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1 al coupling (evidenced by direct response to potassium chloride).
2 of cortical spreading depressions induced by potassium chloride.
3 nadate sensitive, and slightly stimulated by potassium chloride.
4 bition of protein kinase C or high levels of potassium chloride.
5 mide, and by a depolarizing concentration of potassium chloride.
6 but normal with non-nutrient stimuli such as potassium chloride.
7 naptosomes were stimulated by veratridine or potassium chloride.
8 oupling of diaryliodonium salts, alkenes and potassium chloride.
9 increase in current is observed with aqueous potassium chloride.
10  skipped following depolarisation induced by potassium chloride.
11 rine isotopes in gaseous sodium chloride and potassium chloride.
12 hat are composed of eutectic silver chloride-potassium chloride.
13 large white pigs by intravenous injection of potassium chloride.
14 d control individuals were depolarized using potassium chloride.
15 SR was enhanced by chemical stimulation with potassium chloride.
16 port, and disassemble upon depolarization by potassium chloride.
17 g mixtures of kappa and iota carrageenan and potassium chloride.
18 the effective supersaturation of the aqueous potassium chloride.
19 ous agarose gel prepared with supersaturated potassium chloride.
20 ministration of thiopental, pancuronium, and potassium chloride.
21 e partial substitution of sodium chloride by potassium chloride (0%, 25%, and 50%) and addition of ar
22 ases in perfusion pressures (delta mm Hg) to potassium chloride (30-300 mmol/L) of in vitro perfused
23 tive humidity (ERH) of potassium sulfate and potassium chloride aerosol as well as the separation RH
24 e analysis, including particles comprised of potassium chloride and organic nitrogen during the begin
25                                              Potassium chloride and sodium nitrate also potentiate fi
26 not enhance pressure responses to 125 mmol/L potassium chloride, and failed to increase perfusion pre
27 molecular dynamics to model sodium chloride, potassium chloride, and sodium bromide solutions at diff
28  response to treatment with sodium chloride, potassium chloride, and sorbitol.
29 n (BUN) and creatinine, chemistries (sodium, potassium, chloride, and bicarbonate), complete blood ce
30                                      Sodium, potassium, chloride, and creatinine excretion were analy
31 l-time concentration measurements of sodium, potassium, chloride, and pH in a small volume of biologi
32 easures correlated with wastewater (sulfate, potassium, chloride, and sodium) and road runoff (chromi
33 ulation of nonischemic ipsilateral cortex by potassium chloride application (KCl group; n = 7) and sa
34 s shown that simple sugars, amino acids, and potassium chloride are abundant in Belgian endive extrac
35 salts sodium bromide, potassium bromide, and potassium chloride are employed.
36 ine and divided into four groups: diets plus potassium chloride as control, potassium citrate, chlort
37 lcium and phosphorus diet, supplemented with potassium chloride (as control), potassium citrate, chlo
38 hermodynamic chemical activity of sodium and potassium chloride, as well as the effect of the salts o
39 undling serum metabolic panel tests (sodium, potassium, chloride, bicarbonate, glucose, blood urea ni
40    The fluids are dominantly sodium chloride-potassium chloride brines, but they also contain divalen
41 n physiologically relevant concentrations of potassium chloride, calcium chloride, and manganese chlo
42 d carbohydrates) and micronutrients (sodium, potassium, chloride, calcium, phosphorus, magnesium, vit
43 aily potassium nitrate (12 mmol) or placebo (potassium chloride) capsules for 5 days.
44                         While the sodium and potassium chloride carbenoids showed high stabilities in
45                         While the sodium and potassium chloride carbenoids showed high stabilities in
46 pithelial sodium channel (gammaENaC), sodium-potassium -chloride co-transporter 2 (NKCC2), sodium chl
47 ome type I, showing an absence of the sodium-potassium-chloride co-transporter 2, NKCC2.
48  prevented by overexpression of the neuronal potassium-chloride co-transporter KCC2 in principal cort
49                                          The potassium-chloride co-transporter KCC2, encoded by SLC12
50  dysregulation caused by hypofunction of the potassium-chloride co-transporter KCC2.
51  were determined under varying conditions of potassium chloride concentration using a surface plasmon
52 tion at unchanged cytosolic Ca(2+) levels in potassium chloride-constricted intact arteries isolated
53 ransport in the erythrocyte is attributed to potassium chloride cotransporter 1 (KCC1).
