ライフサイエンスコーパス: ptosis

1 with distinctive facial features, including ptosis.

2 6 (14.9%) of the 107 patients with childhood ptosis.

3 were less likely to have postoperative brow ptosis.

4 cell proliferation, differentiation, and apo ptosis.

5 tions and new proposals in the management of ptosis.

6 ocomotion, grooming, diarrhea, tachypnea and ptosis.

7 e use of frontalis suspension for correcting ptosis.

8 ive ophthalmoplegia, often with accompanying ptosis.

9 al mortality, scoliosis, resting tremors and ptosis.

10 ve paralytic ophthalmoplegia with or without ptosis.

11 in vivo by reversal of tetrabenazine-induced ptosis.

12 nonproptotic ophthalmoplegia with or without ptosis.

13 in the left upper eyelid, causing mechanical ptosis.

14 dy criteria, of which 142 eyelids had severe ptosis.

15 nital, neurogenic, traumatic, or aponeurotic ptosis.

16 ongenital ptosis and 30 patients aponeurotic ptosis.

17 g injections appears to increase the risk of ptosis.

18 ve method for patients with mild-to-moderate ptosis.

19 n the eyelids with and without postoperative ptosis.

20 s of levator resection surgery in congenital ptosis.

21 ant effect on the frequency of postoperative ptosis.

22 d symmetric high myopia, normal stature, and ptosis.

23 ctive surgery of extraocular muscles to ease ptosis.

24 ted with a higher frequency of postoperative ptosis.

25 ed improvement in muscle strength and eyelid ptosis.

26 at a tertiary medical center for congenital ptosis.

27 ds and feet, scoliosis, ophthalmoplegia, and ptosis.

28 y sympathetic nervous system defects causing ptosis.

29 episodes of facial hypotonia, jaw drop, and ptosis.

30 a levator advancement was required to repair ptosis.

31 0 (18.7%) of the 107 patients with childhood ptosis.

32 ; 3.05%), and granuloma (N = 15/393; 3.82%), ptosis 2.6% (N = 9/343).

33 ures, 5 (11%) permanent and 4 (9%) transient ptosis, 5 (11%) thermoregulation difficulties, 4 (9%) a

34 epicanthic folds (84%), hypertelorism (68%), ptosis (56%), high upper eyelid crease (64%), lower eyel

35 retro-orbital pain and later with diplopia, ptosis, 6th nerve and pupil-sparing partial 3rd nerve pa

36 diplopia, abnormal eyelid signs (retraction, ptosis, absent crease), ocular asymmetry (hypoglobus, en

37 r degeneration (AMD) and those who developed ptosis after intravitreal injections.

38 The mean degree of ptosis after surgery was 0.9 +/- 0.8 mm.

39 The mean degree of ptosis after surgery was 0.9 0.8 mm.

40 Four patients were diagnosed with congenital ptosis and 30 patients aponeurotic ptosis.

41 lar muscles (CFEOM1) are born with bilateral ptosis and a restrictive infraductive external ophthalmo

42 icial to patients with both mild to moderate ptosis and an asymmetric crease height.

43 form neurofibromas, most commonly because of ptosis and anisometropia.

44 ome but that some specific features, such as ptosis and blepharophimosis, are mostly driven by BRPF1

45 rgeons have made a change in the delivery of ptosis and blepharoplasty surgical services after the re

46 ia eliminates needle risk as well as risk of ptosis and bruising.

47 Dysfunction of ocular muscles, ptosis and cataract are the most common ophthalmologic m

48 Ptosis and diplopia developed in 2 patients despite Medp

49 er of late onset that commonly presents with ptosis and dysphagia.

50 In a consanguineous family with congenital ptosis and elevation of the ptotic eyelid with ipsilater

51 Eighteen months post initial presentation ptosis and eye movements returned normal and choroidal e

52 ngestion, 46% conjunctival injection and 40% ptosis and facial flushing.

53 tion, 58% rhinorrhoea, 54% nasal congestion, ptosis and facial flushing.

54 (9.9%) were diagnosed with simple congenital ptosis and had strabismus, of which there were 4 (4.9%)

55 he 10-year results of surgery for congenital ptosis and identify factors associated with excellent ou

56 vances in the anatomic understanding of brow ptosis and in the procedures used to correct the resulti

57 ovel technique in treating severe congenital ptosis and introduces an innovative approach to Silicone

58 nority of individuals have been upper eyelid ptosis and midline dermoid cysts of craniofacial structu

59 ess was 77.4% and 85% in eyelids with severe ptosis and mild/moderate ptosis, respectively (P = .15).

