ライフサイエンスコーパス: postinfectious

1 recently was likely considered idiopathic or postinfectious.

2 iology was found in 76%: 37% infectious, 16% postinfectious, and 23% noninfectious.

3 ng diseases with a wide array of infectious, postinfectious, and noninfectious causes.

4 d by etiological classification: infectious, postinfectious, and noninfectious.

5 ystemic Th17 immunity in the pathogenesis of postinfectious (antibiotic-refractory) Lyme arthritis.

6 e possibility that this phenotype could be a postinfectious autoimmune CNS disorder, and therefore si

7 These findings support a new subgroup of postinfectious autoimmune inflammatory disorders associa

8 Guillain-Barre syndrome (GBS) is a postinfectious autoimmune neuropathy and anti-gangliosid

9 Postinfectious autoimmune processes are hypothesized to

10 Postinfectious autoimmune processes have been proposed a

11 virulence and provide a molecular basis for postinfectious autoimmunity associated with M. pneumonia

12 c fever (ARF) is one of the best examples of postinfectious autoimmunity due to molecular mimicry bet

13 asma adhesins may play in molecular mimicry, postinfectious autoimmunity, and immune-mediated damage.

14 vation, and neural circuit impairment during postinfectious BGE.

15 Patients with postinfectious bronchiolitis obliterans generally have c

16 Infectious and postinfectious cases had higher median cerebrospinal flu

17 therwise, antibiotics are given at 120 hours postinfectious challenge.

18 demiology, pathophysiology, and treatment of postinfectious chronic fatigue syndrome (CFS) in adolesc

19 lar hemolysis, initially attributed to acute postinfectious cold hemagglutinin (immunoglobulin M) dis

20 inflammatory syndrome in children (MIS-C), a postinfectious complication.

21 what form, or if it may play a role in other postinfectious complications after Lyme disease.

22 forts to improve diagnosis and management of postinfectious complications of SARS-CoV-2 infection in

23 (143 +/- 62 cells/microl); and patients with postinfectious complications showed low EBV load (3.0 +/

24 f human enterocolitis and is associated with postinfectious complications, including irritable bowel

25 ndrome in children (MIS-C) is a novel severe postinfectious condition associated with severe acute re

26 S-C and long COVID within a vast spectrum of postinfectious conditions characterized by intestinal dy

27 al grafts, pseudophakic bullous keratopathy, postinfectious corneal scarring and thinning and keratoc

28 otrienes are involved in the pathogenesis of postinfectious cough and whooping cough (pertussis).

29 However, the burden of postinfectious cough in primary care is high, making it

30 Postinfectious cough is common in primary care, but has

31 al, non-smoking adults aged 16-49 years with postinfectious cough of 2-8 weeks' duration were recruit

32 ene receptor antagonist, in the treatment of postinfectious cough.

33 ontelukast is not an effective treatment for postinfectious cough.

34 ion and underpinning multiple autoimmune and postinfectious diseases.

35 s, acute disseminated encephalomyelitis, and postinfectious disorders should be exclusions in the NMO

36 ions in rare cases with concomitant para- or postinfectious encephalitis.

37 sentation compared to COVID-19, suggesting a postinfectious entity.

38 om keratoplasty restores vision in eyes with postinfectious, full-thickness, central corneal scars.

39 inflammation is suggested to play a role in postinfectious functional gastrointestinal disorders (PI

40 Postinfectious glomerulonephritis (PIGN) is a rare etiol

41 Postinfectious glomerulonephritis (PIGN) is primarily a

42 s, membranoproliferative glomerulonephritis, postinfectious glomerulonephritis, thin basement membran

43 plement activation in 34 children with acute postinfectious GN and low C3 level at onset.

44 pathophysiologic mechanisms underlying acute postinfectious GN by identifying anti-factor B autoantib

45 All children with acute postinfectious GN had activation of the alternative path

46 ase and C3 GN), or clinical course (atypical postinfectious GN).

47 In acute postinfectious GN, anti-factor B autoantibodies were tra

48 With regard to postinfectious GN, C4d staining was negative in six (46%

49 t-mediated glomerulopathy that, unlike acute postinfectious GN, has a poor prognosis.

50 d in human diseases presenting as nephritis (postinfectious GN, IgA nephropathy, antiglomerular basem

51 ng cause of pediatric acute nephritis, acute postinfectious GN, including mechanisms of the pathognom

52 ecimens of C3 GN, and 13 biopsy specimens of postinfectious GN.

53 tients with sepsis, frequently included in a postinfectious hemophagocytic lymphohistiocytosis settin

54 r of life; 5 of 32 (16%) survivors developed postinfectious hydrocephalus (PIH) and 1 (3%) additional

55 oth posthaemorrhagic hydrocephalus (PHH) and postinfectious hydrocephalus (PIH).

56 Paenibacillus thiaminolyticus is a cause of postinfectious hydrocephalus among Ugandan infants.

57 us was the most enriched bacterial genera in postinfectious hydrocephalus CSF (91 [44%] of 209 patien

58 Postinfectious hydrocephalus in infants is a major healt

59 from Paenibacillus spp-related meningitis to postinfectious hydrocephalus over 1-3 months.

