ライフサイエンスコーパス: puff

1 placebo or active cannabis 'puffs' ($0.25-$5/puff).

2 ard (mealworms) and one with punishment (air puff).

3 icipation of drug-related rewards (cigarette puff).

4 or by focal application of glutamate (20 mM puffs).

5 ictive relationship between the tone and the puff.

6 arousal responses to hypoxia, sound, and air puff.

7 ist methysergide and mimicked by a serotonin puff.

8 d, a small liquid reward, or an aversive air-puff.

9 ng pre-test sucrose compared to pre-test air-puff.

10 to light mechanical stimulation from an air puff.

11 causally increased impatience for cigarette puffs.

12 wave-like releases interspersed with several puffs.

13 constitute elementary signals called calcium puffs.

14 ing the startle response with unexpected air puffs.

15 ntrations by taking longer and more frequent puffs.

16 amplitudes, kinetics, and spatial spread of puffs.

17 e evident within the durations of individual puffs.

18 ceptor (IP3R) channels underlying local Ca2+ puffs.

19 and rewards than after punished CSs and air puffs.

20 e activation can be visualized as chromosome puffs.

21 re risk-averse than those receiving many air-puffs.

22 smic reticulum to generate repetitive Ca(2+) puffs.

23 nd puffs with sixty-second intervals between puffs.

24 eavy smokers due to more frequent, intensive puffing.

25 reliable indices of heat load applied during puffing.

26 and pyrolysis regions of a cigarette during puffing.

27 in wheats were used for extrusion and kernel puffing.

28 0.15 to 0.43]), reduced mean daily albuterol puffs (-0.27 puff/d [CI, -0.49 to -0.04 puff/d]), and de

29 or decline 1-12 placebo or active cannabis 'puffs' ($0.25-$5/puff).

30 e-liquid consumed doubled from 3.7 to 7.5 mg puff(-1) and the total aldehyde emission rates tripled f

31 de emission rates tripled from 53 to 165 mug puff(-1), with acrolein rates growing by a factor of 10.

32 2000 ms before and coterminated with an air puff (100 ms) directed at the cornea as the US.

33 neurons, locomotion (44%-56%) and facial air puffs (50%) were more commonly and reliably encoded than

34 punishers of different types (sucrose vs air-puff; 50 kHz vs 22 kHz ultrasonic vocalisations) to rats

35 s vs 289.5 s) with a higher total number of puffs (58.6 vs 17.3), however this may be attributable t

36 In both species, hyperpolarizing HCs by puffing a glutamate antagonist, 6,7-dinitro-quinoxaline-

37 the venom gland transcriptome of the African puff adder Bitis arietans using paired-end reads sequenc

38 , Cerastes cerastes cerastes (Egypt) and the puff adder, Bitis arietans (Nigeria).

39 mug) of once-daily tiotropium or placebo (2 puffs) administered through the Respimat device every ev

40 2.5 mug (2 puffs of 1.25 mug), or placebo (2 puffs), administered through the Respimat device as add-

41 ced E-to Z-isomerization of all-E-lutein and puffing also of all-E-zeaxanthin.

42 ion time of puffs lengthened with increasing puff amplitude size, consistent with independent closing

43 nation time with cluster size independent of puff amplitude.

44 Mean puff amplitudes scale with cluster size, but individual

45 correlations between interpuff intervals and puff amplitudes.

46 hannel recruitment by CICR further determine puff amplitudes.

47 Several parameters, including puff and blip durations, puff latency and frequency, and

48 ture geometries on performance of CO2 Huff-n-Puff and CO2 continuous injection.

49 erent ocean regions and colony morphologies (puffs and rafts).

50 Puffs and sparks are localized intracellular Ca(2+) elev

51 onstant of Ca(2+) inactivation and find that puffs and sparks are often less sensitive to variations

52 zed intracellular Ca(2+) elevations known as puffs and sparks arise from the cooperative activity of

53 We study how the statistics of simulated puffs and sparks depend on the kinetics and dissociation

54 raction of inactivated channels that exhibit puffs and sparks that are nearly time-reversible and ter

55 c Ca(2+) excitability" reminiscent of Ca(2+) puffs and sparks where channels open and close in a conc

56 nactivation often leads to time-irreversible puffs and sparks whose termination is facilitated by the

57 sively fast Ca(2+) inactivation can preclude puffs and sparks, moderately fast Ca(2+) inactivation of

58 ation of IP(3)Rs and RyRs in the dynamics of puffs and sparks, we formulate and analyze Markov chain

59 py production rate of Markov chain models of puffs and sparks.

