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1 encies of less than 44.1% were assumed to be postzygotic.
2         We conclude that CLOVES is caused by postzygotic activating mutations in PIK3CA.
3                       Mosaicism results from postzygotic alterations during embryogenesis leading to
4 ndencies that govern a cell's entry into the postzygotic and exconjugant developmental programs.
5 kage disequilibrium between loci involved in postzygotic and premating isolation must often be built
6 relates to its effect on the pathway between postzygotic and prezygotic isolation.
7  mating may play a dual role as an important postzygotic as well as a premating reproductive barrier
8 two members, AGL62 and AGL90, attenuated the postzygotic barrier between A. thaliana and A. arenosa.
9 nd, indirect role of assortative mating as a postzygotic barrier to gene flow.
10 portion of mate choice's overall effect as a postzygotic barrier to gene flow.
11 ne of these behaviors that can also act as a postzygotic barrier to reproduction and allow temporal p
12 sting studies confirm the polygenic basis of postzygotic barriers and demonstrate the utility of gene
13 is challenge, we engineer SPECIES (Synthetic Postzygotic barriers Exploiting CRISPR-based Incompatibi
14 levation resident populations, with decisive postzygotic barriers to gene flow.
15                                  We assessed postzygotic barriers up to F(2) hybrids and backcrosses.
16           Little is known about how pre- and postzygotic barriers will affect the prospects of "admix
17 prezygotic barriers tend to be stronger than postzygotic barriers, but most studies are based on the
18 rs contributed more to reduce gene flow than postzygotic barriers, but their contributions were more
19 rns are due to assortative mating and strong postzygotic barriers, rather than recent population hist
20 e revealed an unexpectedly high incidence of postzygotic chromosomal abnormalities, which might arise
21 essian fly sex determination system in which postzygotic chromosome elimination is used to establish
22 constructed cell lineages for the first five postzygotic cleavages and calculated a mutation rate of
23 ibitor actinomycin D disrupts many events of postzygotic conjugation (cycloheximide causes indistingu
24 Igf2 and H19 and of other imprinted genes to postzygotic de novo methylation may be the molecular bas
25                                              Postzygotic de novo mutations lead to the phenomenon of
26                 The decision to proceed with postzygotic development is governed by a developmental c
27  established in oogenesis, maintained during postzygotic development on the maternal allele, and eras
28 sequences of events: prezygotic development, postzygotic development, and exconjugant development.
29        Early treatment causes a block of all postzygotic development, suggesting a transcription requ
30 re to replicate DNA, and failure to initiate postzygotic development.
31 ts to pass a checkpoint, allowing entry into postzygotic development.
32 dably perpetuates an epigenetic state during postzygotic development.
33                          Of 10 reconstructed postzygotic divisions, none resulted in balanced contrib
34 understood function, we identified imprinted postzygotic DNA methylation maintenance, likely by direc
35                  Age-related accumulation of postzygotic DNA mutations results in tissue genetic hete
36                                Two rounds of postzygotic DNA replication occur normally in progeny of
37                       Wide crosses result in postzygotic elimination of one parental chromosome set,
38                This finding unveils an early postzygotic event which can explain both the renal invol
39                This finding unveils an early postzygotic event which can explain both the renal invol
40         Expansion has been suggested to be a postzygotic event with the germline protected.
41 arose during MI, 29% during MII and 16% were postzygotic events; a further 7% were meiotic but could
42 may already exist in the maternal oocyte, or postzygotic expansion, if it occurs, arises quite early
43 st vascular anomalies with overgrowth harbor postzygotic gain-of-function mutations in oncogenes.
44  we observed are the result of inhibition of postzygotic gene expression, presumably in anlagen.
45 onditions, and considers the role of somatic postzygotic genetic lesions in brain maldevelopment.
46 lated species Arabidopsis arenosa results in postzygotic hybrid incompatibility, manifested as seed d
47 rent ploidy levels are separated by a strong postzygotic hybridization barrier that is established in
48 pment, we identify three PEGs that establish postzygotic hybridization barriers in the endosperm, rev
49 6% of de novo single-nucleotide variants are postzygotic in origin with no paternal bias, including e
50                            Here we show that postzygotic inactivating mutations of RHOA cause a neuro
51    Recent studies showed remarkable pre- and postzygotic incompatibilities between sympatric colour m
52 number of genes involved in these "intrinsic postzygotic incompatibilities" should increase faster th
53 g allopatry, premating behavioral isolation, postzygotic inviability and Wolbachia-induced cytoplasmi
54 oward understanding the genetic basis of the postzygotic isolating mechanisms of hybrid sterility and
55 oward understanding the genetic basis of the postzygotic isolating mechanisms of hybrid sterility and
56      Hybrid sterility is one of the earliest postzygotic isolating mechanisms to evolve between two r
57 ogress in understanding the genetic basis of postzygotic isolating mechanisms, little is known about
58 icient amount of time for the development of postzygotic isolation among the three species that hybri
59 that are predicted to be involved in pre-and postzygotic isolation and thus affect hybridization.
