ライフサイエンスコーパス: postnatal day

1 cutaneously (s.c.) at 2nd,4th, 6th, 8th,10th postnatal day.

2 for NaV1.4 did not survive beyond the second postnatal day.

3 entials (APs), but only during the first few postnatal days.

4 Here, we show that tau haploinsufficiency in postnatal day 0 (P0) heterozygous (Mapt(+/-)) pups, but

5 hy in wildtype (WT) and AVPR1A knockout (KO) postnatal day 0 (P0) male and female mice on a C57BL/6J

6 hippocampal CA1 region pyramidal neurons at postnatal day 0 (P0) or day 21 (P21) and measured synapt

7 ive dose for 2 hrs, and pups were studied at postnatal day 0 (P0) or P30.

8 The MOC-IHC synapse, functional from postnatal day 0 (P0) to hearing onset (P12), undergoes d

9 e molar in K14-Cre;Wnt10a(flox/flox) mice at postnatal day 0 (PN0), just before the initiation of roo

10 Older NPCs (postnatal day 0) exit the progenitor niche at a higher r

11 uscularis of the small intestine of newborn (postnatal day 0) wild-type C57BL/6 mice as well as from

12 global homozygous knockout (KO) mice die at postnatal day 0, and conditional deletion of Snrk in car

13 rly VV Prox1(hi) endothelial organization at postnatal day 0, and this likely underlies the VV defect

14 At birth (postnatal day 0, PND0), four males and four females were

15 -cre;ROSA:YFP mice at embryonic day 12.5 and postnatal day 0, representing opposite ends of the notoc

16 , on cultured primary neurons harvested from postnatal day 0-1 mouse brains.

17 In wild-type neonatal mice (postnatal day 0-1), we show that intravenous injection o

18 nization") of Prox1(hi) endothelial cells by postnatal day 0.

19 evels of apoptotic proteins were elevated at postnatal day 0.

20 roepithelium was observed in tvrm360 mice at postnatal day 0.

21 nt nerve terminals in mouse utricles between postnatal days 0 (P0) and P17.

22 volume postnatally; growth is fastest during postnatal days 0-4 (P0-P4), preceding most myelination.

23 Specifically, postnatal days 0-7 in mice show no significant change in

24 and anterior cingulate cortical thickness at postnatal Day 1 (P1) in HC and LC voles.

25 riostin (Postn)+ lineage CFs were found from postnatal day 1 (P1) to P11 but were not detected at P30

26 neration after myocardial infarction (MI) on postnatal day 1 (P1), but this ability is lost by postna

27 tically tested, including intraperitoneal at postnatal day 1 (P1), intramuscular at P12, and retro-or

28 hen treated during a critical period between postnatal day 1 and 5, respectively.

29 ried out RNA-seq on 20,424 single cells from postnatal day 1 mouse kidneys, comparing the results of

30 cord was normal in presymptomatic SMA mice (postnatal day 1), but failed to match subsequent postnat

31 We focused our analyses on postnatal day 1, a late stage of cortical neurogenesis w

32 s are smaller and developmentally delayed by postnatal day 1, and have about half the number of glome

33 Male and female mice were infected on postnatal day 1, considered comparable with exposure lat

34 applied chronically, or as a single bolus at postnatal day 1, markedly worsened AAA outcomes in XY in

35 creased within 10 min after oral delivery in postnatal day 1-7 mice.

36 rm single cell RNA analyses of mouse lung on postnatal day 1.

37 after 15 days of treatment that started from postnatal day 1.

38 tnatal maternal separation (180 min/day from postnatal day 1: MS180) whilst control pups remained unh

39 Carotid arteries were isolated from newborn (postnatal day 1; P1), postnatal day 7 (P7) and postnatal

40 S, the limited bedding paradigm (LB) between postnatal days 1-10, we previously documented that LB ma

41 ed with profound ocular anomalies evident by postnatal days 1-4, including severe cryptophthalmos, mi

42 Ultrastructure of postnatal day 10 (P10) central (ret)Arl13b(-/-) photorec

43 NG2(+) progenitors similar to VSMCs prior to postnatal day 10 (P10), and from a previously unknown Pa

44 /-)x GalNAc-T (-/-) mice develop normally to postnatal day 10 (P10), but all die between P20 and P25,

45 y of Agrp neurons during breastfeeding using postnatal day 10 mice.

