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1 f glucose (fetal life) to the use of lipids (postnatal life).
2 yp26b1 in regulating retinoid homeostasis in postnatal life.
3 tions with nonmotor brain regions throughout postnatal life.
4 inuously generated from NG2 cells throughout postnatal life.
5 d, cell-specific ablation of SCs in pre- and postnatal life.
6 enewal of oocyte-containing follicles during postnatal life.
7 s induced during late embryogenesis or early postnatal life.
8 esses that take place during fetal and early postnatal life.
9 ting the amygdala during the first 3 days of postnatal life.
10 re critical for neurotransmission throughout postnatal life.
11 s and contribute to organ homeostasis during postnatal life.
12 ong consequences for metabolic health during postnatal life.
13 e internodes than OLs generated during early postnatal life.
14  during development but rapidly decreases in postnatal life.
15 em is critical for embryonic development and postnatal life.
16 ioral milestones, to the third year of human postnatal life.
17 vailability of energy-dense foods throughout postnatal life.
18 h quantitative and qualitative ways in early postnatal life.
19 f hematopoietic progenitor and stem cells in postnatal life.
20 l-2-associated X)-dependent apoptosis during postnatal life.
21 essential functions throughout gestation and postnatal life.
22  to the maintenance of neuronal viability in postnatal life.
23 ctional maintenance of the epithelium during postnatal life.
24 progenitor cells in embryogenesis and during postnatal life.
25 iods of heightened brain plasticity in early postnatal life.
26 s many as half in a specific period of early postnatal life.
27 velopment, growth and metabolism in pre- and postnatal life.
28 etal development which are reversed in early postnatal life.
29  capacity to renew cardiomyocytes throughout postnatal life.
30  well as in growth and tissue homeostasis in postnatal life.
31 aintenance of the blood-brain barrier during postnatal life.
32 f LCPUFA during the critical first months of postnatal life.
33 n is compatible with development to term and postnatal life.
34 e absence of PIPKIgamma is incompatible with postnatal life.
35 fects brain development during both pre- and postnatal life.
36  and angiogenesis during embryonic and early postnatal life.
37 is become mature and functional during early postnatal life.
38 ibuting to the gammadelta lineage throughout postnatal life.
39  fate in many tissues during development and postnatal life.
40 vival until birth, but it is dispensable for postnatal life.
41 loss of photoreceptors if performed early in postnatal life.
42 ogenesis and during hair follicle cycling in postnatal life.
43 ady committed, either prenatally or early in postnatal life.
44  retina from fetal development through early postnatal life.
45 in the removal of axon branches during early postnatal life.
46 s after injection or on day 15, 30, or 60 of postnatal life.
47  for bone formation during embryogenesis and postnatal life.
48  that myogenin has separate functions during postnatal life.
49 l circuits are shaped by experience in early postnatal life.
50 n from multiple to single innervation during postnatal life.
51 ernal separation during the first 2 weeks of postnatal life.
52 evelopment and its phenotypic maintenance in postnatal life.
53 hermogenesis develops during the 1st week of postnatal life.
54 ate brain development during fetal and early postnatal life.
55 or maintaining tissue homeostasis throughout postnatal life.
56 ic stages and through the first few weeks of postnatal life.
57 stereotyped behaviors that manifest in early postnatal life.
58 t regulates cell fate during development and postnatal life.
59 ponse during the transition from prenatal to postnatal life.
60 embryonic cusp remodeling and continues into postnatal life.
61  for fetal survival but is incompatible with postnatal life.
62 and function are critical in development and postnatal life.
63 e role of the myogenic regulator myogenin in postnatal life.
64 ich is not manifest until the second week of postnatal life.
65 m late embryonic stages through to day 13 of postnatal life.
66 during the later part of gestation and early postnatal life.
67 mental factors during intrauterine and early postnatal life.
68 ic decrease in their expression during early postnatal life.
69 lpha-SKA) that, in mice, occurs during early postnatal life.
70 iss-Webster mice during the first 2 weeks of postnatal life.
71 normally during embryogenesis and survive to postnatal life.
72 oral manner during embryonic development and postnatal life.
73 ontinuously produces male gametes throughout postnatal life.
74  is guided by visual experience during early postnatal life.
75 e formation was significantly reduced during postnatal life.
76  embryos but closes during the first 48 h of postnatal life.
77 ecomes vulnerable to ethanol insult in early postnatal life.
