ライフサイエンスコーパス: primary motor cortex

1 cal circuit (i.e., primary sensory cortex to primary motor cortex).

2 the larynx and the jaw muscles in the human primary motor cortex.

3 nhancements triggered by gamma tACS over the primary motor cortex.

4 edicted by neural activation patterns within primary motor cortex.

5 beta and low gamma desynchronization in the primary motor cortex.

6 tion activity of neurons in the hand area of primary motor cortex.

7 provide such a view from dorsal premotor and primary motor cortex.

8 vergence of sensory information in the mouse primary motor cortex.

9 unctional responses in the thumb area of the primary motor cortex.

10 ptic motor-evoked potential evoked by TMS of primary motor cortex.

11 or how preparatory activity is attenuated in primary motor cortex.

12 ng control region analogous to human layer 5 primary motor cortex.

13 ndent LTP-like and LTD-like effects in human primary motor cortex.

14 y modulating neural activity patterns in the primary motor cortex.

15 lysis of pERK expression in the striatum and primary motor cortex.

16 ed connectivity between the thalamus and the primary motor cortex.

17 tial relationships and its projection to the primary motor cortex.

18 h transcranial magnetic stimulation over the primary motor cortex.

19 lum and basal ganglia, with decreases in the primary motor cortex.

20 acaque monkeys whilst TMS was performed over primary motor cortex.

21 with one of the stimulation protocols to the primary motor cortex.

22 ast conducting transcortical pathway via the primary motor cortex.

23 tomic location of the hand-motor knob in the primary motor cortex.

24 on) explored through paired-pulse TMS of the primary motor cortex.

25 rm potentiation (LTP)-like effect within the primary motor cortex.

26 citability and GABA synaptic activity in the primary motor cortex.

27 ery of motor function after injury in monkey primary motor cortex.

28 l movements were uniquely represented in the primary motor cortex.

29 that only single elements are represented in primary motor cortex.

30 y been shown to reflect motor control in the primary motor cortex.

31 tability and physiological inhibition in the primary motor cortex.

32 creases BDNF in the nigrostriatal system and primary motor cortex.

33 arget selection) altered neural responses in primary motor cortex ~65 ms after the limb disturbance,

34 1) suprathreshold TMS over the contralateral primary motor cortex 70 ms prior to their mean response

35 tivities of layer 5 pyramidal neurons in the primary motor cortex, a cortical region important for th

36 l betweenness centrality of the ipsilesional primary motor cortex, a measure that characterizes the i

37 We studied the influence of primary motor cortex activity on primary somatosensory c

38 A recent study in which primary motor cortex activity was imaged with sub-lamina

39 hat require both vibrissal sensory input and primary motor cortex activity.

40 ich we show that disrupting left PPC but not primary motor cortex after learning has been allowed to

41 ease in connectivity between the putamen and primary motor cortex after levodopa intake during moveme

42 The primary motor cortex allows independent control of joint

43 ely correlated with cortical excitability in primary motor cortex and are predicted by motor tic seve

44 other interconnected structures, such as the primary motor cortex and cerebellum, as well as the cont

45 question, we recorded neural activity in the primary motor cortex and dorsolateral striatum during re

46 Furthermore, inactivation of the primary motor cortex and dorsolateral striatum had disti

47 -related cortical activity in the stimulated primary motor cortex and functionally interconnected reg

48 e found that large numbers of neurons in the primary motor cortex and in several motor areas on the m

49 us, and raphe nuclei (phase II), followed by primary motor cortex and precerebellar nuclei (phase III

50 ty between the pre-supplementary motor area, primary motor cortex and putamen when patients suppresse

51 t psALS) compared with controls in the right primary motor cortex and right caudate.

52 Unaffected siblings showed abnormal primary motor cortex and supplementary motor area co-act

53 odes bilaterally, and especially between the primary motor cortex and supplementary motor area.

54 tients showed increasing coactivation of the primary motor cortex and supplementary motor area.

55 tonia group showed abnormal increases in the primary motor cortex and thalamus compared with controls

56 coupling of beta-phase to gamma-amplitude in primary motor cortex and that deep brain stimulation fac

57 We investigated coherence between primary motor cortex and the dorsal striatum as rats lea

58 anscranial magnetic stimulation (TMS) of the primary motor cortex and the measurement of motor evoked

59 tor cortices (P < 0.05) and between the left primary motor cortex and the right premotor area (P < 0.