46 Mice were exposed to HI on postnatal day 10 using unilateral carotid ligation and g

47 [I:C] to dam at gestational day 12.5), HI at postnatal day 10, and the combined MIA/HI insult in muri

48 results in mice displaying rapid tremors at postnatal day 10, followed by death at postnatal week 3.

49 elevated hypothalamic ER stress as early as postnatal day 10, i.e., prior to the development of obes

50 lso had abnormal ultrasonic vocalizations at postnatal days 10 to 12 and impaired social behavior at

51 ological methods in mouse organs of Corti at postnatal day 11 (P11)-P13, hearing onset in altricial r

52 r to identify the miRNAs in adult and young (postnatal day 11/12) Muller glia of the neural retina, w

53 outcomes between control and PNE rat pups at postnatal days 11-14: 1) the cardiorespiratory responses

54 inctive laminar distribution was observed by postnatal day 12 (P12), when we first identified ChCs by

55 ongly upregulated in the cochlea from around postnatal day 12 (P12), which corresponds to the onset o

56 ese receptors before the onset of hearing at postnatal day 12 (P12).

57 IHCs, which normally occurs at approximately postnatal day 12, was partially prevented.

58 ion of IHCs, which normally occurs at around postnatal day 12, was partially prevented.

59 Disruption of cilia function before postnatal day 12-14 results in rapid cyst formation; how

60 At postnatal days 12 and 18, mild lung disease was evident

61 yer 5 of the developing mouse visual cortex (postnatal days 12-21).

62 -specific disruption of Ezh1 and/or Ezh2, at postnatal day 14 and 2 months after birth.

63 p2(ZFN/+ )rats were significantly reduced by postnatal day 14 and 21, respectively.

64 l window, ranging from embryonic day 18.5 to postnatal day 14 in mice, in which the maturation proces

65 Intranasal inoculation in postnatal day 14 mice with VSVDeltaG-CHIKV or VLV evoked

66 ly 70-fold increase in retrotransposition in postnatal day 14 Mov10l1(-/-) germ cells compared with t

67 rentiate prematurely and to express genes at postnatal day 14 that would normally be induced by month

68 in hepatocytes during liver maturation from postnatal day 14 to month 2 after birth.

69 In neonatal transgenic mice, up to postnatal day 14, a similar increase in steady-state IL-

70 ower when cilia dysfunction is induced after postnatal day 14.

71 mined that the critical period closes around postnatal day 14.

72 selective breeding (postnatal day 7: n = 22; postnatal day 14: n = 49; postnatal day 21: n = 21; adul

73 imulation in young Fmr1 knock-out (KO) mice (postnatal days 14-16), a model of FXS.

74 ess this question, we analyzed synapses from postnatal day 15 (P15) and adult rat hippocampus that ha

75 l stimulation in rat hippocampal area CA1 at postnatal day 15 (P15).

76 anide treatment restored network activity by postnatal day 15 but failed to restore social behavior b

77 Hypothalamic gene expression profiling in postnatal day 15 F2 descendants on the paternal lineage

78 On postnatal day 15 pups were sacrificed and skulls underwe

79 w an abrupt onset of spontaneous seizures at postnatal day 15 recapitulating human EIEE.

80 Immature (postnatal day 15) but not juvenile (postnatal day 30) KC

81 st of cardiac myocyte cell cycle activity at postnatal day 15.

82 pression of both NEEP21/Nsg1 and P19/Nsg2 at postnatal day 16 as well as in the CA1-3 regions of the

83 However, by postnatal day 16, mouse MG lose neurogenic capacity, des

84 Postnatal day 17, 25, 35, 49, and 70 were examined to co

85 On postnatal day 17, eyeballs were enucleated.

86 This dWAT phenotype was established around postnatal day 18 and did not depend on the hair growth c

87 At postnatal day 180 (P180), FF and S NMJs of SOD1 already

88 ckout (KO) mice develop hair cell defects by postnatal day 2 (P2) and are deaf by P21-P25.

