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1 of ICU admission was found as performant as posttraumatic amnesia (area under the curve, 0.81; diffe
2 at ICU admission (phase 1) and compared with posttraumatic amnesia duration and the initial Glasgow C
3 s of ICU admission could be as performant as posttraumatic amnesia for predicting traumatic brain inj
9 xiety, panic, phobia, obsession, compulsion, posttraumatic, care management, case management, collabo
10 e of Coping Status-A), posttraumatic stress (Posttraumatic Checklist-Specific Stressor), anxiety (Hos
18 with mild traumatic brain injury (mTBI) with posttraumatic migraines (PTMs) and those without PTMs on
20 posure, a substantial subset develop adverse posttraumatic neuropsychiatric sequelae (APNS) such as p
27 udies that both evaluate a broad spectrum of posttraumatic sequelae (to overcome fragmentation) and a
28 nce strongly affects treatment approaches in posttraumatic spectrum disorders, their etiology remains
29 re also significant differences in scores of posttraumatic stress (AMD, -5.86; 95% CI, -8.53 to -3.19
30 y Adjustment Schedule [WHODAS]), symptoms of posttraumatic stress (measured by the Posttraumatic Stre
31 vised), coping (Measure of Coping Status-A), posttraumatic stress (Posttraumatic Checklist-Specific S
32 gnificant levels of depression, anxiety, and posttraumatic stress among patients and levels of burden
33 ttle is understood about how the symptoms of posttraumatic stress develop over time into the syndrome
34 conferences and showed reduced prevalence of posttraumatic stress disorder (45% vs 69%; p = 0.01), an
35 ence letters may even increase prevalence of posttraumatic stress disorder (52.4% vs 37.1%; p = 0.03)
36 ich remained significant after adjusting for posttraumatic stress disorder (adjusted RR = 1.34, 95% c
37 roup analysis revealed a lower prevalence of posttraumatic stress disorder (odds ratio, 0.23; 95% CI,
38 02), bipolar I (OR, 1.5; 95% CI, 1.06-2.05), posttraumatic stress disorder (OR, 1.6; 95% CI, 1.27-2.1
39 ant-reported MVC characteristics with 8-week posttraumatic stress disorder (PTSD) adjusting for pre-M
40 s the prevalence of anxiety, depression, and posttraumatic stress disorder (PTSD) after injury and th
41 earlier trauma exposure is known to predict posttraumatic stress disorder (PTSD) after subsequent tr
42 f exposure to stressful experiences, such as posttraumatic stress disorder (PTSD) and depression, are
43 ales disproportionately have higher rates of posttraumatic stress disorder (PTSD) and experience grea
44 ibitory avoidance-based "2-hit" rat model of posttraumatic stress disorder (PTSD) and identified pred
45 orders with hyperarousal symptoms, including posttraumatic stress disorder (PTSD) and major depressio
46 ticipants (N=558) were assessed for probable posttraumatic stress disorder (PTSD) and probable depres
47 ticipants (N=558) were assessed for probable posttraumatic stress disorder (PTSD) and probable depres
48 associated memories is relevant for treating posttraumatic stress disorder (PTSD) and related syndrom
49 ical systems are altered in association with posttraumatic stress disorder (PTSD) and resilience.
50 ration (FDA) as "breakthrough therapies" for posttraumatic stress disorder (PTSD) and treatment-resis
51 s), obsessive-compulsive disorder (OCD), and posttraumatic stress disorder (PTSD) are common mental d
52 e biomarkers of the presence and severity of posttraumatic stress disorder (PTSD) are elusive, yet ba
54 d was a significant risk for a high level of posttraumatic stress disorder (PTSD) arousal symptoms.
