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1  a highly abundant, pentameric, light-driven proton pump.
2 ein on earth and functions as a light-driven proton pump.
3 r-type ATPase (V-ATPase), a heteromultimeric proton pump.
4 esembles bacteriorhodopsin (BR), an archaeal proton pump.
5     Bacteriorhodopsin (bR) is a light-driven proton pump.
6 r protons, limiting luminal acidification by proton pumps.
7 ay for proton uptake and presumably also for proton pumping.
8 bbs energy, in order to perform work such as proton pumping.
9 lycolysis is essential for V-ATPase-mediated proton pumping.
10 driven rotation by blocking ATPase-dependent proton pumping.
11 ane subunits are found to be responsible for proton pumping.
12 models describing the molecular mechanism of proton pumping.
13 ain their optical properties associated with proton pumping.
14 ia two processes, electrogenic chemistry and proton pumping.
15 ) intermediate that couples this reaction to proton pumping.
16 lar vesicles activates V-ATPase activity and proton pumping.
17                                              Proton pumping A-type cytochrome c oxidase (CcO) termina
18 ed in the process to thermodynamically drive proton pumping across its membrane domain.
19          Low cytosolic pH required increased proton pumping across the thylakoid membrane and elevate
20           Thus, the interplay between auxin, proton pump activation and expansin action may be more f
21 ver, forced acidification through artificial proton pump activation inhibits root cell elongation.
22 ver, forced acidification through artificial proton pump activation inhibits root cell elongation.
23           Thus, the interplay between auxin, proton pump activation, and expansin action may be more
24          Pilot experiments revealed that the proton pump activator fusicoccin attenuated SWP duration
25 let simultaneously measured the light-driven proton-pumping activities of each bio-pixel.
26  membrane H(+)-ATPase complex, thus reducing proton pump activity to close stomata.
27 atching, accompanied by the onset of gastric proton pump activity.
28 on of endosomes resulting from inhibition of proton pumping activity of vacuolar-type H(+)-ATPase (v-
29 , biosynthesis of secondary metabolites, and proton-pump activity were mainly affected by calcium.
30 embrane protein that synthesizes ATP through proton-pumping activity across the membrane.
31                                          The proton-pumping activity contributes to the proton electr
32  ATP-hydrolase activity, which is coupled to proton-pumping activity.
33  of the C-terminal domain enabled ATPase and proton-pumping activity.
34 cate a common mechanism of regulation of the proton pump and a potassium channel, two essential eleme
35                      Interestingly, the AHA2 proton pump and the FRO2 reductase, both of which work i
36 e membranes assists kinetic coupling between proton pumping and ATP synthesis.
37 aling during cell expansion that coordinates proton pumping and cellulose synthesis.
38 ) exchangers function as electric shunts for proton pumping and in luminal Cl(-) accumulation.
39  of E286 is increased, which slows down both proton pumping and the chemical catalysis.
40 86 through the D-channel, which impairs both proton pumping and the chemical reaction.
41 ments of isolated vesicles containing single proton pumps and compared these results to solutions fro
42  a trimer arrangement reminiscent of BR-like proton pumps and shows features at the extracellular sid
43  of the studied proteins: DTE type (putative proton pump) and NDQ type (putative sodium pump).
44 , gloeobacter rhodopsin (gR), functions as a proton pump, and binds the carotenoid salinixanthin (sal
45 requires extensive adaptations of membranes, proton pumps, and DNA repair mechanisms.
46               The light-activated inhibitory proton pump Archaerhodopsin (Arch) was expressed under c
47 sly available optogenetic tools, such as the proton pump archaerhodopsin-3 (Arch).