54 of the inhibitory GABAA receptor, the sodium potassium chloride cotransporter 1 (NKCC1) and potassium
55 vity of a bumetanide-sensitive NKCC1 (sodium potassium chloride cotransporter 1)-like chloride cotran
56 tassium chloride cotransporter 1 (NKCC1) and potassium chloride cotransporter 2 (KCC2) and the excita
57 t CA1 pyramidal neurons because of increased potassium chloride cotransporter 2 (KCC2) expression and
58 ride shift caused by significant decrease in potassium chloride cotransporter 2 (Kcc2) mRNA expressio
59  GABAergic transmission are well-documented, potassium chloride cotransporter 2 (KCC2), a key regulat
60 sitive hypotension, with depletion of sodium potassium chloride cotransporter 2 and aquaporin 2.
61  For functional characterization, the mutant potassium chloride cotransporter 3 was modelled in Xenop
62 s associated with reduced dorsal spinal cord potassium chloride cotransporter expression and impaired
63                                          The potassium chloride cotransporter KCC2 plays a major role
64  - is disrupted due to downregulation of the potassium chloride cotransporter KCC2.
65 nds on chloride gradients established by the potassium chloride cotransporter KCC2.
66 aying behavior and discovered mutations in a potassium chloride cotransporter, kcc-2.
67 y gamma-aminobutyric acid and glycine due to potassium chloride cotransporter-2 (KCC2) down-regulatio
68                       This study asks if the potassium-chloride cotransporter (K:Cl) and the calcium-
69                     The furosemide-sensitive potassium-chloride cotransporter (KCC2) plays an importa
70 ession ratios of the K(+)/Cl(-) transporters potassium-chloride cotransporter 2 (KCC2) and sodium-pot
71                                          The potassium-chloride cotransporter 2 (KCC2) is required fo
72  a histidine point variation in the neuronal potassium-chloride cotransporter 2 (KCC2).
73 ium-glucose co-transporter-2 (SGLT2), sodium-potassium-chloride cotransporter 2 (NKCC2), sodium chlor
74 s of Tbx1 or Slc12a2, which encodes a sodium-potassium-chloride cotransporter and is also necessary f
75 ction is downregulated, including the sodium-potassium-chloride cotransporter gene nkcc1 (slc12a2) an
76                Here, we show that the Sodium-Potassium-Chloride Cotransporter Isoform-1 (NKCC1) provi
77 ron microscopy (cryo-EM) structures of human potassium-chloride cotransporter KCC1 in potassium chlor
78 w that specific markers of excitability, the potassium-chloride cotransporter KCC2 and GABAA receptor
79 n associated with dysfunction or loss of the potassium-chloride cotransporter KCC2 in a subset of pyr
80 decrease in the functional expression of the potassium-chloride cotransporter KCC2 in spinal cord dor
81      Once in the cortex, upregulation of the potassium-chloride cotransporter KCC2 is both necessary
82 -2 ex vivo via functional enhancement of the potassium-chloride cotransporter KCC2.
83 s is largely mediated by the neuron-specific potassium-chloride cotransporter KCC2.
84 ion ([Cl(-)](i)), which is maintained by the potassium-chloride cotransporter KCC2.
85 (hepatocyte nuclear factor 4alpha), SLC12A5 (potassium-chloride cotransporter member 5), CDH22 (cadhe
86  receptors can physically associate with the potassium-chloride cotransporter protein, KCC2, which se
87 novel association between the GABABR and the potassium-chloride cotransporter protein, KCC2.
88 by clearance of Cl(-) coupled to K(+) by the potassium-chloride cotransporter, KCC2.
89 sociated with decreased expression of sodium-potassium-chloride cotransporter-1 (NKCC1) and aquaporin
90 crease; claudin-5, 2.7-fold increase; sodium-potassium-chloride cotransporter-1, 2.8-fold increase; a
91 s.Significance statementImpaired function of potassium-chloride cotransporter-2 (KCC2), a key regulat
92                            Disruption of the potassium/chloride cotransporter 3 (KCC3), encoded by th
93 1 consumed 0.4 +/- 0.06 mEq (mean +/- SD) of potassium chloride daily, and group 2 ate 4.8 +/- 1.0 mE
94                                              Potassium chloride depolarization caused sustained relea
95 icantly reduced in hippocampal neurons after potassium chloride depolarization.
96  diet supplemented with potassium citrate or potassium chloride (each 4 mmol/d) for 18 weeks.
