ライフサイエンスコーパス: pulmonary stenosis

1 outlet right ventricle, overriding aorta and pulmonary stenosis.

2 is important in the management of peripheral pulmonary stenosis.

3 autograft was reoperated on for inflammatory pulmonary stenosis.

4 's anomaly and two with single ventricle and pulmonary stenosis.

5 those with diminutive pulmonary arteries and pulmonary stenosis.

6 rther stratified by the presence of residual pulmonary stenosis.

7 even in the presence of significant residual pulmonary stenosis.

8 primary transcatheter therapy for peripheral pulmonary stenosis.

9 Complications included supravalvar pulmonary stenosis (16 of 21 patients) and baffle leaks

10 ho underwent a VS procedure without residual pulmonary stenosis (2.0 interventions [95% CI, 1.5-2.7 i

11 ses were tetralogy of Fallot (71%), valvular pulmonary stenosis (21%), and other (8%).

12 Thirty-one patients had TOF with pulmonary stenosis, 24 had TOF with pulmonary atresia, a

13 duction abnormalities, Ocular hypertelorism, Pulmonary stenosis, Abnormal genitalia, Retardation of g

14 monary blood supply in patients with complex pulmonary stenosis and atresia and can be considered a n

15 Echocardiography has documented acquired pulmonary stenosis and cardiomyopathy in recipient fetus

16 In patients with tetralogy of Fallot, severe pulmonary stenosis and diminutive pulmonary arteries, in

17 abnormal cartilage calcification, peripheral pulmonary stenosis and midfacial hypoplasia.

18 One affected recipient also had pulmonary stenosis and tricuspid valve dysplasia.

19 We developed an in vitro model of mild pulmonary stenosis and wide-open PR that mimics the patt

20 e accepted therapies for isolated peripheral pulmonary stenosis, and have been shown to increase vess

21 ith cyclopamine exhibited pulmonary atresia, pulmonary stenosis, and persistent truncus arteriosus.

22 ar septal defects; patent ductus arteriosus; pulmonary stenosis; aortic stenosis; coarctation of the

23 intervention for severe isolated peripheral pulmonary stenosis at </= 5 years of age.

24 Supravalvar pulmonary stenosis developed in 1 of 11 patients after d

25 leads to myeloid progenitor hyperplasia and pulmonary stenosis due to the leaflet thickening, respec

26 (n=42), primary repair (aged <3 months) with pulmonary stenosis (early-PS group; n=44), primary repai

27 l right ventricular strain increased for the pulmonary stenosis group only (P=0.05).

28 r therapy for infants with severe peripheral pulmonary stenosis has become safer, regardless of genet

29 in 4, innocent murmur in 3, major VSD in 2, pulmonary stenosis in 2 and subaortic membrane, atrial s

30 spectively, and more than mild neoaortic and pulmonary stenosis in 3.2% and 10.3%.

31 t ventricular outflow tract or supravalvular pulmonary stenosis, including optimal use of multimodali

32 Defects include pulmonary stenosis, interrupted aortic artery, retroesop

33 ith diminutive pulmonary arteries and severe pulmonary stenosis is rare and resembles tetralogy of Fa

34 f PR by increased airway pressure and branch pulmonary stenosis may be relevant to the acute postoper

35 ho underwent a VS procedure without residual pulmonary stenosis (MR, 0.22 [95% CI, 0.12-0.43]; P < .0

36 maging (n=111) or inferred from a peripheral pulmonary stenosis murmur (n=41) in 76% of subjects.

37 ubjects (75%), and 37 (19%) had a peripheral pulmonary stenosis murmur with either a normal echocardi

38 Associated diagnoses included pulmonary stenosis (n = 433), pulmonary atresia (n = 121

39 ulmonary atresia (n = 72), congenital branch pulmonary stenosis (n = 9), status post-Fontan operation

40 ents with tetralogy of Fallot (n=39), valvar pulmonary stenosis (n=10), or pulmonary atresia with int

41 TOF) with pulmonary atresia (n=13), TOF with pulmonary stenosis (n=4), post-Fontan palliation (n=5),

42 eat arteries, ventricular septal defect with pulmonary stenosis (n=8).

43 cuspid valve, ventricular septal defect, and pulmonary stenosis, occur in the majority of patients.

44 Thirty-two patients with pulmonary stenosis or atresia (median age: 4.7 years, ra

45 In patients with complex pulmonary stenosis or atresia, a detailed delineation of

46 h subgroups (pulmonary regurgitation P=0.01; pulmonary stenosis P=0.02).

47 Experience of balloon dilation of peripheral pulmonary stenosis (PPS) in Williams syndrome (WS) is li

48 uctures in patients with critical and severe pulmonary stenosis (PS) after balloon dilation, and to d

49 ion (PR) following balloon dilation (BD) for pulmonary stenosis (PS) and to investigate its impact on

50 e in tetralogy of Fallot (TOF) patients with pulmonary stenosis (PS) or pulmonary atresia (PA).

51 with corrected tetralogy of Fallot (TOF) or pulmonary stenosis (PS) referred for pulmonary valve rep

52 ll defects or larger defects associated with pulmonary stenosis, pulmonary hypertension, or aortic re

53 Supravalvar pulmonary stenosis (SVPS) is frequently observed after a

54 t syndrome, patent ductus arteriosus, valvar pulmonary stenosis, tetralogy of Fallot, transposition o

55 t congenital heart defects ranging from mild pulmonary stenosis to severe pulmonary atresia.

56 retrospective review of 99 children with TOF pulmonary stenosis (TOF/PS) or TOF pulmonary atresia (TO

57 Valvar pulmonary stenosis (vPS) accounts for 8% to 12% of conge

58 An initial diagnosis of critical pulmonary stenosis was made in 6 cases, at a mean of 22.

59 e with coarctation of the aorta, or valvular pulmonary stenosis), which have not been previously asso

60 Of the 18 patients with TOF and pulmonary stenosis who had abnormal coronary arteries du

61 ded in the majority of patients with TOF and pulmonary stenosis who have a major coronary artery cros

62 t ventricular septum (PAIVS) and/or critical pulmonary stenosis with reversal of ductal flow (CPS) us