ライフサイエンスコーパス: pulmonary artery

1 ing along the posterior surface of the right pulmonary artery.

2 quency delivery at the root of the aorta and pulmonary artery.

3 and sex, and the relative area change of the pulmonary artery.

4 lly between the subclavian artery and either pulmonary artery.

5 and sub-mesothelial cells at the base of the pulmonary artery.

6 present in the endothelium of mesenteric and pulmonary arteries.

7 ion that leads to high blood pressure in the pulmonary arteries.

8 P-selectin in the medial layer of the small pulmonary arteries.

9 ng in mesenteric arteries and its absence in pulmonary arteries.

10 partial excretion of inert gas across small pulmonary arteries.

11 cterized by the remodelling of pre-capillary pulmonary arteries.

12 s an obstructive disease of the precapillary pulmonary arteries.

13 These EC clones engrafted in the pulmonary arteries.

14 facement of HA from the vessel wall of small pulmonary arteries.

15 nary embolism and vasorelaxation in isolated pulmonary arteries.

16 nels in mesenteric arteries and with eNOS in pulmonary arteries.

17 esting diameter of resistance mesenteric and pulmonary arteries.

18 n alpha favour TRPV4(EC) -eNOS signalling in pulmonary arteries.

19 significantly (P < .001) higher than in the pulmonary arteries (0.15 L/min +/- 0.10) and descending

20 the proximal pulmonary artery (P), a distal pulmonary artery 1 cm proximal to the wedge position (us

21 ses in the proximal pulmonary artery, distal pulmonary artery (1 cm proximal to the wedge position) a

22 7, 30%), and abnormal branching of the right pulmonary artery (ABRPA) (14/47, 30%) were most commonly

23 Pulsed-wave Doppler determination of the pulmonary artery acceleration time (PAAT) as a surrogate

24 raphic parameters revealed shortening of the pulmonary artery acceleration time associated with eleva

25 ect link between a structural abnormality of pulmonary arteries and a response to targeted treatment

26 sented predisposition to vasoconstriction of pulmonary arteries and a severe loss of sildenafil-induc

27 onclassical monocytes are recruited to small pulmonary arteries and differentiate into pulmonary inte

28 ated with increased muscularization of small pulmonary arteries and disease severity.

29 owing to isolated narrowing of right or left pulmonary arteries and is also associated with various o

30 Histologically, lungs showed ectatic pulmonary arteries and pulmonary veins.

31 (PAH) is characterized by remodelling of the pulmonary arteries and right ventricle (RV), which leads

32 with abnormal muscularization of peripheral pulmonary arteries and right ventricular hypertrophy.

33 lmonary gas exchange occurs within the small pulmonary arteries and the extent is similar between oxy

34 within small (1.7 mm in diameter or larger) pulmonary arteries and this was quantitatively similar f

35 After electro anatomic rendering of the pulmonary artery and branches, either a circular or bask

36 fic medial and adventitial thickening of the pulmonary artery and is commonly reproduced in animal mo

37 delity flow (pulmonary artery) and pressure (pulmonary artery and left atrium) in 18 healthy minipigs

38 This study tested the hypothesis that pulmonary artery and pulmonary artery wedge pressures ar

39 tetralogy of Fallot with poor anatomy (small pulmonary arteries) and adverse factors (multiple comorb

40 We measured simultaneous high-fidelity flow (pulmonary artery) and pressure (pulmonary artery and lef

41 in the lung sense hypoxia, infiltrate small pulmonary arteries, and promote vascular remodeling and

42 Pressure measurements from the right atrium, pulmonary artery, and pulmonary capillary wedge pressure

43 ding aorta (aAo), mid-descending aorta, main pulmonary artery, and superior vena cava.

