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1 APOE secretion from BALF macrophages through protease-activated receptor 2.
2 nduces A549 IL-8 secretion via activation of protease-activated receptor 2.
3 against transforming growth factor beta1 and protease-activated receptor 2.
4 ates nociceptors to induce visceral pain via protease-activated receptor-2.
5 ic nociceptors, which required expression of protease-activated receptor-2.
6                                              Protease-activated receptor 2 activates airway apical me
7                                  SLIGRL, the protease-activated receptor 2 activating peptide, enhanc
8     Furthermore, stimulation of cells with a Protease-Activated Receptor-2 activating peptide led to
9              Here we show that modulation of protease-activated receptor 2 activation affects melanos
10                                Inhibition of protease-activated receptor 2 activation by synthetic se
11             The use of RWJ-50353 to modulate protease-activated receptor 2 activation could lead to a
12      We also provide molecular evidence that protease-activated receptor 2 activation followed by PI3
13 s of pooled and recombinant human tryptases, protease activated receptor 2 agonist and antagonist pep
14 ected protective mechanisms, mediated by the protease activated receptor 2 and heat shock protein 70,
15 sm that was dependent upon the activation of protease activated receptor-2 and adenosine triphosphate
16                             Der p1 activates protease-activated receptor 2 and induces the release of
17 al cell monolayers through the activation of protease-activated receptor-2 and histone deacetylase 11
18                   Two genes, F2RL1 (encoding protease-activated receptor-2) and RBFOX2 (encoding RNA-
19 nization of the skin could be blocked by the protease-activated receptor 2 antagonist ENMD-1068.
20 -activated receptor is distinct from that of protease-activated receptor-2, because the response to t
21  inhibitor and Bowman-Birk inhibitor inhibit protease-activated receptor 2 cleavage, affect cytoskele
22  also exhibited a dose-dependent increase in protease-activated receptor-2 cleavage activity.
23 s)-coupled receptor Taar1, respectively, and protease-activated receptor-2 could negatively regulate
24 ficantly reduced in STAT6-deficient, but not protease-activated receptor 2-deficient mice.
25  KK modulates endothelial cell function in a protease-activated receptor-2-dependent manner, leading
26            The mechanism involved epithelial protease-activated receptor-2-dependent production of le
27                                    In vitro, protease-activated receptor-2-dependent vascular permeab
28                                    In vitro, protease-activated receptor-2-dependent vascular permeab
29                                          The protease-activated receptor 2, expressed on keratinocyte
30 tors on skin pigmentation and found that the protease-activated receptor 2, expressed on keratinocyte
31 d human keratinocytes showed upregulation in protease-activated receptor 2 expression as determined b
32                                 We show that protease-activated receptor 2 expression in human skin i
33                       In nonirradiated skin, protease-activated receptor 2 expression was confined to
34 iated skin 24 and 96 h after irradiation and protease-activated receptor 2 expression was detected us
35                After ultraviolet irradiation protease-activated receptor 2 expression was observed in
36 h skin type I showed delayed upregulation of protease-activated receptor 2 expression, however, compa
37 type 2 innate lymphoid cells, which required protease-activated receptor-2 expression.
38 e dependent on subtilisin protease activity, protease-activated receptor-2, IL-33R ST2, and MyD88 sig
39  These results suggest an important role for protease-activated receptor-2 in pigmentation in vivo.
40 onse to activation of the B-cell receptor or protease-activated receptor 2, intracellular dialysis wi
41         Previous studies have shown that the protease-activated receptor 2 is involved in skin pigmen
42 imental arthritis model was not dependent on protease-activated receptor-2, it was dependent on the c
43 ion in MI requires myeloid cell signaling of protease-activated receptor 2 linked to the cytoplasmic
44 ydroxytryptamine [5-HT]), histamine, SLIGRL (protease-activated receptors 2/mas-related G-protein-cou
45            Although in vitro studies suggest protease-activated receptor 2 may be a substrate for TMP
46 ators and suppressed the activation of EGFR, protease-activated receptor 2, nucleotide-binding domain
47 lammatory mediators, and activation of EGFR, protease-activated receptor 2, nucleotide-binding domain
48 se the response to trypsin was unaffected by protease-activated receptor-2 overexpression or knockdow
49 her protease activated receptor-1 (PAR-1) or protease activated receptor-2 (PAR-2) alone did not affe
50 her protease activated receptor-1 (PAR-1) or protease activated receptor-2 (PAR-2) on nonhematopoieti