89 of Ube3a sense and antisense transcripts by postnatal day 2 (P2) in hypothalamus and day 9 (P9) in c

90 By postnatal day 2 (P2), SCN oscillators displayed the dail

91 mice, in which Tshz3 is deleted in CPNs from postnatal day 2 to 3 onward.

92 ered by intracerebroventricular injection at postnatal day 2, followed in some cases by stereotactic

93 On Postnatal Day 2, polyethylenimine-(5) myristic acid/poly

94 L2 was detected by immunohistochemistry from postnatal day 2.5 (P2.5) throughout adulthood.

95 ue, and/or if urine output was <1 mL/kg/h on postnatal days 2 to 7.

96 ly maternal separation (MS) of rat pups from postnatal days 2-10 (PND2-10) on neurobehavioural respon

97 Following limited bedding at postnatal days 2-9, adult (postnatal day 70) cerebellar

98 discrete window in development, beginning at postnatal day 20 in mice.

99 Synaptic transmission in the LSO matures by postnatal day 20, with EPSCs and IPSCs having fast kinet

100 embryonic day 9.5 (primordial heart tube) to postnatal day 21 (mature heart).

101 stnatal day 1; P1), postnatal day 7 (P7) and postnatal day 21 (P21) mice and assessed in a pressure m

102 are evident in arg(-/-) animals as early as postnatal day 21 (P21), a time that precedes any observa

103 ally in Tiam1 KO mice, resembling WT mice at postnatal day 21 (P21), but fail to stabilize, leading t

104 ued by pancellular reinstatement of Ube3a at postnatal day 21 (P21), but not during adulthood.

105 trols, which consisted of 5-6 cell layers at postnatal day 21 (P21), the mutant corneal epithelium co

106 arbofuran exposure from gestational day 7 to postnatal day 21 altered expression of genes and transcr

107 Male offspring were weaned at postnatal Day 21 and then fed a HFD for 9 weeks.

108 tigated if acidified water administered from postnatal day 21 has therapeutic benefits in Cln3(-/-) m

109 SC frequency compared with wild-type SPNs in postnatal day 21 mice.

110 tion in the width of the dendritic spines in Postnatal day 21 to 12-month-old LD animals.

111 ontrolled cortical impact or sham surgery at postnatal day 21, approximating a toddler-aged child.

112 ring gestation and lactation; once weaned at postnatal day 21, Gen-1 mice were then kept on the contr

113 phaly and corpus callosum deficiency, and by postnatal Day 21, microcephaly; the mice died at an earl

114 Upon tamoxifen administration at postnatal day 21, the floxed mHtt-exon1 was removed and

115 /y)) were noticeably symptomatic as early as postnatal day 21, with most dying by postnatal day 55, w

116 electrophysiological effects of prepubertal (postnatal day 21-40) EE on DA neurons, pyramidal neurons

117 e detected in the liver of recipient rats at postnatal day 21.

118 arameters, and resulted in shorter femurs on postnatal day 21.

119 ftment was immunohistochemically analyzed on postnatal day 21.

120 seizures, muscle stiffness, and morbidity by postnatal day 21.

121 cope, a total of 685,673 cells in 56 mice at postnatal day 21.

122 orated all morphological defects in liver by postnatal day 21.

123 ce and controls during development and up to postnatal day 21.

124 d have about half the number of glomeruli at postnatal day 21.

125 tal day 7: n = 22; postnatal day 14: n = 49; postnatal day 21: n = 21; adult: n = 46; all male).

126 xoneme and reduced the CC length as early as postnatal day 22 (P22).

127 s in vitro in the left and right amygdala of postnatal days 22-28 male and female offspring from norm

128 that starts prenatally and continues through postnatal day 25 has a major impact on the structure of

129 HC during an early-middle adolescent window (postnatal days 27-45) in which the brain may be particul

130 s in PV neurons at the critical period peak, postnatal day 28 (P28) after monocular deprivation and d

131 apid cementum growth window from the ages of postnatal day 28 (P28) to P56.

