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1 lls also inhibits local growth of metastatic pulmonary tumor.
2 e delivery but not higher vascularization in pulmonary tumors.
3 eatment of s.c. tumors as well as metastatic pulmonary tumors.
4 ffects were observed in established s.c. and pulmonary tumors.
5 gression that enhanced survival in mice with pulmonary tumors.
6 onged survival times in mice with metastatic pulmonary tumors.
7 of peptide hormones in the growth biology of pulmonary tumors.
8 e delivery but not higher vascularization in pulmonary tumors.
9 r order radiomic features (n = 1212) between pulmonary tumors.
10  as a methodology for the detection of small pulmonary tumors.
11 y for the detection and serial monitoring of pulmonary tumors.
12  of the lung is a subtype of highly invasive pulmonary tumors and is associated with decreased or abs
13                  However, detection of mouse pulmonary tumors and their subsequent response to therap
14    In this case series study, neuroendocrine pulmonary tumors associated with Cushing syndrome had in
15  used to detect submillimeter CEA-expressing pulmonary tumors before they become visible to the naked
16 tion algorithms described by our laboratory, pulmonary tumor burden can be quantitatively measured in
17 q gain is a superior prognostic indicator to pulmonary tumor burden in patients with FHWT with pulmon
18                                              Pulmonary tumor burden was significantly (P < .01) lower
19                   We evaluated the impact of pulmonary tumor burden within subgroups of similarly tre
20 d die at 4 months as a result of progressive pulmonary tumor burden.
21  CD4+ cells were prevented from infiltrating pulmonary tumors by pretreatment with pertussis toxin.
22  that the appearance and regression of these pulmonary tumors can be readily monitored in anesthetize
23 s virus type 11 (HPV-11) genome, whereas the pulmonary tumor cells contained a 10.4-kb genome.
24                                Submillimeter pulmonary tumor colonies could be visualized with both s
25 urethane exhibits a profound specificity for pulmonary tumors driven by an oncogenic Q(61)L/R mutatio
26 ntly differed between primary and metastatic pulmonary tumors (FDR-adjusted p = 0.015, AUC 0.69).
27 res differentiated primary versus metastatic pulmonary tumors, fewer features demonstrated good indiv
28 nide resulted in more than 80% inhibition of pulmonary tumor formation compared to the aerosol contro
29 ical abnormalities found in the proximity of pulmonary tumors from a series of 59 patients.
30                                              Pulmonary tumors from Arf-knockout mice exhibited increa
31 re hyperinvasive to EGF and showed increased pulmonary tumor growth upon tail vein injection.
32 rchitecture, or primary (s.c.) or secondary (pulmonary) tumor growth.
33 , but not with oncogenic EGFR(L858R), caused pulmonary tumors in transgenic mice that were phenotypic
34                          The pathogenesis of pulmonary tumors induced by a tobacco carcinogen, 4-(met
35  COOH-terminal ADAMTS-1 fragment can inhibit pulmonary tumor metastasis.
36 enic organizing pneumonia ([COP] n = 4); and pulmonary tumor (n = 4).
37                                      Because pulmonary tumor necrosis factor-alpha expression is know
38 oarrestin in HT1080 tumor cells and measured pulmonary tumor nodule formation in nude mice.
39 2- and IL-18-cultured TDLN cells infiltrated pulmonary tumor nodules and eradicated established tumor
40 (n = 5) within the lungs with BLI-detectable pulmonary tumor nodules as compared with controls (n = 4
41 xpressing exogenous CYLD were unable to form pulmonary tumor nodules following tail-vein injection.
42 y invasive melanoma cell line, B16F10, forms pulmonary tumor nodules in normal C57BL6 mice.
43 ith influenza virus, increased the number of pulmonary tumor nodules.
44 d iodine analysis for the differentiation of pulmonary tumors on contrast-enhanced dual-energy CT (DE
45 , ROC area = 0.75; CT, ROC area = 0.75), and pulmonary tumor (radiography, ROC area = 0.73; CT, ROC a
46 orthotopic RENCA cell renal transplantation, pulmonary tumor spread was inhibited by pharmacologic bl
47 alectomy significantly reduced the extent of pulmonary tumor spread, whereas aldosterone infusion rec
48  infiltrated and proliferated extensively in pulmonary tumors, while also stimulating tumor antigen-s