48 ction of two different component opsins: the proton pump, Archaerhodopsin and a chloride channel opsi
49 t, following activation of a light-activated proton pump, Archaerhodopsin-3 (Arch), proton transients
50 ing together, we proposed that two tonoplast proton pumps are required for vacuole morphology and PIN
51                   Retinal bound light-driven proton pumps are widespread in eukaryotic and prokaryoti
52 gal toxin that activates the plasma membrane proton pump) are impaired, demonstrating that the transp
53  acceptor and donor, a hallmark of rhodopsin proton pumps, are conserved in these cryptophyte protein
54 opy shows a greater accumulation of V-ATPase proton pumps at the apical surface of A-ICs in KO mice c
55 ubic phase SFX structure of the light-driven proton pump bacteriorhodopsin (bR) to 2.3 A resolution a
56 o haloarchaeal rhodopsins, in particular the proton pump bacteriorhodopsin (BR), than to earlier know
57 elease and uptake events in the light-driven proton-pump bacteriorhodopsin and correlate these to oth
58         Shifting the action spectra of these proton pumps beyond 700 nm would generate new prospects
59 ichia coli F1Fo ATP-synthase and the primary proton pump bo3-oxidase, into synthetic lipid vesicles w
60 n can convert the MastR chloride pump into a proton pump but cannot in HRs.
61 he HIGD1 proteins cannot be due to decreased proton pumping by CIV because CIII, operating alone, als
62 f the HIGD1 proteins may be due to decreased proton pumping by complex IV (CIV) or enhanced leak of p
63  ATP regeneration module, the utility of the proton pump can be extended to other applications in the
64 cts on the abundance of sequences related to proton pumps, carbohydrate metabolism, modifications of
65 presented by the interplay between enzymatic proton pumping, carbon influx and the exchange of calcif
66                             MBS contains two proton pumps compared to one in MBH and likely conserves
67                                              Proton-pumping complex I of the mitochondrial respirator
68 e triphosphatases (V-ATPases) are ATP-driven proton pumps comprised of a cytoplasmic V(1) complex for
69 w stochastic elements, such as variations in proton-pump copy number and cycling between on and off s
70 nt role in understanding how these elements (proton pumps, counter-ion fluxes, membrane potential, bu
71 and possibly physically associated with, the proton-pumping cytochrome bcc-aa (3) supercomplex.
72 w-micromolar levels of sulfide inhibited the proton-pumping cytochrome bo oxidase that is regarded as
73 gh the D-channel is kinetically favored over proton pumping due to the loss of a kinetic gate in the
74 internal proton transport events that enable proton pumping during first steps of oxidation of the fu
75 he D-channel is imperative to achieving high proton-pumping efficiency in the WT CcO.
76 d Ca(2+) channels and activates H(+) -ATPase proton pump efflux that dissociates periplasmic AGP-Ca(2
77        Coxs studied to date are redox-driven proton-pumping enzymes belonging to one of three subfami
78                               This essential proton pump exists in two activity states: an autoinhibi
79 f the Vacuolar H+ ATPase (V-ATPase), the key proton pump for endo-lysosomal acidification, and two pr
80 oichiometry of complex I (i.e. the number of protons pumped for each two electrons transferred) under
81 opsin (BR) is a heptahelical light-dependent proton pump found in the purple membrane of the archaeon
82     Proteorhodopsin (PR), a light-responsive proton pump from marine bacteria, was used as a model to
83 channel that impair, and sometimes decouple, proton pumping from the chemical catalysis.
84                        Besides its canonical proton-pumping function, V-ATPase's membrane sector, Vo,
85     These conditions affect the light-driven proton pumping functional exertion as well.
86                                  The gastric proton pump H(+),K(+)-ATPase acidifies the gastric lumen
87                          In AIG, the gastric proton pump, H(+)/K(+) ATPase, is the major autoantigen
88                       Two types of tonoplast proton pumps, H(+) -pyrophosphatase (V-PPase) and the H(
89                                 Electrogenic proton pumps have been implicated in the generation of s
90  enough to afford electrogenic chemistry and proton pumping, i.e. efficient energy conservation.
