ライフサイエンスコーパス: prolactin

1 only by a very high dose of unlabeled ovine prolactin.

2 rons that normally suppress the secretion of prolactin.

3 likely mediated by elevations of the hormone prolactin.

4 wo independent BCL6 small hairpin RNAs or by prolactin.

5 the nursing mother is induced by the hormone prolactin.

6 turn, serotonin stimulates the secretion of prolactin.

7 for epigenetically regulated genes, such as prolactin.

8 poral reduction in Pit1, growth hormone, and prolactin.

9 and in human islets treated with recombinant prolactin.

10 by investigating transport of (125)I-labeled prolactin ((125)I-prolactin) into the brain of female mi

11 [3.19] vs 4.56 [2.01] mIU/mL; P < .001), and prolactin (14.14 [9.48] vs 9.97 [3.12] ng/mL; P = .01) a

12 o model to characterize the effects of DHEA, prolactin, 17beta-estradiol on insulin-growth factor-1 a

13 phere), CA-125 (53 mum diameter sphere), and prolactin (63 mum diameter sphere) in a single PBS assay

14 Prolactin, a key regulator of 20alpha-HSD, was lower (P=

15 udy also demonstrated that AFAP1 responds to prolactin, a lactogenic hormone, by forming a complex wi

16 tly reduced the concentration of circulating prolactin, a pituitary hormone influencing maternal care

17 is circuit markedly reduced the capacity for prolactin action both in the MPOA and throughout the net

18 Here, we evaluated the role of prolactin action in the MPOA using complementary genetic

19 l mothers abandoned their pups, showing that prolactin action on MPOA neurons is necessary for the no

20 nduced phosphorylation of STAT5, a marker of prolactin action.

21 Compliant matrices promoted physiological prolactin actions and activation of STAT5, whereas stiff

22 We report here for the first time that prolactin activates extracellular signal-regulated kinas

23 Prolactin also suppressed 3-ketosteroid induction of CK5

24 ignificantly reduced secretion of IGFBP1 and prolactin and altered the expression of endometrial rece

25 that enables inducible hepatic production of prolactin and its cleavage product.

26 Prolactin and placental lactogen signal through the prol

27 tudies investigating the association between prolactin and type 2 diabetes beyond pregnancy are rare

28 ment of endometrial decidualization (IGFBP1, prolactin) and c) endometrial receptivity (SPP1, MAOA, E

29 es decreased the expression of Pit1, Zbtb20, prolactin, and growth hormone, while its overexpression

30 driver pathways, including Wnt/beta-catenin, Prolactin, and insulin-like growth factor (IGF)1 signali

31 correlates of severity, including JAK/STAT, prolactin, and interleukin 9 signaling.

32 We assessed plasma levels of progesterone, prolactin, and lipids and placental expression of genes

33 Across multiple human cancers, COX-2, prolactin, and prolactin receptor show consistent differ

34 estosterone, dehydroepiandrosterone sulfate, prolactin, and sex hormone-binding globulin (SHBG) impro

35 ted hormones: estrone sulfate, testosterone, prolactin, and SHBG; change in AUC, 8.8 [P < .001] for G

36 t inhibition of JAK2 significantly decreases prolactin- and interferon gamma (IFN-gamma)-induced tyro

37 ons of the ECM to the physiologic actions of prolactin are increasingly understood, little is known a

38 e orphan nuclear receptor NR4A (Nur77), with prolactin as a major transcriptional target for the NR4A

39 Collectively, these data unveiled prolactin as a retinal trophic factor that may regulate

40 tiangiogenic fragment of the nursing hormone prolactin as playing an important role.

41 We aimed to examine whether prolactin associates with type 2 diabetes prospectively

42 androgen activity) correlated with serotonin/prolactin, but posited aromatase activity correlated sig

43 Secretion of the ligand prolactin by adjacent lung stromal cells is induced by t

44 Prolactin caused no significant change in IGF-1 levels a

45 Tg mice were fed chow containing IC3, plasma prolactin concentrations increased threefold, BP increas

46 Therefore, serum prolactin concentrations reflect endogenous serotonin.

47 In the lactating women only, higher prolactin concentrations were associated with greater su

48 he Tg mice were fed normal chow (NC), plasma prolactin concentrations were comparable to those in fem

49 Prolactin controls the development and function of milk-

50 nsulin-like growth factor binding protein-3, prolactin, cortisol, adrenocorticotropic hormone, thyroi

51 Here we report that prolactin counteracts induction of the CK5-positive popu

52 However, the mechanism by which prolactin crosses the blood-brain barrier and enters the

53 yroid stimulating hormone, gonadotropin, and prolactin deficiencies, whereas for ACTH, growth hormone

54 Trpc5 mutant females show severe prolactin deficiency or hypoprolactinemia that is associ

55 activation, progesterone receptor activity, prolactin effects, and aspects of estrogen receptor beta

56 rrelated these properties with their EPS and prolactin-elevating liabilities at therapeutic doses.

