ライフサイエンスコーパス: pulmonary blood flow

1 e to cyclic interruption and exaggeration of pulmonary blood flow.

2 ty, particularly for children with increased pulmonary blood flow.

3 striction was assessed as the change in left pulmonary blood flow.

4 by CPB is augmented in lambs with increased pulmonary blood flow.

5 shunt or a DS in infants with duct-dependent pulmonary blood flow.

6 a noninvasive, on-line monitor of changes in pulmonary blood flow.

7 SVHD) experience morbidity due to inadequate pulmonary blood flow.

8 nary arterial vasculature, thereby providing pulmonary blood flow.

9 utflow tract obstruction or ductal-dependent pulmonary blood flow.

10 ive option for infants with ductal-dependent pulmonary blood flow.

11 functional single ventricle and very limited pulmonary blood flow.

12 of rat were reduced in shunt rats with high pulmonary blood flow.

13 r remodeling causing increased resistance to pulmonary blood flow.

14 ulmonary hypertension secondary to increased pulmonary blood flow.

15 t (BTS), physiologically distinct sources of pulmonary blood flow.

16 pertension in this animal model of increased pulmonary blood flow.

17 ns, particularly for children with increased pulmonary blood flow.

18 after CPB in lambs with normal and increased pulmonary blood flow.

19 eled form as an imaging modality to evaluate pulmonary blood flow.

20 mics congenital heart disease with increased pulmonary blood flow.

21 lowering cerebral, superior vena caval, and pulmonary blood flows.

22 41 (25, 57); descending aorta, 55 (35, 75); pulmonary blood flow, 16 (0, 34); umbilical vein, 29 (11

23 At rest, TR subjects had lower pulmonary blood flow (3.6+/-0.4 versus 5.1+/-1.9 L/min;

24 sus 13.8+/-4.2 mL/min per kg; P=0.02), lower pulmonary blood flow (6.4+/-1.3 versus 10.3+/-3.3 L/min;

25 T shunts; P=0.001) and presence of antegrade pulmonary blood flow (61% of PDA stents versus 38% of BT

26 109, 265); descending aorta, 252 (160, 344); pulmonary blood flow, 77 (0, 160); umbilical vein, 134 (

27 In the absence of pulmonary blood flow, acid instillation led to a 50% dec

28 d BT shunt for infants with ductal-dependent pulmonary blood flow adjusted for differences in patient

29 palliation for infants with ductal-dependent pulmonary blood flow, adjusted for baseline differences,

30 tored in 14 1-month-old lambs with increased pulmonary blood flow (after in utero placement of an aor

31 Preexisting increased pulmonary blood flow alters the response of the pulmonar

32 ed in 1-month-old lambs (n=7) with increased pulmonary blood flow and 6 age-matched control lambs.

33 s compared with the measured distribution of pulmonary blood flow and evaluated for correlation, accu

34 ncreases the annulus Z score and anterograde pulmonary blood flow and facilities simultaneous coiling

35 ove oxygenation by two mechanisms: increased pulmonary blood flow and improved ventilation-perfusion

36 structions and improve cyanosis by enhancing pulmonary blood flow and oxygen saturation.

37 work using a large animal model of increased pulmonary blood flow and pressure, we have previously de

38 were consistent with comparable increases in pulmonary blood flow and therefore stroke volumes.

39 To determine cerebral and pulmonary blood flow and to establish the hierarchy of c

40 , NPV brought about a marked increase in the pulmonary blood flow and, hence, cardiac output of Fonta

41 Over time, increased pulmonary blood flow and/or pressure results in the emer

42 ukostasis and markedly improved oxygenation, pulmonary blood flow, and graft survival.

43 provements in pulmonary vascular resistance, pulmonary blood flow, and right ventricular contractilit

44 rcise in CHF, to examine its relationship to pulmonary blood flow, and to consider its functional sig

45 ntroversy exists regarding whether accessory pulmonary blood flow (APBF) should be left at the time o

46 ing hyperpnea, and therefore that changes in pulmonary blood flow are not associated with HIB.

47 causing dependence on the arterial duct for pulmonary blood flow are often palliated with a shunt us

48 se patients are influenced by restriction of pulmonary blood flow, arrhythmia, and pacemaker requirem

49 high continuous distending pressure impedes pulmonary blood flow as evidenced by reduced lung volume

50 stent implantation in the arterial duct for pulmonary blood flow augmentation.

