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1 -proline type are cleaved by the dipeptidase prolidase.
2  Sp1 is required for the basal expression of prolidase.
3                                Knock-down of prolidase abrogated cocaine-mediated increased permeabil
4  This study deeply elucidates a link between prolidase activity and neuronal maturation shedding new
5                          Mutations impairing prolidase activity lead in humans to the rare disease pr
6                                      Optimal prolidase activity with Met-Pro as the substrate occurre
7 sed to investigate whether the deficiency of prolidase affects the neuronal maturation during develop
8                                              Prolidase, also known as Xaa-Pro dipeptidase or peptidas
9 t protein in pBM, collagen Type IV, requires prolidase, an exopeptidase cleaving the imidodipeptides
10  understanding of ligand recognition and the prolidase and organophosphorus hydrolase catalytic activ
11             Also measured were the levels of prolidase and total sulfhydryl groups.
12 fects of the compounds on X-Pro dipeptidase (prolidase) and leucyl aminopeptidase are also presented.
13 c enzymes, such as cathepsin B, cathepsin D, prolidase, and plasmin.
14                      The classical method of prolidase assay relied on the colorimetric estimation of
15                                              Prolidase catalyzes the final and rate-limiting step of
16                                              Prolidase deficiency (PD) is a multisystem disorder caus
17  activity lead in humans to the rare disease prolidase deficiency characterized by severe skin ulcers
18 broblasts derived from patients with genetic prolidase deficiency exhibited decreased IFNAR1 surface
19                      Subjects homozygous for prolidase deficiency had a ratio ranging from 0.006 to 0
20 molecular basis of functional aspects in the prolidase deficiency.
21 ied subjects with homozygous or heterozygous prolidase deficiency.
22 n the P. furiosus genome database a putative prolidase-encoding gene with a product corresponding to
23                                          The prolidase exhibited a narrow substrate specificity and h
24                Cocaine exposure also induced prolidase expression and activity in HBMECs.
25 operates with Sp1 in the nucleus to activate prolidase expression and enhance collagen biosynthesis.
26         However, the molecular regulation of prolidase expression remains largely unknown.
27 tion and describe the molecular mechanism of prolidase expression.
28 ate that KLF6/Sp1 transcriptionally regulate prolidase expression.
29  KLF6 as well as Sp1 inhibition also reduced prolidase expression.
30 d in the induction of PEPD transcription and prolidase expression.
31 e indistinguishable from those of the native prolidase from P. furiosus.
32 ase has significant similarity with those of prolidases from mesophilic organisms, but the enzyme dif
33  abnormal metabolism or loss of nonenzymatic prolidase function.
34       The amino acid sequence of P. furiosus prolidase has significant similarity with those of proli
35 sed permeability suggesting a direct role of prolidase in BBB breach.
36 e transmigration, establishing a key role of prolidase in cocaine-induced breach in endothelial cell
37  knowledge provides insight into the role of prolidase in the maintenance of self-tolerance and highl
38 hus, the dark-like (dal) mouse, in which the prolidase is knocked-out, was used to investigate whethe
39 y understanding flavivirus IFN-I antagonism, prolidase is revealed as a central regulator of IFN-I re
40 e, arylesterase, and ceruloplasmin activity, prolidase level, and total sulfhydryl groups were evalua
41                       In addition, increased prolidase levels in patients with RA and CP may be relat
42                                          The prolidase levels of the RA-C, RA-CP groups and the CP gr
43 by evaluating serum oxidative parameters and prolidase levels.
44       CQ31 inhibits the M24B aminopeptidases prolidase (PEPD) and Xaa-Pro aminopeptidase 1 (XPNPEP1),
45                                              Prolidase (PEPD) is the only hydrolase that cleaves the
46 nding of the viral IFN-I antagonist, NS5, to prolidase (PEPD), a cellular dipeptidase implicated in p
47   Subsequently, inhibition of iNOS decreased prolidase phosphorylation and reduced cocaine-mediated p
48 ed iNOS expression concurrent with increased prolidase phosphorylation in cocaine treated cells.
49            We show that the cellular enzyme "Prolidase" plays a key role in cocaine-induced disruptio
50                                Knock-down of prolidase reduced cocaine-mediated monocyte transmigrati
51  synthase (iNOS) mediated phosphorylation of prolidase since mRNA levels of the protein were not alte
52                                              Prolidase was required for IFNAR1 maturation and accumul
53 terminal amino acid sequence of the purified prolidase was used to identify in the P. furiosus genome
54                         Proline dipeptidase (prolidase) was purified from cell extracts of the proteo
55 her the mechanism by which cocaine regulates prolidase, we probed the inducible nitric oxide synthase
56               It also belongs to a family of prolidases, with significant activity against various Xa