57 ne) to 3.21 kg (2.10 to 4.31; zotepine), for prolactin elevation (21 569 participants) from -77.05 ng

58 tor, while dissociation rates correlate with prolactin elevation.

59 Trpc5 is also required for eliciting prolactin-evoked tonic plateau potentials in these neuro

60 The anterior pituitary hormone prolactin exerts important physiologic actions in the br

61 ere some differentiate into nonhematopoietic prolactin-expressing decidual cells.

62 High mobility group box 1-induced prolactin expression in the pancreas maintained FAK1 and

63 h a strong transcriptional activation of the prolactin family 2 subfamily c of growth factors.

64 s evidenced by significantly lower levels of prolactin, fasting glucose, total cholesterol, and trigl

65 ne (LH), follicle-stimulating hormone (FSH), prolactin, fasting plasma glucose, and insulin levels we

66 cl-2, HER2, p53, E-cadherin, Ki67, survivin, prolactin, FOXA1) for survival impact.

67 M is driven in part by the 16-kDa N-terminal prolactin fragment (16K PRL), the underlying molecular m

68 et al. explore signaling downstream of this prolactin fragment and demonstrate that miR-146a is a cr

69 y inhibit the release of the protein hormone prolactin from lactotropic cells in the anterior pituita

70 at CSF is not the major route by which blood prolactin gains access to neurons in the brain.

71 a in the vicinity of an integrated multicopy prolactin gene array.

72 ctions allowed local spatial coordination of prolactin gene expression.

73 -imaging, the authors recently revealed that prolactin gene transcription is highly dynamic and stoch

74 ith an anti-Prl antibody, or deletion of the prolactin gene.

75 We have shown previously that the murine prolactin/growth hormone family member proliferin plays

76 unknown role for the cation channel Trpc5 in prolactin homeostasis of female mice and provide strateg

77 uitary gland and thus play a central role in prolactin homeostasis of the body.

78 normal function of dopamine ARC neurons and prolactin homeostasis.

79 nfluramine significantly increased serotonin/prolactin in all groups (p < .0001).

80 resents a novel prodifferentiation effect of prolactin in breast cancer.

81 s, which may be explained by lower levels of prolactin in males.

82 k sodium influx, consistent with the role of prolactin in osmoregulation.

83 periments demonstrated the essential role of prolactin in promoting collagen deposition and fibrosis.

84 n hypothesized that the receptor may bind to prolactin in the blood and translocate it into the cereb

85 Fenfluramine-induced serotonin/prolactin in the T-treated group was significantly highe

86 for maternal behavior, the specific role of prolactin in this brain region has remained elusive.

87 Prolactin, in turn, orchestrates numerous important biol

88 In stiff matrices, prolactin increased SRC family kinase-dependent phosphor

89 Prolactin increases proliferation and cell motility, pro

90 In vivo mouse studies found that local prolactin induced the amplification of prostate epitheli

91 , downstream signaling mechanisms underlying prolactin-induced adult neurogenesis are completely unkn

92 and critical signaling mechanism underlying prolactin-induced adult neurogenesis.

93 Our data establish a critical role for prolactin-induced behavioral responses in the maternal b

94 tivator of Transcription (STAT) 5A/B mediate prolactin-induced mammary development during pregnancy.

95 r, non-redundant functions are restricted to prolactin-induced mammary gland development and function

96 n compared to wild-type (WT) PRLR, increased prolactin-induced pAkt signaling (>1.3-fold, P < 0.02) a

97 dly activates brain neurons, as evidenced by prolactin-induced phosphorylation of signal transducer a

98 This was confirmed by a significant loss of prolactin-induced phosphorylation of STAT5, a marker of

99 its neurogenesis in the SVZ and OB following prolactin infusion or mating/pregnancy.

100 s-of-function mutation in PRLR, resulting in prolactin insensitivity.

