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1                      The IH switched between pseudohyphal and filamentous growth.
2 ogical transition from single yeast cells to pseudohyphal and hyphal filaments (elongated cells attac
3 tches between growth as budding yeast and as pseudohyphal and hyphal filaments in host organs and in
4 sible morphological transition from yeast to pseudohyphal and hyphal filaments, which is required for
5 nd in significantly greater numbers than did pseudohyphal and hyphal forms, respectively, contrasting
6 e morphogenetic transitions between budding, pseudohyphal and hyphal growth forms that promote the vi
7 gulatory mechanisms that determine growth in pseudohyphal and hyphal morphologies are largely unknown
8 gulatory mechanism functions to specify both pseudohyphal and hyphal morphologies in a dosage-depende
9 thways in yeast, including those involved in pseudohyphal and invasive growth, as well as mating.
10                    Deletion of SFL1 enhances pseudohyphal and invasive growth.
11 vidence concerning the role played by yeast, pseudohyphal, and hyphal forms of C. albicans in adhesio
12         The ability to switch between yeast, pseudohyphal, and hyphal growth forms (polymorphism) is
13 east budding pathway, we used enhancement of pseudohyphal budding in S. cerevisiae by human proteins
14 ansition from a bipolar (sated) to unipolar (pseudohyphal) budding mechanism, where cells elongate an
15 dohyphal growth, and a forking mechanism for pseudohyphal cell proliferation are the key features dri
16 medium and demonstrated a propensity to form pseudohyphal cells on prolonged culture in vitro and in
17 n a ring at the mother-bud neck of yeast and pseudohyphal cells.
18 cerevisiae, S. mikatae, and S. bayanus under pseudohyphal conditions.
19 ound only in S. mikatae and S. bayanus under pseudohyphal conditions.
20 dding cells, was also able to complement the pseudohyphal defect characteristic of the mutant yeast.
21                                          The pseudohyphal defect of Deltamep2/Deltamep2 strains was s
22 t or exogenous cAMP suppresses the Deltagpa2 pseudohyphal defect.
23 ationship between sporulation efficiency and pseudohyphal development and correlates with variation i
24   Activated alleles of RAS2 and CDC42 induce pseudohyphal development and FG(TyA)-lacZ signaling in B
25 n1 cln2 double mutants are more defective in pseudohyphal development and haploid invasive growth tha
26 quired for RAS/MAPK cascade signaling during pseudohyphal development in S. cerevisiae.
27 ed the transcriptional circuitry controlling pseudohyphal development in Saccharomyces cerevisiae.
28 mentous fungus Aspergillus nidulans, induces pseudohyphal development in the yeast Saccharomyces cere
29 Flo11, a cell surface flocculin required for pseudohyphal development, is transcriptionally regulated
30 nents controls mating, haploid invasion, and pseudohyphal development.
31 ons in a positive feedback loop required for pseudohyphal development.
32 ctions in co-operation with STE12p to induce pseudohyphal development.
33  Mos10 is limited to the maturation stage of pseudohyphal development.
34 at they have dramatically different roles in pseudohyphal development: Tpk2 is essential, whereas Tpk
35 ascade signaling, but they are essential for pseudohyphal-development MAPK cascade signaling and othe
36 genic pathways for which Ste20 is essential, pseudohyphal differentiation and haploid-invasive growth
37            The tap42-11 mutation compromised pseudohyphal differentiation and rendered it resistant t
38 he nitrogen sensing machinery that regulates pseudohyphal differentiation by modulating cAMP levels.
39  affinity ammonium permease, is required for pseudohyphal differentiation in response to ammonium lim
40                                              Pseudohyphal differentiation in the budding yeast Saccha
41        Our findings support a model in which pseudohyphal differentiation is controlled by a nutrient
42  that the mutant tRNA(CUG)(Gln) constitutive pseudohyphal differentiation phenotype correlates strong
43               However, Swe1 is essential for pseudohyphal differentiation under a number of nonstanda
44                                              Pseudohyphal differentiation, a filamentous growth form
45  selecting a developmental program--budding, pseudohyphal differentiation, quiescence or sporulation-
46 glucose sensitive, but also affected diploid pseudohyphal differentiation, thereby unexpectedly impli
47 factor Sok2 was found to negatively regulate pseudohyphal differentiation.