60 of 40-100 Gy to 5-7.5 mm fields in the left primary motor cortex and the right subcortical white mat

61 in the rostral versus caudal regions of the primary motor cortex and to its underlying somatotopic o

62 ar proximal arm muscle or isolated neuron in primary motor cortex and two targets were placed so that

63 Most work to date has focused on primary motor cortex, and although there is ample eviden

64 ion to either the dorsolateral prefrontal or primary motor cortex, and induced motor skill improvemen

65 e supplementary motor area, premotor cortex, primary motor cortex, and midcingulate cortex, as well a

66 to either the dorsolateral prefrontal or the primary motor cortex, and neither memory was impaired.

67 d increased metabolism in the preoptic area, primary motor cortex, and the amygdala, and decreased me

68 trality in the anterior cingulate cortex and primary motor cortex; and (iii) abnormal CSF phosphoryla

69 premotor cortices and those associated with primary motor cortex are differentially affected in prim

70 a (area 45), frontoinsular cortex (area FI), primary motor cortex (area 4), primary auditory cortex (

71 y transcranial magnetic stimulation over the primary motor cortex arrive at corticospinal-motor neuro

72 This mechanism supports a viewpoint of primary motor cortex as a site of dynamic integration fo

73 utput from supplementary motor area and left primary motor cortex as the source of signal modificatio

74 e thalamus, entrains the excitability of the primary motor cortex, as reflected by the phase-amplitud

75 ncies (theta, alpha, beta, and gamma) on the primary motor cortex at rest and during motor imagery.

76 regions a unique activation was found in the primary motor cortex (BA4) at MNI coordinates -38 -26 56

77 ntly greater brain activity than controls in primary motor cortex (BA4), supramarginal gyrus (BA40),

78 excitatory coupling between the thalamus and primary motor cortex (Bayesian model selection; winning

79 How can neural activity in the primary motor cortex become dissociated from the movemen

80 V pyramidal and gigantopyramidal neurons in primary motor cortex between 11 carnivore and 9 primate

81 We show that: (i) primary motor cortex broadband gamma power is increased

82 he bilateral hand amputation, we studied the primary motor cortex by using combined task and resting

83 f a brain-computer interface, neurons in the primary motor cortex can be intensely active even though

84 citability in both the angular gyrus and the primary motor cortex caused the reported time of conscio

85 e function of giant pyramidal neurons of the primary motor cortex ('cells of Betz') with the cortical

86 response of the iCSP from the contralesional primary motor cortex (cM1) hand/arm area to spinal level

87 parietal cortex, dorsal premotor cortex, and primary motor cortex contralateral to the acting hand.

88 excitability, a mechanism through which the primary motor cortex controls motor output, is unknown.

89 suggesting that a tonic inhibition state in primary motor cortex could prevent the affordance from p

90 l recordings from the forelimb region of the primary motor cortex demonstrated reduced, training-indu

91 models that can emulate changes seen in the primary motor cortex during learning.

92 s) showed sequential activation in the mouse primary motor cortex during motor skill learning.

93 rea (preSMA), subthalamic nucleus (STN), and primary motor cortex during response inhibition.

94 TMS of the primary motor cortex elicits a sequence of TMS-evoked EE

95 areas, with the most pronounced loss in the primary motor cortex, especially in lateral primary moto

96 We found that neurons in primary motor cortex exhibited a change in the amplitude

97 was revealed: whereas only the contralateral primary motor cortex exhibited unique patterns for each

98 ioglossus (GG) muscles within the rat's face primary motor cortex (face-M1) and adjacent face primary

99 loring the neuroplastic capacity of the face primary motor cortex (face-M1) and adjacent primary soma

100 Hz local field potential oscillations in the primary motor cortex following levodopa treatment.

101 eurostimulation therapies in addition to the primary motor cortex for patients who do not respond ade

102 whereas stimulation of a control region-the primary motor cortex-had no effect on adaptive control.

103 the activity of corticospinal neurons in the primary motor cortex hand area during the use of a tool

104 However, for rats, only the primary motor cortex has been well described.

105 troencephalography (EEG) recordings over the primary motor cortex have been associated with the prepa

106 oth at rest and during a movement task; (ii) primary motor cortex high beta (20-30 Hz) power is incre

107 e role of subcomponents other than that from primary motor cortex, however, is not well understood.