91        Complex I functions as a redox-driven proton pump in aerobic respiratory chains.
92    Bacteriorhodopsin (bR), a light-activated proton pump in Archae, has served for many years as a mo
93 lar simulations to study the function of the proton pump in complex I from Thermus thermophilus The s
94 dressed this by expressing a light-activated proton pump in mitochondria to spatiotemporally "turn of
95 vity, suggesting involvement of the V-ATPase proton pump in the phenotype.
96        Complex I functions as a redox-linked proton pump in the respiratory chains of mitochondria an
97 tion step that could thermodynamically drive proton pumping in complex I.
98     We make use of the physical mechanism of proton pumping in the so-called Complex I within mitocho
99 we investigated the function of two types of proton pumps in Arabidopsis embryo development and patte
100 es maintenance of an acidic pH maintained by proton pumps in combination with a counterion transporte
101  with esophageal disease, but the effects of proton pump inhibitor (PPI) drugs are incompletely chara
102 t in vitro and animal studies have found the proton pump inhibitor (PPI) lansoprazole to be highly ac
103 esponse (SVR) included age, race, cirrhosis, proton pump inhibitor (PPI) prescription, prior HCV trea
104               Esophageal eosinophilia can be proton pump inhibitor (PPI) resistant or responsive, rep
105 RD) commonly starts with an empiric trial of proton pump inhibitor (PPI) therapy and complementary li
106                                              Proton pump inhibitor (PPI) therapy fails to provide ade
107 mfort persists despite maximal (double-dose) proton pump inhibitor (PPI) therapy taken appropriately
108      Epidemiological studies have associated proton pump inhibitor (PPI) therapy with osteoporotic fr
109       Demographics, duration of symptoms and proton pump inhibitor (PPI) therapy, GERD-Health Related
110  present in BE patients are reversible after proton pump inhibitor (PPI) treatment.
111  4.61; 95% CI, 1.42 to 15.0; P = 0.011), and proton pump inhibitor (PPI) use (OR, 3.50; 95% CI, 1.19
112 : Studies have reported associations between proton pump inhibitor (PPI) use and dementia.
113  When erlotinib is taken concurrently with a proton pump inhibitor (PPI), stomach pH increases, which
114     Between 1997 and 1999, 177 patients with proton pump inhibitor (PPI)-refractory GERD were randomi
115 symptoms can be medicated empirically with a proton pump inhibitor (PPI).
116 de concomitant nonbismuth quadruple therapy (proton pump inhibitor [PPI] + amoxicillin + metronidazol
117                        Administration of the proton pump inhibitor omeprazole can reduce both esophag
118                                          The proton pump inhibitor omeprazole is administered to dogs
119 g methods, enhancing antibiotic and possibly proton pump inhibitor stewardship, and prescribing proph
120 agement considerations (potential indefinite proton pump inhibitor therapy and/or surveillance endosc
121 rch Datalink was additionally used to assess proton pump inhibitor therapy for at least 6 months (med
122 -based triple and high-dose dual amoxicillin proton pump inhibitor therapy for subsequent treatment a
123 imal acid suppression (twice daily dosing of proton pump inhibitor therapy) in 8-12 weeks.
124 urgical reintervention and 1192 (59.5%) used proton pump inhibitor therapy, with risk factors for the
125  GERD therapy are a promising alternative to proton pump inhibitor therapy.
126 et agents, had a medical condition requiring proton pump inhibitor treatment, or had already received
127 tburn (mean score 3.2 vs 1.4, p = 0.001) and proton pump inhibitor use (41.7% vs 17.1%, p = 0.023) we
128                                              Proton pump inhibitor use increases the risk of developi
129                              Modification of proton pump inhibitor use may increase rates of SVR.
130          Manometric changes, pH testing, and proton pump inhibitor use were assessed preoperatively a
131 tinal bleeding and a possible association of proton pump inhibitor use with Clostridium difficile and
132 ores, quality of life metrics, and change in proton pump inhibitor use) and objective metrics (pH par
133                         Lower baseline eGFR, proton pump inhibitor use, and combination immune checkp
134  included Model for End-Stage Liver Disease, proton pump inhibitor use, and lower length of stay (c-s
135  g/dL), absence of cirrhosis, and absence of proton pump inhibitor use.
