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1 no between-group differences in severity of psychiatric symptoms.
2 abrupt onset of seizures and/or movement and psychiatric symptoms.
3 behaviors and their impairments give rise to psychiatric symptoms.
4 ly developing group and the group with other psychiatric symptoms.
5 tem, which could affect the brain and induce psychiatric symptoms.
6 ic effect in causing both motor and specific psychiatric symptoms.
7 xes, occasionally spasticity, and frequently psychiatric symptoms.
8 s no longer significant after adjustment for psychiatric symptoms.
9 on positive group to 27 of whom 21 (77%) had psychiatric symptoms.
10 ay underlie other aspects of functioning and psychiatric symptoms.
11 , core autism symptomatology, and associated psychiatric symptoms.
12 d had significantly improved functioning and psychiatric symptoms.
13 ng with the treatment regimen as a result of psychiatric symptoms.
14 how mind and brain work together to produce psychiatric symptoms.
15 f a deleterious association between lead and psychiatric symptoms.
16 s, lead was nonsignificantly associated with psychiatric symptoms.
17 nigmatic relationships between dizziness and psychiatric symptoms.
18 iginous dizziness, subjective imbalance, and psychiatric symptoms.
19 abuse, by themselves and in combination, on psychiatric symptoms.
20 th hepatic disease, neurological disease, or psychiatric symptoms.
21 imprinted gene that is often associated with psychiatric symptoms.
22 with PTSD, substance use disorder, and other psychiatric symptoms.
23 nd often presents with cognitive decline and psychiatric symptoms.
24 l, or dysmorphic conditions co-occurred with psychiatric symptoms.
25 rlie the frequent comorbidity of colonic and psychiatric symptoms.
26 Negative Syndrome Scale was used to measure psychiatric symptoms.
27 nd structured interview measures of PTSD and psychiatric symptoms.
28 reater impairment) than did patients with no psychiatric symptoms.
29 tes were randomly selected for screening for psychiatric symptoms.
30 stand the pathways that link genetic risk to psychiatric symptoms.
31 -acting, and long-lasting treatment for some psychiatric symptoms.
32 e evaluations and dimensional assessments of psychiatric symptoms.
33 ioral alterations, including memory loss and psychiatric symptoms.
34 ease characterized by cognitive deficits and psychiatric symptoms.
35 k for psychosis and perhaps other classes of psychiatric symptoms.
36 ircuit function to precipitate or exacerbate psychiatric symptoms.
37 the neurobiological alterations involved in psychiatric symptoms.
38 r effectiveness, side effects, and impact on psychiatric symptoms.
39 tanding fluctuations in emotional memory and psychiatric symptoms.
40 by diseases exhibiting comorbid visceral and psychiatric symptoms.
41 can induce distinct behavioral outcomes and psychiatric symptoms.
42 system to fully address either individual's psychiatric symptoms.
43 ot specific to working memory abnormality or psychiatric symptoms.
44 ss and trauma, leading to increased risk for psychiatric symptoms.
45 presents one of the earliest and most common psychiatric symptoms.
46 significant after controlling for concurrent psychiatric symptoms.
47 s were associated with both self and partner psychiatric symptoms.
48 donepezil and rivastigmine for cognitive and psychiatric symptoms.
49 w they may be influencing the development of psychiatric symptoms.
50 atients had cognitive deficits, 20 (83%) had psychiatric symptoms, 14 (58%) had insomnia, 12 (50%) ha
51 cannabis were more likely to be due to acute psychiatric symptoms (18.0% vs. 10.9%), intoxication (48
53 sistently poor and ex-poor children had more psychiatric symptoms (4.38 and 4.28, respectively) than
54 als, but was more frequently associated with psychiatric symptoms (58 [25%] vs 34 [15%]) and weight g
55 ome carriers of the translocation who had no psychiatric symptoms-a pattern found in other families w
56 view for DSM-III-R; level of functioning and psychiatric symptoms according to the Global Assessment
58 elve women (14-44 years) developed prominent psychiatric symptoms, amnesia, seizures, frequent dyskin
60 tates and eating disorders are comorbid with psychiatric symptoms and altered emotional responses.