78 nic development and low bone mass throughout postnatal life.
79  in the offspring during gestation and early postnatal life.
80 epigenetic patterning during development and postnatal life.
81 tion at sites of injury and tumorigenesis in postnatal life.
82 ontribute significantly to vasculogenesis in postnatal life.
83 y and physiology over the first few weeks of postnatal life.
84  temporarily deprived of vision during early postnatal life.
85 ory may influence cardiovascular function in postnatal life.
86 and tissues during embryonic development and postnatal life.
87 roliferation observed as early as 6 weeks of postnatal life.
88 mber may be developmentally regulated during postnatal life.
89  occurring during a critical period in early postnatal life.
90  of this gene in maintaining heart rhythm in postnatal life.
91 hylation nor influenced cell survival during postnatal life.
92 ing, postmitotic neurons and persisting into postnatal life.
93 cisions throughout embryonic development and postnatal life.
94 was normal during embryogenesis and in early postnatal life.
95 ted quickly from the brain within 3 weeks of postnatal life.
96 ng one eye during a critical period in early postnatal life.
97 lian gerbils throughout the first 3 weeks of postnatal life.
98 t to marginal contribution in late fetal and postnatal life.
99 al at birth and remained elevated throughout postnatal life.
100 gan development and function during pre- and postnatal life.
101 trioventricular (AV) conduction block during postnatal life.
102 spring birth weight and body weight in early-postnatal life.
103         Vaginal birth prepares the fetus for postnatal life.
104 stem is critical for sustaining prenatal and postnatal life.
105 y, yet few cases are documented in mammalian postnatal life.
106 g-term control of carbohydrate metabolism in postnatal life.
107 man disease, either during development or in postnatal life.
108 ration units, the nephrons, is essential for postnatal life.
109 observed during the critical period in early postnatal life.
110 ully replace fetal chondrocytes during early postnatal life.
111 daptation that occur throughout neonatal and postnatal life.
112  during development, both in utero and early postnatal life.
113 d changes in the transition from prenatal to postnatal life.
114 t (FMR1-KO) mice during the first 5 weeks of postnatal life.
115 verall numbers of precursor cells throughout postnatal life.
116  endocytosed OVA for delayed presentation in postnatal life.
117 rvival and maturation of DA neurons in early postnatal life.
118 lity of lung clocks in priming the fetus for postnatal life.
119  (telogen), and regeneration (anagen) during postnatal life.
120  normally assemble during the first 2-3 y of postnatal life.
121 sis and for cardiac contractility to support postnatal life.
122 de mammalian heart regeneration during early postnatal life.
123 igh fat diet during early development and in postnatal life.
124  of large quantities of embryonic globins in postnatal life.
125 rcuitry is becoming established during early postnatal life.
126 ts observed during the second week of murine postnatal life.
127 normal airway smooth muscle (SM) function in postnatal life.
128      Here, we show that, at the beginning of postnatal life, 0- to 3-d-old neonates reacted to a simu
129 icted period of sensory deprivation in early postnatal life (1) impairs intracortical relay of depriv
130 come in not having experienced adjustment to postnatal life, a potentially important determinant of o
131 elopment is synapse elimination during early postnatal life, a process known to depend on neuronal ac
132  L-type Ca2+ channels and occur during early postnatal life, a time when retinogeniculate connections
133 an neuromuscular junction occur during early postnatal life: acetylcholine receptors (AChRs) disappea
134                                       During postnatal life, activated cells gradually become quiesce
135 dicate that the quality of dietary FA during postnatal life affects the development of the central re
136 ystem to coevolve with the microbiota during postnatal life allows the host and microbiota to coexist
137 sion is not established until early in their postnatal life, AMPARs are expressed early in developmen
138 elial precursors and vascular development in postnatal life and a possible novel therapeutic target.
139 euromodulator during the earliest periods of postnatal life and acts at developing striatal neurons a
140 he developing lung during prenatal and early postnatal life and adult respiratory morbidity or reduce
141 ific cell lineages from NSCs in vivo, during postnatal life and adulthood, as well as in regenerative
142 rain growth in different periods of pre- and postnatal life and cognitive function in 221 9-year-old
143 irth in these infants persists through their postnatal life and contributes to adverse pulmonary outc
144 these processes are rudimentary during early postnatal life and develop only gradually thereafter.
145 e show that MICU1 is vital for adaptation to postnatal life and for tissue repair after injury.