51 op pulmonary hypertension or to redistribute pulmonary blood flow away from the edematous lung region

52 S for cardiac conditions with duct-dependent pulmonary blood flow between January 2012 and December 3

53 was determined by Doppler echocardiography, pulmonary blood flow by inert gas re-breathing, and vaso

54 Pulmonary blood flow can be assessed on ventilation-perf

55 nificantly augmented in lambs with increased pulmonary blood flow compared with control lambs (P < .0

56 Pulmonary blood flow continued to improve, with a total

57 In the animals in group 1, left pulmonary blood flow decreased by 62 +/- 8 (SEM)% during

58 After 24 hrs of sepsis, left pulmonary blood flow decreased from 56 +/- 10% to 26 +/-

59 In the Normal-PLV piglets, pulmonary blood flow decreased from baseline (before inj

60 In the OA-Control piglets, pulmonary blood flow decreased in the most dependent are

61 We conclude that reduction of pulmonary blood flow decreases eNOS mRNA and protein exp

62 stulated to document hyperventilation of the pulmonary blood flow due to a right-to-left EIS were (1)

63 Pulmonary blood flow during cardiac arrest and cardiopul

64 HPV was assessed as the decrease in left pulmonary blood flow during hypoxia, measured with an ul

65 would demonstrate increased heterogeneity of pulmonary blood flow during hypoxia.

66 y on CPET demonstrated higher LV reserve and pulmonary blood flow during incremental CMR-ET.

67 eatures included haemodynamic alterations of pulmonary blood flow ejection and wave reflection, mild

68 A brief period of NPV increased pulmonary blood flow from 2.4 to 3.5 L x min(-1) x /m(-2

69 HAPE-susceptible individuals have increased pulmonary blood flow heterogeneity in acute hypoxia, con

70 spin labeling) was used to quantify spatial pulmonary blood flow heterogeneity in three subject grou

71 Nonpulsatile pulmonary blood flow in Fontan circulation results in pu

72 Maldistribution of pulmonary blood flow in patients with congenital heart d

73 Changes in pulmonary blood flow in response to Ach were determined

74 required reoperation related to the BCPS or pulmonary blood flow in the early postoperative period:

75 tery pressure due to increased resistance to pulmonary blood flow in the setting of portal hypertensi

76 and a higher likelihood of absent upper lobe pulmonary blood flow in these patients.

77 one early death, and procedures to decrease pulmonary blood flow in three patients.

78 In a rat model of high pulmonary blood flow-induced pulmonary vascular collagen

79 In infants with ductal-dependent pulmonary blood flow, initial palliation with patent duc

80 VEC MRI has the ability to quantify pulmonary blood flow inside the lumen of nitinol stents.

81 ingle-ventricle anatomy and ductal-dependent pulmonary blood flow, interstage outcomes, hemodynamics

82 Increased pulmonary blood flow is believed to contribute to the de

83 The pulsatile nature of pulmonary blood flow is important for shear stress-media

84 The spatial distribution of regional pulmonary blood flow is preserved during partial liquid

85 on emission tomography to measure fractional pulmonary blood flow, lung water concentration (LWC), an

86 ingle ventricle anatomy and ductal-dependent pulmonary blood flow may be initially palliated with eit

87 Infants with ductal-dependent pulmonary blood flow may undergo palliation with either

88 e purpose of consistency and makes sense, as pulmonary blood flow measurements are not corrected for

89 blood only to the systemic circuit, whereas pulmonary blood flow occurs passively.

90 stigate the effects of preexisting increased pulmonary blood flow on these changes; and to better def

91 Infants with ductal-dependent pulmonary blood flow palliated with either a PDA stent o

92 ingle ventricle anatomy and ductal-dependent pulmonary blood flow palliated with either DAS or BTS fr

93 ctive study of infants with ductal-dependent pulmonary blood flow palliated with PDA stent (n=104) or

94 Infants with ductal-dependent pulmonary blood flow palliated with PDA stent between 20

95 In infants with ductal-dependent pulmonary blood flow palliated with PDA stent implantati

96 Redistribution of pulmonary blood flow (PBF) away from edematous regions o

97 trated that computed tomography (CT)-derived pulmonary blood flow (PBF) heterogeneity is greater in s

98 Pulmonary blood flow (PBF) is a critical determinant of

99 is determined regional perfusion parameters, pulmonary blood flow (PBF), and mean transit time (MTT).