61 c steroid (AAS) use has been associated with psychiatric symptoms and cognitive deficits, yet we have
64 ons require adaptation in this group because psychiatric symptoms and cognitive impairment are highly
67 dy well-documented phenotypic comorbidity of psychiatric symptoms and diagnoses, which can be indexed
71 with having been tested before, more severe psychiatric symptoms and drug problems, level of worry a
74 cipants every 4 months for 2 years to assess psychiatric symptoms and functional status, and we colle
76 njection drug, and alcohol use.Reductions in psychiatric symptoms and hospitalizations are important
77 mechanisms through which genes contribute to psychiatric symptoms and how pharmacological and psychol
78 on is achieved are associated with decreased psychiatric symptoms and improved functioning in the off
79 en steroid abuse is associated with multiple psychiatric symptoms and is a significant public health
80 ated the association between lead burden and psychiatric symptoms and its potential modification by a
81 eductions in associated eating disorders and psychiatric symptoms and maintenance of gains through fo
84 eimer's disease but who manifest significant psychiatric symptoms and neuroleptic-induced extrapy-ram
85 ine the relationship of estrogen levels with psychiatric symptoms and neuropsychological function in
86 this investigation was to determine whether psychiatric symptoms and other characteristics of the in
88 Dissociation may be a critical mediator of psychiatric symptoms and risk-taking behavior among sexu
90 antibodies includes dominant behavioural and psychiatric symptoms and seizures that often interfere w
91 history of stressful life events, 4) current psychiatric symptoms and substance use, and 5) lifetime
93 vels of alcohol use, injection drug use, and psychiatric symptoms and were one-fifth as likely as tho
94 981 participants endorsing significant other psychiatric symptoms and with 1963 typically developing
95 need for obstetricians to assess history of psychiatric symptoms and, with pediatric and psychiatric
96 ency between model animal behavior and human psychiatric symptoms, and 3) the possibility that model
97 in terms of neuropsychological functioning, psychiatric symptoms, and ability to provide informed co
100 is prevalent, especially among children with psychiatric symptoms, and constitutes a major concern wo
104 involuntary movements, extrapyramidal signs, psychiatric symptoms, and medical and drug treatment his
105 Results were similar in patients without psychiatric symptoms, and the increased risk persisted i
106 scores (PRS) and a broad range of childhood psychiatric symptoms, and to quantify the extent to whic
107 programs, experienced greater reductions in psychiatric symptoms, and were more satisfied with their
108 ated with altered neural processing and with psychiatric symptom (anxiety) in humans, which provides
111 sentations, and the frequent assumption that psychiatric symptoms are an inherent part of the underly
112 rmalities and corresponding neurological and psychiatric symptoms are frequently observed in lysosoma
113 system leading to cognitive, autonomic, and psychiatric symptoms are not sufficiently treated by cur
118 ng the 3 medication groups in improvement of psychiatric symptoms as measured by the PANSS total scor
120 mechanistic insights into the cognitive and psychiatric symptoms associated with a schizophrenia-pre
121 for dystonia-associated genes indicates that psychiatric symptoms associated with dystonia are likely
122 een assumed to underlie the neurological and psychiatric symptoms associated with neurodevelopmental
123 e adjusted by sex, family factors, and child psychiatric symptoms at 8 years of age, we found indepen
124 ed-effects regression models, differences in psychiatric symptoms at baseline and over time between t
127 to 7 had a rate of psychotic, but not other psychiatric, symptoms at age 23 that was nearly seven ti
128 tends these findings by examining changes in psychiatric symptoms, behavioral problems, and functioni
131 fferences were not explained by nonpsychotic psychiatric symptom burden, multimorbidity, or substance
132 ication compliance was associated with fewer psychiatric symptoms but not with better housing placeme
133 icant reductions in substance use, PTSD, and psychiatric symptoms, but community care participants wo
134 est whether systematic patterns of change in psychiatric symptoms can be recovered across weekly asse
135 learning difficulties and seizures and later psychiatric symptoms, cerebellar ataxia, extrapyramidal
136 selective serotonin reuptake inhibitors for psychiatric symptoms, cholinesterase inhibitors for cogn
141 maternal depression severity and children's psychiatric symptoms continued to decrease over time.