44 uctus arteriosus, ventricular septal defect, pulmonary artery anomalies, pulmonary valve stenosis, hy

45 Considering the dilation of the pulmonary arteries as a paramount sign of PAH, we hypoth

46 eter z score, the right/left ventricular and pulmonary artery/ascending aorta diameter ratios were hi

47 89] versus 0.77 [95% CI, 0.75-0.8]; P=0.008] pulmonary arteries at the time of subsequent surgical re

48 atrial switch operation) or 2-stage repairs (pulmonary artery band followed by arterial switch operat

49 ly diminished RV fibrosis progression in the pulmonary artery banding model, without improving RV fun

50 ent also supported the pressure-loaded RV in pulmonary artery banding rats.Conclusions: RVX208, a cli

51 nd rats with RV pressure overload induced by pulmonary artery banding were treated with RVX208 in thr

52 heart failure was induced in athymic rats by pulmonary artery banding, and cells were implanted into

53 ents with pulmonary hypertension, the murine pulmonary artery banding, and rat monocrotaline and Suge

54 ascending (Ao-A) and descending aorta at the pulmonary artery bifurcation (Ao-P) and diaphragm (Ao-D)

55 measured the PA:A ratio at the level of the pulmonary artery bifurcation on CT scans.

56 of the aortic arch in mice as well as human pulmonary artery branches.

57 ies have observed an increase in the rate of pulmonary artery catheter (PAC) use in heart failure adm

58 effectively by hemodynamic monitoring with a pulmonary artery catheter (PAC).

59 ardiac output was measured simultaneously by pulmonary artery catheter and aortic transpulmonary ther

60 atheter for blood pressure measurement and a pulmonary artery catheter for bolus thermodilution.

61 e agreement between echocardiography and the pulmonary artery catheter was moderate (Cohen's Kappa, 0

62 by transducing a peripheral intravenous and pulmonary artery catheter, respectively, after zeroing a

63 ve alternative access route commonly used in pulmonary artery catheterization.

64 tients were enrolled in Economic Analysis of Pulmonary Artery Catheters in whom 328 had functional me

65 condary analysis of the Economic Analysis of Pulmonary Artery Catheters study, a long-term observatio

66 ial and pulmonary artery pressures, improved pulmonary artery compliance, and enhanced left ventricul

67 n bath studies revealed severe alteration of pulmonary artery contraction and relaxation associated w

68 cardiac output reserve and right ventricular pulmonary artery coupling, reduced right atrial and pulm

69 an pulmonary artery SMC and smLRP1(-/-) main pulmonary artery (CTGF and NOX4) and reversed PAH in smL

70 Pulmonary artery denervation (PADN) procedure has not be

71 eviewed for recording variables such as main pulmonary artery diameter and right ventricle (RV)/left

72 Conversely, the mean pulmonary artery diameter z score, the right/left ventri

73 modynamic monitor-derived baseline estimated pulmonary artery diastolic pressure (ePAD) and change fr

74 underwent a 6 min recording of beat-by-beat pulmonary artery diastolic pressure (PAD), stroke volume

75 In the lung, early pulmonary artery dilation observed in untreated MFS mice

76 ne, by measuring these gases in the proximal pulmonary artery, distal pulmonary artery (1 cm proximal

77 tive study analyzed all CT angiograms of the pulmonary arteries done in patients with suspected pulmo

78 ependent stimulation of baroreceptors in the pulmonary artery elicits reflex sympathoexcitation.

79 syltransferase (NAMPT) upregulation in human pulmonary artery endothelial cells (hPAECs) is associate

80 ID1, BMPR2, HEY1 and HEY2 gene expression in pulmonary artery endothelial cells (hPAECs), and this ac

81 cally disrupted or when BMPR2 was reduced in pulmonary artery endothelial cells (PAECs) subjected to

82 eas current data support the notion that, in pulmonary artery endothelial cells (PAECs), expression o

83 Human pulmonary artery endothelial cells cultured with serum f

84 ated the metabolic abnormalities observed in pulmonary artery endothelial cells from patients with id

85 lysis of CLIC4-interacting proteins in human pulmonary artery endothelial cells identified regulators

86 is study show that an 18 h exposure of human pulmonary artery endothelial cells to the different nano

87 s-of-function assays were performed in human pulmonary artery endothelial cells using siRNA and lenti

88 In cultured hypoxic pulmonary artery endothelial cells, lung from human pati

89 y abnormal growth and enhanced glycolysis of pulmonary artery endothelial cells.

