ライフサイエンスコーパス: promote proliferation

1 ion that maintains cell health, but does not promote proliferation).

2 t ER-positive breast cancers rely on CDK4 to promote proliferation.

3 ng with nutrients, to preserve viability and promote proliferation.

4 interactions mediated by Nanog dimerization promote proliferation.

5 of KLF5 and enhances the ability of KLF5 to promote proliferation.

6 tical RNA binding proteins, FBF-1 and FBF-2, promote proliferation.

7 1 overexpression, however, was sufficient to promote proliferation.

8 , the resulting chimeric protein efficiently promoted proliferation.

9 maturation and cell cycle exit and, instead, promoted proliferation.

10 CMZ, whereas a glucagon-receptor antagonist promoted proliferation.

11 rotects beta cells against glucotoxicity and promotes proliferation.

12 ng, which prevents their differentiation and promotes proliferation.

13 FOG-1 specifies the sperm fate and that FBF promotes proliferation.

14 Here, we report that FOG-1 also promotes proliferation.

15 its stimulation of the PI-3K/p70 S6K cascade promotes proliferation.

16 K transformation, with the loss of autophagy promoting proliferation.

17 levels directed antiapoptotic signals while promoting proliferation.

18 s, supporting their functional importance in promoting proliferation.

19 s/astrocytes, expression of signal molecules promoting proliferation (activated Notch1 and its downst

20 en frequently expressed in carcinomas, which promotes proliferation after regulated intramembrane pro

21 is is beneficial for cancer cells because it promotes proliferation against normal cells.

22 xosomes released by injured epithelial cells promote proliferation, alpha-smooth muscle actin express

23 MUC4 promotes proliferation, anchorage-dependent and-independ

24 most notably the stem cell factor SOX9, that promote proliferation and a metastatic phenotype.

25 on renal tubular epithelial cells (TEC) will promote proliferation and antiapoptosis during regenerat

26 or, Snail, in recipient epithelial cells and promote proliferation and drug resistance.

27 onal mechanisms to rapidly induce genes that promote proliferation and efficiently attenuate their ex

28 How caspases promote proliferation and how cells are protected from t

29 IMP2's ability to promote proliferation and IGF action requires IMP2 phosp

30 Whereas other Th2 effectors promote proliferation and IL-4 production by naive T cel

31 We sought to identify cytokines that can promote proliferation and induce or maintain IL-22 produ

32 Tnrc6a was previously reported to promote proliferation and inhibit differentiation of myo

33 crophages produce canonical Wnt ligands that promote proliferation and inhibit differentiation.

34 genic effects in part through its ability to promote proliferation and inhibit p53-dependent apoptosi

35 ATMs were sufficient to promote proliferation and interferon-gamma production fr

36 absence of stromal cells, was sufficient to promote proliferation and invasion characteristic of a m

37 roblasts can be induced by agonists known to promote proliferation and invasion in vivo.

38 tivate ITCH to maintain BRAF activity and to promote proliferation and invasion of melanoma cells, wh

39 and that basal JNK activity is necessary to promote proliferation and maintain diploidy in breast ca

40 s the Hippo pathway effector Yorkie (Yki) to promote proliferation and maintenance of FSCs, but Hh al

41 ells secrete BMP4 in response to hypoxia and promote proliferation and migration of vascular smooth m

42 mutations potentiate the ability of Ack1 to promote proliferation and migration, suggesting that poi

43 endocardium interacts with the myocardium to promote proliferation and morphogenesis during the later

44 c epithelial and stromal cells, where it can promote proliferation and play a role in tissue regenera

45 larval somatic gonad functionally overlap to promote proliferation and prevent early meiosis.

46 ates Nmyc1 and suppresses Tbx2 expression to promote proliferation and specification of the atrial an

47 cells, IL-2 triggers signaling pathways that promote proliferation and survival by activating the STA

48 tly through cell surface GRP78 (CS-GRP78) to promote proliferation and survival of cancer cells; howe

49 wo oncogenic drivers of Burkitt lymphoma, to promote proliferation and survival of primary human B ce

50 t signaling pathway to dictate cell fate and promote proliferation and survival, the role of Pg in th

51 esyl protein transferase (FTase) activity to promote proliferation and survival.

52 ivate beta-catenin-mediated transcription to promote proliferation and tissue expansion.

53 s suggested that apoptotic caspases can also promote proliferation and tumor growth under certain con

54 lates EGF signaling at low cell densities to promote proliferation and, therefore, may be beneficial

55 t disrupts adhesion and induces signals that promote proliferation and/or migration.

