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1 increased phloem delivery and growth of the primary root.
2 s responsible for acropetal transport in the primary root.
3 educed vascular development, and a shortened primary root.
4 of information from the lateral roots to the primary root.
5 where lateral roots begin to emerge from the primary root.
6 appear to have reduced cell division in the primary root.
7 g phloem cells that appear to connect to the primary root.
8 nduced by auxin in the basal meristem of the primary root.
9 root primordia and their emergence from the primary root.
10 ed root hair formation and developed shorter primary roots.
11 del of growth-sustaining water potentials in primary roots.
12 nrecognized role in regulating the growth of primary roots.
13 te to different levels in wild-type and rth1 primary roots.
14 ath of cells in the tips of both lateral and primary roots.
15 ngation or metabolism of maize (Zea mays L.) primary roots.
16 vitropic behavior of seedling hypocotyls and primary roots.
17 and elongation zone of Arabidopsis thaliana primary roots.
18 We examined hydrotropism in maize (Zea mays) primary roots.
19 ition in the meristem and elongation zone of primary roots.
20 image the morphology of Medicago truncatula primary roots.
21 on and cell size control in both lateral and primary roots.
22 ral roots and precocious secondary growth in primary roots.
23 CCRC-M1 labels all cell walls at the apex of primary roots 2 d and older and the apices of mature lat
24 of the Arabidopsis WPP family causes shorter primary roots, a reduced number of lateral roots, and re
25 MAD2 loss-of-function mutants have a shorter primary root and a smaller root meristem, and this defec
26 h stimulation in cortex and epidermis of the primary root and a strong repression in LRPs and to a lo
27 development is a traceable process along the primary root and different stages can be found along thi
28 A insertion mutant of LAC2 displayed a short primary root and high lignin deposition in root vascular
31 ts accumulated very low to no auxin in their primary root and LR tips as observed through expression
33 in increases the expression of XBAT32 in the primary root and partially rescues the lateral root defe
34 relate to the gene expression domains in the primary root and suggest that different GLV signals trig
35 hat control the cellular architecture of the primary root and the initiation of new lateral root orga
36 ized Pi sensing that regulates growth of the primary root and therefore delineates it from sugar-depe
37 mutated, prevents the formation of hairs on primary roots and causes a seedling lethal phenotype.
39 ressed in the endodermal and phloem cells of primary roots and in the vascular tissues of leaves, ste
40 tion-related phenotypes, including shortened primary roots and increases in the number and length of
41 ssion of MtNPF6.8 in the pericycle region of primary roots and lateral roots, and in lateral root pri
42 move defined cells in the cap of Arabidopsis primary roots and quantified the response of the roots t
43 sis (Arabidopsis thaliana) mutant with short primary roots and root hairs was identified from a forwa
44 asticity of the proteomic landscape of maize primary roots and thus provide a starting point for gain
46 s of division and differentiation within the primary root, and describe how their cross-regulation ma
48 ular mechanisms that control this process in primary roots, and discuss recent insights into the regu
49 starved wild-type plants also produce longer primary roots, and lateral root growth is suppressed.
