1 sed of phospholipids and proteins that lines pulmonary alveoli.

2  of delicate gas-exchanging airspaces called pulmonary alveoli.

3 rge venules and epithelial cells (type-I) of pulmonary alveoli.

4               As the main immunocytes lining pulmonary alveoli, alveolar macrophages (AM) are critica

5  phospholipids (DPPC and POPG), would expand pulmonary alveoli and improve gas exchange in premature

6 urfactant activity as judged by expansion of pulmonary alveoli and improvement of gas exchange in inf

7 he esophageal mucosal-submucosal separation, pulmonary alveoli and intestinal villi.

8 ons mainly develop in serum-free spaces (eg, pulmonary alveoli) and open options for new therapeutic

9 d lipoproteins accumulate excessively within pulmonary alveoli, causing severe respiratory distress.

10 f Vgamma2Vdelta2(+) T cells in the blood and pulmonary alveoli following BCG infection and reinfectio

11 tudies, it has not been fully understood how pulmonary alveoli, the elementary gas exchange units in