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1 d-type mice (n=6), we found less bacteria in postcapillary (60+/-6 versus 32+/-5 bacteria) and collec
3 ecommended to differentiate between isolated postcapillary and combined pre-/postcapillary pulmonary
4 ary pulmonary hypertension (IpcPH), combined postcapillary and precapillary (CpcPH), or exercise PH.
5 ith preserved ejection fraction and combined postcapillary and precapillary pulmonary hypertension we
6 ith preserved ejection fraction and combined postcapillary and precapillary pulmonary hypertension, w
7 ith preserved ejection fraction and combined postcapillary and precapillary pulmonary hypertension.
9 alciparum-infected erythrocytes (Pf-IRBC) in postcapillary brain endothelium is a hallmark of cerebra
10 diately improved after TAVI in patients with postcapillary combined (57.8+/-14.1 versus 50.4+/-17.3 m
11 helial venules, a compartment of specialized postcapillary endothelial cells that are characterized b
13 od cells (RBCs) and leukocytes as they enter postcapillary expansions, but the details of the fluid d
15 de lymphocytes (LNCs) tethered and rolled in postcapillary high endothelial venules (HEVs) and to a l
16 left ventricular end-diastolic pressure into postcapillary (left ventricular end-diastolic pressure,
17 at parasitized erythrocytes can sequester in postcapillary microvessels of critical tissues such as t
19 cohort of 255 patients with PH from pre and postcapillary pathogeneses was assembled from 2 centers.
29 een isolated postcapillary and combined pre-/postcapillary pulmonary hypertension (Cpc-PH) in left he
31 (PH) is classified as precapillary, isolated postcapillary pulmonary hypertension (IpcPH), combined p
34 arterial hypertension, and in patients with postcapillary pulmonary hypertension because of left hea
35 sion of various causes, including those with postcapillary pulmonary hypertension due to left heart d
36 lmonary capillary wedge pressure (PCWP) with postcapillary pulmonary hypertension even at rest, which
37 re than those with combined precapillary and postcapillary pulmonary hypertension HFpEF (n=31; intera
39 es a subset of PH-LHD patients from isolated postcapillary pulmonary hypertension to Cpc-PH, which is
40 stcapillary pulmonary hypertension, combined postcapillary pulmonary hypertension, and pulmonary arte
41 ined 4 hemodynamic groups: control, isolated postcapillary pulmonary hypertension, combined postcapil
46 nt of left ventricular failure in those with postcapillary pulmonary hypertension; and hydroxyurea or
49 increased leukocyte rolling and adhesion in postcapillary skin venules that were both inhibited afte
52 was widespread basal plasma extravasation in postcapillary venular endothelia in NEP-/- mice, which w
54 926 (derived from human umbilical vein), and postcapillary venular endothelial cells (derived from bo
55 Duffy Ag expressed on RBCs, capillaries, and postcapillary venular endothelial cells binds selective
59 rs of vasoconstriction, edema formation, and postcapillary venule leakage, followed by ex vivo functi
61 d cells as they flow from a capillary into a postcapillary venule using a lattice Boltzmann approach.
62 We constructed composite models of the human postcapillary venule, combining ECs with PCs or PC-depos
66 of 478 microbubbles (13.6%) observed in six postcapillary venules 11 to 30 microm in diameter and 24
67 s and vascular permeability were measured in postcapillary venules after 4-hour and 1-hour reperfusio
68 TDLN cells began migrating across pulmonary postcapillary venules and first appeared within metastas
69 e blood were investigated in mouse cremaster postcapillary venules and in flow chambers coated with P
71 ity begins in the superficial arterioles and postcapillary venules and progresses to the capillary be
72 ross HEVs is faster than across conventional postcapillary venules and requires a unique set of adhes
74 ation of 40-kDa dextran from capillaries and postcapillary venules but had no effect on extravasation
75 ting leukocytes were labeled and observed in postcapillary venules for adhesion before and up to 120
77 theless, the number of leukocytes rolling on postcapillary venules in an E-selectin-dependent manner
78 bone marrow (BM) endothelium and to inflamed postcapillary venules in an E-selectin-dependent manner.