146 y circuits are shaped by experience in early postnatal life and in many brain areas late maturation o
147 of stem cell-driven neurogenesis persists in postnatal life and is reduced in large-brained species.
148 , integrate them into CNS circuitry in early postnatal life and maintain them in adulthood.
149 an survive through embryonic development and postnatal life and MEF cells from the Ppp5-deficient mic
150  at each junction increases rapidly in early postnatal life and more slowly in young adults.
151 sults clarify the complex roles for RIPK1 in postnatal life and provide insights into the regulation
152 dequacies in nutrient intake may extend into postnatal life and such individuals could be at increase
153 essential for the survival of animals during postnatal life and that its ablation leads to distinct b
154 l muscle defects in Myog-deleted mice during postnatal life and the efficient differentiation of cult
155 tress of maternal separation examined during postnatal life and young adulthood exhibited enhanced hi
156 ural stem/progenitor cells, divide late into postnatal life, and can generate both neurons and astroc
157 ogenesis was extended into the third week of postnatal life, and ectopic neurons and progenitors coll
158 es undergoes pronounced alterations in early postnatal life, and environmental cues may be responsibl
159 is the major carrier for circulating IGFs in postnatal life, and has been shown to have IGF-independe
160 VSMCs, is activated from late fetal to early postnatal life, and is required to maintain the morpholo
161  of pulmonary vascular function during early postnatal life, and its production in the pulmonary vasc
162 n a thermocline, as early as the 1st week of postnatal life, and these pups can also produce heat met
163 o suggest that interactions with V1 in early postnatal life are critical for establishing stimulus se
164 d differentiation during embryonic and early postnatal life are prerequisites for selective synaptic
165                                 At 6 days of postnatal life, arterial blood pressure and heart rate w
166 iculus neurons emerge gradually during early postnatal life as a consequence of experience with cross
167  Gata6 alters atrioventricular conduction in postnatal life as assessed by surface and invasive elect
168 ained in these cells into the second week of postnatal life, at which time Prox1 is dynamically down
169  systematically interrelated at the start of postnatal life, before acquisition of language and cultu
170 ents increased considerably throughout early postnatal life beginning shortly after the peak of the c
171                                           In postnatal life, BMP-7 is expressed primarily in the kidn
172     These differences were apparent in early postnatal life but decreased during progression into adu
173 infants demonstrated growth deficit early in postnatal life but had greater percentage body fat at te
174 ation during beta-cell development and early postnatal life but not for maintenance of adult mass.
175  decreases slightly during the first week of postnatal life, but increases between 1 and 6 weeks of a
176 cardiomyocyte proliferation during the early postnatal life, but it is largely ineffective in driving
177 th occurs throughout embryogenesis and early postnatal life, but its foundation is laid in the primit
178 ontinue to arise from Sox9(+) cells in early postnatal life, but no endocrine or acinar cell neogenes
179 whole length from their time of formation to postnatal life, but some have more complex age-dependent
180 ctional maturation during the first month of postnatal life by developing faster response kinetics, h
181 tory cortex (A1) is easily degraded in early postnatal life by raising rat pups in the presence of pu
182                  The newborn heart adapts to postnatal life by shifting from a fetal glycolytic metab
183 tical periods of embryonic, fetal, and early postnatal life can affect the development of body weight
184 thymocytes during human late fetal and early postnatal life can be reproduced in humanized mice, and
185 aling in forebrain excitatory neurons during postnatal life can evoke persistent mood-related behavio
186  view that environmental events during early postnatal life can influence the formation of neural cir
187 uring periods of developmental plasticity in postnatal life, can also 'programme' function.
188 ows that Abeta overexpression, even early in postnatal life, can perturb plasticity in cerebral corte
189                                     In early postnatal life, cardiomyocyte exit from the cell cycle w
190 development of FC during the first months of postnatal life compared with wild-type animals, resultin
191 eripersonal spatial representations in early postnatal life, despite the crucial roles of peripersona
192 x genes at several time points from 1 day of postnatal life (dpn) to 20 dpn.
193                  Sensory experience in early postnatal life, during so-called critical periods, restr
194 ambs of control ewes between days 6 and 9 of postnatal life, earlier than reported in rodents.