136 duction, or pharmacologically treated with a proton pump inhibitor, the ability of pGP3-deficient C.
137 cy as the positive control of free drug plus proton pump inhibitor, the micromotors can function with
138 py and 8 weeks of maintenance therapy with a proton pump inhibitor; and 4) patients receiving follow-
139 cessful endoscopic therapy receive high-dose proton-pump inhibitor (PPI) therapy (intravenous loading
140 nic hepatitis C virus (HCV) are on prolonged proton-pump inhibitor (PPI) therapy and wish to remain o
141              Heartburn that persists despite proton-pump inhibitor (PPI) treatment is a frequent clin
142 ority of children with EoE not responsive to proton-pump inhibitor (PPI), inflammation is driven by s
143 d received either 10-day sequential therapy (Proton-Pump Inhibitor + Amoxicillin 1 g bid for 5 days a
144 hibitor + Amoxicillin 1 g bid for 5 days and Proton-Pump Inhibitor + Clarithromycin 500 mg + Metronid
145                              Using full-dose proton-pump inhibitor and high-dose Metronidazole in gro
146                              Using full-dose proton-pump inhibitor and higher doses of Metronidazole
147 dose Metronidazole in group A, and full-dose proton-pump inhibitor and prescription from a Gastroente
148                              Strikingly, the proton-pump inhibitor omeprazole similarly altered the m
149  that were either treated with omeprazole, a proton-pump inhibitor that suppresses acid secretion in
150 ephalosporin antibiotic) and lansoprazole (a proton-pump inhibitor) will prolong the QT interval.
151 ndamycin (odds ratio [OR]: 1.23, P = .01) or proton pump inhibitors (OR: 1.20, P < .001) in the 90 da
152 r initial response to any therapy, including proton pump inhibitors (P < .001).
153                           BACKGROUND & AIMS: Proton pump inhibitors (PPI) are among the top 10 most p
154                                              Proton pump inhibitors (PPI) are an invaluable therapy o
155                           BACKGROUND & AIMS: Proton pump inhibitors (PPI) have been associated with a
156                      The association between proton pump inhibitors (PPI) use and risk of acute inter
157 (NMA) was conducted to compare the different proton pump inhibitors (PPI) within triple therapy.
158 ion using standard triple therapy (STT) with proton pump inhibitors (PPI), amoxicillin and clarithrom
159 eported on the effects of concomitant use of proton pump inhibitors (PPIs) and dual antiplatelet ther
160                           BACKGROUND & AIMS: Proton pump inhibitors (PPIs) and histamine-2 receptor a
161                                              Proton pump inhibitors (PPIs) and histamine-2 receptor a
162                           BACKGROUND & AIMS: Proton pump inhibitors (PPIs) are commonly used medicati
163                                              Proton pump inhibitors (PPIs) are frequently used after
164                                              Proton pump inhibitors (PPIs) are popular drugs for gast
165                                              Proton pump inhibitors (PPIs) are used for the long-term
166                                              Proton pump inhibitors (PPIs) are widely used for the tr
167                                              Proton pump inhibitors (PPIs) are widely used to treat g
168 reflux esophagitis successfully treated with proton pump inhibitors (PPIs) began 24-hour esophageal p
169                                              Proton pump inhibitors (PPIs) can contribute to small-bo
170  have hypothesized that the long-term use of proton pump inhibitors (PPIs) can increase the risk of d
171                                              Proton pump inhibitors (PPIs) have been known to induce
172                                              Proton pump inhibitors (PPIs) have been recognized as a
173                Safety issues associated with proton pump inhibitors (PPIs) have recently attracted wi
174                                              Proton pump inhibitors (PPIs) may be a risk factor for h
175                  Recent studies suggest that proton pump inhibitors (PPIs) may increase the risk for
176                                              Proton pump inhibitors (PPIs) might reduce the risk of s
177  dyspepsia (FD), the effect and mechanism of proton pump inhibitors (PPIs) or first-line therapy rema
178                                              Proton pump inhibitors (PPIs) or histamine-2 receptor bl
179                                              Proton pump inhibitors (PPIs) predispose to bacterial ov
180 ncreasing incidence of chronic liver disease.Proton pump inhibitors (PPIs) reduce gastric acid secret
181         Retrospective studies have suggested proton pump inhibitors (PPIs) reduce the need for phlebo
182 of patients with GERD who receive label-dose proton pump inhibitors (PPIs) still have symptoms.
