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1 l endothelial inflammation at the collecting postcapillary venule.
2 rteries and veins, most arterioles, and some postcapillary venules.
3 utrophil emigration across murine mesenteric postcapillary venules.
4 ophil but not eosinophil rolling in inflamed postcapillary venules.
5 and in a flow system to mirror conditions at postcapillary venules.
6 o the relatively low-oxygen and low-velocity postcapillary venules.
7 hat activate leukocytes to transmigrate from postcapillary venules.
8  and E-selectin-dependent adhesion in airway postcapillary venules.
9 ualize rolling of endogenous Tregs in dermal postcapillary venules.
10  pial vessels and in deep ascending cortical postcapillary venules.
11 y channels connecting terminal arterioles to postcapillary venules.
12 displayed on endothelial cells in intestinal postcapillary venules.
13 shear stress levels similar to that found in postcapillary venules.
14 selectins to roll and integrins to arrest in postcapillary venules.
15 tructure and increases leukocyte adhesion in postcapillary venules.
16 sults in vasculitic lesions predominantly in postcapillary venules.
17 enous malformations, and focal dilatation of postcapillary venules.
18  gap formation, and protein extravasation in postcapillary venules.
19 small lymphocytes for the endothelium of the postcapillary venules.
20 rolling effector and central memory cells in postcapillary venules.
21 e and continue to roll as elongated cells in postcapillary venules.
22 es as the main L-selectin ligand in inflamed postcapillary venules.
23 minants of the initiation of cell rolling in postcapillary venules.
24 nd pericytes (PCs) that form the wall of the postcapillary venules.
25 eukocyte rolling and adherence in mesenteric postcapillary venules.
26 ma that tumor vessels arise exclusively from postcapillary venules.
27 erial/venous characteristics and derive from postcapillary venules.
28 re weeks exhibit an inflammatory response in postcapillary venules.
29  occur primarily via capillaries rather than postcapillary venules.
30 nomethyl-L-arginine (NMA) on skeletal muscle postcapillary venules.
31 deficient in in vivo crawling and TEM in the postcapillary venules.
32 rolling eosinophils, but not neutrophils, to postcapillary venules.
33 y designed to mimic physiologic flow through postcapillary venules.
34 ophages and erythrocytes) in capillaries and postcapillary venules.
35 leukocyte rolling and adhesion in mesenteric postcapillary venules.
36  of 478 microbubbles (13.6%) observed in six postcapillary venules 11 to 30 microm in diameter and 24
37  increased in the superficial arterioles and postcapillary venules, 2 weeks after the onset of diabet
38 sed luminally by endothelial cells that line postcapillary venules, a primary site of leukocyte recru
39 s and vascular permeability were measured in postcapillary venules after 4-hour and 1-hour reperfusio
40  TDLN cells began migrating across pulmonary postcapillary venules and first appeared within metastas
41 e blood were investigated in mouse cremaster postcapillary venules and in flow chambers coated with P
42 of neutrophils along the endothelial wall of postcapillary venules and integrin-mediated arrest.
43 ity begins in the superficial arterioles and postcapillary venules and progresses to the capillary be
44 ross HEVs is faster than across conventional postcapillary venules and requires a unique set of adhes
45  borders between endothelial cells that line postcapillary venules at that site.
46 ation of 40-kDa dextran from capillaries and postcapillary venules but had no effect on extravasation
47 ed adhesion of leukocytes in capillaries and postcapillary venules, but no such adhesion in arteriole
48 about midway between terminal arterioles and postcapillary venules, challenging the classical concept
49 We constructed composite models of the human postcapillary venule, combining ECs with PCs or PC-depos
50              Our results show that capillary-postcapillary venule diameter ratio, RBC configuration,
51  adhesion of leukocytes to discrete sites on postcapillary venules, followed by upregulation of adhes
52 ting leukocytes were labeled and observed in postcapillary venules for adhesion before and up to 120
53 l cells from human renal arteries as well as postcapillary venules from lymphoid tissue.
54  in tissues that possess receptors for SP in postcapillary venules; (ii) liposome material in these t
55 theless, the number of leukocytes rolling on postcapillary venules in an E-selectin-dependent manner
56 bone marrow (BM) endothelium and to inflamed postcapillary venules in an E-selectin-dependent manner.
57 phocyte-associated Ag tether on the walls of postcapillary venules in inflamed skin via interaction w
58 othelial venules (HEV) are specialized plump postcapillary venules in lymphoid tissues that support h
59  (PfEMP1) mediates parasite sequestration in postcapillary venules in P. falciparum malaria.
60 es can be found marginalized in the lumen of postcapillary venules in postmortem brain tissue derived
61 nd sialyl Lewis(x) (sLe(x)) to interact with postcapillary venules in the absence of PSGL-1.