50 ts have longer leaves, petioles, hypocotyls, primary roots, and root hairs than wild-type plants, whe
52 he of the Arabidopsis (Arabidopsis thaliana) primary root apical meristem is composed of the quiescen
54 d to defects in endodermal patterning in the primary root arising from impaired timing of periclinal
55 tivates both cell division and elongation in primary roots as well as auxin-responsive and stem cell
56 ctivation of cell division and elongation in primary roots, as well as auxin-responsive and stem cell
57 n a dwarf phenotype in dark-grown shoots and primary roots, as well as the loss of anisotropic cell e
59 ell elongation in the apical region of maize primary roots at low water potentials (psi(w)) was assoc
60 in the growing region of maize (Zea mays L.) primary roots at low water potentials (psiw), largely as
66 3 was expressed in most cells of the growing primary root but was not enriched in the phloem, includi
67 sed abundantly in the vascular system of the primary root, but not in newly formed lateral root primo
69 n factor), suppresses cell elongation of the primary root by activating the cell surface receptor FER
71 s and that these signals are correlated with primary root development, including meristem development
73 indings establish the chemical nature of the primary root-diffusion barrier in Arabidopsis and enable
75 , we show that inactivation of CRK5 inhibits primary root elongation and delays gravitropic bending o
77 we show that depletion of NO in noa1 reduces primary root elongation and increases flavonol accumulat
78 idopsis (Arabidopsis thaliana) by inhibiting primary root elongation and promoting lateral root and r
83 t architecture of these plants is that their primary root elongation is inhibited when grown on P-def
87 ollowing germination, moderate IBA-resistant primary root elongation, and severe defects in IBA-induc
88 , but on high nitrate, arm exhibited reduced primary root elongation, radial swelling, increased numb
89 s study, the inhibitory effect of nitrate on primary root elongation, via inhibition of elongation of
92 in specifying the three main regions of the primary root (elongation, transition and division zones)
96 an growth rate, we used Arabidopsis thaliana primary roots grown vertically at 20 degreesC with an el
97 nitrogen source, nitrate, acting to suppress primary root growth (vertical dimension) in concert with
99 ro grown plants resulted in an inhibition of primary root growth and a proliferation of lateral and a
104 condition but shows increased inhibition of primary root growth and enhanced production of lateral r
106 HMT1) in a PPSB-deficient background resumed primary root growth and induced a change in the plant me
107 e constitutive effect of the arm mutation on primary root growth and its conditional impact on root a
108 tational analysis showed a role for TET13 in primary root growth and lateral root development and red
109 t growth and a reduced sensitivity to ABA on primary root growth and lateral root formation compared
110 miRNA-deregulated) showed less inhibition of primary root growth and less induction of a Pi transport
113 y thus indicates that ZAT6 is a repressor of primary root growth and regulates Pi homeostasis through
115 Stomatal closure, seed germination, and primary root growth are well-known ABA responses that we
117 d PDGLP2 appear to be involved in regulating primary root growth by controlling phloem-mediated alloc
118 HRE2 inhibits root bending, suggesting that primary root growth direction at hypoxic conditions is a
120 while cytokinin treatment repressed further primary root growth in Arabidopsis plants under As(III)
126 psis, an iron-dependent mechanism reprograms primary root growth in response to low Pi availability.
129 ion factor and a close homologue repress the primary root growth in response to P deficiency conditio
130 moted by traits that synergistically allowed primary root growth in seedlings, the expansion of xylem
131 reased lateral root initiation and inhibited primary root growth in the transformants at 10 pM, sever
133 gene expression, seed germination arrest and primary root growth inhibition) compared with ABI5 expre
136 hermore, we show that increased pearl millet primary root growth is correlated with increased early w
142 ed increased root hair formation and reduced primary root growth that could be rescued by the applica
143 exogenous auxin and increased sensitivity of primary root growth to exogenous auxin, indicates that T
144 ced hypocotyl length in red light, inhibited primary root growth under different light conditions, an
146 silon also promoted root hair elongation and primary root growth under severe nitrogen deprivation.
