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1 different behaviors at the level of a single postsynaptic cell.
2 reams even where they converge onto the same postsynaptic cell.
3 t not by the same inhibitors loaded into the postsynaptic cell.
4 he information that can be communicated to a postsynaptic cell.
5 ion of their receptor fields within a shared postsynaptic cell.
6 e-sensitive K+-Cl- transport activity in the postsynaptic cell.
7 time courses of Ca(2+) introduction into the postsynaptic cell.
8 tes to the collection of signals sent to the postsynaptic cell.
9 ynaptic inputs with action potentials in the postsynaptic cell.
10 he number of motor axons that innervate each postsynaptic cell.
11 ted animals form synapses with more than one postsynaptic cell.
12 synaptic cleft, eliciting a response in the postsynaptic cell.
13 between PKA activation in the presynaptic or postsynaptic cell.
14 potentials or constant depolarization in the postsynaptic cell.
15 ein synthesis in the presynaptic but not the postsynaptic cell.
16 en if the transgene is expressed only in the postsynaptic cell.
17 of synapses that results from injury to the postsynaptic cell.
18 nergic input may trigger axonogenesis in the postsynaptic cell.
19 als from interneuron axons that surround the postsynaptic cell.
20 mental and functional states of the pre- and postsynaptic cell.
21 cells can induce direct photocurrents in the postsynaptic cell.
22 nd vacuolar-type H(+)-ATPase activity in the postsynaptic cell.
23 ation via regulation of Wnt signaling in the postsynaptic cell.
24 fined by the identity of the presynaptic and postsynaptic cell.
25 firing was paired with depolarization of the postsynaptic cell.
26 s insensitive to chelation of calcium in the postsynaptic cell.
27 ct quenching of this phosphoinositide at the postsynaptic cell.
28 ll body, the central nervous system, and the postsynaptic cell.
29 eptors are expressed on both presynaptic and postsynaptic cells.
30 requires proper interaction between pre- and postsynaptic cells.
31 istinct sources of perisomatic inhibition to postsynaptic cells.
32 on regulatory interactions between pre- and postsynaptic cells.
33 of GABA release following depolarization of postsynaptic cells.
34 formation and stabilization with individual postsynaptic cells.
35 and physiological properties of the pre- and postsynaptic cells.
36 Held terminal and compare them with those of postsynaptic cells.
37 by bidirectional signaling between pre- and postsynaptic cells.
38 otentials by hyperpolarizing the membrane of postsynaptic cells.
39 es reciprocal communication between pre- and postsynaptic cells.
40 alignment and attachment of presynaptic and postsynaptic cells.
41 voked IPSCs (eIPSCs) by activating mAChRs on postsynaptic cells.
42 selection between individual presynaptic and postsynaptic cells.
43 s regulated by interactions between pre- and postsynaptic cells.
44 multiple synapses with the same or different postsynaptic cells.
45 ound in both presynaptic nerve terminals and postsynaptic cells.
46 ubcellular structures occur in both pre- and postsynaptic cells.
47 ted to impose a transient temporal filter on postsynaptic cells.
48 may be detected by either neuronal or glial postsynaptic cells.
49 e resting potentials of both presynaptic and postsynaptic cells.
50 determined from random samples of unlabelled postsynaptic cells.
51 ts while completely disconnecting from other postsynaptic cells.
52 rmation exchange between the presynaptic and postsynaptic cells.
53 CN1 knockout mice also show reduced IPSCs in postsynaptic cells.
54 ften release multiple neurotransmitters onto postsynaptic cells.
55 ptic tracers were used to mark terminals and postsynaptic cells.
56 iod of viral replication reduces toxicity to postsynaptic cells.
57 lar synaptomatrix separating presynaptic and postsynaptic cells.
58 of different kinds of inputs onto individual postsynaptic cells.
59 al effects on the excitability of respective postsynaptic cells.
60 ng phase locking between the presynaptic and postsynaptic cells.
61 in leading to transcriptional alterations in postsynaptic cells.
62 unoreactivity (IR) and AP frequency in these postsynaptic cells.
63 ter upon a discrete combination of different postsynaptic cells.
64 ecreted BDNF can act on both presynaptic and postsynaptic cells.