90 t, impaired vasoreactivity through increased pulmonary artery endothelial dysfunction and remodeling,

91 uction index (CTOI) and iii) PBV relative to pulmonary artery enhancement (PBV/PAenh); PBV/PAenh was

92 ure and flow sensors were placed at the main pulmonary artery for measuring pulmonary artery resistan

93 atresia growth were similar, although right pulmonary artery growth was better with DAS (change in z

94 g pharmacomechanical means to recanalize the pulmonary arteries have recently been cleared by the US

95 g right ventricle dysfunction or failure and pulmonary artery hypertension by using pressure or volum

96 Pulmonary artery hypertension decreased 8[13, 4] mmHg, a

97 transcription factor FRA2 was restricted to pulmonary artery hypertension.

98 mprehensively map the stepwise remodeling of pulmonary arteries in a robust, chronic inflammatory mou

99 essure and flow velocity measurements in the pulmonary artery in control subjects and patients with p

100 nsporter (5-HTT) play important roles in the pulmonary artery in pulmonary hypertension.

101 excess pressure analyses were applied in the pulmonary artery in subjects with and without pulmonary

102 , distension of baroreceptors located in the pulmonary artery induces a reflex increase in sympatheti

103 icular afterload can be analysed in terms of pulmonary artery input impedance, which we were able to

104 ures performed were fenestration closure and pulmonary artery intervention.

105 mechanical interaction with the dilated main pulmonary artery (MPA).

106 asured in the ascending aorta (AAo) and main pulmonary artery (MPA).

107 Ms) (n = 5/24), and proximal interruption of pulmonary artery (n = 2/24).

108 zed by profound vascular remodeling in which pulmonary arteries narrow because of medial thickening a

109 all-vessel arteriopathy in addition to major pulmonary artery obstruction has been suggested to play

110 racterized by defective thrombus resolution, pulmonary artery obstruction, and vasculopathy.

111 lar Imaging guidelines for assessment of the pulmonary artery occlusion pressure (a frequent surrogat

112 diastolic dysfunction), only 17 (71%) had a pulmonary artery occlusion pressure greater than or equa

113 Imaging guidelines do not accurately assess pulmonary artery occlusion pressure in ventilated critic

114 rade I diastolic dysfunction) had a measured pulmonary artery occlusion pressure less than 18 mm Hg.

115 iovascular Imaging guidelines, the predicted pulmonary artery occlusion pressure was indeterminate in

116 est echocardiographic predictors of a normal pulmonary artery occlusion pressure were a lateral e'-wa

117 r Imaging guidelines for predicting elevated pulmonary artery occlusion pressure were both 74%.

118 Using the pulmonary artery occlusion technique, we sought to asses

119 Pulmonary artery occlusion waveform analysis with estima

120 Based on pulmonary artery occlusion waveforms yielding an estimat

121 The remodelled pulmonary arteries of PAH patients harboured CD117(+) EC

122 ls, smooth muscle cells isolated from distal pulmonary arteries of patients with PAH, rats with Sugen

123 lusion can lead to increased pressure in the pulmonary arteries, often resulting in right ventricular

124 ources, which may be either from systemic or pulmonary arteries or cardio-pulmonary fistulas.

125 Silencing SIRT7 in pulmonary artery or microvascular endothelial cells atte

126 iratory and inert gas levels in the proximal pulmonary artery (P), a distal pulmonary artery 1 cm pro

127 tients in the validation cohort had enlarged pulmonary arteries (PA:A>1).

128 othesized that IASD would improve indexes of pulmonary artery (PA) function at rest and during exerci

129 of LMCA extrinsic compression from a dilated pulmonary artery (PA) in patients with PAH and angina or

130 Elevated pulmonary artery (PA) pressures in patients with heart f

131 Remote monitoring of pulmonary artery (PA) pressures provides clinicians with

132 y hemodynamic monitoring with an implantable pulmonary artery (PA) sensor is approved for patients wi

133 Although exposed to stressful conditions, pulmonary artery (PA) smooth muscle cells (PASMCs) exhib

134 Purpose To investigate whether the pulmonary artery (PA)-to-ascending aorta (Ao) ratio is a

135 e-vessel anastomosis (superior vena cava and pulmonary artery [PA] or bidirectional Glenn operation e

