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1 th sickle cell disease (SCD) risk developing priapism.
2 sive activation of these pathways results in priapism.
3 lation of opiorphin at a life stage prior to priapism.
4  molecular mechanisms for penile fibrosis in priapism.
5 e fibrosis, a dangerous complication seen in priapism.
6  with sickle cell disease (SCD), 40% display priapism.
7 dysregulation is a fundamental mechanism for priapism.
8 employed for patients with SCA and prolonged priapism.
9 ed and explored for use in the prevention of priapism.
10 ients with sickle cell disease (SCD) display priapism.
11 aemic (high-flow or arterial) and stuttering priapism.
12 , pneumonia (2), portal vein thrombosis (1), priapism (1), hemolytic uremic syndrome (1), diaphragm p
13                                              Priapism, abnormally prolonged penile erection in the ab
14 fibrosis in two independent animal models of priapism, adenosine deaminase (ADA)-deficient mice and S
15                                              Priapism, although uncommon in the general population, i
16 e molecular hallmarks and pathophysiology of priapism, an important but poorly characterized erectile
17 Disease, we assembled 273 case subjects with priapism and 979 control subjects.
18  method of choice for treatment of high-flow priapism and for restoration of penile erectile function
19  shown a strong positive correlation between priapism and high levels of intravascular hemolysis in m
20 int to a strong positive correlation between priapism and high levels of intravascular hemolysis in m
21  provide new insight for the pathogenesis of priapism and novel therapies for the disease.
22 atologic characteristics of individuals with priapism and SCD.
23 ide insight regarding the molecular basis of priapism and suggest that strategies to either reduce ad
24 ogical erectile signaling in mouse models of priapism and suggests novel approaches to human therapy.
25 tic sequestration, splenic sequestration, or priapism) and the acute chest syndrome, and patient-repo
26 ted with stroke, pulmonary hypertension, and priapism, and cf-PWV was associated with microalbuminuri
27 nary hypertension, cutaneous leg ulceration, priapism, and ischemic stroke.
28 ephropathy, retinopathy, avascular necrosis, priapism, and leg ulcers.
29 s disease, prior prosthesis explantation and priapism, and men who have undergone construction of a n
30 e of pulmonary hypertension, leg ulceration, priapism, and risk of death in patients with sickle cell
31         The mechanisms underlying stuttering priapism are complex, and involve dysregulation of the n
32                                              Priapism can be subcategorized into three types: ischaem
33 ncluding pulmonary hypertension, leg ulcers, priapism, chronic kidney disease, and large-artery ische
34                                              Priapism data were obtained via daily text messages to t
35                                      Because priapism demonstrates high prevalence in patients with h
36 hs (interquartile range, 3-12), 2.5 and 3.02 priapism events per participant-month were recorded in t
37                            For non-ischaemic priapism following failed conservative treatment, select
38          The optimal management of prolonged priapism for patients with sickle cell anemia (SCA) has
39 lgesics failed to produce detumescence or if priapism had lasted >4 hours, the protocol was activated
40  molecular pathophysiology of SCD-associated priapism has led to the identification of new potential
41 nd answers) on 5 urology CPGs (hematuria and priapism [HP]; staghorn calculi, infertility, and antibi
42  erections occurred in 5 percent of the men, priapism in 1 percent, penile fibrotic complications in
43 pression of genes previously associated with priapism in animal models.
44 ORA2B) signaling, we successfully attenuated priapism in both ADA(-/-) and SCD mice by restoring peni
45 ars) with at least 3 episodes of SCA-related priapism in the past 12 months.
46 e solution under local anesthesia to relieve priapism in young patients with SCA.
47 single-arm, pre-post analysis showed a 58.3% priapism incidence rate reduction in the usual-care arm
48          The investigation and management of priapism involve a stepwise approach.
49                           Recurrent ischemic priapism is a common complication of sickle cell anemia
50                                              Priapism is a condition of persistent penile erection in
51                                              Priapism is a disorder of persistent penile erection unr
52                                    High-flow priapism is a rare condition characterized by a prolonge
53                                              Priapism is an abnormal prolonged penile erection that p
54                                              Priapism is featured with prolonged and painful penile e
55                  The molecular mechanism for priapism is not well characterized.
56                                    Ischaemic priapism is the most common subtype and is associated wi
57  interesting and important, is the fact that priapism of one month's duration could well be treated b
58 her in patients who experienced skin ulcers, priapism, or renal dysfunction.
59                         The 2 patients whose priapism persisted after aspiration and irrigation prese
60 ssion fails, or if a patient presents with a priapism persisting longer than 36-48 h depending on the
61 nt surgery (within the first 24-48 h) if the priapism persists.
62  that sickle cell disease mice (which show a priapism phenotype) evince dysregulated PDE5A expression
63                      All previous trials for priapism prevention have failed to demonstrate clear eff
64            A randomized controlled trial for priapism prevention is feasible in men with SCA.
65  3.4; 95% CI, 1.1-5.8; P = .005) and a 66.3% priapism reduction in the experimental arm (8.9-3.02 eve
66 emergency department with a 3-day history of priapism requiring a surgically performed distal penile
67                 All evaluable patients whose priapism resolved after aspiration and irrigation self-r
68 tions, we hypothesized that the mechanism of priapism rests in aberrant downstream signaling of this
69  in Mrc1(-/-)Asgr2(-/-) mice may account for priapism seen in males.
70 elocity [TRV], microalbuminuria, leg ulcers, priapism, stroke, and osteonecrosis) by clinical examina
71 tiology and pathophysiology of the different priapism subtypes help to determine the management of sp
72                 Current strategies to manage priapism suffer from a poor fundamental understanding of
73         A case of delayed painless high-flow priapism that occured after blunt straddle-type perineal
74 ine accumulation in the penis contributes to priapism through increased A2BR signaling in both Ada -/
75 ent of Mrc1(-/-)Asgr2(-/-) male mice develop priapism when mating due to thrombosis of the penile vei
76     These findings suggest an association of priapism with increased hemolysis.