132 ictable stress (CUS) for 12 days starting at postnatal day 28 (PND28).

133 mice resulted in onset of proteinuria around postnatal day 28, accompanied by foot process effacement

134 loses its neurotrophic potential at or near postnatal day 28.

135 on/off) or intermittent normal saline during postnatal days 28 to 41 and allowed to grow to adulthood

136 doses (2.5-10 mg/kg/ip) through adolescence (postnatal day 29-43).

137 results demonstrate that hindpaw incision at postnatal day 3 (P3) significantly decreased the strengt

138 tion End Products (RAGE) knockout mice after postnatal day 3, an identical OT increase was not observ

139 t observed at the onset of alveologenesis at postnatal day 3.

140 ative effects on ultrasonic vocalizations at postnatal day 3.

141 are transiently recruited to the lung during postnatal days 3-14, which specifically corresponds to t

142 On postnatal day 30 (P30) mutant mice had lower SGN density

143 bryonic day 15.5 fetuses, and persists until postnatal day 30 in cerebellar Purkinje neurons.

144 In cerebellar Purkinje neurons from postnatal day 30 Snord116p-/m+ mice the reduction in neu

145 mmature (postnatal day 15) but not juvenile (postnatal day 30) KCC2(E/+) mice exhibited altered GABAe

146 s prefer to interact with stressed juvenile (postnatal day 30, PN30) conspecifics but avoid stressed

147 stem are linked with high ABP in both young (postnatal day 30-58) and adult SHRs (4-6 months).

148 the high ABP are measureable in young SHRs (postnatal day 30-58) and become greater in adult SHRs.

149 in dynamic environments in rats as young as postnatal day 30.

150 ng acuity in treated animals is unaltered at postnatal day 30.

151 d in some cases by stereotactic injection at postnatal day 30.

152 hock/restraint stress in either adolescence (postnatal day 31-40) or adulthood (postnatal day 65-74).

153 pre- and perinatally, but is not detected at postnatal day 35 (P35).

154 At postnatal day 36-38, the rats were tested in the open fi

155 At postnatal day 4 (P4), conventionalized mice and Bifidoba

156 BM samples were short-term pasteurized from postnatal day 4 to discharge.

157 iet (0.35 mg retinol/kg diet) and treated on postnatal day 4 with an oral dose of either VA (6 mug re

158 We focused on postnatal day 4, shortly after a transient perinatal and

159 ressed in preodontoblast and odontoblasts at postnatal day 4.

160 mas containing GDNF or saline (control) from postnatal days 4 through 8.

161 e;ndufs4(loxP/loxP) retinas commenced around postnatal day 45 (P45) and progressed to loss of two-thi

162 la circuitry and anxiety-related behavior by postnatal day 45 (P45), when AEA levels begin to decreas

163 tal time points between embryonic day 10 and postnatal day 45.

164 us-dependent spatial memory was evaluated on postnatal days 49-60.

165 rgy patterns may be determined early, before postnatal day 5 (P5), and remain largely unaltered by su

166 upted by thoracic spinal cord transection at postnatal day 5 (P5TX).

167 nt with dantrolene in 4L;C* mice starting at postnatal day 5 delayed neurological pathology and prolo

168 the mouse brain between embryonic day 13 and postnatal day 5 in order to identify transcriptional net

169 rimary cultures of GCPs from Jdp2-KO mice at postnatal day 5 were more resistant to apoptosis than GC

170 n of these circuits is abnormal after early (postnatal day 5) removal of descending systems, inducing

171 o C57BL/6J dams from gestational day 11.5 to postnatal day 5.

172 ally formed normally, then degenerated after postnatal day 5; large numbers of vesicles invaded the c

173 supporting presensory spiking formed between postnatal days 5 (P5) and P7, including Ankyrin-G, NaV1.

174 ing positive pressure respiratory support on postnatal days 5 to 14 improves the rate of survival wit

175 Inhaled nitric oxide, initiated at 20 ppm on postnatal days 5 to 14 to high-risk preterm infants and

176 or positive pressure respiratory support on postnatal days 5 to 14.