51                       We studied the role of protease-activated receptor 2 (PAR(2)) and its activatin
52 We investigated trafficking and signaling of protease-activated receptor 2 (PAR(2)) in colitis.
53                                              Protease-activated receptor 2 (PAR(2)) is a central regu
54 erated during injury and inflammation cleave protease-activated receptor 2 (PAR(2)) on primary sensor
55                                              Protease-activated receptor 2 (PAR(2)), a receptor for i
56 ubiquitinates the G protein-coupled receptor protease-activated receptor 2 (PAR(2)), which is require
57            The E3 ligase c-Cbl ubiquitinates protease-activated receptor 2 (PAR(2)), which is require
58 n the mechanisms that terminate signaling by protease-activated receptor 2 (PAR(2)), which mediated t
59 ed to analyze compartmentalized signaling of protease-activated receptor 2 (PAR(2)); the propensity o
60 st the hypothesis that in vivo activation of protease-activated receptor 2 (PAR-2) in humans would me
61                                              Protease-activated receptor 2 (PAR-2) is activated by se
62                                              Protease-activated receptor 2 (PAR-2) is expressed in va
63                                              Protease-activated receptor 2 (PAR-2), a receptor for tr
64 ptase released from these cells can activate protease-activated receptor 2 (PAR-2), which was recentl
65 n was induced by compound 48/80 in wild-type protease-activated receptor 2 (PAR-2)- and MC-deficient
66  neutrophils, T lymphocytes, mast cells, and protease-activated receptor 2 (PAR-2).
67 tivation of two Galpha(q)-coupled receptors, protease-activated receptor-2 (PAR(2)) and neurokinin-1
68                        Proteases that cleave protease-activated receptor-2 (PAR(2)) at Arg(36) downwa
69       We report that legumain (Lgmn) cleaves protease-activated receptor-2 (PAR(2)) in the acidic OSC
70                                              Protease-activated receptor-2 (PAR(2)) is a G-protein co
71                                              Protease-activated receptor-2 (PAR(2)) is one of four pr
72 mal activity in acidic environments, cleaves protease-activated receptor-2 (PAR(2)) on neurons to pro
73 stain hyperexcitability and pain by cleaving protease-activated receptor-2 (PAR(2)) on nociceptors th
74                                              Protease-activated receptor-2 (PAR-2) is a G-protein-cou
75                                              Protease-activated receptor-2 (PAR-2) is activated by tr
76                                              Protease-activated receptor-2 (PAR-2) mediates pro-infla
77               The G protein-coupled receptor protease-activated receptor-2 (PAR-2) plays a key role i
78                     Evidence is growing that protease-activated receptor-2 (PAR-2) plays a key role i
79  dependent on the matriptase activation of a protease-activated receptor-2 (PAR-2) signaling pathway
80 tumor and developmental angiogenesis through protease-activated receptor-2 (PAR-2) signaling.
81 re we show that a serine protease acting via protease-activated receptor-2 (PAR-2) stimulates the dev
82                                              Protease-activated receptor-2 (PAR-2), a Galpha(q/11)-co
83             We have reported previously that protease-activated receptor-2 (PAR-2), a proinflammatory
84 rthermore, matriptase elicited signaling via protease-activated receptor-2 (PAR-2), and promoted fibr
85 ombosis and activates cell signaling through protease-activated receptor-2 (PAR-2).
86 include serotonin (5-HT) and agonists of the protease-activated receptor-2 (PAR-2).
87  activated by trypsin-like proteases, termed protease-activated receptor-2 (PAR-2).
88                                              Protease activated receptor 2 (PAR2) is a G-protein coup
89 odels, we identified the interaction between protease-activated receptor 2 (PAR2) and serine protease
90                                              Protease-activated receptor 2 (PAR2) and single-chain ur
91 n-independent tumor growth by activating the protease-activated receptor 2 (PAR2) and the phosphoryla
92                                            A protease-activated receptor 2 (PAR2) antagonist and PAR2
93 lation, and the EPCR-dependent activation of protease-activated receptor 2 (PAR2) by the ternary TF-V
94          Previous studies have revealed that protease-activated receptor 2 (PAR2) contributes signifi
95                                              Protease-activated receptor 2 (PAR2) is a G protein-coup
96                                              Protease-activated receptor 2 (PAR2) is expressed by vas
97  which regulates liver TICs through the CTSS/protease-activated receptor 2 (PAR2) loop.