60 was 97.2% and 90.9% in patients with severe ptosis and mild/moderate ptosis, respectively (P = .42).

61 cally characterized by exercise intolerance, ptosis and muscle weakness.

62 Patients with severe ptosis and nearly total absence of levator muscle functi

63 months, with little or no facial weakness or ptosis and no ophthalmoplegia.

64 acial videos (kappa >= 0.845 for strabismus, ptosis and nystagmus, and kappa = 0.801 for thyroid-asso

65 rom retrochiasmal visual pathway damage, and ptosis and ocular dysmotility from extraocular muscle in

66 t (90.3% and 85.1%, respectively), including ptosis and ocular movement deficits.

67 sarthria, gross motor regression, hypotonia, ptosis and ophthalmoplegia and had abnormal signals in b

68 probands from 4 families who presented with ptosis and ophthalmoplegia as well as other clinical man

69 of classic myasthenic manifestations such as ptosis and ophthalmoplegia or facial weakness, and links

70 spiratory and generalized limb weakness with ptosis and ophthalmoplegia.

71 a late-onset ocular myopathy beginning with ptosis and progressing slowly.

72 utosomal recessive disorder characterized by ptosis and progressive external ophthalmoplegia, periphe

73 in the fifth or sixth decade with dysphagia, ptosis and proximal limb weakness.

74 in the fifth or sixth decade with dysphagia, ptosis and proximal limb weakness.

75 dure, a historical knowledge of involutional ptosis and ptosis repair is necessary.

76 CFEOM presents with congenital ptosis and restricted eye movements, and can be caused b

77 Affected individuals are born with bilateral ptosis and restrictive ophthalmoplegia with the globes "

78 either progressive external ophthalmoplegia/ptosis and spastic ataxia, or a progressive ataxic disor

79 patients in this case series documented mild ptosis and striking orthostatic reductions in intraocula

80 al dysmorphism, colobomatous microphthalmia, ptosis and syndactyly with or without nephropathy, assoc

81 surgical options depends on the cause of the ptosis and the amount of levator function.

82 A man in his late 60s had ptosis and tremor on standing for 30 years, followed by

83 rbital rejuvenation, addressing lateral brow ptosis and upper eyelid excess.

84 Unfortunately, her ptosis and upward gaze restriction was not improved afte

85 amblyopia who were diagnosed with childhood ptosis and were residents of Olmsted County, Minnesota,

86 -five percent (15 of 23) of our patients had ptosis and/or diplopia, each present in 11 individuals.

87 gical data, indication for surgery, previous ptosis and/or eyelid surgeries and trauma histories, pre

88 ial nerve branches, which can result in brow ptosis and/or orbicularis oculi weakness.

89 haracterized by progressive eyelid drooping (ptosis) and dysphagia although muscles of the limbs can

90 cular muscles and results in droopy eyelids (ptosis) and progressive external ophthalmoplegia.

91 cular muscles and results in droopy eyelids (ptosis) and progressive external ophthalmoplegia.

92 10 patients diagnosed with simple congenital ptosis, and a predominance of isolated horizontal deviat

93 cted the symptoms of tearing, lagophthalmos, ptosis, and diplopia and measured margin-to-reflex dista

94 se the unusual combination of optic atrophy, ptosis, and encephalomyopathy leading to intractable sei

95 physical appearance secondary to proptosis, ptosis, and facial disfigurement, leading to social emba

96 a neurodevelopmental syndrome with epilepsy, ptosis, and hypothyroidism that differs from JBS.

97 ve approach in treating patients with severe ptosis, and it may offer superior outcomes to ELR.

98 We studied a woman who presented with PEO, ptosis, and weakness of pharyngeal, facial, neck, and li

99 lasia, decreased innervation of the gut, and ptosis are consistent with impaired Ret signaling.

100 ture on the classification and management of ptosis are reviewed here.

101 options for correction of specific types of ptosis are reviewed.

102 The treatment options for ptosis are strictly surgical.

103 Isolated unilateral ptosis as a presenting sign of SLE is uncommon and diagn

104 tive drop of MRD1 and clinically significant ptosis as MRD1 drop of >=2 mm.

