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1 the gold standard for the treatment of PCO (Posterior Capsule Opacification).
2 , cystoid macular oedema (CMO), cataract and posterior capsule opacification.
3 nt, cystoid macular oedema, glare, halos and posterior capsule opacification.
4 an effective capsular bend as a deterrent to posterior capsule opacification.
5 ng mechanism for the aberrant cell growth in posterior capsule opacification.
6 pressure spikes, cystoid macular edema, and posterior capsule opacification.
7 and could be important in the development of posterior capsule opacification.
8 practical for use in longitudinal studies of posterior capsule opacification.
9 mmunotoxin may be effective in prevention of posterior capsule opacification.
10 e, and effective measure in the treatment of posterior capsule opacification.
11 d Pande et al. for precise quantification of posterior-capsule opacification.
12 y (0.9%), cataracts (3.6%), intraocular lens posterior capsule opacification (1.8%) and a posterior c
14 ally valuable in prevention and treatment of posterior capsule opacification, a dreaded complication
15 tion (between piggyback intraocular lenses), posterior capsule opacification and lens epithelial cell
16 and pharmacologic methods); 2) treatment of posterior capsule opacification; and 3) characteristics
17 how resistant an intraocular lens will be to posterior capsule opacification as a consequence of rege
18 ept that sharp optic edges markedly decrease posterior capsule opacification as compared with round-e
20 d with lower rates of clinically significant posterior capsule opacification compared to treatment wi
25 stance visual acuity (CDVA), IOL centration, posterior capsule opacification, glaucoma, and retinal c
26 rban residency and were found in 60.4%, with posterior capsule opacification in 29.6% and posterior c
27 ecial interest are techniques for inhibiting posterior capsule opacification in pediatric patients by
28 d strength, which may decrease resistance to posterior capsule opacification in the face of a regener
29 sterior capsulorhexis is required to inhibit posterior-capsule opacification in pediatric patients; w
36 cantly higher rates of hypotony (p = 0.043), posterior capsule opacification (p = 0.047), and surgica
37 I(2) = 0%; n = 2 studies, 161 participants), posterior capsule opacification (P = 0.46; I(2) = 0%; n
39 le is known about the long-term incidence of posterior capsule opacification (PCO) after cataract sur
42 provements in intraocular lens (IOL) design, posterior capsule opacification (PCO) arising from lens
46 antification of After-Cataract [AQUA II]) of posterior capsule opacification (PCO) in high-resolution
50 ct surgery is common, routine and effective, posterior capsule opacification (PCO) occurs in 30-50% o
51 tients gradually develop the complication of posterior capsule opacification (PCO) or secondary catar
52 dy were to determine the 5-year incidence of posterior capsule opacification (PCO) requiring Nd:YAG l
53 utive pseudophakic patients with symptomatic posterior capsule opacification (PCO) underwent Nd:YAG l
54 DVA), subjective refraction, IOL centration, posterior capsule opacification (PCO), and investigators
55 oped yttrium aluminum garnet capsulotomy for posterior capsule opacification (PCO), and visual acuity
56 ts due to a wound-healing response, known as posterior capsule opacification (PCO), following catarac
61 srupting complication of cataract surgery is posterior capsule opacification (PCO; secondary cataract
62 re instrumental in reducing the incidence of posterior capsule opacification, (PCO, secondary catarac
63 the eye this can cause blindness because of posterior capsule opacification, proliferative vitroreti
65 ge design appreciably improves resistance to posterior capsule opacification, significant factors rem
68 active lens exchange suggest higher rates of posterior capsule opacification than with standard catar
70 th and angle width in pseudophakic eyes with posterior capsule opacification were measured with anter