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1 PAH was seen as a disease restricted to the pulmonary circulation.
2 or absent in some SMC tissues, including the pulmonary circulation.
3 ically like the native valve leaflets in the pulmonary circulation.
4 cetylcholine and sodium nitroprusside in the pulmonary circulation.
5 rtant mediator of vascular resistance in the pulmonary circulation.
6 analysis may provide novel insights into the pulmonary circulation.
7 t upon recruitment and distensibility of the pulmonary circulation.
8 ally relevant targets in the right heart and pulmonary circulation.
9 fusion of blood from the splanchnic into the pulmonary circulation.
10 nd function along with perfusion through the pulmonary circulation.
11 dure while preventing adverse effects on the pulmonary circulation.
12 fetus that directs blood flow away from the pulmonary circulation.
13 tunities to manipulate blood pressure in the pulmonary circulation.
14 and can lead to unpredictable effects on the pulmonary circulation.
15 the molecular diagnosis of disorders of the pulmonary circulation.
16 ent on reactions with deoxyhemoglobin in the pulmonary circulation.
17 right ventricle supporting both systemic and pulmonary circulation.
18 of the iron chelator desferrioxamine on the pulmonary circulation.
19 particles (2 x 10(8)) were infused into the pulmonary circulation.
20 eling and persistent vasoconstriction of the pulmonary circulation.
21 a-alveolar to alveolar vessels in the intact pulmonary circulation.
22 ors that escape degradation in bypassing the pulmonary circulation.
23 (P=0.03) as hemoglobin is oxygenated in the pulmonary circulation.
24 nce of normal hepatic venous drainage to the pulmonary circulation.
25 tion of sickled erythrocytes in the inflamed pulmonary circulation.
26 fetus that directs blood flow away from the pulmonary circulation.
27 5 mg/kg) produced selective dilation of the pulmonary circulation.
28 mouse by permanently obstructing 40% of the pulmonary circulation.
29 olerance by impairing blood flow through the pulmonary circulation.
30 rtension (PHTN) are unique properties of the pulmonary circulation.
31 /kg/min), the dilation was selective for the pulmonary circulation.
32 tes the membrane step in PG clearance by the pulmonary circulation.
33 and -independent dilators in the ovine fetal pulmonary circulation.
34 ound in the heart and adjoining coronary and pulmonary circulations.
35 unting of blood between the systemic and the pulmonary circulations.
36 ve vascular disease in both the systemic and pulmonary circulations.
37 an resistance arteries from the systemic and pulmonary circulations.
38 transitions to being solely responsible for pulmonary circulation after birth when the left ventricl
39 1 (31 microgram/min) selectively dilated the pulmonary circulation, an effect again blocked after inh
40 resulted from (129)Xe transport through the pulmonary circulation and diffusion across the blood-gas
42 ata uncovering the cellular diversity of the pulmonary circulation and mechanisms governing vascular
44 zing blood-O2 affinity for O2 loading in the pulmonary circulation and O2 unloading in the systemic c
46 e echocardiography is used for assessment of pulmonary circulation and right ventricular function, bu
47 hanisms underlying development of the normal pulmonary circulation and the essential relationship of
48 he afterload imposed by a combination of the pulmonary circulation and the retrograde contribution of
51 the fetus which directs blood away from the pulmonary circulation and towards the placenta where oxy
52 aternal hyperoxygenation affects human fetal pulmonary circulation and whether there is a gestational
53 ways and lung parenchyma are supplied by the pulmonary circulation, and changes in the pulmonary circ
54 ly sensitive to the afterload imposed by the pulmonary circulation, and the left heart (LH) retrograd
55 low changes on eNOS expression in the normal pulmonary circulation, and to determine whether the incr
56 lar function and, hopefully, its coupling to pulmonary circulation; and 4) searching for effective th
59 sess the osmotic effectiveness of Het in the pulmonary circulation as judged by its exclusion from lu
60 t to provide adequate blood flow through the pulmonary circulation at a normal central venous pressur
61 d in some part to lack of pulsatility in the pulmonary circulation because of altered flow characteri
62 dulating vascular tone and remodeling in the pulmonary circulation, but its role in the pathogenesis
63 n (PAH), a proliferative vasculopathy of the pulmonary circulation, but the origin of vascular injury
64 tructure and function of the right heart and pulmonary circulation can be challenging, due to the com
66 sults demonstrate that although the existing pulmonary circulation can supply the metabolic needs for
67 effects on lung hyperinflation (and possibly pulmonary circulation) can explain the effects on exacer
68 functional and structural alterations of the pulmonary circulation causing marked increase in pulmona
70 majority of which affect the right-sided or pulmonary circulation, contribute significantly to morta
71 he pulmonary circulation, and changes in the pulmonary circulation could alter airway resistance or t
72 In arterial sections from the systemic and pulmonary circulation, CREB content was high in prolifer
74 se (OR = 4.8), metastatic cancer (OR = 4.6), pulmonary circulation disorders (OR = 2.9), congestive h
75 al disorders, peripheral vascular disorders, pulmonary circulation disorders, renal failure, solid tu