195  Monocular deprivation (MD) imposed early in postnatal life elicits profound structural and functiona
196 rt the colonization of marrow space early in postnatal life, expanding progressively and influencing
197 e the association between prenatal and early postnatal life exposure to UFPs and development of child
198 ate retinogeniculostriate pathway throughout postnatal life, extending into adulthood.
199 ctroencephalography in the first 72 hours of postnatal life, focusing on the electrical burst activit
200  samples collected during the first 30 mo of postnatal life from eight pairs of mono- and dizygotic M
201 nvironment during a critical period in early postnatal life has lifelong effects on the structure and
202 , a complete absence of both proteins during postnatal life has little or no effect on the survival a
203 onal status during intrauterine and/or early postnatal life has substantial influence on adult offspr
204                                       In the postnatal life, hematopoietic stem cell (HSC) niches are
205 egulate the maturation of enteric neurons in postnatal life, however, are poorly understood.
206 itional status during intrauterine and early postnatal life impacts the risk of chronic diseases; how
207 on, chronic administration of ghrelin during postnatal life impaired the normal development of ARH pr
208 dergo activity-dependent maturation in early postnatal life in a manner analogous to sensory systems.
209 essive loss of enteric neurons occurs during postnatal life in Hipk2(-/-) mutant mice that preferenti
210 inc intake influences growth in utero and in postnatal life in humans.
211  observed in humans and is incompatible with postnatal life in mice, thus limiting the ability to stu
212  in the cortex during the first few weeks of postnatal life in mice.
213 They also underscore the importance of early postnatal life in the rat, which corresponds to late ges
214 ession of myelin-related proteins throughout postnatal life in the somatosensory (areas 3b/3a/1/2), m
215 tain sequences that confer downregulation in postnatal life in vivo.
216 feration during development and in the early postnatal life; in adult zebrafish, reactivation of this
217 ternal obesity during intrauterine and early postnatal life increases the risk of low bone mass and f
218 mouse brain during embryonic development and postnatal life indicate that it may be involved in the r
219 Rs1 was expressed after the first 4 weeks of postnatal life, indicating an exquisite temporal sensiti
220 ility to anoxic stress at a specific time in postnatal life induced by a partial defect in raphe func
221 e tissue expansion progresses rapidly during postnatal life, influenced by both prenatal maternal fac
222 indings imply that growth in both foetal and postnatal life influences cognitive performance, little
223 any parts of the nervous system during early postnatal life is a competitive process called 'synapse
224 xpenditure in females and suggest that early postnatal life is a critical period during which nutriti
225                                        Early postnatal life is a key time for development of the immu
226  homeostasis during both gestation and early postnatal life is crucial for the development of the fet
227 cation of neuronal architecture during early postnatal life is essential for refining the precision o
228 eptive sensitivity in the first few hours of postnatal life is influenced by birth experience and end
229                           In contrast, early postnatal life is marked by the rapid accumulation of ce
230 GUS transport into brain parenchyma in early postnatal life is mediated by the mannose 6-phosphate/in
231 ut its effect on diet-induced obesity during postnatal life is not known.
232 ts that nutrition during pregnancy and early postnatal life is one of the most important environmenta
233 ific regions of the dorsal horn during early postnatal life is shown to have multiple, deep-rooted un
234 e genome level, the transition from fetal to postnatal life is typified by a reversal of direction, f
235 espite its important role in development and postnatal life, little is known about the signaling path
236                            In utero or early postnatal life (< 7 weeks), before onset of hypertension
237  recurrent, unprovoked seizures during early postnatal life (<P19).
238 adrenal (HPA) axis during prenatal and early postnatal life may explain, in part, the association bet
239 as expression of therapeutic proteins during postnatal life may limit the pathologic consequences and
240 or hypertension; moreover, in utero or early postnatal life may represent a possible therapeutic wind
241                              Stress early in postnatal life may result in long-term memory deficits a
242 ummarize the evidence suggesting that during postnatal life, monocytes can replace resident macrophag
243                                     In early postnatal life, multiple motor axons converge at individ
244              During late embryonic and early postnatal life, neuromuscular junctions undergo synapse
245 he developing embryo but also throughout the postnatal life of mammals from neuronal precursor cells
246 how that a dietary modification in the early postnatal life of the 1-HC female rat sets up a vicious
247 maintained in the skin epithelium throughout postnatal life of the mouse.