48 ntiate to a filamentous growth form known as pseudohyphal differentiation.
49  kinase, protein kinase A (PKA), to regulate pseudohyphal differentiation.
50 n alpha subunit homologs in yeast, regulates pseudohyphal differentiation.
51 es including metabolism, stress response and pseudohyphal differentiation.
52 ap42-regulated phosphatases had no effect on pseudohyphal differentiation.
53 gen starvation or growth on solid medium for pseudohyphal differentiation.
54  and oval cells surrounding and covering the pseudohyphal filament.
55 ays a critical and tightly regulated role in pseudohyphal filamentation.
56       We report here that rapamycin inhibits pseudohyphal filamentous differentiation of S. cerevisia
57 gen deprivation, wherein yeast colonies form pseudohyphal filaments of elongated and connected cells.
58 a role in invasive disease: while hyphal and pseudohyphal filaments penetrate host cells and tissues,
59 ein cells elongate and interconnect, forming pseudohyphal filaments.
60  when FLO11 is expressed, diploid cells form pseudohyphal filaments; when FLO11 is silent, the cells
61 ME6 expression specify growth largely in the pseudohyphal form and that increasing UME6 levels is suf
62 MNL-mediated fungicidal activity against the pseudohyphal form of C. albicans, thereby suggesting pot
63 east organism, both phenotypic switching and pseudohyphal formation have recently been identified in
64 phal growth on UFA-supplemented medium agar, pseudohyphal formation in the OLE1 KO cells was severely
65                         Mutants deficient in pseudohyphal formation were tested in vivo; flo11Delta m
66 ective in nitrogen signaling associated with pseudohyphal formation, sporulation, and NCR.
67  encodes a cell surface protein required for pseudohyphal formation.
68 c switching from the yeast to the hyphal and pseudohyphal forms under certain conditions.
69                Over the last 15 years, yeast pseudohyphal growth (PHG) has been the focus of intense
70                 They were also defective for pseudohyphal growth and agar invasion, and displayed red
71      Mutants deleted of UMP1 were blocked in pseudohyphal growth and development of biofilm-like comp
72 n of the UPR, and subsequent derepression of pseudohyphal growth and meiosis.
73 by the UPR, was sufficient for repression of pseudohyphal growth and meiosis.
74              An activated UPR then represses pseudohyphal growth and meiosis.
75 yclin, prevented fkh1Delta fkh2Delta-induced pseudohyphal growth and modulated some of the fkhDelta-i
76 ing were isolated from an alpha strain using pseudohyphal growth as an assay.
77 egions of the previously known regulators of pseudohyphal growth as well as those of many additional
78  Here, we address the genetic basis of yeast pseudohyphal growth by implementing a systematic analysi
79 gulated protein phosphatase Sit4 exhibited a pseudohyphal growth defect and were markedly hypersensit
80 MEP2 expression or stability, also conferred pseudohyphal growth defects.
81                  Finally, we have found that pseudohyphal growth exhibited by wild-type (HOG1) strain
82 m, but eliminated their abilities to provide pseudohyphal growth for the S. cerevisiae triple mutant.
83 cerevisiae differentiate into a filamentous, pseudohyphal growth form.
84 cerevisiae differentiate into a filamentous, pseudohyphal growth form.
85 rom planktonic single cells to a filamentous pseudohyphal growth form.
86 r-daughter cell junction distinguishes yeast/pseudohyphal growth from hyphal growth in C. albicans.
87 volve processes related to those controlling pseudohyphal growth in budding yeast.
88 of a Gbetagamma dimer to negatively regulate pseudohyphal growth in budding yeast.
89 ein phosphatase regulatory subunit, restored pseudohyphal growth in cells exposed to rapamycin.
90                   This defect in FLO8 blocks pseudohyphal growth in diploids, haploid invasive growth
91                                       Unlike pseudohyphal growth in Saccharomyces cerevisiae, hyphal
92  among ascomycetes and regulates meiosis and pseudohyphal growth in Saccharomyces cerevisiae.
93 c subfamily (Cph2) by its ability to promote pseudohyphal growth in Saccharomyces cerevisiae.
94                    Swe1 is also required for pseudohyphal growth in the absence of Tec1 and for the i
95 s MEP genes capable of efficiently restoring pseudohyphal growth in yeast.