60                                       First, postzygotic isolation appears to involve a modest number
61  epistatic incompatibilities contributing to postzygotic isolation behave as X-linked partial recessi
62 ting isolation with the absence of intrinsic postzygotic isolation between parentals.
63             We analyzed the genetic basis of postzygotic isolation between the Bogota and USA subspec
64                        We go on to study how postzygotic isolation caused by epistatic incompatibilit
65                              This incomplete postzygotic isolation could allow ongoing interploid gen
66             Sex linkage of genes involved in postzygotic isolation generally increases the strength o
67 fying and characterizing factors involved in postzygotic isolation in Drosophila has remained slow, m
68  nothing is known about the genetic basis of postzygotic isolation in the genus.
69 we show that the number of genes involved in postzygotic isolation increases at least as fast as the
70                                      Whereas postzygotic isolation is now complete because of sterili
71  shown that in Drosophila, the mechanisms of postzygotic isolation may evolve more rapidly in males t
72  have sex chromosomes evolve lower levels of postzygotic isolation than taxa with sex chromosomes, at
73                                              Postzygotic isolation was calculated for each cross as t
74                                              Postzygotic isolation was significantly lower in tetrapl
75                                              Postzygotic isolation was weaker among higher order poly
76 ation) or are due to reduced hybrid fitness (postzygotic isolation).
77 arge X-effect, describe the genetic basis of postzygotic isolation, and have led to the realization t
78                                   Additional postzygotic isolation, caused, for example, by either ep
79                               Despite strong postzygotic isolation, ecological niche differentiation
80 ntifying genes and mutations responsible for postzygotic isolation, little is known about the genetic
81             Unlike the genetic components of postzygotic isolation, the loci for prezygotic isolation
82                                    Extrinsic postzygotic isolation, where hybrids experience reductio
83 eproductive isolation--especially intrinsic, postzygotic isolation--does not seem to be the rate-limi
84 n reflect the recessivity of alleles causing postzygotic isolation.
85 ule and the large effect of X chromosomes on postzygotic isolation.
86 e character displacement may have reinforced postzygotic isolation.
87 ontribute to a faster evolution of intrinsic postzygotic isolation.
88 x chromosomes for the evolution of intrinsic postzygotic isolation.
89 solation also appears to result in increased postzygotic isolation.
90 r genomic forces that drive the evolution of postzygotic isolation.
91 rangements appear to contribute to intrinsic postzygotic isolation.
92 ive in the evolution of the genes that cause postzygotic isolation.
93 ic) groups meet after having evolved partial postzygotic isolation; they are selected to evolve or en
94 thaliana genome that affect the frequency of postzygotic lethality and the phenotypes of surviving se
95 f X-bearing sperm, with limited evidence for postzygotic lethality of female progeny.
96                                              Postzygotic lethality of interspecies hybrids can result
97 tween prezygotic nuptial gifts and exclusive postzygotic male care and support the hypothesis that, i
98 ickle cell trait has mild SCD resulting from postzygotic mitotic recombination leading to UPD.
99  stage (meiosis I [MI], meiosis II [MII], or postzygotic mitotic) of the chromosomal error.
100       Sex ratio distortion is independent of postzygotic mortality, and is not associated with an obv
101  blood, indicating that these conditions are postzygotic mosaic disorders.
102                                              Postzygotic mosaic mutations (PMMs) have been implicated
103 vo variants and could be used to distinguish postzygotic mosaic variants from prezygotic de novos.
104 evere developmental disorders for pathogenic postzygotic mosaicism (PZM) in the child or a clinically
105                                  While early postzygotic mosaicism is the predominant mechanism, gene
106 ure by segmental Darier's disease induced by postzygotic mosaicism.
107 ypic and genetic heterogeneity combined with postzygotic mosaicism.
108  in four unrelated patients that a recurrent postzygotic mutation in GNA13 is responsible for a recog
109 ve recently been shown to be due to a single postzygotic mutation in NRAS [neuroblastoma RAS viral (v
110                We hypothesized that a single postzygotic mutation in NRAS could be responsible for mu
111                            The occurrence of postzygotic mutation in the early mouse embryo suggests
112                       Somatic mosaicism is a postzygotic mutation that occurs in the soma, and it may
113                                              Postzygotic mutations (PZMs) begin to accrue in the huma
114                   We systematically analyzed postzygotic mutations (PZMs) in whole-exome sequences fr
115                                        These postzygotic mutations arise from intrinsic errors in DNA
116 1 unrelated families with MPPH and 15 mostly postzygotic mutations in PIK3CA in 23 individuals with M
117 tially through an influence on the number of postzygotic mutations in the embryo.