79 phocyte-associated Ag tether on the walls of postcapillary venules in inflamed skin via interaction w
80 othelial venules (HEV) are specialized plump postcapillary venules in lymphoid tissues that support h
82 es can be found marginalized in the lumen of postcapillary venules in postmortem brain tissue derived
84 ) increases clearance of macromolecules from postcapillary venules in the in situ oral mucosa and, if
85 duced neutrophil rolling and adhesion to the postcapillary venules in the mouse ears is significantly
87 retinal perivascular macrophages resided on postcapillary venules in the superficial vascular plexus
90 arkedly reduced (>60%) leukocyte adhesion to postcapillary venules in wild type and Fpr1(-/-), but no
91 ting therapies evoke transdifferentiation of postcapillary venules into inflamed high-endothelial ven
92 Transendothelial migration of neutrophils in postcapillary venules is a key event in the inflammatory
93 d that the abundance of leukocyte rolling in postcapillary venules is due to interactions between red
95 ncreased expression of P-selectin protein in postcapillary venules of all vital organs after trauma.
99 ng and adhesion were measured in cremasteric postcapillary venules of septic and control rats using i
100 med soluble ICs are rapidly deposited in the postcapillary venules of the cremaster microcirculation,
101 nt microbeads flowing within mildly inflamed postcapillary venules of the cremaster muscle in vivo.
102 in vivo, studied by intravital microscopy in postcapillary venules of the cremaster muscle, was marke
103 dothelial Tie-2 results in leak formation in postcapillary venules of the inflamed cremaster muscle a
104 was restricted to focal areas of the retinal postcapillary venules of the inner vascular plexus.
105 s leukocyte-endothelial cell interactions in postcapillary venules of the mouse cremaster muscle.
106 yte-endothelial cell adhesion in cremasteric postcapillary venules of wild-type (WT) mice, CuZn-super
107 sed to monitor L/E and P/E adhesion in brain postcapillary venules of wild-type (WT), SOD1 transgenic
108 mber of adherent and emigrated leukocytes in postcapillary venules of WT HCD mice was significantly h
109 n is important in inflammation and occurs in postcapillary venules over a wide range of wall shear st
110 se model suggest that adherent leukocytes in postcapillary venules play a critical role in vaso-occlu
112 that P-selectin and E-selectin expressed on postcapillary venules play overlapping roles in the recr
113 d leukocyte-endothelial cell interactions in postcapillary venules revealed that CXCL1-induced neutro
114 h endothelial venules (HEVs) are specialised postcapillary venules that specifically serve this funct
115 thelium may regulate neutrophil migration in postcapillary venules through the presentation of variou
116 ltered adhesive interactions within inflamed postcapillary venules under conditions of blood flow by
117 reveal that the basement membrane of dermal postcapillary venules undergoes changes in structure and
122 hodamine-stained leukocytes were observed in postcapillary venules with analysis for adhesion and rol
124 ntibody fluorescence intensity in submucosal postcapillary venules with the use of intravital microsc
127 increased in the superficial arterioles and postcapillary venules, 2 weeks after the onset of diabet
128 sed luminally by endothelial cells that line postcapillary venules, a primary site of leukocyte recru
129 ed adhesion of leukocytes in capillaries and postcapillary venules, but no such adhesion in arteriole
130 about midway between terminal arterioles and postcapillary venules, challenging the classical concept
131 adhesion of leukocytes to discrete sites on postcapillary venules, followed by upregulation of adhes
132 mbranes that arise from inflamed venules and postcapillary venules, increase in size as the disease p
133 f leukocyte-endothelial cell interactions in postcapillary venules, leading to leukocyte recruitment
135 including endothelial cells of capillary and postcapillary venules, the epithelial cell of kidney col
136 r physiological shear stress consistent with postcapillary venules, we found a significant increase i
137 designed to approximate physiologic flow in postcapillary venules, we have characterized a rolling i
139 ill offer an effective, direct access to the postcapillary venules, where the target event (leukocyte
174 in tissues that possess receptors for SP in postcapillary venules; (ii) liposome material in these t
175 ery catheterization with blood sampling from postcapillary (wedged balloon) and precapillary (unwedge