101 Reduction of prolactin, insulin-like growth factor binding protein-3,

102 rolactin signals and that stiff matrices and prolactin interact in a feed-forward loop in breast canc

103 s the first reported evidence of altered ECM-prolactin interactions in breast cancer, suggesting the

104 The transport of prolactin into the brain was saturable, with transport e

105 ransport of (125)I-labeled prolactin ((125)I-prolactin) into the brain of female mice in the presence

106 Prolactin is a key factor in the cross-talk between the

107 Prolactin is a major hormone product of the pituitary gl

108 This corresponds with biologic data that prolactin is etiologically important in tumor promotion.

109 Prolactin is internalised into early endosomes, where th

110 In the presence of ECM, prolactin is internalised via a clathrin-dependent, but

111 We now report that autocrine prolactin is required for terminal mammary epithelial di

112 Thus, a threefold increase in plasma prolactin is sufficient to increase BP significantly and

113 By using human prolactin knock-in mice, we demonstrate that prolactin-S

114 e was an increased risk for higher proximate prolactin levels [RR, >15.7 vs. </=8.1 ng/mL (i.e., top

115 Clinically, circulating prolactin levels and density of the extracellular matrix

116 ive neural circuit, indicating that changing prolactin levels can act at multiple sites and thus, imp

117 Following lactation, prolactin levels decline and mammary-specific STAT5-depe

118 No significant change was observed in serum prolactin levels following Opra Kappa administration, bu

119 Serum prolactin levels increased significantly, and to a compa

120 est that pregnant women with abnormally high prolactin levels may need special attention.

121 Our data suggest that prolactin levels measured <10 years before diagnosis are

122 on of 20alpha-HSD, which may be due to lower prolactin levels observed in these women.

123 s (RR) and 95% confidence intervals (CI) for prolactin levels on samples collected <10 (proximate) ve

124 These drugs are well known to elevate serum prolactin levels to varying degrees.

125 d high aldosterone, growth hormone (GH), and prolactin levels, thereby presumably fostering efficient

126 nd induces molting, with no change in plasma prolactin levels.

127 nical chemistry laboratory values, including prolactin levels.

128 hile CORT-KOs displayed higher GH- and lower prolactin-levels than controls under both diets.

129 et conditions as SST-KOs presented higher GH/prolactin-levels, while CORT-KOs displayed higher GH- an

130 Prolactin may be a mediator in the pathogenesis of type

131 neurons express prolactin receptors (Prlr), prolactin may regulate GABA secretion from TIDA neurons,

132 Overall, 2,468 cases and 4,021 controls had prolactin measured <10 years and 953 cases and 1,339 con

133 ficantly higher maximum mean levels of serum prolactin (men, 34.56 microg/L [95% CI, 29.75-39.37] vs

134 Increased levels of a cleaved form of prolactin (molecular weight 16 kDa) have been associated

135 free triiodothyronine, parathyroid hormone, prolactin, N-terminal pro-brain natriuretic peptide, 25-

136 To study the effects of prolactin on blood pressure (BP), we generated male mice

137 DA neurons, potentially mediating actions of prolactin on hypothalamic function.

138 cing the PrP(C) signal sequence with that of prolactin or osteopontin.

139 dy temperature, pupil size, plasma cortisol, prolactin, oxytocin, and epinephrine.

140 atase activity correlated significantly with prolactin (p < .0008; r(2) = 0.95).

141 ver, which includes a brief, but pronounced, prolactin peak, oscillation frequency remains stable.

142 Prolactin plays an important role in maintaining a norma

143 Most conspicuously, mutations in prolactin (PRL) and its receptor (PRLR) have an impact o

144 d mediated mainly by the lactogenic hormones prolactin (PRL) and placental lactogens.

145 We identified prolactin (PRL) as a candidate autocrine factor.

146 The hormone prolactin (PRL) contributes to breast cancer pathogenesi

147 The hormone prolactin (PRL) frequently increases in the circulation

148 The hormone prolactin (PRL) has long been debated as a potential imm

149 Accumulating evidence supports a role for prolactin (PRL) in the development and progression of hu

150 estigated the role of the lactogenic hormone prolactin (PRL) in the regulation of ABCG2.

151 nts with mutant prolactinomas display higher prolactin (PRL) levels (p = 0.02) and shorter progressio

152 s during pregnancy and lactation, when serum prolactin (Prl) levels are highly elevated.