136 re, there was a significant reduction in (1) Pulmonary artery(PA) flow-velocity and pulmonary ejectio

137 es and adventitial fibroblasts isolated from pulmonary arteries (PAAF) of idiopathic pulmonary arteri

138 In both series, enhancement in the main pulmonary artery (PAenh), the descending aorta (DAenh),

139 PH due to LHD display vascular remodeling of pulmonary arteries (PAs) associated with poor prognosis.

140 nnels were abundantly expressed in the large pulmonary arteries (PAs) of healthy lung tissues from hu

141 sistance-sized mesenteric arteries (MAs) and pulmonary arteries (PAs) were used as prototypes for art

142 e Contegra conduit in the right ventricle to pulmonary artery position.

143 POPH patients had mean pulmonary artery pressure >25 mm Hg, pulmonary vascular

144 mm Hg predicted pulmonary hypertension (mean pulmonary artery pressure >= 25 mm Hg) with 100% sensiti

145 RHC confirmed PH (mean pulmonary artery pressure >= 25 mmHg) in 46 of 64 (71.9%

146 py at least 6 months after PEA, who had mean pulmonary artery pressure >=25 mm Hg or pulmonary vascul

147 vs 0.22 +/- 0.03, p < 0.001) and lower mean pulmonary artery pressure (26 +/- 3 vs 34 +/- 7 mm Hg; p

148 Median values for mean pulmonary artery pressure (49 mmHg) and pulmonary vascul

149 , including the following mean 24H measures: pulmonary artery pressure (6.8 vs 9.0 mm Hg), reservoir

150 rdiovascular metrics were correlated to mean pulmonary artery pressure (mPAP) and pulmonary vascular

151 Pulmonary hypertension (PH) exists when mean pulmonary artery pressure (mPAP) is 25 mm Hg or greater.

152 Recent advances have clarified the mean pulmonary artery pressure (mPAP) range that is above nor

153 At baseline, mean pulmonary artery pressure (mPAP) was 44 +/- 10 mm Hg (ra

154 was responsible for a 20% reduction of pulse pulmonary artery pressure (P < 0.001 vs. a theoretical p

155 lowed by the invasive measurements of a mean pulmonary artery pressure (PAP) >/=25 mm Hg and mean wed

156 best correlation with the invasive systolic pulmonary artery pressure (r = 0.87) with a small bias (

157 women, ejection fraction 36+/-10%, systolic pulmonary artery pressure 41+/-14 mm Hg with 20% atrial

158 vivo antagonized hypoxia-induced increase in pulmonary artery pressure and distal arteriole musculari

159 Diastolic pulmonary artery pressure and mean PAWP were measured to

160 enous pressure accurately estimates systolic pulmonary artery pressure and mean pulmonary artery pres

161 es (LVADs) has decoupling of their diastolic pulmonary artery pressure and pulmonary capillary wedge

162 d as a >5 mm Hg difference between diastolic pulmonary artery pressure and pulmonary capillary wedge

163 racy of Doppler echocardiography to evaluate pulmonary artery pressure and to predict pulmonary hyper

164 Systolic pulmonary artery pressure assessed from the tricuspid re

165 n left atrial enlargement and lower systolic pulmonary artery pressure compared with left-sided heart

166 Systolic pulmonary artery pressure decreased from 40.5 +/- 1.47 m

167 fraction<50%, with FTR grading and systolic pulmonary artery pressure estimation by Doppler echocard

168 A Doppler echocardiography systolic pulmonary artery pressure greater than 39 mm Hg predicte

169 and Notch inhibition significantly improves pulmonary artery pressure in animals with pulmonary hype

170 systolic pulmonary artery pressure and mean pulmonary artery pressure in ICU patients receiving mech

171 s indicated development of mild elevation of pulmonary artery pressure in the early PAH group (27.00

172 After pulmonary artery pressure information became available t

173 Interdependence was enhanced as pulmonary artery pressure load increased (P for interact

174 on showed severe precapillary PH with a mean pulmonary artery pressure of 45 (10) mm Hg and a pulmona

175 oman with acute respiratory failure and mean pulmonary artery pressure of 65 mm Hg.