105 ren who were diagnosed with childhood eyelid ptosis as residents of Olmsted County, Minnesota, from J

106 ble, the simultaneous onset of OT and eyelid ptosis at a much younger age than usually observed for O

107 ical records of the patients with unilateral ptosis between October 2015 and December 2020 were revie

108 elta translocates to mitochondria during apo-ptosis,but its mitochondrial target remains unclear.

109 In IXT patients, the presence of coexisting ptosis can have a further deleterious impact on binocula

110 Bilateral ptosis cases with documented Hering's dependency yield b

111 Demographic data, baseline ptosis characteristics and postoperative results at 1st

112 group, 18 of 1100 patients (1.6%) developed ptosis, compared with 52 of 2258 patients (2.3%) in the

113 (2.0%) injected without a speculum developed ptosis, compared with 8 of 444 patients (1.8%) injected

114 The attacks were associated with ipsilateral ptosis, conjunctival injection, lacrimation, rhinorrhoea

115 gle exotropia, vertical gaze deficiency, and ptosis consistent with congenital fibrosis of the extrao

116 the 1 mm correction effect (p-value = 0.67), ptosis correction (p-value = 0.60), and post-operation d

117 MMCR is an effective surgical method for ptosis correction as it can not only correct the eyelid

118 s study was to assess outcomes of unilateral ptosis correction based on parameters including degree o

119 he increase in Muller's muscle fibrosis, the ptosis correction effect of MMCR surgery increases, but

120 ontalis sling surgery is a common method for ptosis correction for both pediatric and adult populatio

121 ures should be regarded as a last resort for ptosis correction in adults due to the elevated risk of

122 scle hypertrophy and 1 mm correction effect, ptosis correction, and post-operation difference accordi

123 eyelid surgeries, such as blepharoplasty and ptosis correction, are commonly performed procedures wor

124 surgical time; speculum width; incidence of ptosis (defined as a decrease in MRD1 by 2 mm) postopera

125 patients diagnosed with a congenital form of ptosis demonstrated amblyopia.

126 After pterional craniotomy, ptosis, diplopia, and vertical gaze limitation can resul

127 ith exotropia, vertical gaze limitation, and ptosis do not have classic Moebius syndrome and may have

128 mon etiology in the adult group was myogenic ptosis due to a systemic condition (47%) (p < 0.001).

129 with the classical features of OPMD, namely ptosis, dysphagia and cytoplasmic inclusions on muscle b

130 M) is an inherited myopathy manifesting with ptosis, dysphagia and distal weakness.

131 a rare late onset genetic disease leading to ptosis, dysphagia and proximal limb muscles at later sta

132 is an adult-onset disorder characterized by ptosis, dysphagia and proximal limb weakness.

133 minant disorder characterized by progressive ptosis, dysphagia, and extremity weakness.

134 of acute cranial nerve dysfunction including ptosis, dysphagia, blurred vision, and motor weakness.

135 Blepharophimosis, ptosis, epicanthus inversus syndrome (BPES) is an autoso

136 Blepharophimosis, ptosis, epicanthus inversus syndrome type I (BPES; OMIM

137 Blepharophimosis-Ptosis-Epicanthus Inversus Syndrome (BPES) is a rare con

138 the lacrimal gland (LG) in blepharophimosis-ptosis-epicanthus inversus syndrome (BPES).

139 18 eyes of 9 patients with Blepharophimosis-ptosis-epicanthus inversus syndrome who presented to ocu

140 phology in both sexes (the 'blepharophimosis-ptosis-epicanthus inversus syndrome', BPES).

141 eon and reviewed in detail: blepharophimosis-ptosis-epicanthus inversus syndrome, congenital fibrosis

142 tion disorders--a subset of blepharophimosis-ptosis-epicanthus inversus syndrome, Miller-Dieker lisse

143 ranscription factor, causes blepharophimosis/ptosis/epicanthus inversus syndrome (BPES), a rare devel

144 xL2 are associated with the blepharophimosis/ptosis/epicanthus inversus syndrome characterized with c

145 the literature and in this patient, included ptosis, esotropia, coloboma of the iris, retina, choroid

146 All patients underwent standard preoperative ptosis evaluation with margin-to-reflex distance 1 and 2

147 cluding best corrected visual acuity (BCVA), ptosis evaluation, dilated fundus examination, and ortho

148 tonomic dysfunction in children undergoing a ptosis evaluation.