56 After 1 wk of culture, IL-18 promoted proliferation and accelerated differentiation o

57 IL2RA promoted proliferation and cell-cycle activity and inhib

58 xpression of mutant NRAS and EIF1AX proteins promoted proliferation and clonogenic survival in LGSC c

59 crucial role in LRC fate, loss of p27(kip1) promoted proliferation and differentiation of LRCs in vi

60 , overexpression of a relevant ErbB4 isoform promoted proliferation and disturbed polarization of kid

61 lecules in response to S. aureus and greatly promoted proliferation and gamma interferon (IFN-gamma)

62 exposed epithelial cells activated DC, which promoted proliferation and HIV-1 replication of co-cultu

63 FGF signaling promoted proliferation and induced smooth muscle differe

64 SIRT6, but not SIRT6(H133Y), promoted proliferation and lifespan of mouse VSMCs, and

65 ies, which showed that miR-4707-5p and MYBL2 promoted proliferation and metastasis.

66 lyses showed that overexpression of AK017368 promoted proliferation and restrained differentiation of

67 d on a stiff matrix secreted prosaposin that promoted proliferation and survival of mammary carcinoma

68 ate (ATP) is released from damaged cells and promotes proliferation and activation of a variety of im

69 , regulates the expression of HOX genes, and promotes proliferation and aggressiveness of neoplastic

70 In vitro, loss of REDD1 signaling promotes proliferation and anchorage-independent growth

71 hift in pulmonary arterial hypertension that promotes proliferation and apoptosis resistance in the p

72 The c-Myc oncoprotein promotes proliferation and apoptosis, such that mutation

73 Finally, counteracting R-loops by INO80 promotes proliferation and averts DNA damage-induced dea

74 ions-p73 mediates chemosensitivity while p63 promotes proliferation and cell survival-and are both ov

75 tic genes in inductive UGS mesenchyme, which promotes proliferation and cytodifferentiation of the pr

76 s it favors ECM production and autophagy and promotes proliferation and differentiation by limiting F

77 detection of interleukin-4, a cytokine that promotes proliferation and differentiation of B cells, t

78 eveal an integrin-Wnt7a-Decorin pathway that promotes proliferation and differentiation of neuroepith

79 stained beta-catenin activation sequentially promotes proliferation and differentiation, transient be

80 The cytokine interleukin-15 (IL-15) promotes proliferation and effector capacity of CD8(+) T

81 protein abundance, we suggest that low FOG-1 promotes proliferation and high FOG-1 specifies spermato

82 te that activation of Ras in adult epidermis promotes proliferation and inhibits differentiation and

83 Taken together, lncRNA AK017368 promotes proliferation and inhibits differentiation of m

84 These results suggest that IGF-I promotes proliferation and inhibits osteoblastic differe

85 We further showed that Xrn2 promotes proliferation and inhibits squamous differentia

86 E(2) and vasoactive intestinal polypeptide, promotes proliferation and ion secretion and suppresses

87 epidermis, and a loss of NF-kappaB function promotes proliferation and oncogenesis.

88 requires two classes of mutations, one that promotes proliferation and one that blocks differentiati

89 LMP2A promotes proliferation and protects B cells from MYC-ind

90 In contrast, NOTCH3 promotes proliferation and receptor expression is increa

91 CD40 signaling promotes proliferation and rescues B-cells from apoptosi

92 ctivated in ATII cells after lung injury and promotes proliferation and spreading during repair.

93 ed that hypoxia-inducible factor (HIF)1alpha promotes proliferation and spreading of ATII cells durin

94 P. lncRNA AK017368 promotes proliferation and suppresses differentiation of

95 y controlling a transcriptional program that promotes proliferation and suppresses differentiation, i

96 invasive phenotype switch, although how MITF promotes proliferation and suppresses invasion is poorly

97 In T and B cells, CREB activation promotes proliferation and survival and differentially r

98 showed in WEHI-231 cells that M2 expression promotes proliferation and survival and is associated wi

99 oblast models demonstrated that the receptor promotes proliferation and survival of extravillous trop

100 elial carcinoma cell lines and tumors, which promotes proliferation and survival via activation of th

101 In these neoplasms, HH signaling promotes proliferation and survival, contributes to the

102 aling elicited by growth factors and thereby promotes proliferation and TKI evasion downstream of HER

103 The adenovirus E1A oncoprotein promotes proliferation and transformation by binding cel

104 emonstrate a mechanism by which loss of Par3 promotes proliferation and tumorigenesis, which supports