60 onstant is much greater than the duration of puffs and sparks.

61 s and global Ca(2+) waves, resembling Ca(2+) puffs and waves mediated by inositol trisphosphate (IP(3

62 To elucidate the relation of puffs and waves, we here model a cluster of IP3R channel

63 d InsP3Rs open together giving a local 'Ca2+ puff', and as puffs become more frequent they ignite reg

64 ms macroscopic, fusiform (tufts), spherical (puffs) and raft-like colonies that provide a pseudobenth

65 At P14, puff- and light-evoked NMDAR-mediated responses in both

66 elicited by either electrical stimulation or puff application in labelled PVN neurons was significant

67 and persistence of global signals even when puffs are absent, reveal a second mode of spatiotemporal

68 Puffs are local Ca(2+) signals that arise by Ca(2+) libe

69 Puffs are local Ca(2+) signals that arise by Ca(2+) libe

70 Calcium puffs are local transient Ca(2+) releases from internal

71 Puffs are localized, transient elevations in cytosolic C

72 stic and deterministic simulations show that puffs are not stereotyped events of constant duration bu

73 ), i.e., the waiting time between successive puffs, are found to be well characterized by a probabili

74 g studies have revealed local Ca2+ signals ("puffs") arising from clusters of IP3R, and patch-clamp s

75 s (CS) followed 250 ms later by a 100 ms air puff as an unconditioned stimulus (US) coterminating wit

76 conditioned stimulus (CS) followed by an air puff as an unconditioned stimulus (US) that coterminated

77 ment bias task that employed sucrose and air-puff as decision outcomes.

78 Despite the presence of a puff-associated oscillation in the rate of nicotine accu

79 ulation during cigarette smoking and/or (ii) puff-associated spikes in brain nicotine concentration.

80 10 nondependent smokers (NDS), suggest that puff-associated spikes in the brain nicotine concentrati

81 ected ecdysone-responsive genes reveals that puffing at Eip74EF ceases within an hour or two of cohes

82 to the variability of amplitudes of repeated puffs at a site but the amplitudes of successive puffs w

83 opipette stimulates local, repetitive Ca(2+) puffs at the region of cell contact.

84 ate incremental URF (IURF) of solvent huff'n'puff based on net solvent transfer into ultratight rock,

85 together giving a local 'Ca2+ puff', and as puffs become more frequent they ignite regenerative Ca2+

86 outh of an e-cig user are dependent upon the puffing behavior of the user.

87 that e-cigarette users tend to change their puffing behaviors when using e-liquids with reduced nico

88 owing a HGRAP choice that resulted in an air-puff, but this did not deter chickens from subsequently

89 products (AGEs) and antioxidant capacity of puffed cereals was studied.

90 MF looked like the most appropriate index in puffed cereals.

91 rder; control (C); control with noise of air puff (CN); air puff to conspecific hen (APC); air puff t

92 rom vaporizing PG and GLY under mild, single puff conditions.

93 d transport is driven by individual vortical puffs corresponding to plosive sounds; and 3) a distance

94 Product use and puff count were also assessed.

95 nce, corresponding effectively to a train of puffs, create conical, jet-like flows.

96 ), reduced mean daily albuterol puffs (-0.27 puff/d [CI, -0.49 to -0.04 puff/d]), and decreased mean

97 erol puffs (-0.27 puff/d [CI, -0.49 to -0.04 puff/d]), and decreased mean asthma symptom score (-0.26

98 ypic analyses confirm that sco is allelic to puff daddy(gw1) (pfd(gw1)), a null mutant in fbn2b, and

99 For a high rate of 250 puff day(-1) using a typical vaping regime and popular t

100 solvent into the matrix based on the huff'n'puff design parameters including solvent composition, dr

101 imilarly rapid inhibitory process terminates puffs despite local [Ca(2+)] elevation that would otherw

102 GABA puffs diluted in SM/CSF solution or SM/CSF solution alon

103 responded both to visual stimuli and to air puffs directed at the head and abdomen.

104 of LPA2 disorganizes the gradient of Ca(2+) puffs, disrupts gradient sensing, and reduces the direct

105 n puff volumes (41.6 mL vs 41.3 mL) and mean puff durations (1.4 s vs 1.5 s) were similar to that of

106 Vapers' mean puff durations (2.0 s and 2.2 s) were similar with both

107 cigarette (C651), although puff numbers and puff durations remained similar.

