ライフサイエンスコーパス: preexcitation

1 epicardial asynchrony and QRS vector and LV preexcitation.

2 nimal and endocardial AT vector indicated LV preexcitation.

3 penetrant accessory pathways and ventricular preexcitation.

4 rosus was disrupted resulting in ventricular preexcitation.

5 ical abnormalities, particularly ventricular preexcitation.

6 ovide the anatomic substrate for ventricular preexcitation.

7 lar, resynchronization, and left ventricular preexcitation.

8 elucidating the pathogenesis of ventricular preexcitation.

9 hort (75-millisecond) PR interval to achieve preexcitation.

10 ry care population with electrocardiographic preexcitation.

11 We identified 310 individuals with preexcitation (age range, 8-85 years).

12 tatistically significant association between preexcitation and a higher hazard of death.

13 ndicate sinoatrial node dysfunction, whereas preexcitation and atrioventricular block reveal abnormal

14 e for a syndrome associated with ventricular preexcitation and early onset of atrial fibrillation and

15 cular hypertrophy, and exhibited ventricular preexcitation and sinus node dysfunction.

16 were used to show the mechanistic effects of preexcitation and to determine the optimal stimulation s

17 onset (at 8 to 17 years of age), ventricular preexcitation, and asymptomatic elevations of two serum

18 glycogen-storage cardiomyopathy, ventricular preexcitation, and conduction system degeneration.

19 man cardiomyopathy with cardiac hypertrophy, preexcitation, and glycogen deposition.

20 cterized by cardiac hypertrophy, ventricular preexcitation, and glycogen storage.

21 Wolff-Parkinson-White pattern and persistent preexcitation at maximum exercise undergoing invasive ri

22 sible for a familial syndrome of ventricular preexcitation, atrial fibrillation, conduction defects,

23 glycogen storage eliminated the ventricular preexcitation but did not affect the excessive cardiac g

24 Our results also show that ventricular preexcitation can arise from inappropriate patterning of

25 The human phenotype consists of ventricular preexcitation, conduction abnormalities, and cardiac hyp

26 acute rodent model demonstrated ventricular preexcitation during sinus rhythm.

27 study of patients <21 years of age with HCM/preexcitation from 2000 to 2022.

28 Surface ECG is not adequate to discriminate preexcitation from a benign FVF from that secondary to p

29 d a higher hazard of death for patients with preexcitation >/=65 years (HR, 1.85; 95% CI, 1.07-3.18).

30 e remainder of the population, patients with preexcitation had higher adjusted hazards of atrial fibr

31 In this large ECG study, individuals with preexcitation had higher hazards of atrial fibrillation

32 Young patients with HCM and preexcitation have a high likelihood of underlying stora

33 lar activation (eg, resulting from pacing or preexcitation in patients with Wolff-Parkinson-White syn

34 y glycogen-engorged myocytes as the cause of preexcitation in Pompe, Danon, and other glycogen storag

35 clinical course of patients with ventricular preexcitation in the ECG originates from tertiary center

36 ertrophy followed by dilatation, ventricular preexcitation involving multiple accessory pathways, and

37 Preexcitation is associated with an increased risk of ta

38 Ventricular preexcitation (preexcitation) is well recognized, yet little is known a

39 tified 345 patients with HCM and 28 (8%) had preexcitation (isolated HCM, 10/220; storage disorder, 8

40 was the longest (349+/-6 ms) in the area of preexcitation leading to high average base-to-apex ARI d

41 Preexcitation of the left ventricle after myocardial inf

42 Preexcitation of the RVOT (n=2) resulted in ECGs that su

43 ional BrS patients, we performed decremental preexcitation of the RVOT before endocardial RV mapping.

44 ted by SVT or Wolff-Parkinson-White pattern (preexcitation) on ECG identified MRC2 as a candidate gen

45 AP conduction, as demonstrated by recurrent preexcitation or change in retrograde ventriculoatrial a

46 Electrical preexcitation pacing with appropriate timing of high-str

47 s with an asymptomatic Wolff-Parkinson-White preexcitation persisting at peak exercise.

48 rated the TG(R302Q) mice to have ventricular preexcitation (PR interval 10+/-2 versus 33+/-5 ms in TG

49 Ventricular preexcitation (preexcitation) is well recognized, yet li

50 Preexcitation reduced regional strain in the short term,

51 t of this risk in patients with asymptomatic preexcitation remain controversial.

52 nd electrocardiograms suggesting ventricular preexcitation revealed four LAMP2 and seven PRKAG2 mutat

53 ECGI determined that preexcitation sites were consistent with sites of succes

54 ndle-branch block, ventricular paced rhythm, preexcitation syndrome, or previous revascularization wi

55 tein kinase (AMPK) cause cardiomyopathy with preexcitation syndrome.

56 hough the pathology and electrophysiology of preexcitation syndromes are well characterized, the deve

57 study animals were randomized to 8 weeks of preexcitation (therapy) or no pacing (control).

58 Preexcitation was exclusively FVF mediated in 8 (36%) pa

59 On analysis of ECGs, preexcitation was found in 2 cases.

60 igher hazard of death among individuals with preexcitation when looking across all age groups (HR, 1.

61 Ventricular preexcitation, which characterizes Wolff-Parkinson-White

62 Studies reporting asymptomatic patients with preexcitation who did not undergo ablation were included

63 ted members of both families had ventricular preexcitation with conduction abnormalities and cardiac

64 ectrophysiologic abnormalities, particularly preexcitation (Wolff-Parkinson-White syndrome) and atrio