177 y over 3-fold during a death period spanning postnatal days 5-14.

178 rocnemius and tibialis anterior in mice from postnatal day 55 to 100 and the results obtained were as

179 arly as postnatal day 21, with most dying by postnatal day 55, while females lacking one copy of Mecp

180 In mouse (ret)Arl13b(-/-) central retina at postnatal day 6 (P6) and older, outer segments were abse

181 h of systemic kainic acid administration at postnatal day 6, mRNA levels of Fgf9, Fgf10, Fgfr2c, and

182 cantly lower in the OPN(-) OPN(+/+) group at postnatal days 6 and 8.

183 nally, we identify a developmental window at postnatal Days 6 to 9 when Muller arbors first colonize

184 owever, these responses are diminished after postnatal day-6 (P6), representing a barrier to building

185 etion of Fth in oligodendroglial cells after postnatal day 60 has no effect on myelin production and/

186 Sox10-positive oligodendroglial cells after postnatal day 60 has no effect on myelin production and/

187 Surprisingly, by postnatal day 60 it also caused CA1 histological disorga

188 ays 10 to 12 and impaired social behavior at postnatal day 60.

189 15 but failed to restore social behavior by postnatal day 60.

190 Both male and female (postnatal days 60-90) mice, including 140 D(2)R, 24 D(1)

191 lescence (postnatal day 31-40) or adulthood (postnatal day 65-74).

192 DE10A, PDE11A4 expression begins very low at postnatal day 7 (P7) and dramatically increases until P2

193 neurons projecting to the tooth pulp at both postnatal day 7 (P7) and in the adult.

194 t the basal arbor grew substantially between postnatal day 7 (P7) and P30, undergoing a 45% increase

195 isolated from newborn (postnatal day 1; P1), postnatal day 7 (P7) and postnatal day 21 (P21) mice and

196 We report that the exposure of postnatal day 7 (P7) mice to ethanol generates p25, a CD

197 erving neurovascular integrity, we subjected postnatal day 7 (P7) rats depleted of microglial cells,

198 the mouse lateral superior olive (LSO) from postnatal day 7 (P7) to P96 using voltage-clamp and audi

199 Cs become functionally competent from around postnatal day 7 (P7), before the primary sensory inner h

200 atal day 1 (P1), but this ability is lost by postnatal day 7 (P7).

201 vels were comparable in males and females at postnatal day 7 but were significantly lower in age-matc

202 tly developed model of status epilepticus in postnatal day 7 rat pups that results in widespread neur

203 thy exposing C57BL/6 mice to 75% oxygen from postnatal day 7 to 12.

204 ntified three undifferentiated SG subsets at postnatal day 7, each of which expresses distinct genes,

205 tial gene expression in regenerating (SCI at postnatal Day 7, P7SCI) and nonregenerating (SCI at Day

206 o osteoblasts expressing Col1 and BSP during postnatal day 7-10, when serum levels of thyroid hormone

207 nel beta subunit exhibited NAFLD as early as postnatal day 7.

208 l progenitor cells from embryonic day 9.5 to postnatal day 7.

209 anning 43 generations of selective breeding (postnatal day 7: n = 22; postnatal day 14: n = 49; postn

210 ous injections of 30 mg/kg ketamine (KET) on postnatal days 7, 9, and 11] results in long-lasting alt

211 regions remained stable across development (postnatal days 7-23), there was a developmental emergenc

212 or Scx(flx/flx)] littermates were killed at postnatal days 7-56 (P7-P56).

213 imited bedding at postnatal days 2-9, adult (postnatal day 70) cerebellar and hippocampal endocannabi

214 nied by overt astrogliosis (at approximately postnatal days 70-80).

215 At postnatal day 8 (P8), theta is expressed as brief bursts

216 of dividing cells peaks in the CC lining on postnatal day 8 (P8), with division occurring in 19.2% +

217 he region of mouse first mandibular molar at postnatal day 8 (PN8) induced AI-like pathologies when t

218 ced vulnerability, rat pups were reared from postnatal day 8 (PN8) with a maltreating mother, which p

219 (phosphatase and tensin homolog) protein at postnatal day 8 in mice harboring Nf1 haploinsufficiency

220 c nerve from one hind limb was transected at postnatal day 8 to cause paralysis to that limb.