183  extraesophageal reflux are often prescribed proton pump inhibitors (PPIs) to reduce gastric acid ass
184 ross-sectional study, 8.5% of patients using proton pump inhibitors (PPIs) were rectal carriers of ex
185 monary disease (COPD), ulcer history, use of proton pump inhibitors (PPIs), aspirin, nonsteroidal ant
186 e the risks associated with long-term use of proton pump inhibitors (PPIs), focusing on long-term use
187                                              Proton pump inhibitors (PPIs), frequently prescribed to
188                                              Proton pump inhibitors (PPIs), which are commonly used a
189 f this association is modulated by intake of proton pump inhibitors (PPIs).
190 s with IPF placed on antacid therapy such as proton pump inhibitors (PPIs).
191  and persistent GERD symptoms despite use of proton pump inhibitors (PPIs).
192  2.6, 95% confidence interval [CI] 1.5-4.7), proton pump inhibitors (PPIs; OR 2.1, 95% CI 1.3-3.4), a
193 nocarcinoma coincided with popularization of proton pump inhibitors and has focused attention on gast
194 phylaxis, review the comparative efficacy of proton pump inhibitors and histamine 2 receptor antagoni
195 r histamine 2 receptor antagonists; however, proton pump inhibitors appear to be the dominant drug cl
196 50% treatment-experienced, and 30% receiving proton pump inhibitors at start of treatment.
197 ibitor, the micromotors can function without proton pump inhibitors because of their built-in proton
198                                        While proton pump inhibitors have been widely used for blockin
199 ating the efficacy and safety of withholding proton pump inhibitors in critically ill patients.
200 here is very low certainty for the effect of proton pump inhibitors in patients with esophageal eosin
201 les of several commercial controlled-release proton pump inhibitors in simulated stomach and intestin
202 per gastrointestinal bleeding; the effect of proton pump inhibitors on ventilator-associated pneumoni
203 ally recommended the use of antacid therapy (proton pump inhibitors or histamine-2-receptor antagonis
204 ux, defined as use of antireflux medication (proton pump inhibitors or histamine2 receptor antagonist
205 ot clearly support lower bleeding rates with proton pump inhibitors over histamine 2 receptor antagon
206                           Here, we show that proton pump inhibitors promote progression of alcoholic
207               Further data integration links proton pump inhibitors to circulating metabolites, liver
208  meta-analyzed five trials (604 patients) of proton pump inhibitors versus placebo; there was no stat
209 tegy of stress ulcer prophylaxis with use of proton pump inhibitors vs histamine-2 receptor blockers
210                                  Exposure to proton pump inhibitors was found in 53% of cases (47% in
211                                              Proton pump inhibitors were stopped at least 7 days befo
212 tigation include substituting vonoprazan for proton pump inhibitors, adding probiotics, and vaccine d
213 , change in body mass index, smoking, use of proton pump inhibitors, and anti-diabetic medications, a
214                     Lifestyle modifications, proton pump inhibitors, and laparoscopic fundoplication
215 , GORD, endoscopy, manometry, pH monitoring, proton pump inhibitors, and Nissen fundoplication.