105 Functionally, each promotes proliferation and viability, and they cooperati

106 its loss contributes to oligodendroglioma by promoting proliferation and an OPC-like identity via Ets

107 s critical roles in erythroid progenitors by promoting proliferation and blocking terminal differenti

108 ntrast to the well known activity of Sox2 in promoting proliferation and cell fate determination, our

109 rx1 maintains cells in a stem cell state by promoting proliferation and delaying expression of neura

110 d dendritic cells in addition to its role in promoting proliferation and differentiation of several c

111 h3 interaction constitutes a juxtacrine loop promoting proliferation and dissemination of ovarian can

112 ion exerts opposing behaviors in melanoma by promoting proliferation and expansion and conversely inh

113 ecovery of the mouse hematopoietic system by promoting proliferation and facilitating mobilization of

114 rcinogenesis in a preclinical mouse model by promoting proliferation and increased beta-catenin accum

115 , we establish a novel function of Angpts in promoting proliferation and invasion and inducing Tie-2

116 n the pathogenesis and progression of MF/SS, promoting proliferation and invasion of the malignant ly

117 and inhibits fibrotic remodeling in part by promoting proliferation and migration of endothelial and

118 RC2 maintains small intestinal stem cells by promoting proliferation and preventing differentiation i

119 ole in the response of SCs to limiting BL by promoting proliferation and preventing premature SC diff

120 d activation of TAMs have been implicated in promoting proliferation and survival of cancer cells, as

121 pproximately 92 may mediate these effects by promoting proliferation and through posttranscriptional

122 cted directly on vascular endothelial cells, promoting proliferation and tube formation, possibly thr

123 e growth of triple-negative breast cancer by promoting proliferation and vascular dissemination of ca

124 pression counteracted p21Cip1 up-regulation, promoted proliferation, and drove retinoblastoma formati

125 1 protein expression increased beta-catenin, promoted proliferation, and inhibited p53-dependent apop

126 AP also expands basal epidermal progenitors, promotes proliferation, and inhibits terminal differenti

127 Therefore, FOG-3 normally promotes proliferation, and two copies of the fog-3 gene

128 pression, thereby increasing AKT activation, promoting proliferation, and decreasing c-JUN N-terminal

129 ocking epithelial-mesenchymal transition and promoting proliferation, and provides future directions

130 ely regulated expression of several genes to promote proliferation, apoptosis, and morphogenesis in r

131 Moreover, exposure to 2 Gy IR promotes proliferation arrest and differentiation in the

132 Activation of beta2-AR signaling promotes proliferation associated with increased AKT, ex

133 Acvr1 signaling promoted proliferation at early stages of lens formation

134 E9.5 cortex, suggesting that PACAP normally promotes proliferation at this stage.

135 itors of the canonical BMP signaling pathway promote proliferation but do not affect lineage choice,

136 suggest that signaling through RANK not only promotes proliferation but also inhibits the terminal di

137 dogenous Kras(G12D)-a common AML lesion that promotes proliferation but not self-renewal.

138 Furthermore, FGF promotes proliferation but PDGF favors differentiation.

139 Oncogenes such as HRAS(V12) promote proliferation by upregulating general transcript

140 nases mediates phosphorylation of STAT3, and promotes proliferation by accelerating G(1) --> S progre

141 to modulates myogenic cell determination and promotes proliferation by antagonizing the TGF-beta liga

142 d that overexpression of Id2 in primary AECs promotes proliferation by inhibiting a retinoblastoma pr

143 lts suggest a novel mechanism by which c-Myc promotes proliferation by stabilizing the mitotic spindl

144 Extracellular nucleotides promote proliferation, CXCL12-driven migration, and BM e

145 toward synthetic and proliferative programs, promoting proliferation, cytokine production, and tissue

146 T cell receptor engagement promotes proliferation, differentiation, survival, or de

147 BMP7 thus promotes proliferation directly in nephron progenitors b

148 We find that SREBP1 and 2 promote proliferation downstream of mTORC1, and the acti

149 s string, Cut, and Hedgehog signaling, which promote proliferation during early oogenesis.

150 factor, early growth response gene 3 (Egr3), promotes proliferation during the transition from double

151 NAs encoding growth regulatory proteins that promote proliferation in a variety of cell types.

152 nsfection of p27kip1 siRNA was sufficient to promote proliferation in confluent cultures of HCECs fro

153 Gastrin and its precursors promote proliferation in different gastrointestinal cell

154 Cyclin D proteins promote proliferation in G1 and typically are down-regul

155 Transforming growth factor (ss1TGFss1) can promote proliferation in late stage cancers but acts as

156 nce the dephosphorylated C/EBPalpha does not promote proliferation in Rb-negative cells.