148 teractions of Pi and Fe availability control primary root growth via meristem-specific callose format
150 d type, but no change in auxin inhibition of primary root growth was observed, suggesting that PGGT I
151 f root cap turnover may therefore coordinate primary root growth with root branching in order to opti
152 nsitivity to cold treatment as determined by primary root growth, biomass, and reactive oxygen specie
154 starvation responses, including cessation of primary root growth, extensive lateral root and root hai
155 urce allocation is shifted from secondary to primary root growth, genetic variation exists for this r
156 r high auxin levels, including inhibition of primary root growth, induction of root hairs, and promot
157 altered auxin homeostasis including altered primary root growth, lateral root development, and root
158 lthough auxin supplementation also inhibited primary root growth, loss of meristematic activity was o
160 llow root system architecture (RSA), reduces primary root growth, root apical meristem size, and meri
161 eral root development, as well as defects in primary root growth, root hair initiation, and root hair
162 by rag1 seedlings includes reduced shoot and primary root growth, root tip swelling, and increased la
163 concentrations of Cr (20-40 microM) promoted primary root growth, while concentrations higher than 60
171 interplay between this periodic process and primary root growth; yet, much about this oscillatory pr
173 ergence of new lateral roots from within the primary root in Arabidopsis has been shown to be regulat
174 fluenced the longitudinal growth rate in the primary root in response to Pi deprivation, whereas RGF1
178 ateral root numbers (total lateral roots per primary root) in the mutants to twice the number in the
180 h altered auxin physiology, including longer primary roots, increased number of lateral roots, and in
181 brassinosteroids in hypocotyl elongation and primary root inhibition assays, but it did retain sensit
183 s mutant, root UVB sensitive 1 (rus1), whose primary root is hypersensitive to very low-fluence-rate
184 ablish the spatial expression of LATD/NIP in primary root, lateral root and nodule meristems and the
185 normal function of three meristems, i.e. the primary root, lateral roots and nitrogen-fixing nodules.
186 XS3/4/5/8) resulted in plants with increased primary root length (approximately 25% longer than the w
188 he P-efficient parent Ningyou7 had a shorter primary root length (PRL), greater lateral root density
189 ty were found to have contrasting effects on primary root length and lateral root density, but simila
190 ation, whereas there were reductions in both primary root length and lateral root number in 12-d-old
191 Knockout mutants for athb13 showed increased primary root length as compared with wild-type (Columbia
192 ination rates, survival rates, and increased primary root length compared to control plants under dro
193 iple mutant plants showed markedly increased primary root length compared with wild-type plants.
197 phenotypes [i.e., reduced apical dominance, primary root length, lateral root emergence, and growth;
198 genic tobaccos were observed for increase in primary root length, number of lateral roots, chlorophyl
201 race tool has proved successful in measuring primary root lengths across time series image data.
202 e dwarfness, dark green curled leaves, short primary roots, less lateral roots, and insensitive to ex
203 n increased auxin response including shorter primary roots, longer root hairs, longer hypocotyls, and
204 ated pickle (pkl) were isolated in which the primary root meristem retained characteristics of embryo
205 (2) accumulation to the surface cells of the primary root meristem, (ii) demonstrate the accumulation
207 ethyl arginine reduce the mitotic indices of primary root meristems and inhibit lateral root elongati
209 plays an increased root growth angle, longer primary roots, more adventitious roots and greater nutri
210 multicellular plant tissues, focusing on the primary root of Arabidopsis (Arabidopsis thaliana).
217 ed in the ability to initiate nodules on the primary root of the host plant, Medicago truncatula, ind
218 duced length of the root apical meristem and primary root of the mutant ashr3-1 indicate that synchro
221 s of auxin redistribution across the caps of primary roots of 2-day-old maize (Zea mays, cv Merit) se
222 ck auxin transport and gravitropic growth in primary roots of Arabidopsis (Arabidopsis thaliana).
223 3-acetic acid in both hypocotyl sections and primary roots of Arabidopsis seedlings was measured.