108 ve neuronal stimulations in the ipsilesional primary motor cortex (iM1) can promote functional recove

109 Here we show that inactivation of the mouse primary motor cortex impairs both the acquisition and ex

110 tical inhibition (SICI) of the contralateral primary motor cortex in a sample of 64 healthy right-han

111 determining how striatal activity modulates primary motor cortex in awake head-restrained mice.

112 increase in activity within the ipsilesional primary motor cortex in patients with a wide range of di

113 A recent study demonstrates involvement of primary motor cortex in task-dependent modulation of rap

114 The role of the primary motor cortex in the formation of a comprehensive

115 tructural and functional organisation of the primary motor cortex in the human brain.

116 etitive microstimulation (200 Hz, 500 ms) of primary motor cortex in two rhesus monkeys during perfor

117 To explore the plasticity of the primary motor cortex in upper-extremities amputees and t

118 ndary somatosensory cortex, premotor cortex, primary motor cortex, insula and posterior parietal cort

119 one of two targets by modulating activity in primary motor cortex irrespective of physical movement.

120 The primary motor cortex is a critical node in the network o

121 on's disease, the LTP-like plasticity of the primary motor cortex is impaired, and gamma oscillations

122 Whether and how the primary motor cortex is involved in modulating freezing

123 ear whether the ipsilateral or contralateral primary motor cortex is involved in turning the head rig

124 The primary motor cortex is known to participate in the plan

125 area of the ventral premotor cortex, and the primary motor cortex is necessary for transforming visua

126 While a tumour in or abutting primary motor cortex leads to motor weakness, how tumour

127 ng revealed increased activation in the left primary motor cortex leg area during handgrip and the le

128 ces recovery of function after injury of the primary motor cortex, likely through enhancing plasticit

129 c movement task to reveal cyclic activity in primary motor cortex locked to submovements, and a disti

130 non-discrete premotor signals that drive the primary motor cortex M1 to reflect the movement of the g

131 imary S1 and secondary S2 somatosensory, and primary motor cortex M1) was estimated.

132 y applied iTBS blocks of 600 pulses over the primary motor cortex (M1 stimulation) and the parieto-oc

133 thought to cause dyskinesia, alterations in primary motor cortex (M1) activity are also prominent du

134 Blocking primary motor cortex (M1) activity unilaterally during a

135 Primary motor cortex (M1) almost exclusively controls th

136 d by slower components thought to arise from primary motor cortex (M1) and other supraspinal areas.

137 Here we asked how both primary motor cortex (M1) and PMd represented reach dire

138 Here, we report that the primary motor cortex (M1) and primary somatosensory cort

139 omatomotor areas such as contralateral S1BC, primary motor cortex (M1) and secondary somatosensory co

140 vodopa on induced power in the contralateral primary motor cortex (M1) and STN and on the coherence b

141 We studied movement encoding across the primary motor cortex (M1) and supplementary motor area (

142 sponses to stimulation of different parts of primary motor cortex (M1) and supplementary motor area (

143 a record of the activity in the areas of the primary motor cortex (M1) and the secondary motor cortex

144 inhibitory neurotransmitter GABA within the primary motor cortex (M1) and the strength of functional

145 scranial magnetic stimulation (TMS) over the primary motor cortex (M1) appear to activate distinct in

146 EMENT Connections between the cerebellum and primary motor cortex (M1) are essential for performing d

147 es in the movement-related activation of the primary motor cortex (M1) are thought to be a major cont

148 of local field potentials recorded from the primary motor cortex (M1) arm area in patients undergoin

149 The traditional classification of primary motor cortex (M1) as an agranular area has been

150 We examined neural activity in SMA and primary motor cortex (M1) as monkeys cycled various dist

151 transcranial magnetic stimulation applied to primary motor cortex (M1) at different coil orientations

152 lternating current stimulation (tACS) to the primary motor cortex (M1) at different frequencies durin

153 h transcranial magnetic stimulation (TMS) to primary motor cortex (M1) at specific intervals to induc

154 The proximal representation in the primary motor cortex (M1) controls the arm for reaching,

155 lectrical microstimulation indicate that the primary motor cortex (M1) directly regulates muscle cont

156 orsal premotor cortex onto the contralateral primary motor cortex (M1) during the preparation of a co

157 Classical interpretations of the function of primary motor cortex (M1) emphasize its lack of the gran

158 ents, and communicates those programs to the primary motor cortex (M1) for execution.

