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1                                 We found the pupal Abd-A expression pattern was conserved between spe
2 rphosis, failure to complete larval-pupal or pupal-adult ecdysis and abnormal wing development are am
3    TcADC mRNA was most abundant prior to the pupal-adult molt.
4 tinase, TcCHT5, was found to be required for pupal-adult molting only.
5  TcE93 RNAi in Tribolium castaneum prevented pupal-adult transition and produced a supernumerary seco
6 th the duration of setal pigmentation at the pupal-adult transition.
7                             Thus, Drosophila pupal/adult tissue progenitors can arise both by early a
8 n orchestrated transformation from larval to pupal/adult tissues.
9 ts are responsible for the generation of the pupal and adult abdominal airways.
10 mJHBP), which circulates in the hemolymph of pupal and adult Aedes aegypti males and females.
11 on of their synapses and cell bodies in late pupal and adult animals.
12 cells might be patterned and establishes the pupal and adult midgut as a novel genetic platform for i
13 s intact and that parkin is required only in pupal and adult muscle.
14 et-7-C locus is principally expressed in the pupal and adult neuromusculature.
15 ctions and 3D digital models for the larval, pupal and adult stage, allowed us to describe the morpho
16 rosophila, miR-125, is also expressed during pupal and adult stages of Drosophila development.
17  present only in males (third instar larval, pupal and adult stages) and in adult flies is restricted
18 ous system is sexually dimorphic during both pupal and adult stages.
19                                              Pupal and adult tissues form from imaginal cells, tissue
20 ted by alternative splicing catalyze larval, pupal, and adult cuticle tanning in Tribolium.
21  fascicles formed by lineages during larval, pupal, and adult stages using antibodies against membran
22 vous system were observed in pharate larval, pupal, and adult stages.
23        Within the ovary, stl is expressed in pupal basal stalks and in adult somatic cells of the pos
24 ds the pupa gradually recedes from the inner pupal case (an extracellular layer that encloses the pup
25                    We propose that the inner pupal case induces elongating bristles to bend when they
26                  Following eclosion from the pupal case, wings of the immature adult fly unfold and e
27 rinsic factors-the socket cell and the inner pupal case--and intrinsic factors--actin cytoskeleton as
28 yny, parthenogenesis, and oviposition in the pupal case.
29 hesized some 30 h prior to eclosion from the pupal case.
30 code proteins secreted in the saliva for the pupal case.
31 s a very different phenotype: short indented pupal cases and pupal death with head eversion defects.
32    The morphogenesis of these takes place in pupal cells and is mediated by the actin and microtubule
33 tively spliced RNAs appear in the larval and pupal CNS, but none shows sex specificity.
34 transcription factor that is correlated with pupal commitment (Zhou and Riddiford, 2001).
35                                   Similar to pupal commitment in other tissues at later stages, activ
36 ation of broad expression is correlated with pupal commitment in the adult eye primordia.
37 al instar, MsE74B expression correlates with pupal commitment on day 3 and is induced to maximal leve
38 is study, we show that a molecular marker of pupal commitment, broad, is up-regulated in the wing dis
39                               At the time of pupal commitment, in vitro experiments show that 20E up-
40 mordia initiate metamorphic changes, such as pupal commitment, patterning and cell proliferation.
41 reas E75D has roles both during the molt and pupal commitment.
42 nd pupal molts, with E75A also increasing at pupal commitment.
43 te the spatiotemporal expression of Delta in pupal cone cells.
44 ars to be an irreversible commitment to form pupal cuticle at the next molt.
45 l pupal phenotype, and the resulting grayish pupal cuticle exhibited many small patches of black pigm
46 d stage-specific cuticle genes and activated pupal cuticle genes, showing that br is a major specifie
47 nt phenotype: short indented pupal cases and pupal death with head eversion defects.
