ライフサイエンスコーパス: puerperal

1 usefulness, efficacy, and safety of REBOA in puerperal bleeding due to abnormalities of placental imp

2 thrombotic embolism (4.8; 95% CI, 2.9-7.8), puerperal cerebrovascular disorders (4.7; 95% CI, 3.0-7.

3 A neuronal population with a central role in puerperal changes is the tuberoinfundibular dopamine (TI

4 Puerperal complications represent a diagnostic challenge

5 tcomes comprised chorioamnionitis in labour, puerperal endometritis, wound infection following cesare

6 Puerperal episodes clustered in families.

7 mplicated in susceptibility to triggering of puerperal episodes in women with bipolar disorder.

8 .5%; RD, -0.24 [95% CI, -0.47 to -0.01]) and puerperal fever (0.1% vs 0.3%; RD, -0.19 [95% CI, -0.36

9 vo infection model of the human decidua, the puerperal fever portal of entry.

10 ers during childbirth was approximately 10%; puerperal fever still accounts for over 75,000 maternal

11 ry) and 6.9 percent had minor complications (puerperal fever, a need for blood transfusion, or abdomi

12 00 due to invasive infections and among them puerperal fever.

13 hospital within 42 days of birth, and other puerperal infection varied according to whether she had

14 of maternal mortality (ie, induced abortion, puerperal infection, and pregnancy-induced hypertension)

15 l tolerance, which may increase the risk for puerperal infection.

16 tion of labor (RR, 1.15; 95% CI, 1.03-1.28), puerperal infections (RR, 1.32; 95% CI, 1.02-1.70), pret

17 ffects on bacterial carriage and resistance, puerperal infections, and infant growth.

18 al infections following childbirth-so-called puerperal infections-cause morbidity in 5%-10% of all ne

19 one that, distinct from previously described puerperal modifications, reverses fully on weaning.

20 not correspond to pregnancy, childbirth, the puerperal period, or the perinatal period.

21 idity and mortality during pregnancy and the puerperal period.

22 cations during pregnancy, childbirth, or the puerperal period.

23 rlap in the familial factors predisposing to puerperal psychosis and bipolar disorder.

24 Family studies of puerperal psychosis consistently demonstrate familial ag

25 Episodes of puerperal psychosis followed 81 (26%) of 313 deliveries

26 bipolar disorder who had a family history of puerperal psychosis in a first-degree relative but in on

27 ted a study of the occurrence of episodes of puerperal psychosis in families multiply affected with b

28 Episodes of puerperal psychosis occurred in 74% (N=20) of the 27 par

29 Puerperal psychosis, an episode of mania or psychosis pr

30 udies were identified on eating disorders or puerperal psychosis.

31 fluencing vulnerability to bipolar affective puerperal psychosis.

32 predispose individuals to bipolar affective puerperal psychosis.

33 disorder, 58 (38%) of whom had at least one puerperal psychotic episode.

34 supports the hypothesis that the majority of puerperal psychotic episodes are manifestations of an af

35 ence for familial aggregation of postpartum (puerperal) psychotic episodes in women with bipolar diso

36 = 226, aOR 2.3, 95% CI 1.9-2.8, p < 0.001), puerperal sepsis (0.27%, n = 76 versus 0.17%, n = 78, aO

37 Puerperal sepsis caused by group A Streptococcus (GAS) r

38 um women are at increased risk of developing puerperal sepsis caused by group A Streptococcus (GAS).

39 While outbreaks of puerperal sepsis have been ascribed to Streptococcus pyo

40 Pathogens causing puerperal sepsis include group A Streptococcus (GAS), an

41 Puerperal sepsis is a leading cause of maternal mortalit

42 Puerperal sepsis, a major cause of death of young women

43 lated from invasive infections, particularly puerperal sepsis, a severe infection that occurs during

44 nit admission, severe postpartum hemorrhage, puerperal sepsis, and severe preeclampsia.

45 s events that would favor the development of puerperal sepsis, including adhesion to cervical cells,

46 , malaria, and fever; postpartum infections (puerperal sepsis, mastitis), fever, and malaria; and use

47 tal tract and cause severe diseases, such as puerperal sepsis, neonatal infections, and necrotizing m

48 fections in women after childbirth, known as puerperal sepsis, resulted in classical epidemics and re

49 idemiologically associated with outbreaks of puerperal sepsis, specifically targets the human recepto

50 vide insights into the genesis of modern GAS puerperal sepsis, we reviewed the published cases and ca

51 ase our understanding of their enrichment in puerperal sepsis, we sequenced the genome of a genetical

52 esulting in life-threatening sepsis known as puerperal sepsis.

53 1 and M28, are more commonly associated with puerperal sepsis.

54 nsfusion, intensive care unit admission, and puerperal sepsis.

55 s being nonrandomly associated with cases of puerperal sepsis.

56 ation of serotype M28 isolates with cases of puerperal sepsis.

57 ns of an affective disorder diathesis with a puerperal trigger.

58 lial factors play a role in vulnerability to puerperal triggering itself.