248   Exposure to infectious agents during early postnatal life often alters glucocorticoid responses to
249 l fatty acid deficiency during gestation and postnatal life on the development of visual function in
250 rmacological inhibition of S6K1 during early postnatal life or by reducing the activity of mPFC-BLA c
251             B6.K1 mice became heavier during postnatal life (P < 0.05) and had elevated systolic bloo
252 ation of forebrain excitatory neurons during postnatal life (P2-14), but not in juvenile or adult win
253  ethanol inhalation during the first week of postnatal life (P2-P6).
254 ensive brain development occurs during early postnatal life-particularly within the prefrontal cortex
255 stem cell function in utero and during early postnatal life, PAX3/FOXO1A and PAX7/FOXO1A may subvert
256                   Visual experience in early postnatal life plays a necessary and unique role in the
257 lly to 0, 25 or 50 mg Mn/kg/day during early postnatal life (PND 1-21) or throughout life from PND 1
258 rance toward alloantigens encountered during postnatal life pointing to the existence of a process fo
259 omoting greater neuronal plasticity early in postnatal life preceded the divergence of the human and
260 rt learning and generalization in very early postnatal life, providing evidence that PFC and the fron
261  Stress during the prenatal period and early postnatal life puts male offspring at risk of developing
262  AEP-derived population is eliminated during postnatal life, raising questions about the nature and p
263 conclusion, a GF diet during fetal and early postnatal life reduces the incidence of diabetes.
264 on from the in utero maternal environment to postnatal life requires the activation of thermogenesis
265 kappaB activation in astrocytes during early postnatal life results in hydrocephalus formation and ad
266 situ hybridization, we found that from early postnatal life retinoschisin mRNA is present only in the
267                  Sensory experience in early postnatal life shapes neuronal connections in the brain.
268 ing and grooming (low LG offspring) in early postnatal life show reduced long term potentiation (LTP)
269  mice, mice fed a LOW n-6 or HIGH n-3 during postnatal life showed significant reductions in the dens
270 hat is present in fetal life and persists in postnatal life, suggesting opportunities for early detec
271 hem, are particularly prominent during early postnatal life, suggesting that endogenous CRH may contr
272  these disorders may in fact be treatable in postnatal life, suggests that the scientific community s
273 ore dramatically during the first 5 years of postnatal life than during the entire remaining period.
274 in is strongly influenced by events in early postnatal life that eliminate approximately half of the
275 sine-resistant accessory pathways induced in postnatal life that may rarely disappear later in life.
276              Here, we have shown that during postnatal life, the de novo DNA methyltransferase DNMT3A
277                                     In early postnatal life, the developmental isoforms (embryonic an
278                                           In postnatal life, the function of Hox proteins is less cle
279 at develop throughout embryogenesis and into postnatal life, the generation of differentiated cells t
280 during gametogenesis and through fetal/early-postnatal life, the resultant phenotype is likely the ag
281  deficiency, a disease associated with early postnatal life-threatening hemorrhage.
282 lops early in organogenesis and continues in postnatal life through puberty.
283                                       During postnatal life, thymopoiesis depends on the continuous c
284 al touch and pain processing emerges in late postnatal life to allow flexible and context-dependent b
285 tic interaction in SW mice operates early in postnatal life to influence the expression of GABA(A)R a
286  mammalian heart undergoes maturation during postnatal life to meet the increased functional requirem
287 s switch from strong depression during early postnatal life, to weaker depression and in some cases f
288 n immune development and growth during early postnatal life under different thermal environments.
289 s and interpeak intervals decreased early in postnatal life, values decreased over a delayed and prot
290 essive photoreceptor degeneration throughout postnatal life, via derepression of fetal expression of
291                      Smoke exposure in early postnatal life was also associated with an increased eff
292 t regulates human lymphoid commitment during postnatal life, we used RNA sequencing to assemble the l
293 e directly assess the importance of cilia in postnatal life, we utilized conditional alleles of two c
294                                 During early postnatal life when spontaneous activity of retinal gang
295      Disruption of this pathway during early postnatal life, when olivocochlear axons are forming the
296     Here, we identify a time window in early postnatal life wherein partial amputation culminates in
297  ANG-1 protein levels fall between fetal and postnatal life, while TIE-2 levels increase.
298  cis-regulatory elements are primed in early postnatal life whose functions may be compromised in hum
299  gamma-globin chain production diminishes in postnatal life, with transient production of HbF reticul
300 us system (CNS) germinal zones and, in early postnatal life, within numerous cells throughout the CNS

 
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