108 4), CO(2), N(2), and C(2)H(6)) during huff'n'puff enhanced oil recovery (EOR) improve recovery based

109 Among cigarette experimenters (1 puff), ever e-cigarette use was associated with higher o

110 IP(3), and increased numbers of local Ca(2+) puffs evoked by weaker photorelease.

111 Initial puffs evoked following photorelease of IP3-which would n

112 PFC produced a significant inhibition of air-puff-evoked blinks and reduced the generation of CRs com

113 tration was demonstrated to depend upon both puff flow rate and duration, while nicotine mass ratio w

114 e e-cig was used to collect emissions across puff flow rates and durations spanning the range observe

115 We find that the initial triggering rate of puffs following photorelease of IP(3), and the average f

116 These properties are similar to Ca(2+) "puffs" found in oocytes.

117 nt, including refractoriness and increase of puff frequency during the inter-wave interval.

118 average amount of Ca(2+) released per puff x puff frequency) varies about the square of cluster size,

119 lular signaling and, because of their higher puff frequency, preferentially act as pacemakers to init

120 lution of locally disordered patches (active puffs) from an ordered vortex-lattice flow state.

121 h coordinated activation of localized Ca(2+) puffs generated by stationary clusters of IP(3) receptor

122 da and xi, which represent the basal rate of puff generation and the recovery rate from refractorines

123 molecular complexes of LPA2 trigger a Ca(2+) puff gradient that governs gradient sensing and directio

124 13-Z- and 9-Z-zeaxanthin were identified in puffed grains (2x and 37x on average).

125 ficant positive effect on the pliability and puffing height of chapatti.

126 nt study, we developed a novel compressional-puffing-hydrothermal extraction (CPHE) process which pri

127 we provide evidence that heat shock-induced puffs in JIL-1 null mutant backgrounds are strongly labe

128 ing high-amplitude local Ca signals known as puffs in neurons and sparks in muscle cells.

129 n ASMC, as well as the statistics of calcium puffs in other cell types, can be quantitatively reprodu

130 We imaged puffs in SH-SY5Y cells using total internal fluorescence

131 phosphorylation at transcriptionally active puffs in such polytene squash preparations after heat sh

132 smooth muscle and from IP(3)-mediated Ca(2+) puffs in Xenopus oocytes.

133 The spatial extent of puffs increased with their amplitude, and puffs of simil

134 Puffing influenced starch in vitro digestibility, being

135 creased the volume and duration of cigarette puffs inhaled.

136 of IP3Rs increases, the average duration of puffs initially increases but then becomes saturated, wh

137 pography parameters such as puff time, inter-puff interval and puffs per cigarette.

138 odel, puffing topography (voltage, air-flow, puff interval), and method of collection on 19 elements/

139 volumes (52.2 mL and 83.0 mL) and mean inter-puff intervals (23.2 s and 29.3 s) differed significantl

140 ich was restored by rhythmic delivery of air puffs into the nasal cavity.

141 A point-source model of Ca(2+) puffs is then constructed based on the new IP3R model an

142 Information in the ion "puffs" is received and processed by the cell through res

143 Conversely, depolarizing HCs by puffing kainic acid (KA) into the outer plexiform layer

144 nt antioxidant capacity revealed by digested puffed kernels can be ascribed to both the new formed Ma

145 s CS extracts (CSEs) plus IL-1beta [CSE (6.4 puffs/L)/IL-1beta (2 mug/L)].

146 mal correspondence with experimental data of puff latencies after photorelease of IP3 was obtained wh

147 responses, we then simulated measurements of puff latency after step increases of [IP3].

148 rameters, including puff and blip durations, puff latency and frequency, and frequency of repetitive

149 The mean termination time of puffs lengthened with increasing puff amplitude size, co

150 in (small food quantity), no-risk (of an air-puff) (LGNAP) option.

151 (a P2Y(1) antagonist, 1 muM), ongoing spark/puff like activity and rhythmic intracellular Ca(2+) wav

152 Puffs make only small, transient contributions to global

153 esemble the elementary events ("sparks" and "puffs") mediated by IP(3) receptors and ryanodine recept

154 In this article, using a stochastic puff model and a single-channel data-based IP3R model, w

155 imuli (USs) (liquid rewards and aversive air puffs), newly learned reinforcement-predictive visual st

156 The amplitude of NMDAR currents elicited by puff NMDA application to dorsal horn neurons was also si

157 currents of dorsal horn neurons elicited by puff NMDA application.