143 Clinicians should be aware that serious psychiatric symptoms could be associated with ephedra us
144 relationship between religiosity and current psychiatric symptoms, current substance use, lifetime ps
147 patients with 22q11.2DS to manage associated psychiatric symptoms delayed diagnosis of PD by up to 10
148 eview introduces a classification scheme for psychiatric symptoms, describing them in terms of the st
149 id arthritis in whom subacute neurologic and psychiatric symptoms developed after 3 years of treatmen
152 sociated with quantitative measures of broad psychiatric symptom domains and cognitive functioning.
153 r, the relationship between HM and different psychiatric symptoms domains is not well understood.
154 cal basis for and complex interactions among psychiatric symptoms, drug exposure history, and addicti
155 s of topology of tau lesions with late-onset psychiatric symptoms due to TBI have not been explored.
156 eptococcal infections present with motor and psychiatric symptoms, due to basal ganglia involvement.
157 olescence or early adulthood after age, sex, psychiatric symptoms during childhood and early adolesce
159 ophrenia, neuropsychological functioning and psychiatric symptoms (e.g., apathy and avolition), but n
162 omes with prominent, and sometimes isolated, psychiatric symptoms for which patients are fi rst seen
163 Longitudinal measures for up to 10 years of psychiatric symptoms from the Symptom Checklist-90-Revis
165 nversely related to the magnitude of initial psychiatric symptoms (Hamilton Depression Scale: r = -0.
166 s and atypical presentations, including pure psychiatric symptoms, has shifted scientific interest ba
168 struments designed to collect information on psychiatric symptoms, health, and possible postdeploymen
170 with reductions in use of other substances, psychiatric symptoms, HIV risk behavior, and inpatient s
171 and lives of people suffering from specific psychiatric symptoms, illnesses, and/or disabilities.
172 ata extraction were different neurologic and psychiatric symptoms, imaging results, and age at onset
174 ustly predict general cognitive function and psychiatric symptoms in a large and well-characterized d
175 autonomic reactivity to trauma reminders and psychiatric symptoms in adults who had some degree of di
177 relationship between GMV in the left OFC and psychiatric symptoms in CM, we observed a negative assoc
181 ted with increased risk of the corresponding psychiatric symptoms in HD, suggesting a common genetic
186 ad to cognitive decline, motor deficits, and psychiatric symptoms in patients with Huntington disease
187 cognitive therapy was effective in reducing psychiatric symptoms in people with schizophrenia spectr
190 n the two diagnoses, but the contribution of psychiatric symptoms in primary insomnia appears to be a
191 AP as a useful biomarker for the severity of psychiatric symptoms in response to psychological stress
192 ess because they often present with comorbid psychiatric symptoms in the absence of identifiable vest
194 Manual of Mental Disorders, Fourth Edition, psychiatric symptoms in the never-poor, persistently poo
195 mutation carriers already have cognitive and psychiatric symptoms in the premanifest (premotor) phase
199 m deficits in association with dimensions of psychiatric symptoms in youth using a working memory par
200 t steps in efforts to parse heterogeneity in psychiatric symptoms in youths by identifying abnormalit
201 ington disease (HD) is associated with early psychiatric symptoms including anxiety and depression.
204 t systematic detection and treatment of core psychiatric symptoms, including psychotic and impulsivit
205 also carried this mutation but did not have psychiatric symptoms, indicating that this mutation has
206 l participants and used to obtain convergent psychiatric symptom information for additional first-deg
208 monitoring task and were assessed on current psychiatric symptoms, IQ, and frontal lobe functioning.
209 nce suggests that fetal exposure to maternal psychiatric symptoms is associated with future risk for
210 sure to bullying in the absence of childhood psychiatric symptoms is associated with psychiatric outc
211 o bullying, even in the absence of childhood psychiatric symptoms, is associated with severe adulthoo
212 interventions was a barrier for people with psychiatric symptoms, low premorbid intelligence quotien
213 lateral sclerosis (ALS) patient kindreds and psychiatric symptoms may precede the onset of motor symp
215 g brain, leading to complex neurological and psychiatric symptoms observed in fetal alcohol spectrum
218 There was a significant improvement in the psychiatric symptoms of individuals who were seropositiv
219 gulation, altered stress hormone levels, and psychiatric symptoms of stress-related mental illnesses.