157 These results show that ROS do not promote proliferation in the Nkx3.1-null prostate, but i

158 in ovarian and endometrial cancer cells and promote proliferation in these cells.

159 Stimulation of LD density promoted proliferation in colon cancer cells, whereas si

160 regime reduces mitochondrial metabolism and promotes proliferation in adult mouse cardiomyocytes, re

161 y lesions shortly after HER2 activation, but promotes proliferation in advanced primary tumour cells.

162 ted vascular smooth muscle cell motility but promotes proliferation in association with persistent be

163 wo recent papers now show that Wnt signaling promotes proliferation in both stem cell populations, re

164 Oncogenic MYC activation promotes proliferation in Burkitt lymphoma, but also ind

165 wers endogenous p27(kip1) protein levels and promotes proliferation in confluent cultures of rat CECs

166 Our findings indicate that WNT10B promotes proliferation in human corneal endothelial cell

167 by which the NG2-beta1-integrin interaction promotes proliferation in one case and motility in the o

168 Here we show that the miRNA miR-22 promotes proliferation in primary human cells, and throu

169 KLF5 promotes proliferation in vitro and in vivo and is induc

170 asion in skin cultures without significantly promoting proliferation in vitro and in vivo.

171 erates retinoblastoma development in mice by promoting proliferation, in part by reducing expression

172 silencing by RNA interference significantly promoted proliferation, indicating an inhibitory effect

173 The capacity to promote proliferation, inhibit differentiation and induc

174 cogenic properties, including the ability to promote proliferation, inhibit senescence, and collabora

175 zed route for PKA to activate a pathway that promotes proliferation, inhibits apoptosis, enhances tra

176 -high, but not FAP-low, CAFs to aggressively promote proliferation, invasion and therapy resistance o

177 The fusion gene transcripts promoted proliferation, invasion, and motility with vari

178 ctor 5 (KLF5) is a transcription factor that promotes proliferation, is highly expressed in dividing

179 Although pax6 also promotes proliferation, it differentially regulates neur

180 The ovarian cancer biomarker HE4 is known to promote proliferation, metastasis, chemoresistance, and

181 We also found that FGF-1 and FGF-2 promote proliferation, migration, and survival of cultur

182 We report that RAs promote proliferation, migration, and tube formation of

183 also demonstrated that RGC32 overexpression promoted proliferation, migration and tumorigenic growth

184 Recombinant fibromodulin promoted proliferation, migration, and invasion of HSCs,

185 vesicle-free (S4) human EOC ascites potently promoted proliferation, migration, and invasion of human

186 types, and this aberrant expression strongly promotes proliferation, migration and invasion through m

187 th factor (PDGF)-B to its receptor PDGFRbeta promotes proliferation, migration, and recruitment of pe

188 t role in the bronchiolization of alveoli by promoting proliferation, migration, and attenuation of a

189 se oxidation, while increasing [Ca(2+)]cyto, promoting proliferation, migration, and fission.

190 monstrating that one dose of wild-type fog-1 promotes proliferation more effectively than two doses -

191 , we found CD8+ T cells have the capacity to promote proliferation of BECs in low androgen condition.

192 ltured monocytes had a diminished ability to promote proliferation of both CD4(+) and CD8(+) T cells

193 se disparate signaling pathways cooperate to promote proliferation of cerebellar granule neuron precu

194 we propose that TGF-beta1 dysregulation may promote proliferation of ER-alpha-positive cells associa

195 HIV integration into specific genes may promote proliferation of HIV-infected cells, slowing vir

196 We investigated how inflammation might promote proliferation of LPCs.

197 IL-23 is a two-subunit cytokine known to promote proliferation of memory T cells and T helper typ

198 hibitor, p21(cip1), to inhibit apoptosis and promote proliferation of NCCs, thereby maintaining a mul

199 bundance of serotonin (5-HT), its ability to promote proliferation of neural precursors, and reports

200 ture differentiation of corticotropes and to promote proliferation of pituitary progenitors.

201 growth factors to block differentiation and promote proliferation of primary AML patient blasts.