158 d the increased amplitude of NMDAR-EPSCs and puff NMDA currents in labeled PVN neurons in SHRs but ha

159 or chelerythrine had any effect on mEPSCs or puff NMDA currents in labelled PVN neurons in Wistar-Kyo

160 y increased the amplitude of NMDAR-EPSCs and puff NMDA currents in PVN neurons in WKY rats but not in

161 he basal amplitude of evoked NMDAR-EPSCs and puff NMDA currents in retrogradely labeled PVN neurons w

162 e potentiating effect of AngII on mEPSCs and puff NMDA currents of labelled PVN neurons in SHRs.

163 aptic currents (mEPSCs) and the amplitude of puff NMDA currents of retrogradely labelled spinally pro

164 e basal amplitudes of evoked NMDAR-EPSCs and puff NMDA currents were significantly higher in SHRs tha

165 a larger reduction in evoked NMDAR-EPSCs and puff NMDA-elicited currents of PVN neurons in SHRs than

166 excitatory postsynaptic currents (EPSCs) and puff NMDA-elicited currents were recorded in spinally pr

167 NMDAR-mediated EPSCs and puff NMDA-elicited currents were recorded in spinally pr

168 Both dsGC types responded to puffed NMDA between P7 and P28; and both types exhibited

169 EPSCs and the amplitudes of evoked EPSCs and puff NMDAR currents in spinal dorsal horn neurons.

170 naptic EPSCs evoked from the dorsal root and puff NMDAR currents in spinal dorsal horn neurons.

171 contrast, neither CPD, total daily number of puffs, nor TDPV predicted nicotine metabolite levels abo

172 to the reference cigarette (C651), although puff numbers and puff durations remained similar.

173 ster sizes from the magnitude of the largest puff observed at each site relative to the signal from a

174 , and we investigated how the probability of puff occurrence varies with cluster size.

175 procedure in which a tone was followed by a puff of air to the eye.

176 ng this technique, we found that every 70 mL puff of an e-cigarette deposited 0.019% e-liquid (v/v) i

177 Twice daily beclomethasone treatment was one puff of beclomethasone (40 mug per puff) or placebo give

178 Results: A single puff of E-cig vapor caused the nicotine concentration in

179 Results: After inhalation of a single puff of E-cig vapor, brain nicotine concentration rose q

180 like food body that, when bitten, releases a puff of pollen allowing transfer to stigmas by wind or t

181 ptors in terminals with a spatially confined puff of ryanodine caused a decline in terminal ER [Ca(2+

182 ympathetic nerve activity induced by a 50-mL puff of smoke directed to the external nares of the rabb

183 was scanned following inhalation of a single puff of smoke from a cigarette containing (11)C-nicotine

184 billions of nanoparticles contained in each puff of smoke.

185 ive 5 mug (2 puffs of 2.5 mug) or 2.5 mug (2 puffs of 1.25 mug) of once-daily tiotropium or placebo (

186 ium 5 mug (2 puffs of 2.5 mug) or 2.5 mug (2 puffs of 1.25 mug), or placebo (2 puffs), administered t

187 17 years were randomized to receive 5 mug (2 puffs of 2.5 mug) or 2.5 mug (2 puffs of 1.25 mug) of on

188 ed to receive once-daily tiotropium 5 mug (2 puffs of 2.5 mug) or 2.5 mug (2 puffs of 1.25 mug), or p

189 fs of beclomethasone or placebo for each two puffs of albuterol (180 mug) needed for symptom relief.

190 baseline to week 48 in mean daily number of puffs of albuterol, mean total asthma symptom score, and

191 Rescue beclomethasone treatment was two puffs of beclomethasone or placebo for each two puffs of

192 rats were trained to nose-poke for discrete puffs of CAN(THC), CAN(CBD), or vehicle (VEH) in daily 1

193 he rate of RBC depolarization in response to puffs of CPPG and consequently potentiated the transient

194 rs were recorded in response to 6 successive puffs of GABA diluted in the survival medium (SM), showi

195 ns, and they also responded more strongly to puffs of nicotine.

196 of puffs increased with their amplitude, and puffs of similar size were of similar width, independent

197 The intervention was self-administered 2 puffs of sublingual nitroglycerin (800 mug; intervention

198 creased postsynaptic responsiveness to local puffs of the GABAA receptor agonist isoguvacine.