159 In primates the primary motor cortex (M1) forms a topographic map of the

160 Sensory inputs reaching the primary motor cortex (M1) from the somatosensory cortex

161 ial direct current stimulation (tDCS) of the primary motor cortex (M1) has been found to increase the

162 alternating current stimulation (tACS) over primary motor cortex (M1) has opposite effects on motor

163 l direct current stimulation (tDCS) over the primary motor cortex (M1) has resulted in improved perfo

164 stimulation (TDCS) of the cerebellum and the primary motor cortex (M1) have been found to improve vis

165 Corticomotoneuronal (CM) cells in the primary motor cortex (M1) have monosynaptic connections

166 imulus intracortical microstimulation of the primary motor cortex (M1) in awake animals failed to pro

167 of evidence point to a critical role of the primary motor cortex (M1) in consolidation.

168 ntracortical microstimulation (HFLD-ICMS) to primary motor cortex (M1) in primates produces hand move

169 er, the neural activity patterns and role of primary motor cortex (M1) in these early movements are s

170 The arm region in the primary motor cortex (M1) is assumed to control reaching

171 The primary motor cortex (M1) is highly influenced by premot

172 r movements in M1.SIGNIFICANCE STATEMENT The primary motor cortex (M1) is important for producing ind

173 synaptic inputs engage pyramidal neurons in primary motor cortex (M1) is important for understanding

174 To examine whether primary motor cortex (M1) is involved in maintenance of

175 in macaque ventral premotor cortex (PMv) and primary motor cortex (M1) is modulated by the observatio

176 The primary motor cortex (M1) is strongly influenced by seve

177 Plasticity of synaptic connections in the primary motor cortex (M1) is thought to play an essentia

178 ts with PD, STN spiking is synchronized with primary motor cortex (M1) local field potentials in two

179 complex anatomical networks that include the primary motor cortex (M1) of both hemispheres.

180 forelimb muscle and an unrelated site in the primary motor cortex (M1) of macaques.

181 we analyzed neuronal activity recorded from primary motor cortex (M1) of monkeys performing a 3D arm

182 etween the posterior parietal cortex and the primary motor cortex (M1) of the left-dominant hemispher

183 l direct current stimulation (TDCS) over the primary motor cortex (M1) or the lateral cerebellum can

184 How is the primary motor cortex (M1) organized to control fine fing

185 nd movements is by modulating or shaping the primary motor cortex (M1) outputs to hand muscles.

186 ent intensity between 0.5 and 2.0 mA on left primary motor cortex (M1) plasticity, as well as the imp

187 suggested that antidromic activation of the primary motor cortex (M1) plays a significant role in me

188 The primary motor cortex (M1) possesses a functional somatot

189 Here we analyze data from invasive human primary motor cortex (M1) recordings from patients with

190 CANCE STATEMENT Previous work has shown that primary motor cortex (M1) represents information related

191 Evidence is accumulating that neurons in primary motor cortex (M1) respond during action observat

192 esults show that during the belief of agency primary motor cortex (M1) shows stronger functional conn

193 The whisker primary motor cortex (M1) strongly innervates brain stem

194 Neurons in monkey primary motor cortex (M1) tend to be most active for cer

195 d robustly in the striatum and all layers of primary motor cortex (M1) through a muscarinic-receptor

196 transcranial magnetic stimulation (TMS) over primary motor cortex (M1) to measure corticospinal excit

197 lanted high density microelectrode arrays in primary motor cortex (M1) to record neuronal population

198 is thought to upregulate excitability of the primary motor cortex (M1) using anodal stimulation while

199 elicited from the forelimb representation of primary motor cortex (M1) using this method has not been

200 nnectivity with leads overlying ipsilesional primary motor cortex (M1) was a robust and specific mark

201 g transcranial magnetic stimulation over the primary motor cortex (M1) we examined motor evoked poten

202 stimulation (TMS) over the hand area of the primary motor cortex (M1) when human participants (50% f

203 stimulation (TMS) over the hand area of the primary motor cortex (M1) when humans tracked with the e

