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1 degeneration of the corticospinal tracts and posterior columns.
2 eral fasciculus gracilis and cuneatus in the posterior columns.
3 dorsal root ganglia and degeneration of the posterior columns.
5 substantia gelatinosa, spinothalamic tracts, posterior columns and nuclei and in the neuropil surroun
6 thology in CANVAS, showing the alteration of posterior columns and roots, astrocytic gliosis and axon
7 physiological findings, revealing atrophy of posterior columns and striking neuronal cell loss from t
8 pe of the rare, autosomal-recessive disorder posterior column ataxia and retinitis pigmentosa (PCARP)
9 tion leads to the neurodegenerative syndrome posterior column ataxia and retinitis pigmentosa(7-9), t
10 not only increasing sagittal bending in the posterior column but also high stiffness and increasing
11 ave resulted from intense involvement of the posterior column (gracile fasciculus) in the thoracolumb
13 most abundant in the retina, followed by the posterior column of the spinal cord and other brain regi
14 ed wild-type mouse Flvcr1 mRNA levels in the posterior column of the spinal cord and the retina via q
15 and motor nerves, with loss of myelin in the posterior columns of the spinal cord and loss of anterio
16 bpopulation of neurons in the retina and the posterior columns of the spinal cord via dysregulation o
17 nfiltration of the cerebellum, damage to the posterior columns of the spinal cord, and sparse infiltr
18 munological damage to the cerebellum, to the posterior columns of the spinal cord, and to peripheral
20 of grey matter (GM), white matter (WM), and posterior columns (PC) were measured to determine atroph
21 nisotropy and mean diffusivity in the GM and posterior columns were significantly more abnormal in SP