216         By contrast, patient age, the use of proton pump inhibitors, and the use of primary prophylax
217 f lifestyle and pharmacological therapy with proton pump inhibitors, chemopreventive strategies based
218                            Therapies include proton pump inhibitors, elimination diets, and topical c
219 apies with evidence of effectiveness include proton pump inhibitors, histamine-2 receptor antagonists
220 , GORD, endoscopy, manometry, pH monitoring, proton pump inhibitors, open fundoplication, and laparos
221                                              Proton pump inhibitors, PPIs, are widely prescribed and
222                                          Two proton pump inhibitors, tenatoprazole and esomeprazole,
223                      Among patients who used proton pump inhibitors, there was no significant associa
224                                              Proton pump inhibitors, thyroid hormones, and dihydropyr
225 onomic problem, due to the widespread use of proton pump inhibitors.
226 opy and esophageal pH monitoring, and use of proton pump inhibitors.
227    Consequently, although co-prescription of proton-pump inhibitors (PPIs) reduces upper gastrointest
228 f Gastrointestinal Events Trial) showed that proton-pump inhibitors (PPIs) safely reduced rates of ga
229                                           No proton-pump inhibitors were administered during follow-u
230 antibiotics, opioid analgesics, adrenergics, proton-pump inhibitors, nitroglycerin, diazepam, metoclo
231                                              Proton-pump inhibitors, PPIs, are considered effective t
232 and reminders to reduce inappropriate use of proton-pump inhibitors.
233 hese patients were successfully treated with proton-pump inhibitors.
234 lar-type ATPases (V-ATPases) are ATP-powered proton pumps involved in processes such as endocytosis,
235                          A mechanism for the proton pumping, involving a specific and crucial role fo
236 ur results elucidate the mechanisms by which proton pumping is impaired, thus revealing key kinetic g
237 onstrate that a mutant lacking both of these proton pumps is conditionally viable and retains signifi
238 idoreductase), a membrane-bound redox-driven proton pump, is one of the largest and most complicated
239 ons to bacteriorhodopsin and accelerated the proton pumping kinetics.
240  step plays a central role in activating the proton pumping machinery.
241 complex I has provided new insights into the proton-pumping machinery and a foundation for understand
242 re subunit may be combined with quantitative proton-pumping measurements for mechanistic studies.
243                     To better understand the proton-pumping mechanism of the wild-type (WT) CcO, much
244                                Mitochondrial proton-pumping NADH:ubiquinone oxidoreductase (respirato
245 the D-channel, and not the H-channel, is the proton pump of the yeast mitochondrial enzyme, supportin
246 roton gradient, ultimately maintained by the proton pumps of the respiratory chain, and Ca(2+) bindin
247                         H(+)-ATPase-mediated proton pumping orchestrates cellular homeostasis and is
248              Cytochrome bo3 is a respiratory proton-pumping oxygen reductase that is a member of the
249 olecule level the activity of the prototypic proton-pumping P-type ATPase Arabidopsis thaliana isofor
250 old compared with CEF pathways involving non-proton-pumping plastoquinone reductases.
251  glucose is limiting, however, an ATP-driven proton pump (Pma1) is inactivated, leading to a marked d
252 ptosomal mitochondria and synaptic vesicular proton pump protein (V-ATPase H) levels.
253 near polar or ionic regions in transmembrane proton pump proteins or hydrogen fuel cell materials.
254  to orient the insertion of the light-driven proton pump proteorhodopsin (PR) into liposomes.
255 ein to function as both a redox enzyme and a proton pump, proton transfer into the protein toward the
256 protein to function as both redox enzyme and proton pump, proton transfer out of either of the channe
257                         We conclude that the Proton-Pumping Pyrophosphatase AVP1 localized at the pla
258                                              Proton-pumping pyrophosphatases (H(+)-PPases) are shown
259 ict similar parameter estimates for the mean proton pumping rate, membrane permeability, etc., but, a
260 equence homology with haloarchaeal rhodopsin proton pumps rather than with previously known channelrh
261 /MM) free energy calculations to explore how proton pumping reactions are triggered within its 200 an
262 llular membrane surface by a light-activated proton pump recruited a slow inward ASIC current, which
263                                 However, how proton pumps regulate development remains unclear.
264 tively flat membrane plane regions where the proton-pumping respiratory supercomplexes are situated.