199 c agonist isoproterenol, we observed bright "puffs" of locally increased surface fluorescence intensi

200 Likewise, repetitive iontophoresis (or puffs) of glutamate (or AMPA) onto the dendrites of amac

201 rats learned the location of an aversive air puff on a linear track, as well as during sleep before a

202 pe II fibers was constructed by successively puffing onto OHCs along the cochlear spiral, up to 180 m

203 Focal DHPG puffs onto granule cells or bath application after glome

204 ing GABA(C) receptors to two rapid glutamate puffs onto the bipolar cell terminal.

205 y calcium-induced calcium release to evoke a puff, optimal correspondence with experimental data of p

206 risk (1 in 4 probability of receiving an air-puff) option (HGRAP) or a low-gain (small food quantity)

207 in mean arterial pressure in response to air puff or noise startle.

208 t was one puff of beclomethasone (40 mug per puff) or placebo given in the morning and evening.

209 tive Ca(2+) events (Ca(2+) sparks and Ca(2+) puffs) originating from clusters of Ca(2+) release chann

210 in mL) (P < 0.05) and total daily number of puffs (P < 0.05), but not other topographical measures,

211 s such as puff time, inter-puff interval and puffs per cigarette.

212 respiratory tract infection and less than 2 puffs per day of short-acting beta-agonist; they also ex

213 average consumption of 517 +/- 465 estimated puffs per month, indicating mild to moderate cannabis de

214 Similar results were found for the number of puffs per vaping episode for low (adjusted RR, 2.05; 95%

215 er of vaping episodes per day, and number of puffs per vaping episode) at the 6-month follow-up.

216 y an appearance of large vesicles around the puffing pipette.

217 y responses to aversive stimuli, such as air puff-predictive cues or air puff, remained unimpaired.

218 pattern, observed in raw rice was altered by puffing process and led to the formation of B- and V-typ

219 the antioxidant properties was observed upon puffing process as compared to raw rice samples.

220 ence the present study demonstrates that the puffing process leads to the significant changes in the

221 this study was to investigate the effect of puffing process on the physical, antioxidant properties

222 sity of those peaks was decreased during the puffing process.

223 tivated by spontaneous Ca(2+) events (Ca(2+) puffs/pulsars).

224 We found that motivation to smoke (puff rate) predicted magnitude of DA release in limbic s

225 In contrast, GL DHPG puffs readily increased mIPSCs.

226 Using puffing regimens modelled on puffing topography data fro

227 Differences in collection methods and puffing regimes likely contribute to the variability in

228 uli, such as air puff-predictive cues or air puff, remained unimpaired.

229 in micrograms per kilogram and nanograms per puff, respectively, for easy comparison.

230 Cortical postsynaptic air-puff responses in the trident representation show much l

231 ulated a model to explain the control of the puffing sequence on Drosophila polytene chromosomes init

232 This gives rise to the observed asymmetric puff shape.

233 om single IP3R (blips) and clusters of IP3R (puffs) showed little temperature dependence, whereas the

234 consequence is that the signaling power of a puff site (average amount of Ca(2+) released per puff x

235 The locations of puff sites observed by Ca(2+) imaging remain static over

236 ion coefficient<0.003 microm2 s(-1)), as are puff sites over prolonged periods, suggesting that the a

237 lusters differ appreciably between different puff sites, and we investigated how the probability of p

238 for the IP3R clusters underlying functional puff sites.

239 ated channels throughout the duration of the puff/spark event.

240 luminal depletion in the dynamics of Ca(2+) puff/spark termination in release sites composed of Ca(2

241 In the second case, puff/spark termination is promoted by the local depletio

242 nimal Ca(2+) release site models may exhibit puff/spark-like dynamics in either of two distinct param

243 In one case, puffs/spark termination is due to the process of stochas

244 sity significantly varied (P<0.05) among the puffing stages and was lowest in expanded rice.

245 agnetoencephalographic (MEG) elicited by air puff stimulation of right index finger and recorded usin

246 Tactile experiments using full-body air-puff stimulation suits revealed somatotopic areas of the

247 human cerebellar MEG signals elicited by air-puff stimulation to the eye.

248 rigeminal reflex blinks were evoked with air puff stimuli directed at the cornea in darkness and at t

249 As a proof of principle, we use an air-puff stimulus to the eyeball in order to elicit cerebell

250 the effect of PSEs (minus PNEs) on number of puffs taken from a cigarette (r=0.6, p=0.001).

251 ting the run direction that involved the air puff than for the 'safe' direction.