96                                              Pseudohyphal growth is a developmental pathway seen in s
97                     Our findings reveal that pseudohyphal growth is controlled by the calcineurin sig
98 r all conditions tested, its contribution to pseudohyphal growth is limited to the morphological resp
99  the known role of the MAP kinase pathway in pseudohyphal growth is linked to Mep2 function.
100 tinct from the nutritionally induced form of pseudohyphal growth observed in some strains of S. cerev
101                        Our data confirm that pseudohyphal growth occurs gratuitously in sup70-65 muta
102 PCSTE20), a gene participating in mating and pseudohyphal growth of other fungi, was identified follo
103                                        Early pseudohyphal growth of Saccharomyces cerevisiae is well
104 has, to a large extent, come from studies of pseudohyphal growth of the model organism Saccharomyces
105           A mutant allele, sup70-65, induces pseudohyphal growth on rich medium, an inappropriate nit
106 to wild-type C. parapsilosis, which produced pseudohyphal growth on UFA-supplemented medium agar, pse
107  Sho1p may be a receptor that feeds into the pseudohyphal growth pathway.
108  see text] sup70-65 mutant, which exhibits a pseudohyphal growth phenotype and a 75% slower CAG codon
109 up70-65 background, and ameliorated sup70-65 pseudohyphal growth phenotypes.
110         Classic studies have identified core pseudohyphal growth signaling modules in yeast; however,
111 r of these two proteins specifically induced pseudohyphal growth under noninducing conditions, highli
112 a model in which the Gpr1 receptor regulates pseudohyphal growth via the Gpa2p-cAMP-PKA pathway and i
113                                              Pseudohyphal growth was derepressed in ire1Delta/ire1Del
114 everal criteria, fkh1Delta fkh2Delta-induced pseudohyphal growth was distinct from the nutritionally
115 trogen or glucose limitation, Snf1 regulates pseudohyphal growth, a morphological transition characte
116  calcineurin, are required for C. lusitaniae pseudohyphal growth, a process for which the underlying
117 rmease Mep2 is required for the induction of pseudohyphal growth, a process in Saccharomyces cerevisi
118  colony size at the transition from sated to pseudohyphal growth, and a forking mechanism for pseudoh
119 Mep2 is localized to the cell surface during pseudohyphal growth, and it is required for both filamen
120 rentiation responses to nitrogen starvation, pseudohyphal growth, and meiosis.
121 s yeast genes affecting nitrogen catabolism, pseudohyphal growth, and meiotic sporulation.
122 sed thermotolerance, two TFs that eliminated pseudohyphal growth, and several TFs that increased beta
123 programs in response to nitrogen starvation, pseudohyphal growth, and sporulation.
124 ects spore germination, branching of hyphae, pseudohyphal growth, and transcription in non-symbiotic
125 es cerevisiae RNA-mediated transposition and pseudohyphal growth, Candida albicans filamentous growth
126 ot preclude an sla2-6 mutant from undergoing pseudohyphal growth, highlighting the central role of da
127 ile of differential protein abundance during pseudohyphal growth, identifying a previously uncharacte
128                                  PKA signals pseudohyphal growth, in part, by regulating Flo8-depende
129 logue in yeast, FKH2, caused a form of yeast pseudohyphal growth, indicating that the two genes have
130 e kcs1-S537A,S646A mutant exhibits decreased pseudohyphal growth, invasive growth, and cell elongatio
131 uolar degradation, results in abnormal early pseudohyphal growth, not in the filament maturation defe
132        We identified TF targets relevant for pseudohyphal growth, producing a detailed map of its reg
133 lor formation- increased; another phenotype, pseudohyphal growth, responded to the nutrient limitatio
134 ulence attributes of C. lusitaniae including pseudohyphal growth, serum survival, and growth at 37 de
135                      However, none conferred pseudohyphal growth, showing altered CUG anticodon prese
136 highlights the broad gene set enabling yeast pseudohyphal growth, signaling pathways that regulate th
137                                       During pseudohyphal growth, Snf1 is required for wild-type leve
138 h FKH2 is redundant with FKH1 in controlling pseudohyphal growth, the two genes have different functi
139  abundant at the yeast cell periphery during pseudohyphal growth, we generated quantitative proteomic
140                                       During pseudohyphal growth, yeast cells undergo changes in morp
141  with a role for nuclear Hog1p in repressing pseudohyphal growth.