265 inct pathways, including the vacuolar ATPase proton pump, Retromer, and Commander complexes.
266               Finally, a new clade of likely proton-pumping rhodopsin with non-canonical amino acids
267 use inhibition of the macrophage H(+)-ATPase proton pump significantly decreased O(2) (*-) detection
268                     Some function as outward proton pumps, some as inward chloride pumps, whereas oth
269                                          The proton-pumping stoichiometry of complex I (i.e. the numb
270 describe a simple method for determining the proton-pumping stoichiometry of complex I in inverted me
271                            By virtue of this proton-pumping stoichiometry, we hypothesize that NADPH
272 onsible for the delivery of electrons to the proton pumping subunit.
273       CeMM induced transcription of vacuolar proton pump subunits in hlh-30 mutant worms, and knockdo
274 g an ancestor of many essential redox-driven proton pumps, such as respiratory complex I.
275 on of the vacuolar H(+)-ATPase (V-ATPase), a proton pump that acidifies lysosomes.
276 ATPase; V(1)V(o)-ATPase) is an ATP-dependent proton pump that acidifies subcellular compartments in a
277 r H(+)-ATPase (V-ATPase) is an ATP-dependent proton pump that is essential for cellular homeostasis.
278 complex I is a gigantic (1 MDa) redox-driven proton pump that reduces the ubiquinone pool and generat
279 hodopsin (GR) are retinal-based light-driven proton pumps that absorb visible light (maxima at 520-54
280   Vacuolar ATPases (V-ATPases) are essential proton pumps that acidify the lumen of subcellular organ
281 lar pH is primarily driven by the V-ATPases, proton pumps that use cytoplasmic ATP to load H(+) into
282 ype ATPase and compromised targeting of this proton pump to the plasma membrane upon acid challenge.
283                         Mimicking biological proton pumps to achieve stimuli-responsive protonic soli
284 where it is functionally coupled with apical proton pumps to maintain normal acid-base homeostasis.
285 sensitive alternative oxidase (AOX) is a non-proton-pumping ubiquinol oxidase that catalyzes the redu
286 site in the hydrophilic arm to four putative proton-pumping units.
287 ple allow proton transfer to the BNC, but no proton pumping until a proton has reached E286.
288 (CCs) secrete protons into the lumen via the proton pumping V-ATPase located in their apical membrane
289 y epididymal clear cells is achieved via the proton pumping V-ATPase located in their apical membrane
290   Renal intercalated cells (ICs) express the proton pumping vacuolar H(+)-ATPase (V-ATPase) and are e
291 ear-atomic resolution cryoEM structures of a proton-pumping vacuolar ATPase from human cells, illumin
292 ters into synaptic vesicles uses energy from proton-pumping vesicular- or vacuolar-type adenosine tri
293 he hypothesis that mutants lacking tonoplast proton pumps were defective in auxin transport and distr
294                                              Proton pumps were divided into two subtypes (DTEW and DT
295 ining bacteriorhodopsin (bR), a light-driven proton pump, were arranged on a common hydrogel surface
296  be applicable to the entire family of redox proton pumps, where electron transfer to substrates repl
297  Vps34 lipid kinase complex and the v-ATPase proton pump, whereas Atg genes involved in macroautophag
298 insight enables the utilization of PANI as a proton pump, which is actively tuned through an electroc
299 f the water molecule in the active center of proton pumps, which serves as a bridge for the key proto
300 -2 (KR2), a microbial light-driven sodium or proton pump, with noncovalent mass-spectrometric, electr

 
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