コーパス検索結果 (１語後でソート)
通し番号をクリックするとPubMedの該当ページを表示します
1 pectrum disorders, and 246 youths with other psychopathology).
2 n whether this association is independent of psychopathology.
3 iagnostic risk for general and externalizing psychopathology.
4 present a transdiagnostic feature of general psychopathology.
5 s a risk factor for the development of adult psychopathology.
6 ural conditions influence the development of psychopathology.
7 how we prevent or treat maltreatment-related psychopathology.
8 ot modulated by subthreshold eating disorder psychopathology.
9 hood maltreatment is a major risk factor for psychopathology.
10 abnormalities as maltreated individuals with psychopathology.
11 , diagnosis, and treatment of threat-related psychopathology.
12 erences in brain development associated with psychopathology.
13 sources of human neurodiversity, not merely psychopathology.
14 sources of human neurodiversity, not merely psychopathology.
15 nce-based training to prevent stress-induced psychopathology.
16 of cognition, enculturation, adaptation, and psychopathology.
17 to later stress which triggers or amplifies psychopathology.
18 nvestigate whether these could explain known psychopathology.
19 ed the relationship between maltreatment and psychopathology.
20 agnostic marker of familial vulnerability to psychopathology.
21 chant critique of biological reductionism in psychopathology.
22 sponses bias women and men towards different psychopathology.
23 bstrates and potential biomarkers of general psychopathology.
24 n targets for the treatment or prevention of psychopathology.
25 ical reductionism and common-cause models of psychopathology.
26 he brain circuit-level mechanisms underlying psychopathology.
27 y be associated with divergent dimensions of psychopathology.
28 unction, which is disrupted in some forms of psychopathology.
29 for broad risk to experience common forms of psychopathology.
30 a transdiagnostic factor reflecting general psychopathology.
31 earning, and predictive-processing models of psychopathology.
32 cultural and contextual variables related to psychopathology.
33 to prevent the development of stress-related psychopathology.
34 the network approach blocks reductionism in psychopathology.
35 ladaptive responses that are associated with psychopathology.
36 h with a focus on improving the treatment of psychopathology.
37 d not differ significantly on any measure of psychopathology.
38 sociated with risk for disease morbidity and psychopathology.
39 implicated in decision making, learning, and psychopathology.
40 l yet understudied symptom of trauma-related psychopathology.
41 h indicators, including obesity, injury, and psychopathology.
42 were associated with specific dimensions of psychopathology.
43 jectory and predispose to or protect against psychopathology.
44 ral symptoms) plus a general factor: overall psychopathology.
45 domains of psychological function linked to psychopathology.
46 l therapeutic application for stress-related psychopathology.
47 gence of psychosis in the context of general psychopathology.
48 sights into vulnerability for stress-related psychopathology.
49 l-being likewise predicts the risk for later psychopathology.
50 rain-behavior relationships in children with psychopathology.
51 fer risk for internalizing and externalizing psychopathology.
52 ss a population of adults with heterogeneous psychopathology.
53 a from which to derive conceptualizations of psychopathology.
54 rauma is translated into biological risk for psychopathology.
55 iation is a complex, ubiquitous construct in psychopathology.
56 differences in behavior as well as risk for psychopathology.
57 Childhood trauma is a key risk factor for psychopathology.
58 facilitate the transdiagnostic emergence of psychopathology.
59 lammatory links between childhood trauma and psychopathology.
60 etal growth also predicts the risk for later psychopathology.
61 sequent stressors, and, ultimately, risk for psychopathology.
62 e cognition are vulnerable to broad-spectrum psychopathology.
63 biological marker across different types of psychopathology.
64 extrastriatal areas and predicts subclinical psychopathology.
65 to an increased risk for general features of psychopathology.
66 ism underlying risk and resilience for later psychopathology.
67 may be disrupted in anxiety and post-trauma psychopathology.
68 attributed to a general factor of childhood psychopathology.
69 t stable and generalizable representation of psychopathology.
70 isk-taking observed across multiple forms of psychopathology.
71 ediating the effect of peer victimization on psychopathology.
72 ill showing clinically significant levels of psychopathology.
73 skills, potentially reducing risk for later psychopathology.
74 ehavior in normal adolescent development and psychopathology.
75 e prevention and treatment of stress-related psychopathology.
76 escribe common/shared and unique elements in psychopathology.
77 biota, which may impact opioid tolerance and psychopathology.