149 ssive external ophthalmoplegia (PEO), eyelid ptosis, exercise intolerance and skeletal muscle weaknes

150 other eyelid diseases, such as involutional ptosis, eyelid granulomatous or cancerous lesions.

151 hic data such as age and gender and specific ptosis findings e.g. the cause and duration, MRD-1, and

152 sh a map location for an isolated congenital ptosis gene and demonstrate that this disorder is geneti

153 is study (blepharoplasty group: 23 patients, ptosis group 31 patients).

154 adult patients with dermatochalasis and the ptosis group consisted of adult patients with dermatocha

155 The IXT-ptosis group had worse distance stereoacuity, and a larg

156 Furthermore, the IXT-ptosis group had worse stereopsis at distance (p = 0.01)

157 suppression on the Bagolini test in the IXT-ptosis group than in the IXT-only group (p = 0.04).

158 45 IXT patients with congenital ptosis (IXT-ptosis group) and 58 age-matched IXT patients without pt

159 ance and near were slightly worse in the IXT-ptosis group, the differences were not statistically sig

160 omal-dominant form of mild syndromic ID with ptosis, growth retardation, and hypotonia, and we identi

161 Surgical treatment of congenital ptosis had a high success rate.

162 81 patients diagnosed with simple congenital ptosis had amblyopia, 7 (8.6%) cases of which solely wer

163 Those patients with a history of ptosis had undergone surgery with levator procedure at l

164 Four patients had traumatic ptosis history whereas four patients had previous multip

165 aly, low frontal hairline, facial asymmetry, ptosis, hypertelorism, broad great toes, and clinodactyl

166 ial features, and ocular alterations such as ptosis, hypertelorism, nystagmus, and chorioretinal colo

167 uals with intellectual disability, epilepsy, ptosis, hypothyroidism, and genital anomalies, we uncove

168 50-year-old female had painless progressive ptosis in both eyes for 6 months.

169 d to the noninjection group and incidence of ptosis in patients whose injections were performed with

170 The outcomes were the incidence of ptosis in the injection group compared to the noninjecti

171 Seven patients had ptosis in the left eye whereas one patient had ptosis in

172 al techniques for treating severe congenital ptosis in the paediatric age group: Silicon rods ptosis

173 The most common etiology of ptosis in the pediatric group was simple congenital (70%

174 osis in the left eye whereas one patient had ptosis in the right eye.

175 a occurred in 1 in 7 children diagnosed with ptosis in this population-based cohort.

176 hildren diagnosed with any form of childhood ptosis in this population-based cohort.

177 d sickness behavior (lethargy, piloerection, ptosis) in the GR(dim)-LPS mice was associated with incr

178 rome (KOS, also reported as blepharophimosis-ptosis-intellectual disability syndrome).

179 Hereditary isolated congenital ptosis is an autosomal dominant disorder with incomplete

180 geons have increasingly recognized that brow ptosis is an important contributor to dermatochalasis an

181 Bilateral ptosis is reported with unilateral hemispheric lesions,

182 oup) and 58 age-matched IXT patients without ptosis (IXT only group) who presented for eye examinatio

183 l records of 45 IXT patients with congenital ptosis (IXT-ptosis group) and 58 age-matched IXT patient

184 ain, pain in the arm used for drug infusion, ptosis, leg cramps, and visual and voice changes.

185 The cardinal features were ptosis, limited elevation, and hypotropia.

186 ents (30 per group) with severe involutional ptosis [Margin Reflect Distance 1 (MRD1) <= 0 mm] who un

187 Isolated unilateral ptosis may represent a rare neuro-ophthalmic manifestati

188 Automated ptosis measurements produced by our software algorithm c

189 well as skeletal and eye abnormalities (ie, ptosis, myopia, and retina detachment).

190 er eyelid crease (n = 20, 69%), upper eyelid ptosis (n = 14, 52%), and superior sulcus hollowing (n =

191 d by strabismus (n = 2, 10%), occlusion from ptosis (n = 9, 43%), or anisometropia (n = 9, 43%), or a

192 in 5 patients because of severe ophthalmic (ptosis, n = 2; retinal ischemia, n = 2) or systemic (hyp

193 es were visual acuity, occurrence of induced ptosis, need for further surgery, cosmesis, and quality

194 attern of muscle weakness included bilateral ptosis (non-fatigable in adulthood), myopathic facies an

195 cus often have atypical phenotypes including ptosis, obstructive sleep apnoea, and the occurrence of

196 ealed marked periorbital edema and hematoma, ptosis, ocular movements limitation, an infero-temporal

197 clinic for subacute swelling, reddening, and ptosis of her left upper eyelid.