220 that some patients with rapidly progressive psychiatric symptoms or cognitive impairment, seizures,
221 wide trauma may be different from changes in psychiatric symptoms or general distress after such even
225 aze palsy, rhythmic myoclonus, dementia with psychiatric symptoms, or hypothalamic manifestations).
227 itish birth cohort) underwent assessments of psychiatric symptoms, primarily anxiety and depression,
228 t from other types of depression in terms of psychiatric symptom profile and treatment response.
229 ly, there was no evidence of exacerbation of psychiatric symptoms, psychosis, depression, or suicidal
232 e used to determine the associations between psychiatric symptom ratings and quantitative anatomic an
233 ere were no significant treatment effects on psychiatric symptom ratings or psychiatric adverse event
238 gs indicate that in a young woman with acute psychiatric symptoms, seizures, and central hypoventilat
239 report four young women who developed acute psychiatric symptoms, seizures, memory deficits, decreas
240 a 26% (95% CI=7%-44%) greater improvement in psychiatric symptom severity compared with standard case
241 cortisol secretion and multiple measures of psychiatric symptom severity were also collected on all.
242 with life, neurobehavioral symptom severity, psychiatric symptom severity, and sleep impairment were
243 mary care clinicians who detect subthreshold psychiatric symptoms should consider a broad psychiatric
245 hosocial and functioning measures, including psychiatric symptoms, social interactions, quality of li
246 ed aspects of motivation are associated with psychiatric symptoms such as anergia, fatigue, lassitude
247 ications, and more alcohol-related emotional/psychiatric symptoms such as depression and anxiety.
249 characterized by comorbidity of colonic and psychiatric symptoms, such as irritable bowel syndrome.
250 likely to have prominent speech-related and psychiatric symptoms than patients with classic disease
251 the complexities of evaluating and treating psychiatric symptoms that are concurrent with a seizure
252 mmon and results in several neurological and psychiatric symptoms that are poorly linked to standard
253 In addition, 50 (43%) of the mothers had psychiatric symptoms that did not meet the diagnostic th
254 The authors define 6 groups of subthreshold psychiatric symptoms that do not meet the full criteria
255 ut extrapyramidal symptoms or signs also had psychiatric symptoms that might be related to their carr
256 ase of a woman in her late 30s who developed psychiatric symptoms that progressed to encephalopathy,
257 sive learning processes in the expression of psychiatric symptoms that transcend diagnostic boundarie
258 tempts before treatment may reflect emerging psychiatric symptoms that trigger medical consultations
263 h histories of mood disorders reported their psychiatric symptoms to a medical provider, a substantia
264 ulation study with data on social habits and psychiatric symptoms to compare prevalences of depressio
265 any relationship between brain pathology and psychiatric symptoms, true natural disease entities may
266 manner, with onset of motor, cognitive, and psychiatric symptoms typically occurring in midlife, fol
268 up these children at age 12 years to assess psychiatric symptoms using the Diagnostic Interview Sche
269 nterview for Substance and Mental Disorders; psychiatric symptoms using the Positive and Negative Syn
271 athology of 505 (46%) patients with reported psychiatric symptoms was described in more detailed term
274 nts and abnormalities of voluntary movement, psychiatric symptoms, weight loss, dementia, and a relen
280 re years of education, white race, and fewer psychiatric symptoms were associated with being in the g
281 owing data: 1) whether changes in children's psychiatric symptoms were associated with changes in the
282 hildhood and early adolescence, and parental psychiatric symptoms were controlled statistically.
289 on about bullying, exposure to bullying, and psychiatric symptoms were obtained from parents, teacher
291 y enrolled, participants with high levels of psychiatric symptoms were oversampled for follow-up.
292 Healthy women with no gastrointestinal or psychiatric symptoms were randomly assigned to groups gi
294 Own psychological resilience factors and psychiatric symptoms were strongly correlated for both p
295 measured by the LPP, predispose children to psychiatric symptoms when exposed to higher levels of st
296 n the ICU were significantly associated with psychiatric symptoms, whereas greater severity of illnes
297 ntions are beneficial in reducing iatrogenic psychiatric symptoms while allowing patients to maintain
298 he co-occurrence of various neurological and psychiatric symptoms with DD, including mood disorders,
299 ers, disabling relationship difficulties, or psychiatric symptoms without associated impairment.
300 type of isolated psychiatric episodes (pure psychiatric symptoms without neurological involvement) e