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1 oM) produced marked neurotoxicity in the CA1 pyramidal cell layer.
2 trol levels, most notably, in the CA1 region pyramidal cell layer.
3 m1 expression is restricted to the postnatal pyramidal cell layer.
4 ppocampal neuropil layers and weakly stained pyramidal cell layers.
5 timulation of either the granule cell or CA3 pyramidal cell layers.
6 4) with axons predominantly ramifying in the pyramidal cell layer; (2) Schaffer collateral/commissura
8 umber of mossy fibers that can grow into the pyramidal cell layer and an inhibition of process retrac
9 inhibition after stimulation adjacent to the pyramidal cell layer and inhibited induction of long-ter
10 noreactivity (IR) was most noticeable in the pyramidal cell layer and interspersed interneurons, espe
11 ssed in glutamatergic neurons of the CA1-CA3 pyramidal cell layer and the granular layer of the denta
12 mpi, HCN1 mRNA expression was substantial in pyramidal cell layers and lower in dentate gyrus granule
13 ons with their dendrites confined to the CA3 pyramidal cell layer, and a previously undescribed cell
14 nule and mossy cells, disorganization of the pyramidal cell layer, and early postnatal death of pyram
15 CA3 field, the mossy fibers ran through the pyramidal cell layer, and while near the transition to f
16 n and diffuse ERbeta-ir primarily in the CA1 pyramidal cell layer as well as in a few interneurons.
18 n aberrant and persistent innervation of the pyramidal cell layer by mossy fibers, including excessiv
19 and axons of cells lying within or near the pyramidal cell layer, consistent with their being GABAer
20 are found at lower levels in the hippocampal pyramidal cell layer, dentate granule cell layer and thr
21 increased independent of adrenals in the CA1 pyramidal cell layer, dentate gyrus polymorphic layer, b
22 ces to investigate ROS production in the CA1 pyramidal cell layer during and after transient ischemia
23 piking interneurones with cell bodies in the pyramidal cell layer fired preferentially at input frequ
24 quently traveled mostly perpendicular to the pyramidal cell layer from CA3 (or CA1) but also in all d
26 aphy did not reveal changes in GR protein in pyramidal cell layers; however, increased GR signal was
28 fine dendritic staining was observed in the pyramidal cell layer, in the granule cell layer, and in
29 Furthermore, we find that spiking in the CA1 pyramidal cell layer is modulated in a consistent travel
30 In that Gas6 is expressed throughout the pyramidal cell layer, it may activate these cells in bot
32 d local field potentials from the dorsal CA1 pyramidal cell layer of 7- to 8-month-old wild-type and
33 d local field potentials from the dorsal CA1 pyramidal cell layer of 7-8 month old wild-type and rTg4
34 hemical labeling of GABAergic boutons in the pyramidal cell layer of CA1 was preserved in the mouse m
35 campus in the granule cell layer, hilus, and pyramidal cell layer of CA3, CA2, and CA1 subfields.
36 calcium imaging of neuronal activity, in the pyramidal cell layer of mouse hippocampal in vitro prepa
37 urons that were evoked by stimulation of the pyramidal cell layer of the CA1, but not by depolarizing
38 ngiotensin II receptor mRNA was found in the pyramidal cell layer of the CA1, CA2 and CA3 subfields,
40 us displayed dense CNTFR alpha-ir within the pyramidal cell layer of the hippocampal formation and th
41 tivity was found in a subset of cells in the pyramidal cell layer of the hippocampus and in the granu
42 e [14C]AA incorporation in the claustrum and pyramidal cell layer of the hippocampus compared with ve
43 UNEL-positive cells appeared only in the CA1 pyramidal cell layer of the hippocampus, and DNA gel ele
44 ased intensity of signal is also seen in the pyramidal cell layers of medial prefrontal and anterior
45 heterotopia throughout the granule cell and pyramidal cell layers of mice containing a heterozygous
46 in immunoreactivity was seen, especially in pyramidal cell layers of the ipsilateral (stroked) hippo
47 layers-5 and 6) and the CA1 and CA3 sectors (pyramidal cell layers) of the hippocampus, were analyzed
49 re were ischemia-tolerant neurons in the CA1 pyramidal cell layer that survived delayed neurodegenera
50 rough adulthood, expression decreased in the pyramidal cell layer versus the dentate gyrus granule ce
51 mately 50% in each subregion, though the CA1 pyramidal cell layer was markedly more sensitive to the
53 mage and neurogenesis in the hippocampal CA1 pyramidal cell layer were immunohistochemically studied.
54 mulation spots in the hilus and proximal CA3 pyramidal cell layer were more likely to evoke EPSCs in
55 ions, known as ripples, are prominent in the pyramidal cell layer, where they coincide with increased
56 ocated in the superficial portion of the CA1 pyramidal cell layer, whereas it is absent from deep-lay
57 ells are aligned in radial arrays across the pyramidal cell layer, whereas like-genotype cohorts in t
58 granule cell layer, hilus, and proximal CA3 pyramidal cell layer while measuring evoked EPSCs in nor
59 revealed dark degenerating terminals in the pyramidal cell layer with lingering CCK and CB(1) immuno
60 malformations included disruption of the SRK pyramidal cell layer, with spreading of the CA3 mossy fi