252 ing more strongly after punished CSs and air puffs than after rewarded CSs and rewards.

253 familiar equilibrium (or longtime transient) puffs that are spatially localized and keep their size i

254 es a quantal dissection of the local calcium puffs that constitute building blocks of cellular Ca(2+)

255 large amount of gas is produced in situ to "puff" the droplet during heating, followed by decomposit

256 or and smoking topography parameters such as puff time, inter-puff interval and puffs per cigarette.

257 C); control with noise of air puff (CN); air puff to conspecific hen (APC); air puff to observer hen

258 (CN); air puff to conspecific hen (APC); air puff to observer hen (APH).

259 trigeminal blink reflex by delivering an air puff to one eye as saccades were evoked by sub-optimal s

260 ent of this controversy by delivering an air puff to one eye to invoke the trigeminal blink reflex as

261 r visual stimuli paired with an aversive air-puff to the eye in a trace-conditioning paradigm.

262 ally salient unconditioned stimulus (US; air puff to the eye).

263 93129's inhibitory effect was strongest when puffed to STN-->SNr axon terminals in SNr, indicating a

264 ignificantly due to extrusion (to 25.7%) and puffing (to 31.6%), compared to the content in the raw m

265 ds using a cold trap or impinger and various puffing topographies.

266 purpose was to examine the effect of model, puffing topography (voltage, air-flow, puff interval), a

267 show that product characteristics influence puffing topography and, therefore, the results obtained

268 Using puffing regimens modelled on puffing topography data from 19 experienced e-cigarette

269 We adapted a cigarette puffing topography device for use with e-cigarettes.

270 There were no significant differences across puffing topography measurements observed between smokers

271 We used puffing topography to measure directly product use.

272 Consumer puffing topography was measured for 60 vapers provided w

273 Understanding the factors that affect puffing topography will be important for standardising t

274 This study was undertaken to determine the puffing topography, mouth level exposure (MLE) and avera

275 nts spanning multiple pixels (sparks, waves, puffs, transients, etc.), from which spatiotemporal even

276 ce, we found that while an abrupt facial air puff triggered transient increases in noradrenaline rele

277 al "trigger" channel, and the probability of puff triggering thus increases steeply with increasing n

278 ays/40 IU twice daily) or placebo (8 days/10 puffs twice daily), initiated within 12 days posttrauma.

279 ne inhaled corticosteroids (QVAR 80 mug, two puffs twice per day, equivalent to 800 mug per day beclo

280 l-organic framework (EMOF) nanoparticles are puffed up to submillimeter-scaled ADMS-decorated carbon

281 e average frequency of subsequent repetitive puffs, vary about linearly with cluster size.

282 two times more likely to work for cigarette puffs versus money in a progressive ratio, choice task (

283 l IP(3)R channel within a cluster triggers a puff via Ca(2+)-induced Ca(2+) release.

284 Total daily puff volume (TDPV; in mL) (P < 0.05) and total daily num

285 g the expired carbon monoxide level or total puff volume, suggesting minimal compensation.

286 Amongst smokers, e-cigarette mean puff volumes (41.6 mL vs 41.3 mL) and mean puff duration

287 lar with both types of e-cigarette, but mean puff volumes (52.2 mL and 83.0 mL) and mean inter-puff i

288 Under all regimes, air puff volumes were within 1 mL of the target and aerosol

289 esearch, users took, on average, larger mean puff volumes when using a THP compared to the reference

290 a 500-msec tone; in the trace paradigm, the puff was delivered after a 700-msec empty "trace" interv

291 In the delay paradigm, a mild puff was delivered to the eye at the end of a 500-msec t

292 bility to blink in response to a corneal air puff was monitored weekly for 9 weeks.

293 The average interval between puffs was considerably shorter for THP1.0(T) compared to

294 Formaldehyde intake from 100 daily puffs was higher than the amount inhaled by a smoker con

295 s at a site but the amplitudes of successive puffs were uncorrelated, even though we observed statist

296 eptor-mediated Ca(2+) release events (Ca(2+) puffs) were more frequent in HF atrial myoctes and were

297 Ca(2+) signals is punctuated by a flurry of puffs, which terminate before the peak by a mechanism in

298 eover, the appearance of a new population of puffs with longer latencies, prolonged durations, and at

299 nnected to a pump drawing air for two second puffs with sixty-second intervals between puffs.

300 site (average amount of Ca(2+) released per puff x puff frequency) varies about the square of cluste