224 morphogenesis, we examined the morphology of primary roots of Arabidopsis thaliana and the organizati
225 ed PCD (TUNEL staining and DNA laddering) in primary roots of both Arabidopsis thaliana wild type (Co
228 pattern of longitudinal surface extension in primary roots of maize (Zea mays L.) upon application an
231 epress expression of embryonic traits in the primary roots of pkl seedlings, whereas activation of PK
233 trong knock down of rth6 expression in young primary roots of the mutant rth6, the gene is also signi
236 is correlated with differential elongation, primary roots of Zea mays cv Merit maintained vertically
237 to measure the differential contribution of primary root pericycle cell files to developing lateral
238 from proliferation to differentiation in the primary root, plays a new role in controlling LRP develo
239 during initiation, sometimes encircling the primary root prior to growth in a normal downward direct
240 However, the mechanisms that regulate the primary root response to Pi-limiting conditions remain l
241 ualize the dynamic behavior of nuclei within primary roots, root hairs, and pollen tubes, which are t
243 -TGA and HYPERSENSITIVITY TO LOW PI-ELICITED PRIMARY ROOT SHORTENING1 (HRS1)/HRS1 Homolog family, whi
244 the growth biophysics of maize (Zea mays L.) primary roots suggested that cell walls in the apical 5
245 major axis [i.e. brace, crown, seminal, and primary roots]), suggesting that LRBD has varying utilit
246 study these interactions the proteome of the primary root system of the maize (Zea mays L.) lrt1 muta
248 t development, as trm4b mutants have shorter primary roots than the wild type due to reduced cell div
249 t hypoxia triggers an escape response of the primary root that is controlled by ERFVII activity and m
250 n early adaptive response of the Arabidopsis primary root that precedes the entrance of the meristem
251 ripts are elevated more than 100-fold in pkl primary roots that inappropriately express embryonic tra
256 ponse depended on direct contact between the primary root tip and the NO(3)(-), and was not elicited
258 e study showed that CHL1 is activated in the primary root tip early in seedling development and at th
259 pear to be normal, delivery of phloem to the primary root tip is limited severely in young seedlings.
260 esence of exogenous l-glutamate (Glu) at the primary root tip slows primary root growth and stimulate
262 ence of a NO(3)(-) signalling pathway at the primary root tip that can antagonise the root's response
263 lings, its expression is concentrated in the primary root tip where it activates the gamma-glutamyl c
268 ow Pi medium, the quiescent center domain in primary root tips increases as an early response, as rev
270 rt is aligned with the gravity vector in the primary root tips, facilitating downward organ bending a
271 d in a range of tissues and cells, including primary root tips, root vascular tissue, hydathodes, and
272 cription and probably mRNA stability both in primary root tissues and in LRPs, it acts differentially
274 osphoproteome atlas of four maize (Zea mays) primary root tissues, the cortex, stele, meristematic zo
275 tracted to the simple radial organization of primary root tissues, which form a series of concentric
277 ot systems build in complexity from a single primary root to a network of thousands of roots that col
278 y, we show that hypoxic conditions cause the primary root to grow sidewise in a low oxygen environmen
279 pply of Fe to Cr-treated Arabidopsis allowed primary root to resume growth and alleviated toxicity sy
281 stimulates pericycle cells within elongating primary roots to enter de novo organogenesis, leading to
283 s work on the adaptation of maize (Zea mays) primary roots to water deficit showed that cell elongati
284 both newly emerged lateral roots and in the primary root, ultimately resulting in the selective deat
285 17 and their reciprocal F1 hybrid progeny in primary roots under control and water deficit conditions
286 ior to the determinate growth program of the primary roots under low Pi and unveil a critical functio
289 cation of epidermal cell fate in Arabidopsis primary roots we have isolated 8 new mutants that fall i
290 Maize (Zea mays L.) seedlings with 5-cm primary roots were exposed to anoxic (0% [v/v] O2), hypo
292 s in the accumulation of auxin in the tip of primary root, whereas loss-of-function mutations in thes
293 olved in responses to gravity stimulation in primary roots, whereas on the other, FLP and MYB88 funct
294 expressed in epidermal and cortical cells of primary roots, whereas the TUB8 chimeric gene was prefer
295 y root system features a single fast-growing primary root which we hypothesize is an adaptation to th
296 luorescent protein construct was measured in primary roots whose apyrase expression was suppressed ei
298 tinuous light, and seedlings develop shorter primary roots with an increased number of lateral roots
299 ium is reduced, we observe elongation of the primary root without an increase in P availability or a