198 important contributor to dermatochalasis and ptosis of the upper eyelid.

199 This study aims to evaluate the effect of ptosis on the binocular function of patients with IXT.

200 rns-Sayre syndrome consisting of progressive ptosis, ophthalmoparesis, mitochondrial myopathy, and pi

201 vere gastrointestinal dysmotility; cachexia; ptosis, ophthalmoparesis, or both; peripheral neuropathy

202 report describes monozygotic male twins with ptosis, optic atrophy, and recent-onset intractable myoc

203 lacrimation, nasal congestion, rhinorrhoea, ptosis or eyelid oedema.

204 of the patients required further surgery for ptosis or strabismus.

205 oor levator function (<= 4mm), and syndromic ptosis or systemic diseases were excluded.

206 surgery for cataract, strabismus, nystagmus, ptosis, or nasolacrimal duct obstruction.

207 regulator of the TNF-alpha-mediated anti-apo-ptosis pathways in RA FLSs.

208 ons, and innovative materials in the care of ptosis patients.

209 r suturing techniques in severe involutional ptosis patients.

210 teral optic neuropathy, ophthalmoplegia with ptosis, pigmentary retinopathy, and retrochiasmal visual

211 abnormal signs in the animals: for example, ptosis, piloerection, tremor, ataxia or exophthalmos.

212 Severe congenital ptosis poses a complex challenge for oculoplastic surgeo

213 isystem disorder characterized clinically by ptosis, progressive external ophthalmoplegia, gastrointe

214 onset typically before the age of 30 years; ptosis; progressive external ophthalmoplegia; gastrointe

215 nt are variable and include eyelid swelling, ptosis, proptosis, and loss of vision.

216 ber of patients in Group 1 had mild residual ptosis, proptosis, and movement restriction at 12 weeks,

217 h of hospital stay, and sequelae of disease (ptosis, proptosis, and movement restriction) were evalua

218 utures, inflammation, granuloma formation or ptosis recurrence, were registered.

219 Physical examination revealed mild bilateral ptosis, reduced muscle tone and strength that worsened i

220 atients undergo separate surgical visits for ptosis repair and blepharoplasty, is not desirable to mo

221 d 2 are crucial for the surgical planning of ptosis repair and blepharoplasty.

222 to examine the history of posterior approach ptosis repair and the events that have led to its curren

223 view was performed of patients who underwent ptosis repair between 2012 and 2019.

224 the MMCR, and the current reimbursements for ptosis repair by insurance companies.

225 Posterior approach ptosis repair has made a resurgence over the last decade

226 MMCR ptosis repair is an effective approach in treating patie

227 torical knowledge of involutional ptosis and ptosis repair is necessary.

228 Concomitant blepharoplasty did not affect ptosis repair outcomes in any group.

229 The rate of ptosis repair success in eyelids that underwent MMCR was

230 In eyelids that underwent ELR, the rate of ptosis repair success was 77.4% and 85% in eyelids with

231 ty or having to wait at least 3 months after ptosis repair to have a blepharoplasty.

232 eria were patients who underwent MMCR or ELR ptosis repair, patients with complete documentation of p

233 ontour symmetry is an essential parameter in ptosis repair, yet temporal asymmetry often persists aft

234 important adjuncts for adult and congenital ptosis repair.

235 e studies also included the addition of brow ptosis repair.

236 tant blepharoplasty performed at the time of ptosis repair.

237 d in two favored techniques for involutional ptosis repair: the Muller muscle-conjunctiva resection (

238 There are certain established surgeries in ptosis repair; however, there is no ideal surgical techn

239 g the etiology and appropriate management of ptosis requires a directed evaluation seeking specific s

240 onally, all mutation carriers had congenital ptosis requiring surgery, 4 had myopia, 2 had retinal de

241 The ptosis resolved completely within 24 h of initiating hig

242 eyelids with severe ptosis and mild/moderate ptosis, respectively (P = .15).

243 atients with severe ptosis and mild/moderate ptosis, respectively (P = .42).