48  loss of ACOX1 leads to developmental delay, pupal death, reduced lifespan, impaired synaptic transmi
49 xpression of the appropriate innexins during pupal development (but not later) rescues connection def
50        Photoreceptors form during Drosophila pupal development and acquire elaborate membrane structu
51                                        After pupal development and adult eclosion, unilateral (with o
52 ly expressed in R7 during the second half of pupal development and are necessary for R7 to terminate
53 vered that the critical period begins during pupal development and extends into adulthood, but temper
54   Our results show that amon is required for pupal development and identify a subset of neuronal cell
55  Drosophila Runx protein Lozenge (Lz) during pupal development causes a decrease in cell death in the
56  transition proceeds normally, with extended pupal development compensating for reduced autophagy.
57 system and find that amon is required during pupal development for head eversion, leg and wing disc e
58 sed in MB neuroblasts during later stages of pupal development only, which includes the time when MB
59 ntributes to a transcriptional switch during pupal development that establishes the metabolic state o
60 helial cells surrounding the sex comb during pupal development to promote sex comb rotation, a comple
61 eye disc glia through the earliest stages of pupal development to reveal the counterparts of these ce
62                                       During pupal development, MN5 undergoes de novo dendritic growt
63                     During the first half of pupal development, N-cadherin is required for R7 growth
64  larval atlas and proceeding forward through pupal development, one will be able to reconstruct adult
65 or of developmental timing during larval and pupal development, opposes EGF signaling in testes.
66 ain high levels of Cic throughout larval and pupal development, repressing the expression of vein-spe
67 (139) mutant animals die during mid and late pupal development, respectively, EcR(94) mutants arrest
68                                       During pupal development, the abdominal histoblast cells prolif
69 r of immediate-early genes during Drosophila pupal development, yet is able to orchestrate distinct d
70  unidentified function that is essential for pupal development.
71 o generate three-cell secretory units during pupal development.
72 ed for joint development in the tarsi during pupal development.
73 al with most mutant animals arresting during pupal development.
74 the normalized virus titer during larval and pupal development.
75 go marked changes in their morphology during pupal development.
76 scription factor broad (br), which specifies pupal development.
77 fferentiation program is attenuated prior to pupal development.
78 nd female first and second legs during early pupal development.
79 indirect flight muscles during mid stages of pupal development.
80 alled ommatidia during late larval and early pupal development.
81 ht muscles and central nervous system during pupal development.
82 ins stable throughout larval development and pupal development.
83 ic interneurons of the flight circuit during pupal development.
84 mpairment of proliferation during larval and pupal development.
85 it of Drosophila calcineurin, can suppress a pupal developmental arrest phenotype to adult viability.
86 r during adulthood along with the larval and pupal developmental stages, corrects the olfactory memor
87      In the tobacco hornworm, Manduca sexta, pupal diapause can be induced by exposure of fifth-insta
88  use archived expression data to compare the pupal diapause of S. crassipalpis with the adult reprodu
89 hat we describe also prevents the entry into pupal diapause when administered to larvae that are envi
90 and pathways differentially regulated during pupal diapause, dynamically regulated across diapause de
91 ultural pests, DH prompts the termination of pupal diapause.
92 pendent on the duration of the overwintering pupal diapause.
93 tar larvae on the photoperiodic induction of pupal diapause.
94 s have an important role in the induction of pupal diapause.
95 arvae, and are then downregulated throughout pupal diapause.
96 complex yield hybrid males that die prior to pupal differentiation.
97              By examining how the Drosophila pupal dorsal thorax epithelium responds to morphogenetic
98  segment six [APR(6)s] die by 48 hours after pupal ecdysis (PE; entry into the pupal stage), whereas
99 uron network is remodeled immediately before pupal ecdysis by the emergence of 12 late CCAP neurons.
100 laces the cuticle between larval stages, and pupal ecdysis externalizes and expands the head and appe
101 ired for viability through its regulation of pupal ecdysis in a type II receptor Wishful thinking (Wi
102 anized layers of the network controlling the pupal ecdysis sequence: a modular input layer, an interm
103 ound to be entirely sufficient for wild-type pupal ecdysis, even after targeted ablation of all other
104                This sequence, which mediates pupal ecdysis, is governed by the serial release of seve
105  died at the pupal stage from the failure of pupal ecdysis, whereas larval ecdysis and adult eclosion
106 ysis progression and results in a failure of pupal ecdysis.