60 -center study involving Ugandan infants with postinfectious hydrocephalus showed no significant diffe

61 Patients with postinfectious hydrocephalus with Paenibacillus spp infe

62 pathogen in neonatal sepsis, meningitis, and postinfectious hydrocephalus, we aimed to complete three

63 operitoneal shunting in Ugandan infants with postinfectious hydrocephalus.

64 anda and is the dominant cause of subsequent postinfectious hydrocephalus.

65 , of these, 14% (5 of 37 neonates) developed postinfectious hydrocephalus.

66 Recent data developing from the study of postinfectious IBS has challenged the belief that IBS is

67 ardiasis patients and the pathophysiology of postinfectious IBS is needed.

68 esponsible for diarrheal disease and chronic postinfectious illness.

69 ion of the central nervous system (CNS), and postinfectious immune mediated processes.

70 Guillain-Barre syndrome (GBS) is an acute postinfectious immune-mediated polyneuropathy.

71 sts that SARS-CoV-2 may be associated with a postinfectious, immune-mediated myopathy.

72 occur in patients with COVID-19 secondary to postinfectious inflammatory neuropathy, prone positionin

73 ococcal meningoencephalitis complicated by a postinfectious inflammatory response syndrome (cPIIRS).

74 advantageous T cell response, resulting in a postinfectious, inflammatory arthritis.

75 appears to be a significant risk factor for postinfectious irritable bowel and chronic fatigue syndr

76 stitute an important pathogenic mechanism in postinfectious irritable bowel syndrome (IBS).

77 and Campylobacter, are at increased risk of postinfectious irritable bowel syndrome and inflammatory

78 their impact on long-term effects, including postinfectious irritable bowel syndrome and inflammatory

79 luoroquinolone antibiotics and concurrently, postinfectious irritable bowel syndrome has been associa

80 Postinfectious irritable bowel syndrome may occur in 3%

81 lain-Barre syndrome, reactive arthritis, and postinfectious irritable bowel syndrome) contribute cons

82 whether rifaximin is effective in preventing postinfectious irritable bowel syndrome.

83 s' diarrhea causes substantial morbidity and postinfectious irritable bowel syndrome.

84 determined in 147 patients with LA (87 with postinfectious LA and 60 with antibiotic-responsive LA),

85 resent at greater frequency in patients with postinfectious LA compared to those with antibiotic-resp

86 The message from postinfectious LA for other autoimmune arthritides is th

87 ies develop to ECM proteins in a subgroup of postinfectious LA patients who have specific HLA-DR alle

88 s to these peptides or proteins.RESULTSOf 24 postinfectious LA patients, 58% had CD4+ T cell response

89 lular matrix (ECM) proteins in patients with postinfectious LA, identified potential Borreliella burg

90 Moreover, in patients with postinfectious LA, the levels of these Th17 mediators co

91 Postinfectious Lyme arthritis (LA) is associated with dy

92 regulation of HLA-DR molecules, including in postinfectious Lyme arthritis (LA).

93 is infection and its 2 main adverse outcomes-postinfectious Lyme arthritis and posttreatment Lyme dis

94 have previously reported that patients with postinfectious Lyme arthritis often harbor the peptidogl

95 estigate the impact of infection severity on postinfectious mental disorder sequelae.

96 nt (30-011) who exhibited the characteristic postinfectious mononucleosis phenotype of XLP with hypog

97 are associated with an increase in all-cause postinfectious morbidity and mortality.

98 These findings favor postinfectious myocarditis in children and adolescents w

99 with CNSL, 10 with encephalitis, and 4 with postinfectious neurological complications.

100 Guillain-Barre syndrome (GBS) is a postinfectious neuropathy most frequently caused by Camp

101 d than ever for evidence-based management of postinfectious olfactory dysfunction (PIOD).

102 iated by multiple etiologies but it is often postinfectious or due to persistent cellular immune-medi

103 es of people with acute Lyme disease and its postinfectious outcomes.

104 y hereditary, idiopathic, paraneoplastic, or postinfectious pancerebellitis.

105 o glycans present on glycolipids mediate the postinfectious paralytic disease, Guillain-Barre syndrom

106 technique is also useful in the treatment of postinfectious, posthemorrhagic, and normotensive hydroc

107 bacter infection has also been linked to the postinfectious sequelae of reactive arthritis and Guilla

108 lifetime health outcomes (e.g., death, cure, postinfectious sequelae) for individuals developing TB d

109 ation of the role of Campylobacter jejuni in postinfectious sequelae, a broadened understanding of Ca

110 imicrobial therapy is recommended to prevent postinfectious sequelae.

111 e of diarrhea and is associated with serious postinfectious sequelae.

112 unity and identification of risk factors for postinfectious sequelae.

113 s, providing a new paradigm for the study of postinfectious symptoms in a subset of patients with Lym

114 PCC) is a highly debilitating and persistent postinfectious syndrome that affects millions of people

115 tive development in children, and protracted postinfectious syndromes.

116 re for the treatment of Long-Covid and other postinfectious syndromes.

117 matory immune response leading to persistent postinfectious synovitis with vascular damage, cytotoxic