78 ysregulation disorder, with ADHD, or without psychopathology.
79 vidence accumulation in the Stroop task) and psychopathology.
80 developmental mechanisms underlying risk for psychopathology.
81 n provide further insight into the nature of psychopathology.
82 ign of translational models related to human psychopathology.
83 ing situations, and systematically in social psychopathologies.
84 ence of problematic behaviors and a range of psychopathologies.
85 ally developing youths and youths with other psychopathologies.
86 can contribute to fragility underlying human psychopathologies.
87 ful experiences might result in fear-related psychopathologies.
88 creasing vulnerability to the development of psychopathologies.
89 ipants who experience loneliness and in many psychopathologies.
90 eeking habits, a putative factor in multiple psychopathologies.
91 al health, and its dysregulation may lead to psychopathologies.
92 or and may predispose individuals to various psychopathologies.
93 ly relevant in the context of stress-related psychopathologies.
94 proaches for the treatment of stress-related psychopathologies.
95 rauma-exposed participants had high rates of psychopathology (187 [29.2%] of 641 for major depressive
96 with lifetime PTSD had even higher rates of psychopathology (87 [54.7%] of 159 for major depressive
98 Evaluations assessed hypochondriasis, other psychopathology, adverse events, functional status, and
100 r alterations in DA function associated with psychopathology also extend to explaining associations b
101 derated higher levels of adult internalizing psychopathology among behaviorally inhibited individuals
102 gree relatives (49 relatives had no lifetime psychopathology and 25 had a non-BD mood disorder), and
104 e evaluated for differences in both clinical psychopathology and brain activation during an n-back wo
105 to capture dimensional associations between psychopathology and brain measures across a continuous s
107 imilarly changed brain model is extending to psychopathology and clinical psychology, and partly acco
108 n youths (8-23 years), we formed data-driven psychopathology and cognitive components using a large p
109 ant properties that were associated with the psychopathology and cognitive impairment of psychiatric
111 for understanding the nature and origins of psychopathology and for developing improved intervention
112 he secondary outcomes were general levels of psychopathology and functioning, as assessed by the Brie
113 t (GWC) to age and individual differences in psychopathology and general cognitive ability in youths
114 in supplementation might not improve overall psychopathology and global neurocognition, it may have s
115 cated as key mechanisms across many forms of psychopathology and have given rise to automated mechani
117 nformation processing deficits in delusional psychopathology and in genetic risk for schizophrenia.
118 struct for informing models of developmental psychopathology and individual differences in outcome ov
119 then discusses the validity of experimental psychopathology and introduces a methodological approach
121 uss the utility of integrating developmental psychopathology and neuroscience to facilitate future re
122 sposing to the development of trauma-related psychopathology and provide biologically plausible suppo
123 indicate that genetic discoveries related to psychopathology and psychosocial factors may be limited
125 enotypes to the development of internalizing psychopathology and set the stage for developing improve
126 ations for understanding biased attention in psychopathology and support a role for learning in the e
127 more accurately model the dynamic nature of psychopathology and system change as well as have treatm
128 lications for the evolving classification of psychopathology and the challenges for clinical translat
129 al relevance of epigenetics for the study of psychopathology and to consider the state of current res
130 nt, has training and experience in assessing psychopathology, and is willing to participate in ongoin
131 risk of attempting suicide independently of psychopathology, and should be included in suicide risk
132 In adulthood (26 y; n = 109), personality, psychopathology, and sociodemographics were self-reporte
133 f anxiety disorders and other stress-related psychopathology, and understanding the neural systems un
134 this review, we outline how the Experimental Psychopathology approach, based on experimental models o
136 nduced metabolic change, and improvements in psychopathology are associated with metabolic disturbanc
138 ase entity, encompassing a broad spectrum of psychopathology arising from distinct pathophysiological
139 iatric research may benefit from approaching psychopathology as a system rather than as a category, i
140 end to explain brain differences observed in psychopathology as an underlying (causal) neurobiologica
142 , emerged as a strong contributor to general psychopathology as well as to each individual symptom do
143 presentations of the nature and structure of psychopathology, as exemplified by psychodynamic, DSM/IC
144 ortex (PFC) are especially relevant to human psychopathology, as the PFC, in addition to its function
145 -life adversity (ELA) increases the risk for psychopathologies associated with amygdala-prefrontal co
146 T pathway as a potential target for treating psychopathologies associated with the attribution of exc
148 adulthood are periods of heightened risk for psychopathology associated with emotion dysregulation, a
150 of any severity was associated with greater psychopathology, attention deficits, and impulsiveness,
153 the association between metabolic change and psychopathology change by estimating the correlation bet
155 choice behaviour and may offer insights into psychopathologies characterized by dysfunctional decisio
156 , and functional connectivity with affective psychopathology, cognition, and family environment.