142 ergoes a dimorphic transition to filamentous pseudohyphal growth.
143  Tpk2, but not Tpk1 or Tpk3, is required for pseudohyphal growth.
144 ndergo a dimorphic transition to filamentous pseudohyphal growth.
145 of the osmoresponsive MAPK Hog1p may enhance pseudohyphal growth.
146 of Mks1p or deletion of MKS1 interferes with pseudohyphal growth.
147 c regulator that we show to be essential for pseudohyphal growth.
148 n promotes the initiation of sporulation and pseudohyphal growth.
149 a2/Deltagpa2 mutant strains have a defect in pseudohyphal growth.
150 aughter cell-specific function necessary for pseudohyphal growth.
151 ed morphogenesis during budding, mating, and pseudohyphal growth.
152 here that Ash1 has an essential function for pseudohyphal growth.
153 lized to the nuclei of daughter cells during pseudohyphal growth.
154  anticodon presentation cannot itself induce pseudohyphal growth.
155 tationary phase but opt for pathways such as pseudohyphal growth.
156 d in vivo at Ser residues 537 and 646 during pseudohyphal growth.
157  of a morphological transition, in this case pseudohyphal growth.
158 sly been shown to respond to IAA by inducing pseudohyphal growth.
159  increased expression of a gene required for pseudohyphal growth.
160 CSTE20 suppressed defects in both mating and pseudohyphal growth.
161 logical changes similar to those seen during pseudohyphal growth.
162 t exhibited a constitutive polarized mode of pseudohyphal growth.
163 and transcriptional outputs regulating yeast pseudohyphal growth.
164 cluding hyperpolarization and enhancement of pseudohyphal growth.
165 veal crosstalk between these pathways during pseudohyphal growth.
166  that its dynamics are distinct from that of pseudohyphal growth; during pheromone-induced filament f
167 y, the three A kinases are not redundant for pseudohyphal growth; Tpk2, but not Tpk1 or Tpk3, is requ
168 is sufficient to drive the C. albicans yeast-pseudohyphal-hyphal transition.
169 transition from the normal yeast form to the pseudohyphal, invasive form.
170 rity glycerol (HOG), pheromone response, and pseudohyphal/invasive growth pathways], but its activati
171 cellular signals: mating-pheromone response, pseudohyphal/invasive growth, cell wall integrity, and h
172 Our data suggest that the differentiation of pseudohyphal-like sterigmatal cells during multicellular
173 and changes in budding pattern, leading to a pseudohyphal morphology, even in liquid medium.
174 the fkh1 fkh2 mutant displays a constitutive pseudohyphal morphology, indicating that Fkh1 and Fkh2 m
175 myces cerevisiae that plays a direct role in pseudohyphal or filamentous growth for that organism.
176 c switching and to produce wrinkled (WR) and pseudohyphal (PH) colony types at frequencies of approxi
177 imorphic and switches from a yeast form to a pseudohyphal (PH) form when starved for nitrogen.
178 ific gene expression required for mating and pseudohyphal (PH)/filamentous growth (FG).
179                    In addition, the sup70-65 pseudohyphal phenotype was partly complemented by overex
180              To investigate the basis of the pseudohyphal phenotype, 10 novel sup70 UAG suppressor al
181  young cells differ significantly from their pseudohyphal progenitors, in their shape, and in their t
182 , the divergence in the binding sites of the pseudohyphal regulators Ste12 and Tec1 was determined in
183 l intracellular loop of MEP2 involved in the pseudohyphal regulatory function.
184 cient to cause cells to gradually shift from pseudohyphal to hyphal morphology.
185 2), suggesting that Cln1,2/Cdks regulate the pseudohyphal transcriptional program.
186 on are separable processes controlled by the pseudohyphal transcriptional program.
187                                         This pseudohyphal transition has been studied extensively as
188 species overexpression approach, we used the pseudohyphal transition of Saccharomyces cerevisiae as a
189 teresting regulatory mechanisms enabling the pseudohyphal transition.
190  central to both the mating response and the pseudohyphal transition.
191 al profile of the C. albicans reverse hyphal-pseudohyphal-yeast transition and demonstrate that this

 
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