97          Investigations involve solutions of potassium chloride electrolyte containing potassium ferr
98                                  Addition of potassium chloride enhanced extraction of HgR from LAEA-
99 ed for characterization was lithium chloride/potassium chloride eutectic (LKE), which has potential a
100 s link earlier observations that both sodium/potassium/chloride exchange and Ca(2+) are required for
101  current (I(NaP)) and down-regulation of the potassium/chloride extruder KCC2 lead to spasticity afte
102 system and its regulatory control of sodium, potassium, chloride, hydrogen ion, and water homeostasis
103 ium reduction and its partial replacement by potassium chloride in pizza dough and crusts prepared by
104 n response to capsaicin, cholecystokinin and potassium chloride in TNX-deficient mice.
105 um ferricyanide, and supporting electrolyte, potassium chloride, in the presence of a magnetic field.
106 -deleted (GSNOR(-/-)) mice were subjected to potassium chloride-induced CA and subsequently resuscita
107 type mice underwent a 4, 8, 12, or 16-minute potassium chloride-induced cardiac arrest followed by 90
108                             Within 1 hour of potassium chloride-induced depolarization, independent o
109 us was modulated by focal microinjections of potassium chloride into the nucleus reuniens, during whi
110  nitride membrane dividing water solution of potassium chloride into two compartments connected by th
111 effect of bumetanide, an FDA-approved sodium-potassium-chloride intruder (NKCC1) antagonist, on presy
112 de (an FDA-approved antagonist of the sodium-potassium-chloride intruder, NKCC1) treatment increases
113                                              Potassium chloride ion cotransporters (KCCs) are part of
114 ing TMX with a depolarizing concentration of potassium chloride (K+ -30 mM).
115 rough sodium-potassium-2-chloride (NKCC) and potassium-chloride (KCC) cotransporters is a major deter
116  was sensitive to the level of extracellular potassium chloride (KCl) and depolarizing concentrations
117 itrogen, and creatinine, escalating doses of potassium chloride (KCl) and spironolactone were adminis
118  vasodilator (sodium nitroprusside, SNP), or potassium chloride (KCl) at rest; (2) mild or moderate i
119 splicing events that consistently respond to potassium chloride (KCl) depolarization.
120 (6 mmol 3 times daily) vs equimolar doses of potassium chloride (KCl) for 6 weeks, each with a 1-week
121 egg albumin, with sodium chloride (NaCl) and potassium chloride (KCl) from phosphate-buffered saline
122 ors, 38% diabetes) were treated with 40 mmol potassium chloride (KCl) per day for 2 weeks.
123 acement of added sodium chloride (NaCl) with potassium chloride (KCl) provides a potential strategy t
124 mic K concentration during the conversion of potassium chloride (KCl) salt and wheat straw particles
125 ricans.We sought to determine the effects of potassium chloride (KCl) supplements, at a commonly pres
126                            The flow of 0.2 M potassium chloride (KCl) through the random sphere packi
127           We demonstrate here that replacing potassium chloride (KCl) with potassium acetate (KAc) or
128  exists mainly as potassium hydroxide (KOH), potassium chloride (KCl), and atomic potassium (K) in co
129  evoked in arteries via norepinephrine (NE), potassium chloride (KCl), and caffeine, and in veins thr
130 stitution-mediated DS(-) precipitation using potassium chloride (KCl), and excellent peptide recovery
131 m acetate (NaAc), ammonium chloride (AmmCl), potassium chloride (KCl), and sodium chloride (NaCl).
132 son of transport of monovalent electrolytes [potassium chloride (KCl), sodium chloride (NaCl)], 2:1 e
133                                              Potassium chloride (KCl)-depolarization has been used to
134  mass for either 24 or 65 h showed increased potassium chloride (KCl)-induced and oxytocin-induced co
135 t (+)-N-allylnormetazocine ((+)-SKF10047) on potassium chloride (KCl)-induced calcium influx in RGC-5
136 ma-1 receptor agonist (+)-SKF10047 inhibited potassium chloride (KCl)-induced calcium influx.
137 [Ca2+]i when they were stimulated with 30 mM potassium chloride (KCl).
138 endothelin [ET-1], phorbol ester [PdBu], and potassium chloride [KCl]).
139 ala and responds strongly to stimuli such as potassium chloride, lithium chloride, and protein kinase
140                   Measurements were taken in potassium chloride (moderate ion-lipid binding) and tetr
141 rved ejection fraction to oral KNO3 (n=9) or potassium chloride (n=3).