244 ngenital disorder characterized by bilateral ptosis, restrictive external ophthalmoplegia with the ey

245 Eyelids with persistent postoperative ptosis showed a significantly (P = .03) lower preoperati

246 is in the paediatric age group: Silicon rods ptosis sling and a novel technique involving the use of

247 e study included 34 patients with congenital ptosis subjected to levator muscle plication surgery dur

248 gait ataxia, scoliosis, resting tremors, and ptosis, suggesting a defect in proprioception.

249 ution of the posterior and anterior approach ptosis surgeries has resulted in two favored techniques

250 should be aware of the risk of dry eye after ptosis surgery and discuss dry eye as a complication of

251 f patients who underwent levator advancement ptosis surgery between April 2002 and December 2004 by t

252 ive OSDI score was significantly higher post ptosis surgery compared with the preoperative score (25.

253 e LG scores and in fluorescein staining post ptosis surgery compared with the preoperative values (pa

254 c changes in adult patients following eyelid ptosis surgery is 1 year and a considerable number of pa

255 tear production is recommended especially if ptosis surgery is planned.

256 rithm showing the appropriate/most preferred ptosis surgery techniques is prepared based on the tradi

257 The recent preference for posterior approach ptosis surgery, in particular the MMCR, is multifactoria

258 after individualized amblyopia treatment and ptosis surgery, with at least one year of follow-up, wer

259 stopathological findings and the outcomes of ptosis surgery.

260 cceptable result to evaluate the outcomes of ptosis surgery.

261 late-onset muscle disorder characterized by ptosis, swallowing difficulties, proximal limb weakness

262 h exotropia and aberrant regeneration with a ptosis that improved on adduction.

263 d of adult patients with dermatochalasis and ptosis that showed significant improvement after phenyle

264 eferred for surgical correction of right eye ptosis that was present for 2 years.

265 fering opinions on the cause of involutional ptosis, the mechanism by which the MMCR works, the predi

266 comes of MMCR vs ELR in patients with severe ptosis, there was a statistically significant higher rat

267 multaneous correction of both strabismus and ptosis through a single procedure.

268 Age, gender, ptosis type, type of sling, complications, and re-surger

269 Forty-seven patients with mild to moderate ptosis underwent MMCR surgery and pathological samples i

270 tion, high upper eyelid crease, upper eyelid ptosis, upper and/or lower eyelid retraction, and eyelid

271 The mean degree of ptosis was 3.9 +/- 1.6 mm.

272 The mean degree of ptosis was 3.9 1.6 mm.

273 t hemispheric infarctions, in whom bilateral ptosis was accompanied by impaired upward gaze.

274 Photo-based ptosis was assessed by a masked oculofacial plastic surg

275 Clinical ptosis was defined as any postoperative drop of MRD1 and

276 Severe ptosis was described as an MRD1 of 0 or worse.

277 both ocular and pharyngeal muscle weakness, ptosis was just as likely to occur before or concurrent

278 Within the injection group, ptosis was mostly bilateral, diagnosed on average 22.4 m

279 Persistent clinically significant ptosis was observed in more than 3% of patients undergoi

280 'limb-girdle' pattern of weakness; although ptosis was often present from an early age, eye movement

281 Preoperative significant ptosis was present in 28/31 (90.3%) of the upper eyelid

282 Eyelid ptosis was reported in 1 patient (1 event).

283 Ptosis was the first reported symptom in two-thirds of t

284 cal, clinically significant, and photo-based ptosis were 25.4% (71/279), 3.2% (9/279), and 3.3% (9/27

285 rsions, angle of misalignment, and degree of ptosis were evaluated before surgery and at last follow-

286 ignificant differences in incidence rates of ptosis were observed.

287 rafting and postoperative residual upper lid ptosis were significantly greater in the transconjunctiv

288 Induction of apo-ptosis which causes the insertion of the soluble form of

289 onal case series on patients with congenital ptosis who underwent levator muscle resection in Farabi

290 Patients with unilateral ptosis who underwent surgical correction and levator fun

291 y-three patients diagnosed with involutional ptosis who underwent surgical correction using a small o

292 Eyelids of patients with severe congenital ptosis with poor levator function who underwent either f

293 d effective surgical strategy for congenital ptosis with poor levator function, providing excellent e

294 blepharoptosis, used widely in the repair of ptosis with poor levator function.

295 tory of SLE presented with acute right-sided ptosis without other neurological deficits while on stab

296 ses with good levator function or (amount of ptosis x 3) + 11 mm in cases with fair levator function]

297 culated by a traditional formula [(amount of ptosis x 3) + 9 mm in cases with good levator function o