157 e latent components corresponding to general psychopathology, cognitive dysfunction, and impulsivity.
158 oms, which might miss possible links between psychopathology, cognitive processes, and personality tr
160 nalyses were conducted on structural MRI and psychopathology data collected from 861 members of the p
162 incentives, to ask if symptoms and traits of psychopathology decrease or increase model-based control
165 The authors hypothesized that if general psychopathology drives the convergence of structural alt
166 ital status, social deprivation, severity of psychopathology, duration of index CRT episode, first co
170 lue < 0.0001) of the variance in the general psychopathology factor and ~ 0.50% (p value < 0.0001) in
171 orders were exerted mostly through a general psychopathology factor representing the shared effect ac
172 attempt almost exclusively through a general psychopathology factor representing their shared effect,
173 ty problem by assuming that a single general psychopathology factor underlies multiple disorders.
174 ted associations between ADHD PRS, a general psychopathology factor, and several dimensions of neurod
176 ing and externalizing dimensions), a general psychopathology factor, or a combination of these explan
177 diagnostic research has identified a general psychopathology factor-often called the 'p' factor-that
178 y phenotypic risk to develop anxiety-related psychopathology, for accelerating the development of imp
179 oms, the "trait reductionism" of dimensional psychopathology frameworks suffers from the same limitat
181 eview summarizes the mentalizing approach to psychopathology from a developmental socioecological evo
182 e (g = 0.40; 95% CI, 0.22-0.58), and general psychopathology (g = 0.32; 95% CI, 0.09-0.55), with no d
184 viations in the psychosis spectrum and other psychopathology groups in age-related changes in resting
185 tory, cardiometabolic health, cognition, and psychopathology had the most consistent and replicable a
186 ork approach as a novel way of understanding psychopathology has helped address some of the existing
187 eeking and vulnerability to the emergence of psychopathology, has been difficult to study due to pedi
188 out the limitations of network approaches to psychopathology have emerged, inspiring the development
189 ications inspired by the network approach to psychopathology have increased exponentially in the twen
190 risk factors mediating the susceptibility to psychopathology have led to a heightened focus on both t
191 connecting symptoms in a network approach to psychopathology hinges neither on causality nor on ratio
193 e search for universal, biological causes of psychopathology; however, this is not an inevitable outc
194 sychiatric disorders, specific dimensions of psychopathology (i.e., internalizing and externalizing d
196 altreatment render individuals vulnerable to psychopathology if abnormalities occur in specific nodes
197 HR group, they are associated with affective psychopathology, impairments in verbal memory, and overa
199 the first study to examine multiple forms of psychopathology in association with advanced DNA methyla
201 al genetic liability towards broad childhood psychopathology in the general population, in addition t
204 transmission of risk for the development of psychopathology in their children relies on solid founda
208 ve important implications for stress-related psychopathologies, including post-traumatic stress disor
209 dentifies a novel mechanism that can support psychopathology, including addiction.SIGNIFICANCE STATEM
210 ave extended this approach to other types of psychopathology, including depression, anxiety, and eati
211 L) longevity gene polymorphism rs9315202 and psychopathology, including posttraumatic stress disorder
212 be applied to the emergence (or decline) of psychopathology, including psychosis prediction, as well
214 ar to confer resilience or susceptibility to psychopathologies involving prefrontal cortex (PFC) dysf
215 sharing, mental energy conservation, diverse psychopathologies, irrational decisions in invertebrates
216 nique effects of 2 major dimensions of child psychopathology, irritability and anxiety, on neural res
218 nctive aspect of NMDAR-antibody encephalitis psychopathology is complexity; core aspects of mood and
219 ed to reduced cortical thickness and overall psychopathology is most strongly linked to global reduct
220 roticism with its link to the development of psychopathology is of interest concerning influential fa
223 de have been implicated in a wide variety of psychopathologies, it remains unclear how individual dis
224 ed intercorrelations among the many forms of psychopathology, leading to the identification of a gene
226 nt identified through court records, risk of psychopathology linked to objective measures was minimal
228 hibitory control are associated with various psychopathologies, many of which emerge during adolescen
230 both general cognitive function and general psychopathology (mean correlations between predicted and
231 ical equilibrium." The SOC can represent the psychopathology network structures and additionally sugg
232 o not reflect psychobiological mechanisms of psychopathology, nor the complex interplay of genetic an
233 ffspring of control parents with non-bipolar psychopathology (OCP), and 27 healthy controls (HC) had
234 offspring of comparison parents with non-BD psychopathology (OCP, n = 36), and offspring of healthy
236 suggest that the neural basis underlying the psychopathology of attempted suicide in patients with MD
237 y encephalitis present to psychiatrists, the psychopathology of NMDAR-antibody encephalitis needs to
239 nt inference would partially account for the psychopathology of schizophrenia as measured in terms of
241 ions that appear to duplicate aspects of the psychopathology of several neuropsychiatric disorders.