76 The patients with nonpulsatile group in the pulmonary circulation dropped the PVRI from 2.18+/-0.34
77 genital heart diseases that have compromised pulmonary circulation due to severe stenosis involving p
78 on to allow shunting from either systemic-to-pulmonary circulation (eg, in the first day of life) or
80 lation [FMD] in the brachial artery) and the pulmonary circulation (exhaled nitric oxide [NO] product
81 ss to constrictor agents, at a time when the pulmonary circulation exhibits varying degrees of vasoco
82 assumed that the pulsatile components of the pulmonary circulation exist in predictable and constant
83 lar interdependence) and its coupling to the pulmonary circulation further modulate RV behavior in di
87 o increased leukocyte recruitment within the pulmonary circulation in a mouse acute endotoxemia model
88 contextualize the story of the discovery of pulmonary circulation in ancient Persian and Greek theor
89 ostic modalities for the right ventricle and pulmonary circulation in invasively ventilated patients
90 ecome the first-line modality for imaging of pulmonary circulation in patients suspected of having pu
91 entricular (RV) function and the coupling to pulmonary circulation in patients with heart failure and
93 on for the importance of the right heart and pulmonary circulation in several disease states across t
94 te-of-the-Art Review, we address the role of pulmonary circulation in those with advanced heart failu
99 Thus the hypoxia-induced remodeling of the pulmonary circulation is a highly complex process where
100 atients with chronic heart failure (HF), the pulmonary circulation is a major source of endothelin-1
101 pacted by living at altitude, as the passive pulmonary circulation is dependent on the resistance of
102 evidence suggesting that the recognition of pulmonary circulation is older than the time of Ibn Nafi
103 dvancing gestation; this suggests that fetal pulmonary circulation is under acquired vasoconstriction
104 y C fibre endings, primarily supplied by the pulmonary circulation, is transmitted to this commissura
106 or other processes affecting the developing pulmonary circulation may represent a return to an earli
107 ctively investigate the role of NOS 3 in the pulmonary circulation, mice with targeted disruption of
109 ution from venous splanchnic beds to central pulmonary circulation need to be taken into account in s
110 s an important role in the remodeling of the pulmonary circulation, notably during exposure to hypoxi
111 ich breast cancer cells are infused into the pulmonary circulation of artificially ventilated explant
112 These TE structures have functioned in the pulmonary circulation of growing lambs for up to 4 month
113 forces maintain eNOS content in the normoxic pulmonary circulation of the adult rat, and suggest that
119 flation results in structural changes in the pulmonary circulation, potentially affecting pulmonary p
120 death, hypertension, ischemic heart disease, pulmonary circulation problems, stroke, and type 2 diabe
121 t heart, including diseases of the lungs and pulmonary circulation, promote atrial fibrillation (AF),
122 ardiography with detailed examination of the pulmonary circulation, pulse oximetry, complete blood co
123 pc-PH is amenable to therapies targeting the pulmonary circulation remains to be tested by properly d
124 presystemic pump may limit flow through the pulmonary circulation, restricting ventricular filling a
125 al flow consistency analysis in systemic and pulmonary circulation showed average relative difference
127 by abnormally elevated blood pressure of the pulmonary circulation that results, over time, from exte
129 ortant modulator of tone in the hypertensive pulmonary circulation, the roles of cyclic 3'-5'-guanosi
130 ays and the cardiac septations necessary for pulmonary circulation.This article has an associated 'Th
132 set latency of the excitation was within the pulmonary circulation time, consistent with being activa
133 The typical response of the adult mammalian pulmonary circulation to a low oxygen environment is vas
135 l PVR and limits the capability of the fetal pulmonary circulation to dilate or sustain vasodilation
137 account for the unique vulnerability of the pulmonary circulation to heterozygous mutations of BMP t
138 monary blood flow alters the response of the pulmonary circulation to hypothermic CPB; the increase i
139 the important contribution of the developing pulmonary circulation to lung growth in the setting of p
140 estational age-related response in the fetal pulmonary circulation to maternal hyperoxygenation durin
142 d contrast agents can now safely transit the pulmonary circulation to provide opacification of the le
143 e redistribution of EC-SOD from the lung and pulmonary circulation to the extracellular fluids is ben
144 onal contrast-enhanced MR angiography of the pulmonary circulation was feasible at 3.0 T and provided
145 onal contrast-enhanced MR angiography of the pulmonary circulation was performed with a 3.0-T MR syst
146 On the basis of the unique properties of the pulmonary circulation, we show how all relevant physiolo
149 ve potassium channels in the fetal and adult pulmonary circulation which regulate vascular tone in re
150 ion, including hemodynamic parameters in the pulmonary circulation, which are superior in their ident
151 g modalities interrogate the right heart and pulmonary circulation with greater diagnostic precision.
152 of delivered cells exited the heart into the pulmonary circulation, with 26+/-3% (IM), 47+/-1% (IC),
153 promising agent for molecular imaging of the pulmonary circulation, with abundant specific binding si