57 lex traumatic events are at risk of not only posttraumatic stress disorder (PTSD) but also other ment
58 e resilient to psychiatric disorders such as posttraumatic stress disorder (PTSD) compared with those
61 The limited neurobiological understanding of posttraumatic stress disorder (PTSD) has been partially
63 ncreasing predictability of animal models of posttraumatic stress disorder (PTSD) has required active
64 or (alpha1AR) antagonist prazosin for combat posttraumatic stress disorder (PTSD) in 67 active duty s
66 lable from medical dispensaries for treating posttraumatic stress disorder (PTSD) in many states of t
67 the neurobiological mechanisms that predict posttraumatic stress disorder (PTSD) in recent trauma su
68 re to stress is a risk factor for developing posttraumatic stress disorder (PTSD) in response to trau
69 al blood during the onset and development of posttraumatic stress disorder (PTSD) indicate widespread
81 econsolidation marker.SIGNIFICANCE STATEMENT Posttraumatic stress disorder (PTSD) is characterized by
87 ived from genome-wide association studies of posttraumatic stress disorder (PTSD) may inform risk for
88 interactions between smoking and symptoms of posttraumatic stress disorder (PTSD) on pain intensity,
89 Medication and psychotherapy treatments for posttraumatic stress disorder (PTSD) provide insufficien
92 nced trauma and associated distress-that is, posttraumatic stress disorder (PTSD) severity-more than
94 dicate a prevalence for clinically important posttraumatic stress disorder (PTSD) symptoms of 25%.
96 in white matter organization in people with posttraumatic stress disorder (PTSD) using diffusion MRI
98 s process a potential therapeutic target for posttraumatic stress disorder (PTSD), a mental illness c
99 y of daily living, 20% screened positive for posttraumatic stress disorder (PTSD), all SF-12 physical
100 of NPY with trauma-evoked syndromes such as posttraumatic stress disorder (PTSD), although the exact
102 ent among veterans, especially veterans with posttraumatic stress disorder (PTSD), and poses a major
103 sychiatric disorders, such as depression and posttraumatic stress disorder (PTSD), are inadequate.
104 posure therapy is an effective treatment for posttraumatic stress disorder (PTSD), but a comprehensiv
105 smaller hippocampal and amygdala volumes in posttraumatic stress disorder (PTSD), but findings have
106 psychotherapy is a first-line treatment for posttraumatic stress disorder (PTSD), but its mechanisms
107 posure therapy is an effective treatment for posttraumatic stress disorder (PTSD), but many patients
108 to underlie the pathophysiology of pediatric posttraumatic stress disorder (PTSD), but the few studie
109 al patterns of spontaneous brain activity in posttraumatic stress disorder (PTSD), but the findings a
110 r (GR) sensitivity is present in people with posttraumatic stress disorder (PTSD), but the molecular
111 hippocampal volume is frequently observed in posttraumatic stress disorder (PTSD), but the psychologi
112 n array of pathological conditions including posttraumatic stress disorder (PTSD), characterized by d
113 ism rs9315202 and psychopathology, including posttraumatic stress disorder (PTSD), depression, and al
114 cumented heightened systemic inflammation in posttraumatic stress disorder (PTSD), few studies have a
115 d C-reactive protein, have been described in posttraumatic stress disorder (PTSD), generalized anxiet
116 erapy (CPT), an evidence-based treatment for posttraumatic stress disorder (PTSD), has not been teste
117 stress and stress-related disorders, such as posttraumatic stress disorder (PTSD), is a prominent exa
118 e genetic basis of three disorder categories-posttraumatic stress disorder (PTSD), major depressive d
119 eneralization of fear, a cardinal feature of posttraumatic stress disorder (PTSD), manifests as inapp
120 re-based therapy, an effective treatment for posttraumatic stress disorder (PTSD), relies on extincti
122 or stress-related psychopathologies, such as posttraumatic stress disorder (PTSD), that are character
123 factor for cardiovascular disease (CVD), and posttraumatic stress disorder (PTSD), the sentinel stres
124 for developing anxiety disorders, including posttraumatic stress disorder (PTSD), the underlying mec
125 e well-known peripheral immune activation in posttraumatic stress disorder (PTSD), there are no studi
149 ala reactivity is a vulnerability factor for posttraumatic stress disorder (PTSD); however, our under
150 hose who screened positive for depression or posttraumatic stress disorder after return from deployme
151 rove health-related quality of life, but not posttraumatic stress disorder among ICU survivors and ma
152 r among ICU survivors and may result in less posttraumatic stress disorder among relatives of ICU pat
154 pite significant overlap of symptoms between posttraumatic stress disorder and anxiety disorders, the
155 es an updated review of sleep disturbance in posttraumatic stress disorder and anxiety-related disord
156 elin signaling may enhance susceptibility to posttraumatic stress disorder and comorbid conditions su
157 thasone compared with placebo on symptoms of posttraumatic stress disorder and depression and health-
158 nts, beneficial effects on the occurrence of posttraumatic stress disorder and depression may be pres
160 nea and insomnia), mental health status (eg, posttraumatic stress disorder and depression), nonocular
164 by deficient neurosteroidogenesis, including posttraumatic stress disorder and major depressive disor
166 responses and have been have been linked to posttraumatic stress disorder and other mental health af
167 le target in developing novel treatments for posttraumatic stress disorder and related disorders.
