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1 an elevated TSH level almost always signals primary hypothyroidism.
2 history of treatment with levothyroxine for primary hypothyroidism.
3 yrotropin action, not subclinical autoimmune primary hypothyroidism.
4 ls were described in a preliminary report on primary hypothyroidism.
5 for the accelerated atherosclerosis seen in primary hypothyroidism.
6 low free thyroxine (T4) level indicate overt primary hypothyroidism.
7 tients (62%): 15 (36%) developed persistent, primary hypothyroidism; 4 (10%) developed isolated TSH s
8 adrenocorticotropic hormone deficiency, and primary hypothyroidism at 4 years from diagnosis was 93%
9 fferences in pubertal timing, development of primary hypothyroidism, breast cancer as a second malign
10 es against thyroglobulin, and development of primary hypothyroidism (decreased levels of serum thyrox
11 corticotropin and gonadotropin deficiencies, primary hypothyroidism, gonadal failure and obesity.
12 s compared with survivors not so exposed for primary hypothyroidism (hazard ratio [HR], 6.6; 95% CI,
13 fied and regression models compared risks of primary hypothyroidism, hyperthyroidism, thyroid neoplas
14 itis (an autoimmune disease) is the cause of primary hypothyroidism in up to 85% of patients with hyp
19 fested other features, including ichthyosis, primary hypothyroidism, neurological symptoms, and crypt
21 perform a genome-wide association study for primary hypothyroidism, the most common thyroid disease.
24 pared with levothyroxine alone, treatment of primary hypothyroidism with combination levothyroxine pl