107  LoF mutants exhibit late pupal lethality or pupal eclosion defects.
108 ion affects a locus previously implicated in pupal eclosion.
109 tle specifically during the late development pupal, emerging adult, and newly eclosed adult stages.
110                        Pteromalus puparum, a pupal endoparasitoid of various butterflies, represents
111 e larval endoparasitoids but not to males or pupal endoparasitoids, showing that they maintain specif
112  of wound-induced syncytium formation in the pupal epidermis suggested direct membrane breakdown lead
113 erized actin structures in the ovary and the pupal epithelium.
114 nd Dac at this stage is not dependent on the pupal expression of Distalless (Dll), the main regulator
115 rd femur, but that they do not contribute to pupal expression of Ubx in the second femur.
116 ecycling endosomes in Drosophila postmitotic pupal eye epithelia.
117                            In the Drosophila pupal eye epithelium, Rho1 GTPase regulates AJ remodelin
118 s in the postmitotic Drosophila melanogaster pupal eye epithelium, we demonstrate that Rho1 is requir
119                 Patterning of the Drosophila pupal eye is characterized by precise cell movements.
120 f the fly chimaerin ortholog RhoGAP5a in the pupal eye led to an excess of interommatidial pigment ce
121                               The Drosophila pupal eye provides a sensitive and accessible model for
122                 Reducing Pyd function in the pupal eye resulted in mis-patterning of the interommatid
123 onal analysis in the Drosophila melanogaster pupal eye, we find that Cdc42 is critical for limiting a
124 ial precursor cells (IPCs) of the Drosophila pupal eye.
125 rates that MB neurons, which are born around pupal formation, acquire unique dendritic branching patt
126 do not undergo extensive morphogenesis until pupal formation.
127                                       In the pupal function described here, N signalling activates ta
128                                         This pupal function of dac is separate from its earlier role
129  peptide hormones that coordinate larval and pupal growth and development.
130  the TcCP30 gene had no effect on larval and pupal growth and development.
131 s have defects in larval tracheal growth and pupal head eversion, and Mmp2 mutants have defects in la
132 used global gene expression analysis in late pupal heads to better characterize the post-embryonic fu
133 ion suppresses myofibril assembly defects in pupal indirect flight muscles and dramatically reduces m
134                                       Mutant pupal indirect flight muscles display normal myofibril a
135 olbachia infection were released, with prior pupal irradiation of the released mosquitoes to prevent
136 ) rearrange to generate the highly organized pupal lattice, in which hexagonal ommatidial units pack
137 on of the Dll gene within bract cells of the pupal leg by EGF receptor signaling.
138 d sex-specific gene expression in Drosophila pupal legs.
139                                              Pupal lethal mutants were screened for specific defects
140       The high-protein diet also reduced the pupal lethality and the increased volume of acidic vesic
141  both the larval developmental delay and the pupal lethality caused by loss of KDM5.
142                                          The pupal lethality of these dpp mutants was partially rescu
143              Strong LoF mutants exhibit late pupal lethality or pupal eclosion defects.
144 igned a drug screening strategy based on the pupal lethality phenotype induced by TDP-43 when express
145 s: a >or=50% loss correlated with larval and pupal lethality, disrupted nuclear structures, and in so
146         Strong knockdown of MCO1 resulted in pupal lethality, indicating that MCO1 is an essential ge
147 upariation of starved larvae, which leads to pupal lethality, whereas forced autophagy induction resu
148 eride levels, small fat body cells and early pupal lethality.
149 % of NS1 transcripts also lead to larval and pupal lethality.
150 he appearance of melanotic tumors and larval/pupal lethality.
151 caused muscular atrophy in larval stages and pupal lethality.
152  in the mesodermal derivatives, which led to pupal lethality; or in the central nervous system, which
153 tes that patterning events that occur during pupal life move the ommatidial units an additional 15 de
154  of Eyc during rhabdomere extension early in pupal life results in inappropriate retention of normall
155 ntified for extreme precipitation during the pupal life stage for univoltine species.