204 observed in neuronal population activity in primary motor cortex (M1) when monkeys make reaching mov

205 x, followed by a test stimulus over the left primary motor cortex (M1) with a random interstimulus in

206 (TMS) near ventral premotor cortex (PMv) and primary motor cortex (M1) with a short 8-ms inter-pulse

207 Inactivation of the primary motor cortex (M1) with muscimol affected anticip

208 the motor hotspot (thought to represent the primary motor cortex (M1)) to modulate motor network exc

209 were first measured after stimulation of the primary motor cortex (M1), corticospinal tract (CST), an

210 rsal striatum, which receives input from the primary motor cortex (M1), followed a similar age progre

211 CSP) from the hand/arm representation of the primary motor cortex (M1), high-resolution anterograde t

212 y that imagined movements directly influence primary motor cortex (M1), how this might occur remains

213 ere present in the pars opercularis IFG/PMv, primary motor cortex (M1), IPL/supramarginal gyrus and m

214 After subtotal infarcts of primary motor cortex (M1), motor rehabilitative training

215 sis by disrupting neural activity in SMA, in primary motor cortex (M1), or in a control site by means

216 grade tracers into grasp zones identified in primary motor cortex (M1), PMv, and area 2 with long tra

217 reas including anterior parietal areas, from primary motor cortex (M1), premotor cortex (PM), the sup

218 In the primary motor cortex (M1), residual subperceptual hand t

219 tal areas convey the necessary signal to the primary motor cortex (M1), the cortical site where the f

220 These cortical areas include the primary motor cortex (M1), the rostromedial motor area (

221 Two sites were stimulated [contralateral primary motor cortex (M1), vs ipsilateral cerebellum] wh

222 y in the motor network and, specifically, in primary motor cortex (M1), which results from overall di

223 V), secondary somatosensory cortex (S2), and primary motor cortex (M1), with similar distributions in

224 d the connectivity pattern of the stimulated primary motor cortex (M1), without changing overall loca

225 No changes were observed in the primary motor cortex (M1).

226 s have a complete body representation in the primary motor cortex (M1).

227 e prefrontal (PFC), somatosensory (SS2), and primary motor cortex (M1).

228 datasets from primary visual cortex (V1) and primary motor cortex (M1).

229 itions: left or right DLPFC or left or right primary motor cortex (M1).

230 o synaptic plasticity and E/I balance in the primary motor cortex (M1).

231 c stimulation delivered to the contralateral primary motor cortex (M1).

232 a burst stimulation (iTBS) is reduced in the primary motor cortex (M1).

233 ations of labeled cells included area 3a and primary motor cortex (M1).

234 the posterior parietal cortex (PPC) and the primary motor cortex (M1).

235 Md or LPF and a single pulse TMS (sTMS) over primary motor cortex (M1).

236 odulations of large groups of neurons in the primary motor cortex (M1).

237 ent on changes in local circuitry within the primary motor cortex (M1).

238 In our case, the target was the primary motor cortex (M1).

239 logical processes in the cerebellum (CB) and primary motor cortex (M1).

240 t of motor skills modify the activity of the primary motor cortex (M1)?

241 silateral PMRF (115 cells) and contralateral primary motor cortex (M1, 210 cells).

242 imulation over the leg representation of the primary motor cortex, maximal voluntary contractions (MV

243 imulation over the arm representation of the primary motor cortex, maximal voluntary contractions, th

244 model to predict single neuron responses in primary motor cortex (MI) during a reach-to-grasp task b

245 m: the dorsal premotor cortex (PMd, area 6), primary motor cortex (MI, area 4), and posterior parieta

246 rtical loop in PD in which gamma activity in primary motor cortex, modulated by the phase of low-freq

247 ive projection intensity for cases targeting primary motor cortex (MOp), primary somatosensory cortex

248 ulation, guided by functional mapping of the primary motor cortex, naive participants were awakened f

249 During spindles, primary motor cortex neurons fired at a preferred phase,

250 eparatory activity of dorsal premotor cortex/primary motor cortex neurons in monkey exhibits similar

251 ly corticospinal axons originating in caudal primary motor cortex ("new M1") and area 3a make monosyn

252 We thus recorded activity from the primary motor cortex of 4 male sleeping rats to investig

253 paired associative stimulation (PAS) at the primary motor cortex of healthy humans.