98 rated during inflammation and disease cleave protease-activated receptor 2 (PAR2) on afferent nerves
99 hypothesized that mast cell tryptase cleaves protease-activated receptor 2 (PAR2) on colonocytes to i
100 leased during inflammation and injury cleave protease-activated receptor 2 (PAR2) on primary afferent
101 ses (mast cell tryptase and trypsins) cleave protease-activated receptor 2 (PAR2) on spinal afferent
102                        The G-protein-coupled protease-activated receptor 2 (PAR2) plays an important
103 ger signaling through the G protein-coupled, protease-activated receptor 2 (PAR2) relevant to inflamm
104                                              Protease-activated receptor 2 (PAR2) signaling and downs
105 d scaffold of coagulation proteases cleaving protease-activated receptor 2 (PAR2) that plays pivotal
106 nzyme B did not kill, but instead stimulated protease-activated receptor 2 (PAR2) to cooperate with I
107 tly induce itch in mice but upregulated skin protease-activated receptor 2 (PAR2) transcripts, a key
108              We set out to determine whether protease-activated receptor 2 (PAR2), a sensor of extrac
109  Neutrophils from aPL-treated mice expressed protease-activated receptor 2 (PAR2), and stimulation of
110 nocytes lacking TRPV3 impair the function of protease-activated receptor 2 (PAR2), resulting in reduc
111 F) conditions induced KLK5 and activated the protease-activated receptor 2 (PAR2), resulting in thymi
112 g proinflammatory cytokine secretion through protease-activated receptor 2 (PAR2).
113 gulation factor VIIa-dependent activation of protease-activated receptor 2 (PAR2).
114 avage of a G-protein coupled receptor called protease-activated receptor 2 (PAR2).
115 y was carried out to determine the effect of protease-activated receptor-2 (PAR2) activation on the p
116       The irreversible proteolytic nature of protease-activated receptor-2 (PAR2) activation suggests
117  in turn cleaved the pericellular substrates protease-activated receptor-2 (PAR2) and pro-urokinase.
118 uch as trypsin and mast cell tryptase cleave protease-activated receptor-2 (PAR2) at R(36) downward a
119    This study sought to evaluate the role of protease-activated receptor-2 (PAR2) in coxsackievirus B
120                                              Protease-activated receptor-2 (PAR2) is a 7-transmembran
121                                              Protease-activated receptor-2 (PAR2) is an emerging new
122 at degrades elastic fibers and activates the protease-activated receptor-2 (PAR2) on endothelial cell
123                                              Protease-activated receptor-2 (PAR2) traffics to lysosom
124                                              Protease-activated receptor-2 (PAR2), a cell surface rec
125 ta-Tryptase, but not homotetramer, activates protease-activated receptor-2 (PAR2), which is expressed
126 on of beta2-adrenergic receptor (beta2AR) or Protease-activated-receptor-2 (PAR2) results in relief f
127  like trypsin receptor 1 (F2RL1, also called protease-activated receptor 2 [PAR2]).
128 keratinocyte-melanocyte interactions via the protease-activated receptor 2 pathway affects melanosome
129   These results imply that inhibition of the protease-activated receptor 2 pathway by soymilk may be
130 agents that could exert their effect via the protease-activated receptor 2 pathway.
131  regulate PKA activity in cardiomyocytes via protease-activated receptor 2 proteolysis.
132                               Differences in protease-activated receptor 2 regulation in type I skin
133 d to be unaffected, as calcium signaling via protease-activated receptor 2 remained unaltered.
134 onditions induced kallikrein 5 and activated protease-activated receptor 2, resulting in thymic strom
135                    Furthermore, we show that protease-activated receptor 2 signaling is involved in m
136 IIa (activated factor VII)-integrin B1-PAR2 (protease-activated receptor 2) signaling complex by util
137 er dysfunction, itch, and dermatitis via the protease-activated receptor 2-thymic stromal lymphopoiet
138 muscarinic receptor, P2Y purinoceptor 1, and protease-activated receptor 2) with known allosteric sit

 
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