153 Prolactin (PRL) regulates activity of nociceptors and ca

154 nses: proliferation, caspase activation, and prolactin (PRL) release.

155 (TIDA) neurons are the central regulators of prolactin (PRL) secretion.

156 Emerging evidence reveals that prolactin (PRL) signaling at its cognate prolactin recep

157 Prolactin (PRL) signaling is up-regulated in hormone-res

158 The polypeptide hormone prolactin (PRL) stimulates breast epithelial cell growth

159 opmental potential, but when stimulated with prolactin (PRL) they increased the differentiation of ot

160 The lactogenic hormone prolactin (PRL) transcriptionally increases ZnT2 express

161 The mammotrophic hormone, prolactin (PRL), and/or its receptor are also expressed

162 e distribution of pituitary hormones such as prolactin (PRL), growth hormone (GH), adrenocorticotropi

163 phosphorylated Ser305-ERalpha in response to prolactin (PRL), implying that maximal ERalpha phosphory

164 at the estrogen-responsive pituitary hormone prolactin (PRL), signaling through hepatocyte-predominan

165 lating concentrations of total testosterone, prolactin (PRL), thyroid stimulating hormone (TSH), free

166 We have previously shown that the prolactin (PRL)-activated tyrosine kinase JAK2 phosphory

167 tent by somatolactotropes, the precursors of prolactin (PRL)-producing lactotropes.

168 w expression of pituitary growth hormone and prolactin (prl).

169 xpression pattern of growth hormone (Gh) and prolactin (Prl).

170 so expressed elevated levels of the decidual prolactin Prl8a2, as well as altered levels of transcrip

171 Finally, the signaling cascade downstream of prolactin/PRLR activated STAT3 rather than STAT5 in PDAC

172 on VEGF receptor 2 (VEGFR2) expressed in PD prolactin-producing cells known to impair gonadotrophin

173 cal importance, the cell-level mechanisms of prolactin production are not well understood.

174 an lncRNA that regulates growth hormone and prolactin production in the anterior pituitary.

175 fluorescent protein under the control of the prolactin promoter as a quantifiable visual readout of t

176 The hormone prolactin promotes lactational differentiation of mammar

177 In mouse models, prolactin promotes mammary carcinomas that resemble lumi

178 We explored whether the hormone prolactin provides trophic support to retinal cells, thu

179 Peripherally administered prolactin rapidly activates brain neurons, as evidenced

180 To determine why, we explored the human PRL-prolactin receptor (hPRLR)-Janus kinase 2 (JAK2)-signal

181 male mice in the presence and absence of the prolactin receptor (PRLR(-/-)).

182 in and placental lactogen signal through the prolactin receptor (PRLR) and contribute to adaptive bet

183 ), is significantly reduced due to decreased prolactin receptor (Prlr) and ErbB4 expression in Xbp1-d

184 show that CN/Nfatc1 regulates expression of prolactin receptor (Prlr) and that canonical activation

185 Growth hormone receptor (GHR) and prolactin receptor (PRLR) in jawed vertebrates were thou

186 hat prolactin (PRL) signaling at its cognate prolactin receptor (PRLR) in primary afferents promotes

187 Since the prolactin receptor (Prlr) is regulated by reproductive h

188 led that >98% of ovarian cancers express the prolactin receptor (PRLR), forming the basis of a new mo

189 Prolactinomas develop in mice lacking the prolactin receptor (PRLR), which is a member of the cyto

190 es, including progenitors that expressed the prolactin receptor (PRLR).

191 Many genes that are activated by the prolactin receptor are associated with tumorigenesis and

192 We have scrutinized the prolactin receptor as an archetype model of homodimeric

193 state cancer model, we find up-regulation of prolactin receptor as cancer cells that have disseminate

194 olecular architecture of the monomeric human prolactin receptor by combining experimental and computa

195 Additionally, we demonstrated that prolactin receptor expression in Nav1.8(+) neurons was n

196 t were due to a heterozygous mutation in the prolactin receptor gene, PRLR, resulting in an amino aci

197 Ectopic expression of prolactin receptor in mouse cancer cells enhances microm

198 On the basis of high expression of the prolactin receptor in the choroid plexus, it has been hy

199 In particular, prolactin receptor internalization and localization was

200 The prolactin receptor is an archetype member of the class I

201 Overexpression of the prolactin receptor is seen in more than 95% of human bre

202 Candidate oncogenes include the prolactin receptor PRLR activated by a focal deletion th

203 ultiple human cancers, COX-2, prolactin, and prolactin receptor show consistent differential expressi