244 on solid foundations in the understanding of psychopathology, of development, and of developmental ps
247 tanding of this phenomenon, with the general psychopathology or p-factor model emerging as the most p
249 ing the personalized approach in descriptive psychopathology, precision assessment, and treatment sel
250 ng, is associated with both ACE exposure and psychopathology, providing the basis for an emerging lit
251 hese associations influence genetic risk for psychopathology, psychosocial factors and EDU and/or SES
252 the most important for prediction of general psychopathology, psychotic symptoms, and norm-violating
253 These findings were not affected by non-BD psychopathology, psychotropic medication use, or symptom
254 (i.e., anxiety and depression, beta = 0.20) psychopathology, rather than a transdiagnostic risk for
255 ictimization during adolescence might induce psychopathology-relevant deviations from normative brain
257 ion, the reductionist research strategies in psychopathology research (the Research Domain Criteria [
259 om and colleagues argue that reductionism in psychopathology research has not provided the expected i
260 tem has been implicated in trauma and stress psychopathology, resulting in a growing interest in modu
261 he role of psychosocial adversity in shaping psychopathology risk to present a working conceptual mod
262 interviews for trauma exposure, PTSD, other psychopathology, risk events, functional impairment, and
263 ing emotion perception and identification in psychopathology.SIGNIFICANCE STATEMENT Emotion perceptio
264 including cognitive status, health literacy, psychopathology, social support, medical adherence, and
265 ve identified a general liability factor for psychopathology-sometimes called the 'p factor'- that un
266 Differences in PANSS positive and general psychopathology subscale score, Marder positive factor s
269 he high BMI group had a higher PANSS general psychopathology subscore, higher levels of blood glucose
272 considerable implications for stress-related psychopathologies, such as posttraumatic stress disorder
274 etween healthy controls and individuals with psychopathology suggests that individual difference find
275 Structural covariance networks predicted psychopathology symptoms above and beyond demographic ch
276 l would be associated with reduced levels of psychopathology symptoms at the transition from middle t
277 created to explore the relationship between psychopathology symptoms both within and across anxiety
278 dy investigated the mechanism and changes in psychopathology symptoms throughout the COVID-19 outbrea
279 aluate intervention effects on teacher-rated psychopathology symptoms using the Strengths and Difficu
280 tes that urgency is more robustly related to psychopathology than are other forms of impulsivity.
281 d cognitive performance and higher levels of psychopathology than both subtype 2 and typically develo
282 inking maternal overnutrition to obesity and psychopathology that can be conserved across multiple ge
283 resentational diversity, neurocognition, and psychopathology that provides important constraints and
284 Irritability is an important dimension of psychopathology that spans multiple clinical diagnostic
286 ionally to the study of child and adolescent psychopathology, the nature of the relationship between
287 While subtypes did not differ in clinical psychopathology, they diverged in their activation profi
288 ubstantial fraction of genetic influences on psychopathology transcend clinical diagnostic boundaries
289 and constrain hypotheses about the nature of psychopathology, treatment mechanisms, and treatment tar
290 ychosis spectrum symptoms and 609 with other psychopathologies), using a recently developed, validate
291 the same participants revealed that overall psychopathology was associated with decreased connectivi
293 ical thickness in most networks, and overall psychopathology was associated with globally reduced gra
296 nsive involvement of neuroactive steroids in psychopathology, we hypothesized that the behavioral com
298 rly psychiatric patients had lower levels of psychopathology when assessed while wearing hearing aids
299 This question is of particular importance in psychopathology, where enhanced attention to threat is a
300 disorder, psychosis, depression, and general psychopathology) with the resulting striatal connectivit