戻る
「早戻しボタン」を押すと検索画面に戻ります。 [閉じる]

コーパス検索結果 (１語後でソート)

通し番号をクリックするとPubMedの該当ページを表示します
1 Xs)-1 and -2) catalyze the committed step in prostaglandin biosynthesis.
2 andin H(2) (PGH(2)) in the committed step of prostaglandin biosynthesis.
3 -2 (PGHS-2), catalyzes the committed step in prostaglandin biosynthesis.
4 ooxygenase (COX)-1 and COX-2, which catalyze prostaglandin biosynthesis.
5 COX) isoforms catalyze the committed step in prostaglandin biosynthesis.
6  indicate linkage between LPA signalling and prostaglandin biosynthesis.
7 gous to the enzymatic mechanism proposed for prostaglandin biosynthesis.
8 nd -2 (PGHSs) catalyze the committed step in prostaglandin biosynthesis.
9 ved to function as an important regulator of prostaglandin biosynthesis.
10 ade linking members of the MAPK pathway with prostaglandin biosynthesis.
11 inary excretion of lipids was used to assess prostaglandin biosynthesis.
12 nsaturated fatty acids in a critical step in prostaglandin biosynthesis.
13  serves as a biochemical link between NO and prostaglandin biosynthesis.
14 fications that alter COX functionalities and prostaglandin biosynthesis.
15        Thus, IRE1alpha-XBP1 is a mediator of prostaglandin biosynthesis and a potential target to con
16 2, are responsible for the committed step in prostaglandin biosynthesis and are the targets of the no
17 es shows enrichment of genes associated with prostaglandin biosynthesis and genes that drive T cell c
18 (2) may contribute to negative regulation of prostaglandin biosynthesis and inflammation, suggesting
19  in doing both: transcriptionally inhibiting prostaglandin biosynthesis and its reversibility by an e
20  and infection through the activation of the prostaglandin biosynthesis and regulation pathway.
21 TGS2, which codes for COX-2, a key enzyme in prostaglandin biosynthesis, and AREG, which codes for th
22 wn to acetylate an active site serine, block prostaglandin biosynthesis, and give 15R-hydroxyeicosate
23       Cyclooxygenase catalyses a key step in prostaglandin biosynthesis, and recent work suggests tha
24 ch are responsible for the committed step in prostaglandin biosynthesis, and this has been considered
25 nt in cyclooxygenase-1, a critical enzyme in prostaglandin biosynthesis, are resistant to these rapid
26  a mechanism that has also been proposed for prostaglandin biosynthesis as well as for the formation
27 syndromes where oxidant stress and augmented prostaglandin biosynthesis coincide.
28                          While inhibition of prostaglandin biosynthesis did not affect the ability of
29 COX-2), which is the rate-limiting enzyme in prostaglandin biosynthesis from arachidonic acid and is
30 o vertebrate enzymes known to play a role in prostaglandin biosynthesis have been identified in the o
31 rleukin(IL)-1beta treatment markedly induced prostaglandin biosynthesis in diseased compared to healt
32 ted by an interaction between oestradiol and prostaglandin biosynthesis in the fetal brain.
33          There are reports of stimulation of prostaglandin biosynthesis in these cells by direct inte
34                                              Prostaglandin biosynthesis is catalyzed by two spatially
35        The initial and rate-limiting step in prostaglandin biosynthesis is stereoselective removal of
36 COX-2), an inducible rate-limiting enzyme in prostaglandin biosynthesis, is implicated in various phy
37 ible cyclooxygenase-2 (COX-2) gene regulates prostaglandin biosynthesis,is up-regulated in colorectal
38 hibit the same cyclooxygenase enzymes in the prostaglandin-biosynthesis pathway.
39 and RNA-seq integration revealed upregulated prostaglandin biosynthesis pathways in BAT and apoptotic
40 c profiling identified an estradiol-enhanced prostaglandin biosynthesis signature in Tsc2-deficient (
41 ctions are related to its ability to inhibit prostaglandin biosynthesis, some of its beneficial thera
42  of the different cyclooxygenase isoforms in prostaglandin biosynthesis, surface hydrophobicity, and
43 es are responsible for the committed step in prostaglandin biosynthesis, the generation of prostaglan
44 OX-1 and -2), catalyze the committed step in prostaglandin biosynthesis-the conversion of arachidonic
45 7.4 cells that produce NO. and PGHS inhibits prostaglandin biosynthesis to the same extent as NOS inh
46 ygenase-2 (COX-2), a rate-limiting enzyme in prostaglandin biosynthesis, which was also increased at
47                     Aspirin therapy inhibits prostaglandin biosynthesis without directly acting on li