204 h impaired intercellular junction formation, prolactin receptor trafficking, and alveolar lumen devel

205 igh-affinity ligand-binding interface of the prolactin receptor, resulting in a loss of downstream si

206 Using a novel conditional deletion of the prolactin receptor, we have identified functional subpop

207 Moreover, retinas from prolactin receptor-deficient mice exhibited photorespons

208 st cancer progression and therapy as loss of prolactin receptor-Stat5 signaling occurs frequently and

209 rine system and mediated by sensory neuronal prolactin receptor.

210 , indicating transport is independent of the prolactin receptor.

211 f tissue-resident macrophages, expresses the prolactin-receptor (PRLR).

212 We found that MPOA neurons expressing prolactin receptors (Prlr) form the nexus of a complex p

213 As these neurons express prolactin receptors (Prlr), prolactin may regulate GABA

214 ng through hepatocyte-predominant short-form prolactin receptors (PRLR-S), constrained TNF receptor-a

215 f matrices also increased co-localization of prolactin receptors and integrin-activated FAK, implicat

216 e brain in PRLR(-/-) mice lacking functional prolactin receptors compared to control mice, indicating

217 In this model, the expression levels of prolactin receptors in the retina were upregulated.

218 tinal polypeptide, purinergic, androgen, and prolactin receptors were also expressed in gland of Wolf

219 other malfunctions associated with defective prolactin regulation in humans.

220 Here, hormone release studies show that prolactin release from isolated rat lactotrophs stimulat

221 and 2.5-5 mm dbcAMP) these agents stimulate prolactin release, an inhibition is measured at higher c

222 symptoms, weight gain, sedation, increase in prolactin release, overall functioning, and quality of l

223 ecstasy") and blunted d-fenfluramine-induced prolactin release, substantiating the importance of alph

224 his may serve as a feedforward inhibition of prolactin release.

225 erior to risperidone in terms of increase in prolactin release.

226 e activation of GPR10 by its cognate ligand, prolactin releasing peptide, promotes PI3K-AKT-mTOR path

227 entified in protostome invertebrates, whilst prolactin-releasing peptide (PrRP) and short neuropeptid

228 peptide-1 (GLP-1) neurons and noradrenergic prolactin-releasing peptide (PrRP) neurons participate i

229 n neurons that are immunoreactive for either prolactin-releasing peptide or glucagon-like peptide 1,

230 sufficient for full signal transduction for prolactin-releasing peptide, rather than a deep, membran

231 l, epidermal growth factor, myeloperoxidase, prolactin, resistin and soluble tumor necrosis factor al

232 Castration partially reduced the serotonin/prolactin response and Letrozole partially blocked the e

233 y reduced the fenfluramine-induced serotonin/prolactin response in the presence or absence of DHT.

234 receptors (Prlr) form the nexus of a complex prolactin-responsive neural circuit, indicating that cha

235 We characterized prolactin-responsive neurons within the MPOA at differen

236 factor, STAT5 (pSTAT5), was used to identify prolactin-responsive neurons.

237 efficacy in antagonizing KOR agonist-induced prolactin secretion and in tail-flick analgesia in mice.

238 ion, including the effects of medications on prolactin secretion and the complexities of making contr

239 findings suggest that oxytocin can modulate prolactin secretion by exciting TIDA neurons, and that t

240 with the COX-2 inhibitor celecoxib abrogates prolactin secretion by fibroblasts and reduces tumor ini

241 contribute to the dopaminergic inhibition of prolactin secretion diurnally, as their neuromedin S(+)

242 urons, known as neuroendocrine regulators of prolactin secretion from the pituitary gland, also relea

243 serotonin (5-HT) and SSRIs cause changes in prolactin secretion is not known.

244 eurochemical identity, however, as pituitary prolactin secretion is primarily under monoaminergic con

245 he dynamic dopaminergic control of pituitary prolactin secretion, a key reproductive hormone.

246 ons are involved in the control of pituitary prolactin secretion, and the GABAergic subpopulation may

247 y-thyroid axis in mammals and also regulates prolactin secretion, directly or indirectly via tuberoin

248 amine neurons, the "TIDA" cells that control prolactin secretion, reorganize their trademark oscillat

249 endocrine neurons involved in the control of prolactin secretion, we have shown that approximately ha

250 pamine (TIDA) neurons, the main inhibitor of prolactin secretion.