118            We identified de novo germline or postzygotic mutations in three core components of the ph
119 lar disorder, supporting the hypothesis that postzygotic mutations of developmental disorder genes ma
120 icate the contributions of both germline and postzygotic mutations to the risk of bipolar disorder, s
121 nvestigations evaluated body distribution of postzygotic mutations, their stability over time, and th
122                           In the analysis of postzygotic mutations, we observe significant enrichment
123  differ in terms of the body distribution of postzygotic mutations.
124            These results suggest that single postzygotic NRAS mutations are responsible for multiple
125 r exchange, pronuclear fusion, and anchoring postzygotic nuclear division products to the posterior c
126                   cnj9 eliminates the second postzygotic nuclear division, and subsequently, new macr
127 rd prezygotic nuclear division and the first postzygotic nuclear division.
128 ly neoplasm frequently associated with early postzygotic occurrence of this genomic alteration.
129 male care of offspring is the rarest form of postzygotic parental care among animals and has arisen i
130 . suggests prezygotic aneuploidy followed by postzygotic partial reversion leads to a recurrent form
131  the triploid block, which is an established postzygotic polyploidization barrier.
132                         To determine whether postzygotic rejection of self-fertilized ovules is due t
133  inviable seed formation, revealing a strong postzygotic reproductive barrier separating these two sp
134  determine the strength of 17 prezygotic and postzygotic reproductive barriers between two Lysimachia
135                           Although intrinsic postzygotic reproductive barriers can play a fundamental
136 le insights into the nature and evolution of postzygotic reproductive barriers in diverged species.
137 s of experimental hybrids indicate that most postzygotic reproductive barriers in plants are polygeni
138 etics of phenotypic divergence and intrinsic postzygotic reproductive barriers remains tenuous.
139 of the heterogametic sex is one of the first postzygotic reproductive barriers to evolve during speci
140 enetic incompatibility approach, to generate postzygotic reproductive barriers.
141                 Much evidence has shown that postzygotic reproductive isolation (hybrid inviability o
142                         This study evaluates postzygotic reproductive isolation among three cytotypes
143                           Polyploidy confers postzygotic reproductive isolation and is thought to dri
144  important implications for the evolution of postzygotic reproductive isolation and speciation.
145                                 This form of postzygotic reproductive isolation appears to be highly
146 tural selection might drive the evolution of postzygotic reproductive isolation even when allopatric
147                                              Postzygotic reproductive isolation evolves when hybrid i
148 ly-acting and potentially widespread form of postzygotic reproductive isolation is largely unknown.
149 oid speciation entails substantial and rapid postzygotic reproductive isolation of nascent species th
150      The Dobzhansky-Muller model posits that postzygotic reproductive isolation results from the evol
151  Organisms that quickly evolve prezygotic or postzygotic reproductive isolation should have faster ra
152 obzhansky-Muller model posits that intrinsic postzygotic reproductive isolation--the sterility or let
153 that has been implicated in the evolution of postzygotic reproductive isolation.
154  characterize the mechanisms and strength of postzygotic reproductive isolation.
155  of genes contributing to the maintenance of postzygotic reproductive isolation.
156 s thus suggest that the relative strength of postzygotic RI may be underestimated when its effects on
157                              The strength of postzygotic RI was up to three times lower for F(1) prog
158      In the invasive ant Nylanderia fulva, a postzygotic SAS leads daughters to preferentially carry
159  gene expression play important roles in the postzygotic seed abortion in interspecific hybrids or ne
160                    The mechanism behind this postzygotic seed abortion is poorly understood.
161 t is also known to play an important role in postzygotic selection against hybrids and, thus, the pur
162 stitution is more likely to be the result of postzygotic selection against other unbalanced karyotype
163 ulation, enhancing the efficiency with which postzygotic selection purges introgressed DNA.
164                                              Postzygotic somatic mutations activating the phosphatidy
165                                              Postzygotic somatic mutations have been found associated
166 such as Arabidopsis thaliana and A. arenosa, postzygotic species barriers often affect seed abortion,
167 thal and zygotic-rescue activities to gain a postzygotic survival advantage.
168   Focal malformations are caused by somatic (postzygotic) variants in genes related to cell growth (i
169                                              Postzygotic variation arising during neurogenesis is rec

 
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