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1 as autism spectrum disorders and congenital prosopagnosia.
2 abilities in individuals with developmental prosopagnosia.
3 ed by individuals who suffer from congenital prosopagnosia.
4 d the focal clinical syndrome of progressive prosopagnosia.
5 entify connections common to lesions causing prosopagnosia.
6 erior temporo-occipital damage can result in prosopagnosia.
7 ailing diagnostic criteria for developmental prosopagnosia.
8 reproducible and specific to lesions causing prosopagnosia.
10 ughly 2% of the population has developmental prosopagnosia, a congenital deficit in recognizing other
13 ontal regions were independent predictors of prosopagnosia and predicted subclinical facial agnosia i
15 for covert face recognition in developmental prosopagnosia, and suggest this phenomenon results from
16 mechanisms in individuals with developmental prosopagnosia are not qualitatively different from that
17 ce recognition deficits observed in Acquired Prosopagnosia are typically associated with impaired hol
20 trials where participants with developmental prosopagnosia classified famous faces as known or famili
21 ork derived from clinically evident cases of prosopagnosia could predict subclinical facial agnosia i
23 nising faces that, in contrast with acquired prosopagnosia, develop in the absence of manifest brain
31 results ruled out all extant explanations of prosopagnosia except one that proposed that faces are re
34 as previously been described as memory loss, prosopagnosia, getting lost and behavioural changes.
37 group of 12 participants with developmental prosopagnosia in a task that required them to judge the
40 argue that the focal syndrome of progressive prosopagnosia is one of the clinical presentations of se
41 ses in the ventral temporal cortex (VTC) and prosopagnosia is reported in patients with lesions in th
44 tients to exhibit associative agnosia and/or prosopagnosia: many authors have used the label SD for p
45 hat holistic processing deficits in Acquired Prosopagnosia may be task-specific and persist over time
48 erature search identified 44 lesions causing prosopagnosia, only 29 of which intersected the right fu
52 licits modality-specific impairments such as prosopagnosia, pure word blindness and category-specific
54 de participants with extreme behavior (e.g., prosopagnosia, super-recognizers), and does not accommod
56 visual field defect, visual hallucinations, prosopagnosia, topographical disorientation, disturbance
58 ecognition in individuals with developmental prosopagnosia, we tested a group of 12 participants with
61 variety of neural profiles in developmental prosopagnosia, which is consistent with behavioral studi