168 s potential therapeutic effects for treating posttraumatic stress disorder are related to altering em
169 -year anxiety disorders, mood disorders, and posttraumatic stress disorder by sexual identity (hetero
170 ew Version (PSS-I) and the stressor-specific Posttraumatic Stress Disorder Checklist (PCL-S); seconda
171 oms of posttraumatic stress (measured by the Posttraumatic Stress Disorder Checklist [PCL]), personal
172 outcomes were posttraumatic stress symptoms (Posttraumatic Stress Disorder Checklist for DSM-5), func
174 s 6 months after patient discharge using the Posttraumatic Stress Disorder Checklist-Civilian Version
175 y was assessed at hospital discharge via the Posttraumatic Stress Disorder Checklist-Specific scale;
176 ck women experienced higher risk of lifetime posttraumatic stress disorder compared with white women.
177 ses including mild traumatic brain injury or posttraumatic stress disorder display alterations on ASL
181 co-occurring psychiatric conditions and with posttraumatic stress disorder having the largest impact.
182 polymorphism, rs4523957, is associated with posttraumatic stress disorder in humans, consistent with
184 health, increasing depression, anxiety, and posttraumatic stress disorder in these same villages.
186 nisms of severe sepsis in the development of posttraumatic stress disorder need further examination.
187 There has been relatively little study of posttraumatic stress disorder or obsessive-compulsive di
188 ntify anxiety, stress, caregiver burden, and posttraumatic stress disorder outcomes in informal careg
189 of threat.SIGNIFICANCE STATEMENT Anxiety and posttraumatic stress disorder patients generalize fear t
191 (OR, 1.15; 95% CI, 1.02-1.30; P = .02), and posttraumatic stress disorder score (OR, 1.04; 95% CI, 1
192 fied between overall DE symptom severity and posttraumatic stress disorder scores and tear breakup ti
193 uman postmortem datasets from depression and posttraumatic stress disorder studies have identified ma
196 rlson index of zero) had higher frequency of posttraumatic stress disorder symptoms (adjusted odds ra
197 rrest and subsequent hospitalization-induced posttraumatic stress disorder symptoms (cardiac arrest-i
198 ationship to whole-brain cortical thickness, posttraumatic stress disorder symptoms (PTSD) and depres
200 patient- and family-member risk factors for posttraumatic stress disorder symptoms among family memb
201 e individuals, increased lifetime stress and posttraumatic stress disorder symptoms explained the maj
203 studies reported significant improvement in posttraumatic stress disorder symptoms of relatives of I
206 found no significant reduction in patients' posttraumatic stress disorder symptoms with ICU diaries
208 significant general anxiety, depression, or posttraumatic stress disorder symptoms, and these sympto
212 lthough symptoms of anxiety, depression, and posttraumatic stress disorder were found to be common 1
215 , bipolar disorder, depression, anxiety, and posttraumatic stress disorder) with major CVD outcomes (
216 , 20.2%; generalized anxiety disorder, 7.9%; posttraumatic stress disorder, 9.8%; alcohol dependence,
217 specific psychiatric disorders (depression, posttraumatic stress disorder, addiction, social anxiety
218 s (i.e., sex of the exposed parent, parental posttraumatic stress disorder, age at Holocaust exposure
221 target for treatment of depression, anxiety, posttraumatic stress disorder, and also against age-rela
222 ast 6-month generalized anxiety disorder and posttraumatic stress disorder, and past 12-month alcohol
223 tions, such as generalized anxiety disorder, posttraumatic stress disorder, and social anxiety disord