142 rved ejection fraction to oral KNO3 (n=9) or potassium chloride (n=3).
143  meshwork (TM) cells possess a robust sodium-potassium-chloride (Na-K-Cl) cotransport system that fun
144 udy was to examine the effects of sodium and potassium chloride on lipid oxidation in O/W emulsions.
145           SDs were induced by application of potassium chloride or by oxygen and glucose deprivation
146 man potassium-chloride cotransporter KCC1 in potassium chloride or sodium chloride at 2.9- to 3.5-ang
147 cal administration of the depolarizing agent potassium chloride or systemic administration of the ant
148 e to phenylephrine and serotonin, but not to potassium chloride or U46619.
149 incubation of cells in 100 nm insulin, 30 mm potassium chloride, or 0.25 mm diazoxide, indicating tha
150 trol, potassium citrate, chlorthalidone plus potassium chloride, or potassium citrate plus chlorthali
151                 Addition of sodium chloride, potassium chloride, or sodium sulfate to leptospiral med
152 alemia was induced by continuous infusion of potassium chloride over 45 minutes followed by a bolus.
153           The AG was defined as (sodium plus potassium) - (chloride plus total carbon dioxide).
154                                  Compared to potassium chloride, potassium citrate reduced urinary ca
155 udies were performed under a range of salts (potassium chloride, potassium phosphate, potassium aceta
156                      Brief exposure to 10 mM potassium chloride produced epileptiform bursting and po
157 loganic acid (PubChem CID: 89640), pyridine, potassium chloride (PubChem CID: 4873), ammonium carbona
158 um carbonate, gallic acid (PubChem CID: 370) potassium chloride (PubChem CID: 4873), sodium acetate (
159                   Elevation of extracellular potassium chloride resulted in spontaneous asynchronous
160 on recognition, herein, we present the first potassium chloride-selective heteroditopic ion-pair rece
161                                              Potassium chloride showed similar impact on lipid oxidat
162    The tolerance limits for sodium chloride, potassium chloride, sodium acetate, sodium fluoride, sod
163 artially denaturing TGGE is carried out with potassium chloride, sodium chloride, or magnesium chlori
164 chemical parameters (including electrolytes (potassium, chloride, sodium), creatinine, urea, and rand
165 rontium acetate/sodium fluoride (SrAc2F) and potassium chloride/sodium monofluorophosphate (KCl/MFP)
166 strated by measuring conductivity of aqueous potassium chloride solution and bottled water samples an
167  dissolution kinetics of ZnO NPs in buffered potassium chloride solution using anodic stripping volta
168 M phosphate buffer (pH 7.0) containing 0.1 M potassium chloride solution, and a flow rate of 0.8 ml m
169                                              Potassium chloride-stimulated NO formation was also enha
170                                              Potassium chloride supplementation of the Sulfolobus ass
171  potassium intake to recommended levels with potassium chloride supplementation raises plasma potassi
172 find that trehalose has a specific impact on potassium chloride that is unlike that of other sugars o
173 ements for growth: 9 amino acids, sodium and potassium chloride, thiamine, iron, zinc, magnesium, hyp
174 ol), potassium citrate, chlorthalidone (with potassium chloride to equalize potassium intake), or pot
175 of epileptiform activity induced by elevated potassium chloride to induce multiple forms of LTP in ar
176  episodes were induced by application of 3 M potassium chloride to the cortex of adult anesthetized r
177 m-chloride cotransporter 2 (KCC2) and sodium-potassium-chloride transporter (NKCC1).
178 ter in the kidney loop of Henle and the KCC2 potassium-chloride transporter in neuronal membranes, wo
179 de and ethacrynic acid, which block the KCC1 potassium-chloride transporter in the kidney loop of Hen
180 lobal ischemia caused an upregulation of the potassium-chloride transporter KCC2 in neurons which per
181 bitor, but not by VU01240551 an inhibitor of potassium-chloride transporter KCC2.
182 ression of active sodium transporters-sodium/potassium/chloride transporter type 2 (NKCC2), sodium/ch
183 dly induced by sodium chloride, drought, and potassium chloride treatments.
184                             Perfusion of low potassium chloride Tyrode solution plus quinidine led to
185 lar acidification rate upon stimulation with potassium chloride was significantly reduced in patient
186 ute, and the latency, Km, and sensitivity to potassium chloride were also similar.

 
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