100 Pulmonary blood flow (PBF), first-pass bolus kinetic par

101 ay be related to the pre-iNO distribution of pulmonary blood flow (PBF).

102 onary vascular resistance (PVR) and increase pulmonary blood flow (PBF); more gradual changes occur o

103 Hyperoxia did not change cerebral and pulmonary blood flow; Po2 increased 94% (P=0.01).

104 t intentionally left in place to augment the pulmonary blood flow provided by the BDG.

105 Carbon monoxide transfer factor (TLCO) and pulmonary blood flow (Q(C)) were measured by a rebreathe

106 The attendant increases in pulmonary blood flow (Qp) and oxygen content may alter p

107 Oximetry underestimated CMR-measured pulmonary blood flow (Qp) by an average of 1.1 L/min per

108 n pulmonary artery was ligated distally, and pulmonary blood flow (Qp) was provided through a 5-mm ao

109 RV and left ventricle (LV) volumes and pulmonary blood flow (Qp) were calculated.

110 e measured in vivo the distribution of total pulmonary blood flow (QPA) between the right (QRPA) and

111 dex accounted for the increased cerebral and pulmonary blood flow (R=0.73, P=0.02) and cerebral O2 tr

112 Changes in pulmonary blood flow rate can alter the size of the perf

113 artery shunt or Sano modification to provide pulmonary blood flow rather than the standard modified B

114 e-to-pulmonary artery shunt as the source of pulmonary blood flow, rather than the modified Blalock-T

115 ng injury was evaluated by the left-to-right pulmonary blood flow ratio, the weight gain of the isogr

116 +/-3.3 L/min; P=0.001), and less increase in pulmonary blood flow relative to VO2 (+4.6+/-1.1vs +6.2+

117 Patients had a two-ventricular repair (A) or pulmonary blood flow supplied by an aortopulmonary shunt

118 e at birth, allowing for a rapid increase in pulmonary blood flow that is essential for efficient gas

119 With duct-dependent pulmonary blood flow, the procedure carries high risk, a

120 usion ratio throughout the lung by directing pulmonary blood flow to better ventilated areas of the l

121 Pulmonary blood flow to lung units with a normal VA/Q ra

122 ich were also associated with an increase in pulmonary blood flow, transpulmonary efficiency, and rig

123 further modified by active redistribution of pulmonary blood flow under hypoxic and hyperoxic conditi

124 l of congenital heart disease with increased pulmonary blood flow using in vitro approaches.

125 reserve (PFR) was calculated as the ratio of pulmonary blood flow velocity in response to Ach relativ

126 ite were assessed by measuring the change in pulmonary blood flow velocity with a Doppler-tipped wire

127 unchanged during NPV, and the improvement in pulmonary blood flow was achieved by an increase in stro

128 Pulmonary blood flow was assessed by fluorescent microsp

129 tional experiments were carried out in which pulmonary blood flow was eliminated.

130 Accessory pulmonary blood flow was included in 18 patients.

131 stitution of IPPV, and in a second subgroup, pulmonary blood flow was measured after an extended peri

132 In one subgroup of patients, pulmonary blood flow was measured again after reinstitut

133 Accessible pulmonary blood flow was measured at each workload with

134 Pulmonary blood flow was measured using the direct Fick

135 relationship between foramen ovale shunt and pulmonary blood flow was noted (r=-0.64; P<0.0001).

136 In the OA-PLV piglets, pulmonary blood flow was preserved over time throughout

137 Regional pulmonary blood flow was studied in 21 piglets in the su

138 In 1-month-old lambs with increased pulmonary blood flow, we have demonstrated early alterat

139 Regional lung water and pulmonary blood flow were assessed by positron emission

140 reduces left atrial pressures but increases pulmonary blood flow, which may be poorly tolerated in p

141 PVR (PVRI) using Fick principle to calculate pulmonary blood flow, with respiratory mass spectrometry