221 exposed to an Adversity-Scarcity model from postnatal day 8-12, where insufficient bedding for nest

222 pendent endocytosis occurred at calyces from postnatal days 8-15, suggesting its existence before and

223 isingly, the Cre(+) mice became cachectic by postnatal day 80 due to bilateral GCT.

224 ng also received lipopolysaccharide (LPS) on postnatal day 9 to produce postnatal immune activation (

225 ), which simulates a bacterial infection, on postnatal day 9.

226 birth, but they rapidly worsened and died by postnatal day 9.

227 rikingly, rapamycin treatment in the first 8 postnatal days ameliorates the neurological phenotype of

228 scillatory activity was compromised in early postnatal days as was both feedforward and feedback inhi

229 LA-CeA) develops rapidly during the first 10 postnatal days, before external inputs underlying amygda

230 n [born on embryonic day (E) 29; examined on postnatal day (D) 3 and D7] and term-born (born on E32;

231 Lipopolysaccharide-induced inflammation at postnatal day four induced lasting impairments in two di

232 ovement, increase over the course of several postnatal days in altricial animals.

233 tal lipidomic remodeling during the first 60 postnatal days, including progressive accumulation of ch

234 tress (33-35 postnatal days) then SPS (58-60 postnatal days; n = 14), or (2) no adolescent-stress and

235 , or (2) no adolescent-stress and SPS (58-60 postnatal days; n = 14), or (3) unstressed conditions (n

236 developed kernicterus between days 14 and 45 postnatal days of life; peak B(T) >= 30 mg/dL and B(T) /

237 a single dose of 80 mg ENU/kg body weight on postnatal day one.

238 was deleted after Hensen's cell formation at postnatal day (P) 0/P1 and fate-mapping analysis reveale

239 ncRNA expression profiles in mouse hearts at postnatal day (P) 1, P7 and P28 via microarray.

240 array (Quantum Devices, Barneveld, WI) from postnatal day (p) 10 to p25.

241 perinatal ages from embryonic day (E) 17 to postnatal day (P) 11 and found that cell death peaks jus

242 rom body weight gain and NAFLD in adulthood (postnatal day (P) 112).

243 embrane-bound ephrin-B1 in astrocytes during postnatal day (P) 14-28 period would affect synapse form

244 from hippocampal and neocortical slices from postnatal day (P) 2-P15 mice, photostimulation caused de

245 ild-type mice; these changes were present at postnatal day (P) 20 for PV neurons and P40 for WFA/PNN+

246 NEC was induced in mouse pups between postnatal day (P) 5 and 9.

247 ryptophan hydroxylase 2 (Tph2)(+) neurons by postnatal day (P) 5 through P12: approximately one-third

248 aKO kidneys were hypoplastic and not cystic, postnatal day (P) 7 mutants had proximal tubular-derived

249 are prominent in the somatosensory cortex by postnatal day (P) 7.

250 OZ) causes a neuropathy at 90 d after birth [postnatal day (P) 90], with a subsequent spontaneous reg

251 We used cultures of cortical neurons from postnatal day (P)0-P2 golden Syrian hamsters (Mesocricet

252 In acute cerebellar slices from postnatal day (P)12-14 mice, light-evoked EPSCs were lar

253 haride (LPS; 100 mug/kg, i.p.) or vehicle at postnatal day (P)14, and kept until adolescence (P35-P45

254 amidal cortical neurons from male and female postnatal day (P)28 C57BL/6J mice.

255 Hindpaw incision at postnatal day (P)3 significantly diminished total primar

256 tages (embryonic days 14-18) into adulthood [postnatal day (P)48] using electron microscopy and tomog

257 We explanted testis tissue at postnatal day (P)5.5 and cultured it until P11.5.

258 ndoplasmic reticulum was evident as early as postnatal day (P)6.