65 resynaptic cell types converge onto a common postsynaptic cell, acting together to shape neuronal out
67 e results indicate that signals initiated by postsynaptic cell adhesion molecule ApCAM coupled with t
68 ere we show that conditional deletion of the postsynaptic cell adhesion molecule neuroligin-3 in parv
69 ts of synapses by binding extracellularly to postsynaptic cell adhesion molecules and intracellularly
71 ene family encodes single-pass transmembrane postsynaptic cell adhesion molecules that are important
74 h repeat transmembrane proteins (LRRTMs) are postsynaptic cell adhesion molecules that bind to presyn
78 anism underlying Cdk5-mediated regulation of postsynaptic cell adhesion molecules.SIGNIFICANCE STATEM
79 ized conditional deletion of neuroligin-2, a postsynaptic cell adhesion protein located at gamma-amin
81 ockout (cKO) mice of neuroligin-2 (Nlgn2), a postsynaptic cell-adhesion molecule of inhibitory synaps
86 ynaptic cell-adhesion molecules that bind to postsynaptic cell-adhesion molecules such as neuroligins
91 Neuroligins are evolutionarily conserved postsynaptic cell-adhesion molecules that function, at l
93 Thus, our data suggest that two unrelated postsynaptic cell-adhesion molecules, LRRTMs and neuroli
96 connections of dopaminergic cells and their postsynaptic cells, AII amacrine and melanopsin-containi
98 nnabinoids (eCBs), which are produced in the postsynaptic cell and act on the presynaptic terminal, a
99 r current, decreasing covariance between the postsynaptic cell and afferents activated by the second
100 ane-N,N,N', N'-tetraacetate (BAPTA) into the postsynaptic cell and is similar to long-term potentiati
101 ncreasing covariance between activity of the postsynaptic cell and its afferents that were activated
102 ter accounting for only 70% of the effect on postsynaptic cell and protons released together with the
103 membrane FasII levels in the presynaptic and postsynaptic cell and requires the presence of the fly h
104 calcium-dependent regulatory protein in the postsynaptic cell and suggests that hemichannels on the
105 arity of the responses in different types of postsynaptic cell and the properties of miniature EPSCs
106 polarization, on the influx of Ca2+ into the postsynaptic cell and, at least in part, on the activati
107 , causing the payload to be endocytosed into postsynaptic cells and delivered to the nucleus, where i
108 ll-cell contact between appropriate pre- and postsynaptic cells and is followed by recruitment of pro
109 tional precision-between genetically defined postsynaptic cells and neurotransmitter-defined presynap
110 fusible lipophilic molecules are released by postsynaptic cells and regulate presynaptic neurotransmi
111 Thus, synaptic learning rules vary with the postsynaptic cell, and may require the interaction of di
112 of Agrn mutant mice, demonstrating that the postsynaptic cell, and MuSK in particular, has a potent
113 eptors is homosynaptically controlled by the postsynaptic cell, and that it is not due to constitutiv
114 These results indicate that the activity of postsynaptic cells, and the activation of NMDA receptors
115 hether GABA diffuses from MOC axons to other postsynaptic cells, and the location and function of GAB
116 rocess require interactions between pre- and postsynaptic cells, and which proceed cell-autonomously.
117 ogether, endogenous PSD-95 and SAP102 in the postsynaptic cell appear to regulate transcellularly the
118 ar junction, receptors expressed in a single postsynaptic cell are confronted with an array of hundre
119 e, the integration time and threshold of the postsynaptic cell are matched to the statistics of conve
120 wo synapses sharing the same presynaptic and postsynaptic cells are known to be correlated in size.
122 on of LTP required correlated spiking of the postsynaptic cell as well as the activation of the NMDA
125 er in presynaptic or in both presynaptic and postsynaptic cells at early developmental or postdevelop
126 dings are one of the first demonstrations of postsynaptic, cell-autonomous actions of endocannabinoid
128 t the adhesive mechanisms that link pre- and postsynaptic cells before synapse formation may be diffe
129 ampal cultures, deletion of TrkB in only the postsynaptic cell, before synapse formation, also result
135 m stereotypic connections with an individual postsynaptic cell, but how a single presynaptic cell typ
136 acterized for its function in activating the postsynaptic cell, but the significance of spontaneous r
137 a noncanonical Wnt signaling pathway in the postsynaptic cell by modulating the internalization of t
140 e contact a common interneuron partner, each postsynaptic cell can arrive at a different connectivity
141 la glutamatergic neuromuscular junction, the postsynaptic cell can regulate synaptic strength by both
143 ating individual inputs is difficult because postsynaptic cells can receive thousands of inputs.