156 nd rhodopsin expression, is completed during pupal life.
157 ite puparium stage causes the formation of a pupal-like abdomen with few or no short bristles.
158 , we studied the molecular mechanisms of the pupal melanism in Spodoptera exigua.
159 xpressions of TH and DDC are involved in the pupal melanization of S. exigua.
160 e integument were compared during the larval-pupal metamorphosis process of the S. exigua wild type (
161 ull Nurf301 mutants do not undergo larval to pupal metamorphosis, and also enhance dominant-negative
162 zygotic null mutant, but rarely suffices for pupal metamorphosis, revealing later functions for slpr
163 terocytes and endocrine cells of a transient pupal midgut are selected from within the clusters of ad
164 ence of JH as there was no change during the pupal molt of allatectomized animals.
165 ns full size, concomitant with the larval-to-pupal molt orchestrated by the steroid hormone ecdysone.
166 rgistic mortality occurred during the larval-pupal molt.
167  expressed transiently during the larval and pupal molts as the ecdysteroid titer begins to decline a
168 E74A is expressed late in the larval and the pupal molts when the ecdysteroid titer has declined to l
169 y 20-hydroxyecdysone (20E) during larval and pupal molts, with E75A also increasing at pupal commitme
170 r E75D mRNA expression during the larval and pupal molts.
171  of specific genes during development of the pupal nervous system and emphasizes the relevance of SOC
172 ression changes in the developing Drosophila pupal nervous system.
173  imaging of calcium transients from cultured pupal neurons, we confirmed that Ral does not participat
174  are the primary phagocytic cell type in the pupal neuropil.
175                  Remarkably, however, mosaic pupal ommatidia with three or fewer Dip3(+) photorecepto
176 go metamorphosis, failure to complete larval-pupal or pupal-adult ecdysis and abnormal wing developme
177  a germline cyst with the basal stalk in the pupal ovary contributes to FSC niche formation.
178    This striking phenotype originates in the pupal ovary, where the developing germarium is shaped by
179                             During the early pupal period (P6-P48) these primordia grow in size and d
180 ifferentiates during the first 2 days of the pupal period when terminal branches and synapses of seco
181 e autophagy and reduced viability during the pupal period--a phase when animals rely on autophagy for
182 utant cells degenerate much later during the pupal phase of development.
183 ion of dsTcDDC into larvae produced a lethal pupal phenotype, and the resulting grayish pupal cuticle
184  we used the genetically amenable Drosophila pupal photoreceptor and follicular epithelium.
185     Finally, using live imaging of ingrowing pupal photoreceptor axons, we show that DRA R7 and R8 te
186                                       In the pupal photoreceptor, membrane recruitment of Par6-aPKC a
187                 In contrast, we show that in pupal photoreceptors Arr1-eGFP becomes internalized and
188 pical-basal polarity is compromised in early pupal photoreceptors, and no identifiable apical membran
189 zyme that is required for larval growth, pre-pupal/pupal viability and long-term adult lifespan.
190 vity is tightly regulated across time in the pupal retina and that epithelial cells in this tissue re
191 tion of myosin II in Drosophila melanogaster pupal retina leads to increased cortical tension, apical
192 le pathway that affected caspase activity in pupal retina through hid and Inhibitor of Apoptosis Prot
193 omises normal developmental apoptosis in the pupal retina, while loss of ex has only mild effects.
194 his paper, we used the developing Drosophila pupal retina--looking specifically at the reorganization
195 adherens junction in cells of the developing pupal retina.
196  effects of Par-1 on Baz localization in the pupal retina.
197 elial patterning during morphogenesis of the pupal retina.
198 d abnormal E-cadherin localization in mutant pupal retinas, correlating with aberrant cellular arrang
199  Silencing of CrzR in PTTH neurons increased pupal size, phenocopying the inhibition of Crz neuronal
200 nhibition of Crz neuronal activity increased pupal size, whereas it hardly affected pupariation timin
201       As the larvae entered the pupal stage, pupal sizes reflected the overall larval weights.
202      Here, we analyzed the expression of the pupal specifier gene broad (br), and the effect on br of
203 g essential genes, lethality was high in the pupal stage and also found in the larval stages.