176 Patients were excluded if they had systolic pulmonary artery pressure of more than 60 mm Hg, a previ

177 ary regurgitation velocities and either mean pulmonary artery pressure or diastolic pulmonary artery

178 Pulmonary artery pressure reduction and NO formation as

179 A wireless pulmonary artery pressure sensor (CardioMEMS) is approve

180 ts managed with guidance from an implantable pulmonary artery pressure sensor compared with usual car

181 edicare claims data from patients undergoing pulmonary artery pressure sensor implantation between Ju

182 Stratifying patients by ratio of systolic pulmonary artery pressure to stroke volume and right atr

183 were identified, of which ratio of systolic pulmonary artery pressure to stroke volume was the stron

184 Coupling ratio of systolic pulmonary artery pressure to stroke volume with right at

185 monary arterial elastance (ratio of systolic pulmonary artery pressure to stroke volume) before left

186 ery mean gradient was 11 mm Hg, and systolic pulmonary artery pressure was 32 mm Hg.

187 acceleration time (PAAT) as a surrogate for pulmonary artery pressure was found to be of clinical va

188 nular plane systolic excursion, and systolic pulmonary artery pressure were combined to determine 4 h

189 Pulmonary hypertension (mean pulmonary artery pressure, >/=25 mm Hg) was present in 8

190 or precapillary pulmonary hypertension (mean pulmonary artery pressure, >/=25 mm Hg; pulmonary vascul

191 g 29 (18%) regarded as moderate-severe (mean pulmonary artery pressure, >/=35 mm Hg) and 28 (34%) als

192 e associated with hemodynamic severity (mean pulmonary artery pressure, 35.2 +/- 9.6 vs. 26.9 +/- 10.

193 ystolic pulmonary artery pressure, diastolic pulmonary artery pressure, and mean pulmonary artery pre

194 d invasive central venous pressure, systolic pulmonary artery pressure, diastolic pulmonary artery pr

195 While PH-HFpEF is defined by a high mean pulmonary artery pressure, high left ventricular end-dia

196 sociated with FTR included elevated systolic pulmonary artery pressure, older age, female sex, lower

197 otal trial proved the safety and efficacy of pulmonary artery pressure-guided heart failure managemen

198 Intracardiac and pulmonary artery pressure-guided management has become a

199 ate the DPD as per usual practice (diastolic pulmonary artery pressure-mean PAWP).

200 ex to calculate the QRS-gated DPD (diastolic pulmonary artery pressure-QRS-gated PAWP).

201 iastolic pulmonary artery pressure, and mean pulmonary artery pressure.

202 ng the relevance of this technique to assess pulmonary artery pressure.

203 mean pulmonary artery pressure or diastolic pulmonary artery pressure.

204 .2 SD) using age, EuroSCORE II, and systolic pulmonary artery pressure.

205 of LV function, fixed perfusion defects, and pulmonary artery pressure.

206 technique for the noninvasive evaluation of pulmonary artery pressure; however, little information i

207 nce, left ventricular filling pressures, and pulmonary artery pressures and improved compliance (p <

208 when left ventricular filling pressures and pulmonary artery pressures increase.

209 the treatment group, in which daily uploaded pulmonary artery pressures were used to guide medical th

210 ry artery coupling, reduced right atrial and pulmonary artery pressures, improved pulmonary artery co

211 l characteristics that independently predict pulmonary artery pressures, right ventricular-to-left ve

212 uded elevated right atrial pressure, reduced pulmonary artery pulse pressure, and reduced stroke volu

213 g segments and extensive lobar and segmental pulmonary artery reconstruction.

214 ries and a severe loss of sildenafil-induced pulmonary arteries relaxation.

215 monary hypertension (PH) is characterized by pulmonary artery remodeling that can subsequently culmin

216 nal advanced therapies, typically focused on pulmonary artery reperfusion.

217 d at the main pulmonary artery for measuring pulmonary artery resistance (Z0), effective arterial ela

218 rtery trunk allowed continuous assessment of pulmonary artery resistance, effective elastance, compli

219 is characterized by progressive narrowing of pulmonary arteries, resulting in right heart failure and

220 ed by progressive loss and remodeling of the pulmonary arteries, resulting in right heart failure and

221 voir-excess pressure analysis applied to the pulmonary artery revealed distinctive differences betwee

222 y capillary wedge pressure, 8.1+/-3.1 mm Hg; pulmonary artery saturations 63.6+/-6.8% at 6000 rpm).