145 between neurons, whereas presynaptic versus postsynaptic cell classes dictate the connectivity, effi
146 cause it contains a small number of pre- and postsynaptic cells connected in an invariant pattern.
147 sence of tetrodotoxin, depolarization of the postsynaptic cell consistently produced a broadening of
148 ctions use glutamate as transmitter, and the postsynaptic cells contain both NMDA and AMPA receptors.
150 of BCs along the soma-dendritic axes of the postsynaptic cell could enhance directional tuning at th
152 o the nucleus of the presynaptic but not the postsynaptic cell during 5-HT-induced long-term facilita
155 zed "detonator" synapses that potently drive postsynaptic cell firing through their profound frequenc
156 across dendritic compartments, and (2) that postsynaptic cell-firing is the critical trigger for ind
157 we hypothesized that receptor expression in postsynaptic cells follows changes in transmitter expres
158 eus (PrV), trigeminal afferent terminals and postsynaptic cells form discrete modules ("barrelettes")
159 encing of ZIP3, but not ZIP1, can rescue the postsynaptic cells from kainate-induced neurodegeneratio
162 ich afferent input regulates the survival of postsynaptic cells have received considerably less atten
167 an unexpected nerve-independent role for the postsynaptic cell in generating this topological complex
168 cal antagonists to either the presynaptic or postsynaptic cell in paired whole-cell recordings from h
171 through the release of endocannabinoids from postsynaptic cells in a manner that could not be blocked
174 he dynamic cell surface remodeling needed by postsynaptic cells in coordinating synaptogenesis initia
175 tions of several molecules on the surface of postsynaptic cells in order to choose a particular targe
178 e, but the relative roles of presynaptic and postsynaptic cells in these changes are only beginning t
179 mination in vivo and asked whether the major postsynaptic cells in this circuit, the ganglion cells,
180 we inhibited protein synthesis in individual postsynaptic cells in vivo while monitoring presynaptic
182 ents leading to substrate/Na+ release to the postsynaptic cell, including the structure and dynamics
183 uronal input can lead to profound changes in postsynaptic cells, including atrophy and cell death.
184 elator BAPTA (10 mm) was introduced into the postsynaptic cell, indicating that the tonic inhibition
185 ntral synapses, endocannabinoids released by postsynaptic cells inhibit neurotransmitter release by a
187 The model accounts for LTP and LTD when the postsynaptic cell is voltage clamped and depolarized (LT
188 s to the overall synaptic activity of single postsynaptic cells is essential to our understanding of
189 aptic depression and facilitation, or at the postsynaptic cell level because of subthreshold membrane
190 ssibility that membrane fusion events in the postsynaptic cell may be required for the change in syna
191 d NO donors, suggested that NO released from postsynaptic cells mediated FSI and likely activated pre
192 ntegrin And Metalloproteinase (ADAM) 22, the postsynaptic cell membrane receptor for the glycoprotein
193 synaptic neuron with a depolarization of the postsynaptic cell mimicked the decrease of unitary IPSCs
194 s, excitatory inputs may alter the firing of postsynaptic cells more effectively than inhibitory inpu
195 e activity of presynaptic cells according to postsynaptic cell outputs and to maintain synaptic funct
196 Moreover, infusing a PKA inhibitor into postsynaptic cells produced synaptic depression that occ
198 synaptic junctions on individual, identified postsynaptic cells reflected the overall postsynaptic ta
199 signaling is an important mechanism by which postsynaptic cells regulate the structure and function o
200 the reciprocal interactions between pre- and postsynaptic cells required for the development of matur
201 ise recordings from the CiA interneurons and postsynaptic cells reveal that the Engrailed-1 neurons p
202 ss-of-function experiments in either ChCs or postsynaptic cells revealed that IgSF11 is required for
203 P was prevented by with voltage clamping the postsynaptic cell soma during high-frequency stimulation
204 t of a synapse-specific Hebbian factor and a postsynaptic-cell-specific homeostatic factor, with each
205 namics of excitatory synapses align with the postsynaptic cell subclass, whereas inhibitory synapse d
206 nd those that shared a common presynaptic or postsynaptic cell, suggesting local perisynaptic influen