224 d chemotherapy-related cognitive impairment, posttraumatic stress disorder, and traumatic brain injur
225 l comorbid psychiatric conditions, including posttraumatic stress disorder, anxiety disorders, obsess
227 ive disorders (mean scores >35), followed by posttraumatic stress disorder, borderline personality di
228 of traumatic events is a hallmark symptom of posttraumatic stress disorder, characterized by rich and
230 antify the impact of psychiatric conditions (posttraumatic stress disorder, depression, anxiety, and
231 ty disorders, obsessive-compulsive disorder, posttraumatic stress disorder, depressive disorders, bip
233 rders, such as major depressive disorder and posttraumatic stress disorder, exact enormous socioecono
234 entia among men with stress disorders except posttraumatic stress disorder, for which women had a hig
235 he primary symptoms linked to deployment are posttraumatic stress disorder, mood disorders, GI proble
236 y a role in the formation and persistence of posttraumatic stress disorder, of which sleep impairment
237 nalyses used a priori seeds relevant to TMS, posttraumatic stress disorder, or MDD (subgenual anterio
239 (mood, anxiety, substance use, personality, posttraumatic stress disorder, schizophrenia, and psycho
241 suicidal plan, bipolar disorder, psychosis, posttraumatic stress disorder, substance dependence, cur
268 nd 78 patients (13.8%) who received placebo (posttraumatic stress disorder: odds ratio, 0.82; 95% CI,
269 yment (depression: OR, 1.4; 95% CI, 1.1-1.9; posttraumatic stress disorder: OR, 2.4; 95% CI, 2.1-2.8)
270 in-angiotensin system has been implicated in posttraumatic stress disorder; however, the mechanisms r
271 erapy for anxiety, obsessive-compulsive, and posttraumatic stress disorders and to evaluate whether a
272 ld higher prevalence of major depressive and posttraumatic stress disorders in women compared with me
274 may have high levels of anxiety, depression, posttraumatic stress disorders, and/or complicated grief
275 Other conditions represented were psychotic, posttraumatic stress or anxiety, somatoform, neurocognit
276 equal to 14 (interquartile range, 5-20) and Posttraumatic Stress Scale equal to 22 (interquartile ra
277 tal Anxiety and Depression Scale; p = 0.010, Posttraumatic Stress Scale), resilience (p = 0.012, Hosp
279 ange of postdisaster trajectories of chronic posttraumatic stress symptom (PTSS) and depression sympt
280 t in PTSD vs. controls, were associated with posttraumatic stress symptom (PTSS) severity, or were af
282 screened positive for cardiac arrest-induced posttraumatic stress symptomatology at discharge (median
284 r cardiac arrest, and cardiac arrest-induced posttraumatic stress symptomatology was associated with
285 ss disorder symptoms (cardiac arrest-induced posttraumatic stress symptomatology) at hospital dischar
287 d social support, cognitive functioning, and posttraumatic stress symptoms were also assessed using s
291 gnificant levels of anxiety, depression, and posttraumatic stress, as well as cognitive function and
292 tic neuropsychiatric sequelae (APNS) such as posttraumatic stress, major depression, and regional or
293 e APNS, as traditionally classified, include posttraumatic stress, postconcussion syndrome, depressio
294 cantly different regarding the prevalence of posttraumatic stress-related symptoms (52.3 vs 50%, resp
295 tionnaire and the Impact of Event Scale (for posttraumatic stress-related symptoms) 3 months later.
297 ent negative life events, and depressive and posttraumatic-stress-disorder symptom scores with placen
299 eficits belong to the most prevalent chronic posttraumatic symptoms, most likely due to diffuse axona