259 transcriptomic analysis of single cells from postnatal day (P)7 and P30 murine aortic (AoV) and mitra

260 nd subsynaptic distributions at the immature postnatal day (P)7 and the mature (P21) calyx.

261 trovirally birthdated either early-born [EB; postnatal day (P)7] or adult-born (AB; P60) DGCs.

262 xhibited a mild decrease in vascular loss at postnatal day (P)8 compared with age- and strain-matched

263 sparsely populated with GABAergic neurons at postnatal day (P)8.

264 orded from rat VGN somata of either sex aged postnatal day (P)9-P21.

265 bialis anterior (TA) muscles of live mice at postnatal days (P) 1, 7, 14, 21 and 42, respectively.

266 ere exposed to limited nesting material from postnatal days (P) 2-9.

267 periments in acute mouse brainstem slices at postnatal days (P) 4 and 11 during pharmacological block

268 f10) from Fgf10-expressing beta-tanycytes at postnatal days (P)4-5 results in the generation of signi

269 iod for thalamocortical connectivity between postnatal days P12 and P15, during which tone exposure a

270 eletal muscle atrophy, resulting in death by postnatal day P13.

271 Mice were studied from postnatal day (P15) to 28 weeks by spectral domain optic

272 rcise protocol from the 21(st) to the 60(th) postnatal days (P21-P60), and evaluated at 0 (P60), 30 (

273 During the first postnatal week (postnatal days P4-P7), V1 was not visually responsive an

274 ms of germ and granulosa cells from E16.5 to postnatal day (PD) 3 were reported.

275 radigm, was conducted every thirty days from postnatal day (PD) 30 to PD 180.

276 ketamine (20 mg/kg) for 15 consecutive days (Postnatal Day [PD] 35-49).

277 ontrol mouse dams were suckled from birth to postnatal day (PN) 21 on dams fed either a control (20%

278 ose uptake during odor-shock conditioning in postnatal day (PN)14 rat pups showed that maternal prese

279 of WAT progenitors isolated from pups on the postnatal day (PND) 1 and 21.

280 Six female C57BL/6 mice at postnatal day (PND) 10 were administered a single gavage

281 By postnatal day (PND) 12, communities separated based on e

282 Females were bred at postnatal day (PND) 120.

283 nd proteome of vaginal swab samples taken on postnatal day (PND) 2 and 16 in gilts to determine if te

284 a single 4 Gy whole-brain radiation dose on postnatal day (PND) 21 and were randomized to 0.24% Li2C

285 ization and vascularization were analyzed at postnatal day (PND) 21.

286 In the present study, we fed postnatal day (PND) 24 weanling female rats an SPI diet

287 or was studied using the forced-swim test on postnatal day (PND) 25 in rats either weaned on PND 21,

288 b transgenic (Scnn1b-Tg(+)) mice to SHS from postnatal day (PND) 3-21 and lung phenotypes were examin

289 tic minipumps from gestational day 8 through postnatal day (PND)16.

290 r used two exposure regimes, one stopping at postnatal day (PND)21 (stop-dose) the other continuing u

291 fly exposed to 0.1 to 5,000 mug BPA/kg BW on postnatal days (PND) 1, 3, and 5.

292 ally or in a combination (ABX cocktail) from postnatal days (PND) 14 to 21, followed by ad libitum, l

293 ate whether neonatal alcohol exposure during postnatal days (PND) 2-6 in rats (third trimester human

294 Female mice were exposed to GEN on postnatal days (PND)1-5 and uterine tissues collected on

295 ere exposed to normoxia or hyperoxia through postnatal days (PNDs) 1 to 14, and the hyperoxia-exposed

296 At postnatal-day seven, rat pups underwent moderate or seve

297 Outer segments appeared rapidly at postnatal day six and their appearance coincided with a

298 posed to either (1) adolescent-stress (33-35 postnatal days) then SPS (58-60 postnatal days; n = 14),

299 sis of the effect of feeding in the first 14 postnatal days with own mother's milk, with or without h

300 at three developmental ages (15, 30, and 45 postnatal days) with array tomography three-dimensional