207 olished after dialyzing 40 mm BAPTA into the postsynaptic cell, suggesting that DHPG activated postsy
209 molecular epitope spreading between GluR3, a postsynaptic cell surface protein, and munc-18, a presyn
211 nce of cell bodies from both presynaptic and postsynaptic cells, synaptic efficacy increased for 48 h
214 functional neurotransmitter receptors on the postsynaptic cell that is regulated by interaction with
215 ry machinery is a retrograde signal from the postsynaptic cell that mediates the formation of synapti
216 sed to mediate interactions between pre- and postsynaptic cells that are necessary for synapse format
217 investigated GABAA receptor localization in postsynaptic cells that fail to receive presynaptic cont
219 homeostatic potentiation of inhibition onto postsynaptic cells that show increased levels of excitat
220 h different cells (e.g. myelinating glia and postsynaptic cells), the recruitment and retention of AI
221 in synaptic excitation and inhibition in all postsynaptic cells, the ratio of these two components is
222 Synaptotagmin 4 (Syt4) is transmitted to the postsynaptic cell through anterograde delivery of Syt4 v
223 r clusters but acts transcellularly from the postsynaptic cell through N-cadherin to enhance asynchro
224 By comparing biophysical maturation of the postsynaptic cell to alterations in presynaptic organiza
225 spike frequencies are more likely to cause a postsynaptic cell to fire than are bursts with higher or
227 0-Hz stimulation or by depolarization of the postsynaptic cell to prevent block of NMDA-specific glut
228 w a novel form of plasticity that allows the postsynaptic cell to selectively modulate spontaneous ne
229 f a barcoded rabies virus from its "starter" postsynaptic cell to that cell's presynaptic partners.
230 hich it is released, and the response of the postsynaptic cell to that transmitter all contribute to
231 nections requires a dialogue between pre and postsynaptic cells to coordinate the assembly of the pre
232 ation requires interactions between pre- and postsynaptic cells to establish the connection of a pres
233 uced by synaptic activities are derived from postsynaptic cells to potentiate presynaptic properties,
234 s retrograde messengers that are released by postsynaptic cells to regulate neurotransmitter release
235 can act as retrograde messengers that allow postsynaptic cells to regulate the strength of their syn
236 ons and interactions between presynaptic and postsynaptic cells together promote the segregation of c
237 ment, neuromuscular junctions and some other postsynaptic cells transition from multiple- to single-i
238 ibition was also found to be brain state and postsynaptic cell type dependent but that alone could no
240 precise spike timing, synaptic strength, and postsynaptic cell type in the activity-induced modificat
241 ing the identity of both the presynaptic and postsynaptic cell type is key when analyzing neocortical
242 y fiber inputs to CA3 GABAergic cells on the postsynaptic cell type was correlated with the frequency
243 unique, a function of presynaptic cell type, postsynaptic cell type, environment, developmental stage
246 ble for neurotransmitter release but plays a postsynaptic, cell type-specific role in cerebellar Purk
247 form distinct wiring patterns with multiple postsynaptic cell types during development remains unexp
249 the division of common inputs among multiple postsynaptic cell types to create parallel circuits with
250 individual presynaptic cells contact several postsynaptic cell types, generating divergence of signal
253 orchestrated communication between pre- and postsynaptic cells via coordinated trans-synaptic signal
254 l role in conferring presynaptic patterns to postsynaptic cells via neurotransmitter receptor-mediate
255 e simply a transient influx of Ca2+ into the postsynaptic cell, via either NMDA receptors or voltage-
257 regulated by neurotrophins secreted from the postsynaptic cell was examined in Xenopus nerve-muscle c
258 ed Ca2+ chelators nitr-5 or nitrophen in the postsynaptic cell was sufficient to induce persistent sy
259 the presynaptic cell evoked responses in the postsynaptic cell when presynaptic firing was paired wit
260 take were surprisingly resilient compared to postsynaptic cells, which overall were most vulnerable t
261 We show that ZIP1 mediates zinc influx into postsynaptic cells, while ZIP3 is responsible for zinc r
262 ly be assigned to particular presynaptic and postsynaptic cells without specialized labeling methods.