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1 (HDM) extract derived from Dermatophagoides pteronyssinus.
2 of PBMC with beta-lactoglobulin and with D. pteronyssinus.
3 om the United Kingdom (n = 56) exposed to D. pteronyssinus.
4 32 registered allergens of Dermatophagoides pteronyssinus.
5 sedust mite (odds ratio for Dermatophagoides pteronyssinus 0.20 [0.05-0.81], p=0.02; D farinae 0.20 [
7 f house-dust-mite allergen (Dermatophagoides pteronyssinus 1 [Der p1]) in the management of asthma is
8 5% in response to dust mite Dermatophagoides pteronyssinus, 40% in response to dust mite D. farinae,
9 and of IL-2 in response to Dermatophagoides pteronyssinus allergen 1 (Derp1) was lower in the salmon
14 the development of food allergy, that is, D pteronyssinus allergens in breast milk, which disrupt gu
15 Parental hay fever and early exposure to D pteronyssinus allergens promote IgE polysensitization to
16 ybrid molecules as a therapeutic model in D. pteronyssinus allergic mice led to the reduction of IgE
19 ovocation tests (NAPT) with Dermatophagoides pteronyssinus, Alternaria alternata, Olea europaea and g
20 rgen provocation tests with Dermatophagoides pteronyssinus, Alternaria alternata, Olea europea, and a
21 pecific AIT should rely upon a mixture of D. pteronyssinus and D. farinae extracts, manufactured from
22 nce of mite allergens recorded by IUIS in D. pteronyssinus and D. farinae extracts, with groups 2, 8,
25 th a sublingual solution of Dermatophagoides pteronyssinus and Dermatophagoides farinae extracts or a
27 ophagoides farinae (Df) and Dermatophagoides pteronyssinus and from the mold Aspergillus fumigatus st
33 e peritrophic matrix lining the midgut of D. pteronyssinus as well as on the surface of the fecal pel
34 trated nasal provocation tests (NPT) with D. pteronyssinus at baseline and at the end of the study.
35 th species had a high ratio (> 10) of IgE D. pteronyssinus:B. tropicalis, whereas 68% of patients exp
36 n addition, specific IgE to Dermatophagoides pteronyssinus, Blomia tropicalis and their tropomyosins
37 l infections are common and widespread in D. pteronyssinus, both in natural and culture-based growth
41 otective IgG mainly against Dermatophagoides pteronyssinus (Der p) 1 and Der p 2 and to a lesser exte
42 ls were pulsed with TSLP or Dermatophagoides pteronyssinus (Der p) allergen, before FACS phenotyping
43 ith a whole body extract of Dermatophagoides pteronyssinus (Der p), one of the causative agents of ho
45 early life and analyzed for Dermatophagoides pteronyssinus (Der p1) and Dermatophagoides farinae (Der
47 n the house dust mite (HDM) Dermatophagoides pteronyssinus, Der p 1, 2, 5, 7, 21, and 23 have been id
48 tly related to higher FeNO: Dermatophagoides pteronyssinus, Dermatophagoides farina, Blomia tropicali
49 ngual tablet formulation of Dermatophagoides pteronyssinus:Dermatophagoides farinae 1:1 extract in ad
50 ngual tablet formulation of Dermatophagoides pteronyssinus:Dermatophagoides farinae 1:1 extract in ad
51 A cDNA coding for a new Dermatophagoides pteronyssinus (Dp) allergen, Der p 37, was isolated from
52 were subjected to NAC with Dermatophagoides pteronyssinus (DP), Alternaria alternata (AA), grass (GP
54 en implemented for thirteen Dermatophagoides pteronyssinus enzymatic activities, associated with Der
57 e sensitized against native Dermatophagoides pteronyssinus extract and repeatedly treated by applicat
58 edly treated by application of depigmented D pteronyssinus extract via laser-generated skin micropore
61 ization of RNA viruses from Dermatophagoides pteronyssinus, followed by their detection in different
62 rns of the IgE response to 12 molecules of D pteronyssinus from birth to adulthood and to investigate
65 ns from the house dust mite Dermatophagoides pteronyssinus Identification of human IgE Ab binding epi
66 Survey 2, having information on cat/grass/D. pteronyssinus IgE levels and symptoms on exposure to ani
67 , and skin sensitisation to Dermatophagoides pteronyssinus in 205 cases and 399 controls aged over 16
68 .001) and on the bedroom floor (P=0.004), D. pteronyssinus in the bed (P=0.007), and cockroach allerg
70 The house dust mite (HDM) Dermatophagoides pteronyssinus is one of most important allergen sources
73 f IgE, IgG and IgG(4) to 13 Dermatophagoides pteronyssinus molecules were measured at baseline and af
74 s promote IgE polysensitization to several D pteronyssinus molecules, which in turn predicts current
75 llen (n = 108), followed by Dermatophagoides pteronyssinus (n = 105), Dermatophagoides farina (n = 96
76 sal allergen challenge with Dermatophagoides pteronyssinus (NAC-DP) in mucosal and peripheral B-cell
77 12 purified allergens from Dermatophagoides pteronyssinus or D. farinae were assessed in sera from 1
78 ization to house dust mite (Dermatophagoides pteronyssinus) or the fungal allergen Alternaria alterna
79 change in total IgE, IgE to Dermatophagoides pteronyssinus, or IgE to Blattella germanica between the
80 change in total IgE, IgE to Dermatophagoides pteronyssinus, or IgE to Blattella germanica between the
82 lergens (NAPT-M), including Dermatophagoides pteronyssinus, pollens, alternaria, and dog epithelia, w
83 posures to aerosolized HDM (Dermatophagoides pteronyssinus) powder on 4 consecutive days in an allerg
84 cells (PBMC) to beta-lactoglobulin and to D. pteronyssinus; production of IFN-gamma on stimulation of
85 -old mice breast-fed by mothers exposed to D pteronyssinus, protease-inactivated D pteronyssinus, or
86 risons of IgE responses to B. tropicalis, D. pteronyssinus, rBlo t 5, and rDer p 5, showed that B. tr
87 e were injected with IgE to Dermatophagoides pteronyssinus (rDer p)2 and then exposed intranasally to
89 in silico metatranscriptomic analysis of D. pteronyssinus RNA samples, visualized by electron micros
90 .58-6.61], p for interaction=0.017), where D pteronyssinus sensitisation was common, but unrelated to
92 ent who also had dust mite (Dermatophagoides pteronyssinus)-specific IgE antibodies from time points
94 ints with various ratios of polyclonal to D. pteronyssinus-specific IgE (16:1 to 86:1), we demonstrat
95 ed that increased ratios of polyclonal to D. pteronyssinus-specific IgE did not attenuate basophil se
96 E antibody responses to B. tropicalis and D. pteronyssinus that were related to exposure: only 22% of
97 n from the house dust mite, Dermatophagoides pteronyssinus, that belongs to the papain-like cysteine
98 ne whether oral exposure to Dermatophagoides pteronyssinus through breast milk affects gut mucosal im
101 ities (51.1% and 18.8%, respectively), and D pteronyssinus was the most relevant aeroallergen (51.1%)
103 ermatophagoides farinae and Dermatophagoides pteronyssinus were found to be above the detection limit
104 IgE to the extract and 12 molecules of D pteronyssinus were tested by means of ImmunoCAP and micr
105 concurrently exposed to B. tropicalis and D. pteronyssinus, with patients from the United States and