204 93 is expressed widely in adult cells at the pupal stage and is required for many patterning processe
205 ine this requirement temporally to the early pupal stage and use RNA-sequencing to identify SOCE medi
206  The majority of CCAP KO animals died at the pupal stage from the failure of pupal ecdysis, whereas l
207 ell fate determination, typically during the pupal stage of holometabolan species.
208 up to 99.8% of homozygous females die at the pupal stage when raised on diet that lacks tetracycline.
209 cap cells can develop into GSCs at the early pupal stage while the rest directly differentiate.
210 matic activity was detected at day 13 of the pupal stage with a peak at day 2 adult stage.
211 ours after pupal ecdysis (PE; entry into the pupal stage), whereas APR(4)s survive until adulthood.
212 peratures, which are likely to influence the pupal stage, are important for predicting the timing of
213 lth requires DPTP69D during the mid- to late-pupal stage, eclosion requires DPTP69D during the early
214                    As the larvae entered the pupal stage, pupal sizes reflected the overall larval we
215 eloping (hemimetabolous) insect that lacks a pupal stage, we cloned br from the milkweed bug, Oncopel
216 val muscle defects and semi-lethality at the pupal stage.
217 ocrine cell development takes place at a mid-pupal stage.
218 n of Elp3 results in larval lethality at the pupal stage.
219 r cells contributing to the TDT at the early pupal stage.
220 ene Dchx1 and can be followed until the late pupal stage.
221  showing that br is a major specifier of the pupal stage.
222 arvae while another induced mortality at the pupal stage.
223 f pupariation and lethality during the early pupal stage.
224 ture lethality and arrested development at a pupal stage.
225  expression of TcKr-h1 and TcBR-C during the pupal stage.
226  5' shortvein (shv) regulatory region at the pupal stage.
227 tracts are found and such robo embryos reach pupal stages and die, while robo3 mutant embryos develop
228 lopmental delay, lethality during larval and pupal stages and hyperplasia of the hematopoietic organ,
229 oreceptors lose neuronal markers during late pupal stages but do not re-enter a proliferative state o
230 c4da neurons remains constant from larval to pupal stages but the expression of Fos is specifically a
231           Null animals survive to larval and pupal stages due to a large maternal contribution of CaM
232 atic activity ubiquitously during larval and pupal stages is lethal.
233 arvae, lethality occurs in the late larval - pupal stages of development.
234 reas Tlr is required later during larval and pupal stages of development.
235    The developmental times of the larval and pupal stages on I. batatas than on I. triloba were 37.01
236                         We found that at mid-pupal stages the Drosophila melanogaster CNS neuropil wa
237  error-correction mechanism operating during pupal stages to reposition inappropriately orientated om
238 ction of the cDNA only during the larval and pupal stages was inconsequential to performance in olfac
239 l developmental delay (e.g. prolonged larval/pupal stages) often associated with decreased levels of
240 ecific isoform of troponin I during the late pupal stages, although the incompleteness of this transi
241 of 275,000 single cells at adult and at five pupal stages, and built a machine-learning framework to
242 yogenesis, low expression through larval and pupal stages, and greatly enriched expression in the adu
243 p in isolation from one another during early pupal stages, and that some patterning events are indepe
244 s promote terminal R8 differentiation during pupal stages, including the regulation of rhodopsin expr
245 ing 30% and 42% knockdown for early and late pupal stages, respectively.
246 ted by ecdysone receptor B1 (EcRB1) at early pupal stages, suggesting that ecdysone signaling provide
247 erozygous dDP mutant animals develop to late pupal stages, the analysis of somatic mutant clones show
248                                           At pupal stages, the wing-hinge contraction contributes to
249 r visceral muscles is mainly required during pupal stages, when Hand participates in the proper hormo
250  alters overall dimensions at the larval and pupal stages.