202 vitro-generated B10 cells were also found to promote proliferation of regulatory T cells in coculture

203 lished role in extending telomeres, TERT can promote proliferation of resting stem cells through a no

204 Drugs activating noncanonical Shh promote proliferation of satellite cells, which is aboli

205 netic approach to tease out key kinases that promote proliferation of specific cancer cell types.

206 d to identify exogenous soluble factors that promote proliferation of SSCs.

207 hat steatosis alters the microenvironment to promote proliferation of tumor initiating cells (TICs) a

208 igh levels of proangiogenic cytokines, which promoted proliferation of both endothelial and epithelia

209 NGF promoted proliferation of CD34(+) cells but not HRECs.

210 hereas those that developed into macrophages promoted proliferation of CD8(+) T cells only.

211 IL-22 also activated STAT3 and promoted proliferation of cultured BMOL cells (a mouse L

212 Ras-mediated hyperactivation of LKB1 promoted proliferation of GNMT-deficient hepatoma cells

213 Reducing levels of alpha1-ACT promoted proliferation of HCC cells in vitro.

214 CAP37 promoted proliferation of HCEC in a time- and dose-depen

215 pared with nontumor tissue samples; estrogen promoted proliferation of human primary hepatocytes.

216 ived cells that developed into migratory DCs promoted proliferation of influenza A virus-specific CD4

217 Exogenous Igf2 protein promoted proliferation of MB precursor cells (GNP) and a

218 Increase of PRP concentration promoted proliferation of MSCs, and 2.5% to 10% of PRP g

219 ased the intraprostatic pHe by 0.2 units and promoted proliferation of noninvasive C3 cells, which re

220 expression of WT1 decreased CDC73 levels and promoted proliferation of OSCC cells.

221 m optogenetically stimulated cortical slices promoted proliferation of pediatric and adult patient-de

222 Furthermore, it strongly promoted proliferation of TF1 cells, above the levels ob

223 Schistosome eggs promoted proliferation of the urothelial cells but inhib

224 cer and activator of transcription 5 (STAT5) promotes proliferation of a wide range of cell types, th

225 of patients with AML and that ROS production promotes proliferation of AML cells.

226 reases the number of immature beta-cells but promotes proliferation of both mature and immature beta-

227 Estrogen receptor-alpha (ERalpha) promotes proliferation of breast cancer cells, whereas t

228 (2014) show that microbial-derived butyrate promotes proliferation of cancer-initiated intestinal ep

229 a driver mutation, NTHi-induced inflammation promotes proliferation of early adenomatous lesions in a

230 nal cofactor in the Hippo signaling pathway, promotes proliferation of embryonic cardiomyocytes by ac

231 Human chorionic gonadotropin (hCG) promotes proliferation of endogenous neural stem cells,

232 Activation of IL-4/IL-13 signaling promotes proliferation of FAPs to support myogenesis whi

233 Drosophila melanogaster ovary, Wingless (Wg) promotes proliferation of follicle stem cells located ~5

234 aken together, our results indicate that FAK promotes proliferation of glioblastoma cells by enhancin

235 ic skin lesions, induces differentiation and promotes proliferation of human keratinocytes.

236 stromal-dependent paracrine VEGF-A signaling promotes proliferation of human primary multiple myeloma

237 o those found in human skin cancers, and PMA promotes proliferation of human skin cells.

238 liferation, and increased CTGF in beta-cells promotes proliferation of immature (MafA(-)) insulin-pos

239 uggest a model in which high mTORC1 activity promotes proliferation of immature SCs and antagonizes S

240 ived from adenomas inhibits their growth and promotes proliferation of intestinal stem cells that ret

241 ion, inflammatory cells produce IL-22, which promotes proliferation of LPCs via STAT3.

242 Taken together, our data show that the AhR promotes proliferation of MB cells, suggesting that this

243 from our laboratory has shown that Nef also promotes proliferation of myeloid cells through a signal

244 The Wnt/beta-catenin pathway promotes proliferation of neural progenitor cells (NPCs)

245 esses differentiation of endocrine cells and promotes proliferation of Nkx6-1(+)Ptf1a(+) multipotent

246 CDO1 silencing promotes proliferation of NSCLC by limiting the futile m

247 We show that WNT pathway activation in vitro promotes proliferation of NSCs but not GNPs.

248 regulation of cyclin D1 by HIF-1alpha, which promotes proliferation of ovarian cancer cells.

249 Strikingly, Tip30 deletion promotes proliferation of primary MECs and results in ra

250 ehog signaling in the epidermis in which SHH promotes proliferation of progenitors of the hair lineag

251 creased phosphorylation of histone H3, which promotes proliferation of T lymphocytes.

