ライフサイエンスコーパス: proximal small intestine

1 ried out microarray studies in the colon and proximal small intestine.

2 rated faster-than-normal transit through the proximal small intestine.

3 rily Na(+)/H(+) exchanger 3) activity in the proximal small intestine.

4 immunodominant peptides concentrated in the proximal small intestine.

5 and in absorptive enterocytes located in the proximal small intestine.

6 nvolved in the clearing of reovirus from the proximal small intestine.

7 ls, resulting in inflammatory lesions in the proximal small intestine.

8 spatial expression atlas of the adult human proximal small intestine.

9 window of opportunity for absorption in the proximal small intestine.

10 on the rate of delivery of nutrients to the proximal small intestine.

11 t not other cholesterol transporters, in the proximal small intestine.

12 The RetDN/+ proximal small intestine also had severe hypoganglionosi

13 ly more intestinal tumors, especially in the proximal small intestine and colon, and some of these tu

14 e lipid droplets (LDs) in enterocytes of the proximal small intestine and failed to thrive.

15 peptide produced by K cells of the mammalian proximal small intestine and is a potent stimulant of in

16 beta-galactosidase activity initiated in the proximal small intestine and spread cranially and caudal

17 he duration of infection, including both the proximal small intestine and the colon.

18 CCK cells are concentrated in the proximal small intestine, and hormone is secreted into t

19 Isolating the proximal small intestine, and in particular its luminal

20 ated mice is preferentially delivered to the proximal small intestine as a precursor to fecal excreti

21 the distal gut inhibits transit through the proximal small intestine as the ileal brake.

22 In this group, mucosal blood flow to the proximal small intestine but not to the ileum returned t

23 ated endocannabinoid mobilization in the rat proximal small intestine by altering enzymatic activitie

24 tion and inflammatory tissue immunity in the proximal small intestine by regulating retinol metabolis

25 e of bacterial tyrosine decarboxylase in the proximal small intestine can explain the increased dosag

26 llous differentiated epithelial cells of the proximal small intestine, decreases in expression in the

27 c crypts, and were highest in numbers in the proximal small intestine, decreasing in frequency in a g

28 uminal antigen differ between gLNs, with the proximal small intestine-draining gLNs preferentially gi

29 tinal lesions (one/two calves tested) in the proximal small intestine (duodenum and jejunum) of Gn ca

30 G is removed by FcRn-expressing cells in the proximal small intestine (duodenum and jejunum); remaini

31 stine but are somewhat less effective in the proximal small intestine (especially the clinically impo

32 ross the apical brush-border membrane of the proximal small intestine established by the loss-of-func

33 oxylases at the site of levodopa absorption, proximal small intestine, had a significant impact on le

34 RECENT FINDINGS: Recently, the proximal small intestine has gained attention for its un

35 dose-dependent morphological changes in the proximal small intestine (i.e., duodenum), including wid

36 ounts for their enhanced colonization of the proximal small intestine in an animal model of cholera.

37 acids, a prevalent constituent of the human proximal small intestine, increase intracellular c-di-GM

38 sulting from passage from the stomach to the proximal small intestine induce the functional effect of

39 This segment of proximal small intestine is innervated by the gastroduod

40 Uroguanylin propeptide expression is high in proximal small intestine, low in stomach and distal smal

41 atic protein secretion when infused into the proximal small intestine of conscious rats.

42 igen was also detected in the jejunum of the proximal small intestine of one of two calves tested by

43 logic examination showed mild lesions in the proximal small intestine of only one pig (1/7).

44 ed by immunofluorescent (IF) staining in the proximal small intestine of the WT-PEC-inoculated pigs,

45 gnificant (125)I-STa-binding occurred in the proximal small intestines of GC-C KO and WT mice.

46 interactions regulate host physiology in the proximal small intestine, particularly the duodenum, is

47 tes folate transport into enterocytes in the proximal small intestine; pcft loss-of-function mutation

48 al BaP doses result in adenocarcinoma of the proximal small intestine (PSI) in Cyp1a1(-/-) mice; Cyp1

49 by lipolytic enzymes in the stomach and the proximal small intestine, releasing fatty acids and mono

50 Sensing of dietary triacylglycerol in the proximal small intestine results in physiological, hormo

51 ation that favoured tumour initiation in the proximal small intestine (SI) and blocked tumour growth

52 eflect efficient detoxication of oral BaP in proximal small intestine such that significant amounts o

53 -type of enteroendocrine cell located in the proximal small intestine that produces glucose-dependent

54 e by maintaining the microbial milieu of the proximal small intestine, the clearance of the overgrowt

55 Side-to-side anastomosis of TESI to proximal small intestine was performed or omitted.

56 The velocity through the proximal small intestine was significantly higher in cir

57 The proximal small intestine was the commonest involved segm

58 The proximal small intestine was the most commonly affected

59 carotid artery, as well as enterocytes from proximal small intestine, were obtained at 1.5 hrs after

60 undant on apical enterocyte membranes in the proximal small intestine, where it facilitates FA uptake

61 Uroguanylin mRNA is most prominent in proximal small intestine, whereas guanylin mRNA is predo

62 nsal bacteria inhibit viral infection of the proximal small intestine while simultaneously stimulatin

63 y has a strict axial gradient-highest in the proximal small intestine with no expression in the colon