251 ctional role distinct from the regulation of prolactin secretion.

252 rons had no effect on feedback regulation of prolactin secretion.

253 not necessary for the feedback regulation of prolactin secretion.

254 that the SST/CORT role in the control of GH/prolactin secretions is maintained under LF- and HF-diet

255 neurons that were not previously known to be prolactin-sensitive, notably in the medial amygdala, wer

256 Pregnancy hormones, such as prolactin, sensitize neural circuits controlling parenta

257 adjustment, women in the highest quartile of prolactin showed the lowest risk for diabetes compared w

258 The endoplasmic reticulum-targeting prolactin signal sequence did not affect StAR associatio

259 Prolactin/signal transducer and activator of transcripti

260 Furthermore, enhanced prolactin signaling also led to amplification of a lumin

261 The transcription factor STAT5 mediates prolactin signaling and controls functional development

262 functional relationship between the ECM and prolactin signaling in breast cancer.

263 filing further revealed a potential role for prolactin signaling in regulating BCR editing.

264 hways associated with VEPTB include EGFR and prolactin signaling pathways, inflammation- and immunity

265 in CKO mice without interrupting full-length prolactin signaling, as indicated by normal nursing acti

266 uit is robust with respect to alterations in prolactin signaling.

267 Prolactin signalling depends on a cross-talk with baseme

268 alling, although the role of endocytosis for prolactin signalling is not known.

269 e biological processes, mediated through the prolactin signalling pathway.

270 s is a powerful regulator of the spectrum of prolactin signals and that stiff matrices and prolactin

271 rix stiffness potently regulates a switch in prolactin signals from physiologic to protumorigenic out

272 d consequences of increased ECM stiffness on prolactin signals to luminal breast cancer cells in thre

273 A principal signaling mediator of prolactin, Stat5, promotes cellular differentiation of b

274 prolactin knock-in mice, we demonstrate that prolactin-Stat5a/b signaling promoted metastases formati

275 In the high-prolactin state of lactation, TIDA neurons shift to fast

276 Prolactin-stimulated adult neurogenesis in the subventri

277 by retroviral infection of shRNA attenuates prolactin-stimulated neurogenesis in SVZ-derived adult n

278 Prolactin stimulates milk production, whereas oxytocin i

279 Prolactin suppressed Pg-induced BCL6 through Jak2-Stat5

280 were documented in the MCF-7 cell line, and prolactin synthesis was assessed in GH3 rat pituitary tu

281 Vasoinhibins are N-terminal fragments of prolactin that prevent BRB breakdown during diabetes.

282 drugs to elevate blood levels of the hormone prolactin, the mechanism for this hormonal imbalance is

283 ing mice with chronically elevated levels of prolactin, the rate of (125)I-prolactin transport into t

284 y alveoli during pregnancy are controlled by prolactin through the transcription factors STAT5A and S

285 stages of prostate tumorigenesis induced by prolactin to help determine whether this hormone or its

286 These findings suggest that targeting prolactin together with IL6, a known major activator of

287 in prostate epithelium of prostate-specific prolactin-transgenic mice.

288 There was no change in the rate of (125)I-prolactin transport into the brain in PRLR(-/-) mice lac

289 These data suggest that prolactin transport into the brain involves another as y

290 ated levels of prolactin, the rate of (125)I-prolactin transport into the brain was significantly inc

291 exual stimulation and coitus in female mice; prolactin-triggered oviductal fluid secretion clears the

292 o be involved in mammalian placentation: the prolactins (two clusters), serpins, cathepsins, and the

293 We measured prolactin via immunoassay in cases diagnosed from 1990 t

294 Expression of the decidualisation marker prolactin was decreased in Ct-infected ESC at both mRNA

295 The effects of increased plasma prolactin were abolished by a genetic absence of eNOS.

296 Estrone sulfate, testosterone, and prolactin were selected by stepwise regression and incre

297 e levels of lipids, glycated hemoglobin, and prolactin were similar in the trial groups.

298 Because CSF levels of (125)I-prolactin were very low, even up to 90 min after adminis

299 e weight gain and greater increases in serum prolactin, whereas haloperidol decanoate was associated

300 at control release of the pituitary hormone, prolactin, which triggers key maternal adaptations, incl