62 ) increases clearance of macromolecules from postcapillary venules in the in situ oral mucosa and, if
63 duced neutrophil rolling and adhesion to the postcapillary venules in the mouse ears is significantly
64 , E-selectin, and ICAM-1 are up-regulated on postcapillary venules in the retina.
65  retinal perivascular macrophages resided on postcapillary venules in the superficial vascular plexus
66 s required to sustain neutrophil adhesion in postcapillary venules in vivo.
67 eukocyte trafficking across mouse mesenteric postcapillary venules in vivo.
68 arkedly reduced (>60%) leukocyte adhesion to postcapillary venules in wild type and Fpr1(-/-), but no
69 mbranes that arise from inflamed venules and postcapillary venules, increase in size as the disease p
70 ting therapies evoke transdifferentiation of postcapillary venules into inflamed high-endothelial ven
71 Transendothelial migration of neutrophils in postcapillary venules is a key event in the inflammatory
72 d that the abundance of leukocyte rolling in postcapillary venules is due to interactions between red
73                    Plasma extravasation from postcapillary venules is one of the earliest steps of in
74 f leukocyte-endothelial cell interactions in postcapillary venules, leading to leukocyte recruitment
75 rs of vasoconstriction, edema formation, and postcapillary venule leakage, followed by ex vivo functi
76           In flow chamber experiments, under postcapillary venule-like flow conditions, the pretreatm
77              Most of the leakage occurred in postcapillary venules (< 40-microns diameter), whereas m
78 ncreased expression of P-selectin protein in postcapillary venules of all vital organs after trauma.
79 ks leukocyte extravasation in lung, skin and postcapillary venules of cremaster muscle.
80 s the leukocyte-endothelial cell adhesion in postcapillary venules of HCD mice.
81                         Leukocyte rolling in postcapillary venules of inflamed tissues is reduced in
82 ng and adhesion were measured in cremasteric postcapillary venules of septic and control rats using i
83 med soluble ICs are rapidly deposited in the postcapillary venules of the cremaster microcirculation,
84 nt microbeads flowing within mildly inflamed postcapillary venules of the cremaster muscle in vivo.
85 in vivo, studied by intravital microscopy in postcapillary venules of the cremaster muscle, was marke
86 dothelial Tie-2 results in leak formation in postcapillary venules of the inflamed cremaster muscle a
87 was restricted to focal areas of the retinal postcapillary venules of the inner vascular plexus.
88 s leukocyte-endothelial cell interactions in postcapillary venules of the mouse cremaster muscle.
89 yte-endothelial cell adhesion in cremasteric postcapillary venules of wild-type (WT) mice, CuZn-super
90 sed to monitor L/E and P/E adhesion in brain postcapillary venules of wild-type (WT), SOD1 transgenic
91 mber of adherent and emigrated leukocytes in postcapillary venules of WT HCD mice was significantly h
92 n is important in inflammation and occurs in postcapillary venules over a wide range of wall shear st
93 se model suggest that adherent leukocytes in postcapillary venules play a critical role in vaso-occlu
94          E-selectin and P-selectin on dermal postcapillary venules play critical roles in the migrati
95  that P-selectin and E-selectin expressed on postcapillary venules play overlapping roles in the recr
96 d leukocyte-endothelial cell interactions in postcapillary venules revealed that CXCL1-induced neutro
97       Neutrophil extravasation occurs across postcapillary venules, structures composed of endothelia
98 h endothelial venules (HEVs) are specialised postcapillary venules that specifically serve this funct
99 including endothelial cells of capillary and postcapillary venules, the epithelial cell of kidney col
100 thelium may regulate neutrophil migration in postcapillary venules through the presentation of variou
101 ltered adhesive interactions within inflamed postcapillary venules under conditions of blood flow by
102  reveal that the basement membrane of dermal postcapillary venules undergoes changes in structure and
103        The number of leukocytes in capillary-postcapillary venule units per 4-second image sequence (
104 d cells as they flow from a capillary into a postcapillary venule using a lattice Boltzmann approach.
105                         Wall shear stress in postcapillary venules varies widely within and between t
106   Intravital video microscopy of cremasteric postcapillary venules was performed.
107 r physiological shear stress consistent with postcapillary venules, we found a significant increase i
108  designed to approximate physiologic flow in postcapillary venules, we have characterized a rolling i
109                                              Postcapillary venules were dilated, and up to 70% of the
110                                     Tracheal postcapillary venules were visualized and leukocyte kine
111                                           In postcapillary venules, where shear stress is approximate
112 ill offer an effective, direct access to the postcapillary venules, where the target event (leukocyte
113 hodamine-stained leukocytes were observed in postcapillary venules with analysis for adhesion and rol
114 ed a network of arterioles, capillaries, and postcapillary venules with continuous blood flow.
115 ntibody fluorescence intensity in submucosal postcapillary venules with the use of intravital microsc
116                     Intravital microscopy of postcapillary venules within the cremaster muscle of mic
117 ial permeability and leakage of albumin from postcapillary venules within the dura mater.

 
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