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1 may lead to potentially fatal EBV-associated posttransplant lymphoproliferative disease.
2 D4+ T cells reflects HVL patients at risk of posttransplant lymphoproliferative disease.
3 One EVR/rTAC patient developed posttransplant lymphoproliferative disease.
4 We had no cases of posttransplant lymphoproliferative disease.
5 Burkitt's lymphoma, Hodgkin's lymphoma, and posttransplant lymphoproliferative disease.
6 ew the clinical and pathological spectrum of posttransplant lymphoproliferative disease.
7 as not been previously reported as a form of posttransplant lymphoproliferative disease.
8 ed with an increased risk for development of posttransplant lymphoproliferative disease.
9 One patient in each group developed posttransplant lymphoproliferative disease.
10 patients developed hematologic malignancies (posttransplant lymphoproliferative diseases, 18; Hodgkin
11 allogeneic CTL lines to treat EBV-associated posttransplant lymphoproliferative disease, a response r
13 ve Hodgkin disease but not in EBV-associated posttransplant lymphoproliferative disease and Hodgkin d
14 e first steps of in vivo immune control over posttransplant lymphoproliferative disease and lymphomas
15 tive for Epstein-Barr virus (EBV)-associated posttransplant lymphoproliferative disease and melanoma.
16 led lesions of Epstein-Barr virus-associated posttransplant lymphoproliferative disease and was chara
17 transplantation, (vii) graft disease, (viii) posttransplant lymphoproliferative disease, and (ix) sto
18 re (death or retransplant), acute rejection, posttransplant lymphoproliferative disease, and cardiac
19 cidence of reoperation, vascular thrombosis, posttransplant lymphoproliferative disease, and estimate
20 nt diabetes; 0.2%, avascular necrosis; 0.2%, posttransplant lymphoproliferative disease; and 0%, poly
21 nd reduce the risk of adverse events such as posttransplant lymphoproliferative disease are discussed
22 sia (ALH), that is pathologically similar to posttransplant lymphoproliferative disease associated wi
25 er LT for advanced-stage PSC was 18.7%, with posttransplant lymphoproliferative diseases, colorectal
26 associated with an increased risk of CMV or posttransplant lymphoproliferative disease compared with
30 virus disease, graft-versus-host disease, or posttransplant lymphoproliferative disease has been obse
31 ic HCC and other tumors and various forms of posttransplant lymphoproliferative disease have occurred
32 Nonmelanoma skin cancer, Kaposi sarcoma, and posttransplant lymphoproliferative disease have standard
33 BV), as is demonstrated by the occurrence of posttransplant lymphoproliferative disease in immunosupp
34 ed with several human malignancies including posttransplant lymphoproliferative disease in immunosupp
36 ased risk of Epstein-Barr virus (EBV)-driven posttransplant lymphoproliferative disease, is an import
37 se (focal segmental glomerulosclerosis), and posttransplant lymphoproliferative disease may lead to t
38 pisodes of allograft rejection, incidence of posttransplant lymphoproliferative disease or coronary a
39 te with clinical confounders or a history of posttransplant lymphoproliferative disease or Epstein-Ba
40 Ten patients had no previous diagnosis of posttransplant lymphoproliferative disease (PT-LPD), whi
43 is considered to be a major risk factor for posttransplant lymphoproliferative disease (PTLD) and AI
44 t survival, episodes of rejection as well as posttransplant lymphoproliferative disease (PTLD) and gr
46 ays an important role in the pathogenesis of posttransplant lymphoproliferative disease (PTLD) by per
47 EBV) DNA titers more than 1000 copies/mL and posttransplant lymphoproliferative disease (PTLD) develo
48 non-White ethnicity (p = 0.014) and previous posttransplant lymphoproliferative disease (PTLD) diagno
49 as to determine the incidence and hazard for posttransplant lymphoproliferative disease (PTLD) in a s
50 have previously reported a 10% incidence of posttransplant lymphoproliferative disease (PTLD) in ped
58 nosis of Epstein-Barr Virus (EBV)-associated posttransplant lymphoproliferative disease (PTLD) is imp
59 the subsequent development of EBV-associated posttransplant lymphoproliferative disease (PTLD) is the
63 The incidence, risk factors, and outcome of posttransplant lymphoproliferative disease (PTLD) were e
64 Epstein-Barr virus (EBV) is associated with posttransplant lymphoproliferative disease (PTLD), and E
65 gnancies, including Burkitt's lymphoma (BL), posttransplant lymphoproliferative disease (PTLD), nasop
66 EBV type II and III latency tumors, such as posttransplant lymphoproliferative disease (PTLD), on EB
67 n increased risk of cytomegalovirus (CMV) or posttransplant lymphoproliferative disease (PTLD), we ex
68 s been linked to several diseases, including posttransplant lymphoproliferative disease (PTLD), which
80 ad, EBV serology, and EBV-related morbidity (posttransplant lymphoproliferative disease [PTLD] or sym
81 virus, BK virus, and Epstein-Barr virus) and posttransplant lymphoproliferative disease remained low.
82 gnized cytotoxic T-lymphocyte epitopes in 25 posttransplant lymphoproliferative disease specimens fro
83 antation (OLT) one of the patients developed posttransplant lymphoproliferative disease, successfully
85 e: unknown (seven), poor compliance (three), posttransplant lymphoproliferative disease, (two), hepat
91 (five Hodgkin's-like and one Burkitt's-like posttransplant lymphoproliferative disease) with restric
92 graft disease, (viii) chronic illness, (ix) posttransplant lymphoproliferative disease, (x) intracta