223 m, end-systolic diameter was 6.74+/-0.88 cm, pulmonary artery saturations were 46.7+/-9.2%, and pulmo

224 Phase contrast sequences in the aorta and pulmonary artery showed systemic output of 20 mL and pul

225 between pre-Fontan and 6 years in the RV-to-pulmonary artery shunt group but was stable in the modif

226 the modified Blalock-Taussig shunt or RV-to-pulmonary-artery shunt.

227 with a significantly greater number of small pulmonary artery side branches <300 mum per cm vessel (3

228 f obliterative and plexiform-like lesions in pulmonary arteries, similar to PAH patients.

229 y CT pulmonary angiography metrics with main pulmonary artery size and right-to-left ventricular rati

230 However, other markers of morbidity and pulmonary artery size favored the PDA stent group, suppo

231 iation, demographic, clinical variables, and pulmonary artery size were similar.

232 lar anomalies, and surgical outcomes of left pulmonary artery sling (LPAS) using cardiovascular compu

233 -activated PPARgamma binds to Smad3 in human pulmonary artery SMC (coimmunoprecipitation), thereby bl

234 ed the canonical TGFbeta1-CTGF axis in human pulmonary artery SMC and smLRP1(-/-) main pulmonary arte

235 The normal pulmonary artery SMC population is heterogeneous, and we

236 avioral, and pathogenic heterogeneity within pulmonary artery SMCs.

237 tive vasculopathy characterized by excessive pulmonary artery smooth muscle cell (PASMC) proliferatio

238 Human pulmonary artery smooth muscle cells (HPASMCs) demonstra

239 Altered metabolism in pulmonary artery smooth muscle cells (PASMCs) and endoth

240 pha) is increased, the role of HIF-1alpha in pulmonary artery smooth muscle cells (PASMCs) remains co

241 (PAs) associated with marked accumulation of pulmonary artery smooth muscle cells (PASMCs) represents

242 found to contribute to the proliferation of pulmonary artery smooth muscle cells (PASMCs), and inhib

243 HPV is intrinsic to pulmonary artery smooth muscle cells (PASMCs).

244 ar remodeling and excessive proliferation of pulmonary artery smooth muscle cells (PASMCs).

245 and is a mitogen and migratory stimulus for pulmonary artery smooth muscle cells (PASMCs).

246 vage via the sheddases, ADAM10 and ADAM17 in pulmonary artery smooth muscle cells (PASMCs).

247 ression and activity are strongly reduced in pulmonary artery smooth muscle cells and endothelial cel

248 ysfunction and uncontrolled proliferation of pulmonary artery smooth muscle cells and fibroblasts.

249 d apoptosis-resistant proliferation of human pulmonary artery smooth muscle cells in an HMGB1/RAGE-de

250 lmonary hypertension (defined as an elevated pulmonary artery systolic pressure >40 mm Hg on echocard

251 lasma volume by 11% (P < 0.01) and increased pulmonary artery systolic pressure (19.6 +/- 4.3 vs. 26.

252 ed to controls, patients on PDE5i had higher pulmonary artery systolic pressure (53.4 mm Hg versus 49

253 e (from 23.4 +/- 4.9 to 10.5 +/- 3.1 mm Hg), pulmonary artery systolic pressure (from 60.6 +/- 14.2 t

254 per unit; 95% CI: 1.02 to 1.06; p < 0.001), pulmonary artery systolic pressure (HR: 1.51 per 10 mm H

255 IV/HCV coinfection is associated with higher pulmonary artery systolic pressure (PASP) and prevalent

256 Baseline pulmonary artery systolic pressure (PASP) estimated from

257 0 mg/g) and available echocardiogram-derived pulmonary artery systolic pressure (PASP) from the Jacks

258 significantly up to 1 mo (P < 0.001); median pulmonary artery systolic pressure (PAsP) was 45.9 mm Hg

259 Pulmonary artery systolic pressure (PASP) was determined

260 Echocardiography-derived pulmonary artery systolic pressure (PASP), pulmonary vas

261 5 reduced ejection fraction) with PH (HF-PH; pulmonary artery systolic pressure [PASP] >/=40 mm Hg) w

262 imum left atrial volume [LAV], and estimated pulmonary artery systolic pressure), lead to the presenc

263 clinical trials, despite similar E/e' ratio, pulmonary artery systolic pressure, and event rates.