254 In the primary motor cortex of PD patients, neuronal population

255 al field potential (LFP) activity within the primary motor cortex of three cats during a prism visuom

256 ng-term recordings of neural activity in the primary motor cortex of two female nonhuman primates dur

257 t decreases in basal glutamate levels in the primary motor cortex on the side ipsilateral to the MPTP

258 ster scaling of the number of neurons in the primary motor cortex over the brainstem and spinal cord

259 motor area and functional anticorrelation to primary motor cortex (p < 0.001).

260 ementary motor area (Pcorrected = 0.020) and primary motor cortex (Pcorrected = 0.044), but not feed-

261 inesia is associated with dynamic changes in primary motor cortex physiology, to date, there are no p

262 s on the functional MR imaging signal in the primary motor cortex (PMC), as false-negative blood oxyg

263 e stomach originated overwhelmingly from the primary motor cortex, primary somatosensory cortex, and

264 Transcranial magnetic stimulation over primary motor cortex provided an assay of corticospinal

265 ng of more than 1,300 neurons in adult mouse primary motor cortex, providing a morpho-electric annota

266 tion, respectively, delivered to the area of primary motor cortex representing the hindlimb.

267 primary motor cortex, especially in lateral primary motor cortex, representing the hand and face.

268 ed with song learning in the avian analog of primary motor cortex (robust nucleus of the arcopallium,

269 in three main sources of feedback to rat S1: primary motor cortex, secondary somatosensory cortex, an

270 Primary motor cortex slices were prepared enabling simul

271 mated from high-frequency rTMS targeting the primary motor cortex (SMD, 0.77; 95% CI, 0.46-1.08; P<.0

272 s: SMD, 0.23; 95% CI, -0.02 to 0.48, and low primary motor cortex: SMD, 0.28; 95% CI, -0.23 to 0.78)

273 ey nodes in the motor network, including the primary motor cortex, supplementary motor area, premotor

274 3 sessions exploring the excitability of the primary motor cortex, the response of the primary motor

275 etroinsular area, frontal afferents from the primary motor cortex, the supplementary motor area, and

276 aspects of each hemisphere are involved: the primary motor cortex, the ventral lateral premotor corte

277 Furthermore, the primary motor cortex, the ventral lateral premotor corte

278 In the primary motor cortex, there was a significant loss (57%

279 he primary motor cortex, the response of the primary motor cortex to a plasticity-inducing protocol,

280 suggested that indirect connections from the primary motor cortex to forelimb motoneurons, via brains

281 pulse transcranial magnetic stimulation over primary motor cortex to measure long-interval cortical i

282 utions of the cerebellum, basal ganglia, and primary motor cortex to motor learning can begin to be i

283 re was increased intracortical inhibition in primary motor cortex under high working memory load.

284 es of layer V pyramidal neurons in the mouse primary motor cortex using two-photon microscopy.

285 Two different whisker representations in primary motor cortex (vM1) affect whisker movements in d

286 st, broadband gamma (50-200 Hz) power in the primary motor cortex was greater in the DYST-ARM and PD

287 nscranial magnetic stimulation over the left primary motor cortex was used to determine short-latency

288 e BP's late component (BP2, generated in the primary motor cortex) was found to be linked to motor ex

289 al magnetic theta burst stimulation over the primary motor cortex, was still possible, and even favor

290 Within primary motor cortex, we evoked finger movements involvi

291 imulation over the arm representation of the primary motor cortex, we examined ipsilateral motor-evok

292 scranial magnetic stimulation (TMS) over the primary motor cortex, we investigated whether a placebo

293 cortex, and ventral striatum but also in the primary motor cortex well before the response itself.

294 ificantly greater regional blood flow in the primary motor cortex, whereas psychogenic dystonia was a

295 ranial magnetic stimulation applied over the primary motor cortex, which suppresses voluntary drive a

296 anscranial magnetic stimulation (TMS) on the primary motor cortex with an online-navigated TMS-tACS s

297 al tract (CST) neurons in the subdivision of primary motor cortex within the central sulcus ("new M1"

298 we investigate the function of mouse whisker primary motor cortex (wM1), a frontal region defined by

299 rtices, including both the whisker region of primary motor cortex (wMC) and anterior lateral motor co

300 89, p=0.0302) and of grey matter in the left primary motor cortex (Z score 4.23, p=0.041).