251 r of wounds (<=10) observed on the different pupal stages.
252 evelopment, including embryonic, larval, and pupal stages.
253 hal system where females died at late larval/pupal stages.
254 esulting in female death at larvae and early pupal stages.
255   Homozygous mutant dronc animals die during pupal stages; however, at a low frequency we obtained ho
256 rence) from behavioural observations and for pupal survivorship (related to performance), showing tha
257  tissue fusion, embryonic dorsal closure and pupal thorax closure in Drosophila are useful experiment
258 criptional repressors in multiple larval and pupal tissues, including many DSF-expressing tissues.
259 function as overwintering housing during the pupal to adult developmental period.
260  the timing of the transition from larval to pupal to adult stages.
261 oth br-Z1 and br-Z4 caused the appearance of pupal traits in the adults, but disruption of br-Z5 had
262  morphogenetic processes during the prepupal-pupal transition in Drosophila.
263                            During the larval-pupal transition, a switch from gene activation by EcR/U
264 most Acf1 null animals die during the larval-pupal transition, Acf1 is not absolutely required for vi
265 ch br expression is restricted to the larval-pupal transition, Of'br mRNA is expressed during embryon
266                            During the larval-pupal transition, the levels of CDK8 protein positively
267 mutants proceeds normally until the prepupal-pupal transition, when final leg elongation is delayed b
268 ostmitotic neurons born during the larval-to-pupal transition, when transitions among three MB subtyp
269 ssure during the normal time of the prepupal-pupal transition.
270  and to shorten their bodies at the prepupal-pupal transition.
271 ing key developmental events at the prepupal-pupal transition.
272 ges correlate with the timing for the larval-pupal transition.
273 etabolism (SREBP activity) during the larval-pupal transition.
274 ingiensis toxin, but significantly decreased pupal weight and adult emergence, possibly due to PM str
275                                              Pupal weight, adult emergence and lifetime fecundity of
276 urthermore, ectopic expression of Ubx on the pupal wing activated the eyespot-associated genes spalt
277  F-actin prehair to the distal vertex of the pupal wing cell has been shown to be dependent upon the
278     We show that centrioles are polarized in pupal wing cells as a readout of PCP signalling, with bo
279 ited to a small region at the distal edge of pupal wing cells as in wild type, resulting in multiple
280  show that Tai expression transforms sessile pupal wing cells into an invasive mass that penetrates t
281 he PCP proteins accumulate asymmetrically in pupal wing cells where they are thought to form distinct
282  and for suppression of endoreduplication in pupal wing cells.
283                             Likewise, during pupal wing development, BMPs help to specify vein versus
284 ifferentiation, chromatin closes at a set of pupal wing enhancers for the key rate-limiting cell cycl
285 he core protein Strabismus in the Drosophila pupal wing increases its stability and promotes its clus
286 rowing hairs, and we failed to detect Trc in pupal wing nuclei, implying that in this developmental c
287 ensitivity of the posterior crossvein in the pupal wing of Drosophila to reductions in the levels and
288                                              Pupal wing RNA was isolated from tissue prior to, during
289  the cytoplasm in differentiating larval and pupal wing vein cells, and we show that this cytoplasmic
290  and other tissues, and can diffuse into the pupal wing via the hemolymph.
291 of Ras to maintain vein cell identity in the pupal wing, our results indicate that Ras controls Shg l
292  it affects the range of BMP movement in the pupal wing, probably as part of a lipid-BMP-lipoprotein
293 hairs to distal cell edges in the Drosophila pupal wing.
294 s required for hexagonal cell packing in the pupal wing.
295 ment of the posterior crossvein (PCV) in the pupal wing.
296 ormation along the proximodistal axis of the pupal wing.
297  and inter-vein subregions of the Drosophila pupal wing.
298 ermis of both the embryonic germband and the pupal wing.
299                Experimental manipulations of pupal wings reveal that the bias has been released throu
300 ecovery after photobleaching in prepupal and pupal wings, we have investigated the turnover of two ke

 
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