252 TNF-alpha promotes proliferation of thyrocytes in vitro, and anti-

253 Importantly, we show that E2F3 promotes proliferation of VSMCs leading to increased IH,

254 Ex vivo, BMP inhibits, and Gremlin 1 promotes, proliferation of cultured BCC cells.

255 l capillary endothelial cells (BRCECs) while promoting proliferation of bovine cornea epithelial cell

256 he pathogenesis of hepatocellular carcinoma, promoting proliferation of cancer cells, the inflammator

257 at Zic1 and Zic4 have Shh-dependent function promoting proliferation of granule cell progenitors.

258 identifies p18 and p21 as novel targets for promoting proliferation of human beta cells and demonstr

259 prominent effect on macrophages, while also promoting proliferation of Igm(+) B cells and memory T c

260 enic system increased islet mass at birth by promoting proliferation of immature beta cells, in the a

261 hase of acute tubular necrosis in animals by promoting proliferation of injured tubular cells and dec

262 ated signaling and suggest a role of KLF5 in promoting proliferation of intestinal epithelia in respo

263 FGFs are commonly implicated in promoting proliferation of neural precursor cells, but i

264 lization after B cell activation, as well as promoting proliferation of pro-B cells.

265 Runx1 may act as a skin oncogene by directly promoting proliferation of the epithelial cells.

266 supporting instead a crucial role for SHH in promoting proliferation of these RP progenitors and for

267 116 cells increased 11G5-induced senescence, promoting proliferation of uninfected cells.

268 The E2F1 transcription factor can promote proliferation or apoptosis when activated, and i

269 moterol, a specific partial agonist, did not promote proliferation or migration.

270 These include: promoting proliferation, protecting cells from activatio

271 the immature follicle to inhibit apoptosis, promote proliferation, stimulate production of steroid a

272 ating from the pre-T-cell receptor (pre-TCR) promote proliferation, survival and differentiation of i

273 s at the conclusion of PGC reprograming I to promote proliferation, survival and expression of the go

274 , insulin can synergize with inflammation to promote proliferation, survival, and dissemination of ca

275 ruiting Wingless (Wnt) signaling pathways to promote proliferation, survival, and motility.

276 hese phenotypes are due to CD97's ability to promote proliferation, survival, and the maintenance of

277 Erythropoietin promoted proliferation, survival, and wound recovery in

278 duction of this NIK fragment into normal SCs promotes proliferation, survival, and adhesion while ind

279 of transcription 3 (STAT3) signaling, which promotes proliferation, survival, and metastasis of canc

280 type and induces c-Rel target genes, thereby promoting proliferation, survival, and more invasive bre

281 (AML) involves the cooperation of mutations promoting proliferation/survival and those impairing dif

282 erable evidence supports a role for PRL-1 in promoting proliferation, the biological regulators and e

283 of the graft endothelium to anti-HLA Abs may promote proliferation through the mTOR pathway.

284 miR-211 promoted proliferation through the posttranscriptional a

285 y stromal myofibroblasts of the lower crypt, promotes proliferation through canonical beta-catenin ac

286 hese data show for the first time that ACA11 promotes proliferation through inhibition of NRF2 functi

287 t whereas intestinal cells induce genes that promote proliferation thymocytes induce expression of ge

288 generation of different organs, ranging from promoting proliferation to inhibiting it.

289 n (SPOP) stabilize the TRIM24 protein, which promotes proliferation under low androgen conditions.

290 ecific miR-290 and miR-302 microRNA families promote proliferation whereas let-7 microRNAs inhibit se

291 iomes: alpha-Proteobacteria-rich communities promote proliferation, whereas Bacteroidetes or pathogen

292 protected cells from induced cell death and promoted proliferation, whereas GM-CSF alone was complet

293 We first show that CAV1 expression promotes proliferation, whereas it suppresses migration

294 a membranes assessed by in vivo optogenetics promotes proliferation, whereas pharmacologically or gen

295 Wnt signals act together to synergistically promote proliferation while maintaining the cells in an

296 g apoptosis--decreasing cell number--or that promote proliferation while simultaneously inhibiting ap

297 Both ligands enhance viability, and Tgfbeta1 promotes proliferation while ActB supports maturation.

298 ress in HSPCs from FA mice, showing that MYC promotes proliferation while increasing DNA damage.

299 damage through inhibiting cell apoptosis and promoting proliferation with important implications for

300 Unlike in other cell types in which YAP promotes proliferation, YAP in T cells suppresses prolif