264 g for age, gender, and baseline RV/LV ratio, pulmonary artery systolic pressure, and modified Miller

265 line and postprocedure RV/LV diameter ratio, pulmonary artery systolic pressure, and modified Miller

266 An increase in pulmonary artery systolic pressure, estimated noninvasiv

267 ted R(2) for absolute change in RV/LV ratio, pulmonary artery systolic pressure, modified Miller Scor

268 as improvements in central venous pressure, pulmonary artery systolic pressure, RV/left ventricular

269 This was accompanied by more pulmonary artery thromboses and adherent hMSCs found on

270 ts in the 30 mg group died during the study (pulmonary artery thrombosis and cardiorespiratory failur

271 he development of PTE in COVID-19 might be a pulmonary artery thrombosis because of severe lung infla

272 Unexpected evidence of pulmonary artery thrombus formation was found in 19% of

273 s the ratio of fully or partially obstructed pulmonary arteries to normal arteries.

274 ophysiology catheter was placed in the right pulmonary artery to allow electrical intravascular stimu

275 sence and pattern of emphysema, the ratio of pulmonary artery to ascending aortic diameter, quantitat

276 excess pressure analyses were applied in the pulmonary artery to characterize changes in wave propaga

277 ft ventricular eccentricity index (EI), main pulmonary artery-to-aorta (PA/AO) diameter ratio, and pu

278 involved in the regulation of heart rate(6), pulmonary artery tone(5,7), sleep/wake cycles(8) and res

279 Pressure and flow sensors placed at the pulmonary artery trunk allowed continuous assessment of

280 LT (0 [H0], 6, 12, 24, 48 and 72 hours after pulmonary artery unclamping).

281 e predominantly driven by tricuspid valve or pulmonary artery vasculature damage (Stage 3) and right

282 atrial damage), Stage 3 (tricuspid valve or pulmonary artery vasculature damage), or Stage 4 (right

283 lling pressures with exercise, and depressed pulmonary artery vasodilator reserve.

284 served in LV ejection fraction ( P=0.93) and pulmonary artery velocity ( P=0.07).

285 logy, increase in RV filling time (P=0.002), pulmonary artery velocity time integral (P=0.006), and R

286 ex, brachial flows (ipsilateral to AVF), and pulmonary artery velocity.

287 Isolated abnormality of both right and left pulmonary arteries was also noted.

288 Thrombosis of small and mid-sized pulmonary arteries was found in various degrees in all 1

289 sed; a conventional approach, where only the pulmonary artery was perfused; or a conventional approac

290 ced by temporary, unilateral clamping of the pulmonary artery, was tested before and after induction

291 eous respiration and dynamic stress tests on pulmonary artery wave propagation and reservoir function

292 ascular resistance >240 dyn-sec/cm(-5) , and pulmonary artery wedge pressure <=15 mm Hg without anoth

293 ated, potentially related to the use of mean pulmonary artery wedge pressure (PAWP).

294 The pulmonary artery wedge pressure was </=15 mm Hg in 54%,

295 Hg; pulmonary vascular resistance, >3.0 WU; pulmonary artery wedge pressure, </=15 mm Hg).

296 ted the hypothesis that pulmonary artery and pulmonary artery wedge pressures are higher in SIPE-susc

297 Isolated pulmonary arteries were evaluated ex vivo in a myograph.

298 r the BACS approach, where the bronchial and pulmonary arteries were synchronously perfused; a conven

299 l, cardiac MRI mPAP model 2 (right ventricle pulmonary artery), which excludes the black blood flow s

300 K channels in mesenteric arteries but not in pulmonary arteries, which may explain TRPV4(EC) -IK/SK c