230 rolonged stress, a validated rodent model of post-traumatic stress disorder, in combination with opto

231 treatments of psychiatric conditions such as post-traumatic stress disorder, in which aversive experi

232 for understanding mental disorders, such as post-traumatic stress disorder, little is known about th

233 oradrenergic system, a known risk factor for post-traumatic stress disorder, modulates the stress-ind

234 hosis, n = 129; unipolar depression, n = 92; post-traumatic stress disorder, n = 91; obsessive-compul

235 controls, n=17; major depression, n=38; and post-traumatic stress disorder, n=50), we examine the di

236 activity disorder (ADHD), Tourette syndrome, post-traumatic stress disorder, or psychosis, either as

237 t hyperactivity disorder, Tourette syndrome, post-traumatic stress disorder, or psychosis.

238 atric disorders, including major depression, post-traumatic stress disorder, schizophrenia, and addic

239 raging results as a treatment for refractory post-traumatic stress disorder, social anxiety in autist

240 To elucidate a sensory pathology of post-traumatic stress disorder, we examined intrinsic vi

241 memories in disorders such as addiction and post-traumatic stress disorder.

242 oad, threat-neutral sensory hyperactivity in post-traumatic stress disorder.

243 ing an additional aetiological mechanism for post-traumatic stress disorder.

244 ctive disorders, such as chronic anxiety and post-traumatic stress disorder.

245 its potential for use in pharmacotherapy for post-traumatic stress disorder.

246 and its levels are reduced in patients with post-traumatic stress disorder.

247 associated with excessive stress, including post-traumatic stress disorder.

248 development of anxiety disorders, including post-traumatic stress disorder.

249 for surgery, and the development of acute or post-traumatic stress disorder.

250 rders, such as depression, schizophrenia, or post-traumatic stress disorder.

251 atric disorders such as major depression and post-traumatic stress disorder.

252 bserved in depression, bipolar disorder, and post-traumatic stress disorder.

253 rders, such as depression, schizophrenia, or post-traumatic stress disorder.

254 disorders, including depression, anxiety and post-traumatic stress disorder.

255 anges in emotional arousal that characterize post-traumatic stress disorder.

256 cing social stimuli in models of anxiety and post-traumatic stress disorder.

257 ed protein kinase SGK1 in the development of post-traumatic stress disorder.

258 report symptoms of depression, anxiety, and post-traumatic stress disorder.

259 1), only five (1.1%) of 470 met criteria for post-traumatic stress disorder.

260 ats, a model that captures features of human post-traumatic stress disorder.

261 ICU was associated with reduced symptoms of post-traumatic stress disorder.

262 atments for memory-related disorders such as post-traumatic stress disorder.

263 ptoms of many psychiatric illnesses, such as post-traumatic stress disorder.

264 distortion, and treatment of disorders like post-traumatic stress disorder.

265 xamined in the context of the development of post-traumatic stress disorder.

266 in stress-related memory disorders, such as post-traumatic stress disorder.

267 sociated with psychiatric disorders, such as post-traumatic stress disorder.

268 osed as a novel treatment for depression and post-traumatic stress disorder.

269 ptive plasticities that underlie anxiety and post-traumatic stress disorders in humans.

270 has been demonstrated to be useful to treat post-traumatic stress disorders.

271 than or equal to 24 hours were screened with Post-Traumatic Stress Scale 10 intensive part B after IC

272 tion group versus control group, with a mean Post-Traumatic Stress Scale 10 intensive part B score 39

273 There was a significantly reduced Post-Traumatic Stress Scale 10 intensive part B score fo

274 Mean Post-Traumatic Stress Scale 10 intensive part B score wa

275 Sense of Coherence Scale 13 and Post-Traumatic Stress Scale 10 intensive part B were com

276 tudy has described their potential to reduce post-traumatic stress symptomatology in family members.

277 ess disorder (PTSD), anxiety, depression and post-traumatic stress symptomatology, health-related qua

278 d Post Concussive Symptom Questionnaire) and post-traumatic stress symptoms (Posttraumatic Stress Dis

279 lems were associated with increased level of post-traumatic stress symptoms at 12 months.

280 ough RB survivors were more likely to report post-traumatic stress symptoms of avoidance and/or hyper

281 ons did not reveal any significant effect on post-traumatic stress symptoms or sense of coherence aft

282 No differences in post-traumatic stress symptoms or sense of coherence wer

283 tic brain injury, older age, and more severe post-traumatic stress symptoms provided a good predictio

284 e effects, post-treatment complications, and post-traumatic stress symptoms that can significantly di

285 nd coping strategies and resources predicted post-traumatic stress symptoms three months after the ch

286 Post-traumatic stress symptoms were assessed via self-re

287 For post-traumatic stress symptoms, 48 (67.6%) were at risk.

288 ted negative psychological effects including post-traumatic stress symptoms, confusion, and anger.

289 nship between negative coping strategies and post-traumatic stress symptoms.

290 ure, and this effect was also independent of post-traumatic stress symptoms.

291 ssments revealed heightened post-concussive, post-traumatic stress, and depressive symptoms along wit

292 mes included personally identified problems, post-traumatic stress, depression symptoms, feelings of

293 Health Administration (VA) have a history of post-traumatic-stress-disorder (PTSD), and there exists

294 Patients with post-traumatic-stress-disorder reported higher pain scor

295 Our data suggests that a history of post-traumatic-stress-disorder was correlated with highe

296 Secondary outcomes included history of post-traumatic-stress-disorder, anesthesia type, first o

297 sociated with anxiety, perceived stress, and post-traumatic symptomatology.

298 not show differences in anxiety, stress, and post-traumatic symptoms compared to non-cancer participa

299 strate that CN2097 significantly reduces the post-traumatic synthesis of proinflammatory mediators an

300 , intravital microscopy demonstrated reduced post-traumatic thrombus formation in the pericontusional

301 s not yet strong evidence of benefit for the post-traumatic variant.